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1 enotypes, inactivates mismatch repair in the yeast cell.
2 a level, is then extended to the rest of the yeast cell.
3 , there are 57,000 to 60,000 nucleosomes per yeast cell.
4 n animal feed additive, from a low number of yeast cells.
5 beta (Abeta) peptides accumulate around the yeast cells.
6 nked to cellular growth rate in RP-deficient yeast cells.
7 of the plasma membrane during endocytosis in yeast cells.
8 t sulfate, when heterogeneously expressed in yeast cells.
9 endogenous Sod1 (Sod1-KD), and in sod1Delta yeast cells.
10 ed by living and isolated single bacteria or yeast cells.
11 an overcome resistance of turgor pressure in yeast cells.
12 es that can form viable endosymbionts within yeast cells.
13 f Escherichia coli endosymbionts within host yeast cells.
14 cytoplasmic iron-sulfur cluster assembly in yeast cells.
15 eviate the nystatin-sensitivity of lam2Delta yeast cells.
16 As are localized to distinct granules within yeast cells.
17 escently labeled circular chromosome in live yeast cells.
18 ies of formaldehyde-cross-linking in budding yeast cells.
19 rion fusions to encode synthetic memories in yeast cells.
20 anslational modification found in animal and yeast cells.
21 redominant form of mtDNA replication in rho+ yeast cells.
22 that mitophagy is perturbed in CL-deficient yeast cells.
23 s been demonstrated using both bacterial and yeast cells.
24 accumulation, unlike the OsPCS2b transformed yeast cells.
25 in vitro, and blocked autophagy induction in yeast cells.
26 ggregate activity observed in living budding yeast cells.
27 f m(6)A-modified FAA1 transcripts in haploid yeast cells.
28 to monitor glutathione import into the ER of yeast cells.
29 ry of stress adaptation is encoded in single yeast cells.
30 e joint distributions of mRNA populations in yeast cells.
31 bacteria use lactic acid to communicate with yeast cells.
32 izations as well as phenotypes of expressing yeast cells.
33 artments and cell membrane when expressed in yeast cells.
34 ion is closely guided by experiments in live yeast cells.
35 nodes and contractile rings in live fission yeast cells.
36 isappearance of mitochondria from the mutant yeast cells.
37 f NAD into peroxisomes against AMP in intact yeast cells.
38 reams and lysis of Rhodosporidium toruloides yeast cells.
39 ges in senescing and post-senescent survivor yeast cells.
40 s cytoplasmic hydrogen ion concentrations in yeast cells.
41 near invaginations at the plasma membrane of yeast cells.
42 l2-L-13 induces mitophagy in Atg32-deficient yeast cells.
43 critical role in metabolic energy control in yeast cells.
44 he toxicity of human alpha-synuclein in live yeast cells.
45 m can be visualized on the surface of living yeast cells.
46 our well-understood models of mammalian and yeast cells.
47 hnology can be modified to allow analysis of yeast cells.
48 ions for imaging mEos3.2-tagged molecules in yeast cells.
49 ities, namely, Escherichia coli bacteria and yeast cells.
50 rrested, growing, and synchronously dividing yeast cells.
51 inds to antigenic epitopes on the surface of yeast cells.
52 odulates SOS1 activity by activating SOS2 in yeast cells.
53 ultrastructural changes in the cytoplasm of yeast cells.
54 ntly decreases cellular longevity in diploid yeast cells.
55 in translocation are not directly coupled in yeast cells.
56 ,000 ORFs in exponentially growing wild-type yeast cells.
57 ges at 2 to 24 h after infection with viable yeast cells.
58 e Gal4 target genes GAL3 and GAL10 in living yeast cells.
59 icochemical parameters of replicatively aged yeast cells.
60 rity, endocytosis, adhesion, and invasion in yeast cells.
64 val in response to sudden glucose depletion, yeast cells activate lipid-droplet (LD) consumption thro
67 isolated UPEC was subsequently determined by yeast cell agglutination and immunofluorescence microsco
68 ow that, in wolf lichens, Tremella occurs as yeast cells also in thalli that lack basidiomata and inf
69 , we measure gene network activity in single yeast cells and find that the activity of the compensate
71 s of Rcf1/Rcf2 single and double null-mutant yeast cells and mitochondria, we further explored how Rc
74 s rapid screening of ADAR variants in single yeast cells and provides quantitative evaluation for enz
76 evels of tension at kinetochores in dividing yeast cells and relating these measurements to kinetocho
77 ies to track the replicative aging of single yeast cells and reveal that the temporal patterns of het
78 correlations across hundreds of thousands of yeast cells and reveals ample evidence of both vertical
79 d these antibody libraries on the surface of yeast cells and selected antibodies that strongly recogn
81 sensitive marker of increased ROS levels in yeast cells and suggest that changes in ribosomes may be
82 significant decrease in both phagocytosis of yeast cells and the frequency of nonlytic exocytosis.
84 sed mitochondria and cytoplasm isolated from yeast cells, and [(35)S]cysteine to detect cytoplasmic F
85 d, stabilise the position and orientation of yeast cells, and demonstrate independent control over mu
86 vents of cell division in both mammalian and yeast cells, and in fission yeast a single mitotic cycli
87 events cause genetic instability in diploid yeast cells, and propose that similar, heterozygous muta
88 mplate for DNA double-strand break repair in yeast cells, and Rad52, a member of the homologous recom
92 The trends of measured lateral migrations of yeast cells are similar to the corresponding Clausius-Mo
95 , the continuous separation of live and dead yeast cells as well as the yeast cells with targeted dia
96 be opens new avenues for studies focusing on yeast cells, as well as other cells with a degradable ce
99 As confer a competitive fitness advantage to yeast cells because expression of these non-coding molec
100 f molecular noise that is inevitable in tiny yeast cells, because mistakes in sequencing cell cycle e
101 scriptionally formed G4 DNA in vivo and that yeast cells become highly sensitivity to G4-stabilizing
103 rgo) and demonstrate its utility not only in yeast cells, but also in cultured mammalian cells, Droso
104 on a genomic scale have been investigated in yeast cells, but comparable experiments have not been do
106 ublocations within mitochondria of respiring yeast cells by fusing a pH-sensitive GFP to proteins res
107 MG inhibits the growth of glucose-fermenting yeast cells by inducing endocytosis and degradation of t
109 platform, we measure noise dynamics in aging yeast cells by tracking the generation-specific activity
110 ully folded bioactive cyclotides inside live yeast cells by using intracellular protein trans-splicin
111 ned Nanobody on the surface of an individual yeast cell can be monitored through a covalent fluoropho
115 stributes homogeneously in wild-type fission yeast cells, can be made to concentrate at cell ends by
120 rnal, Stahl et al (2019) reveal that budding yeast cells confer a growth advantage to their daughters
121 duce rejuvenated daughters, dividing budding yeast cells confine aging factors, including protein agg
122 e presence of a proteasome inhibitor or when yeast cells contained mutations in the CDC48 or SSA1 gen
123 nal microscopic diagnosis, as characteristic yeast cells could be observed only in 14 pus samples.
127 we provide a stochastic model of the budding yeast cell cycle that accurately accounts for the variab
128 rom peripheral leukocytes, brain tissue, and yeast cell cycle, revealed novel marker genes that were
133 er investigations of the budding and fission yeast cell-cycle, we identify two generic dynamical rule
142 or single-cell RNA sequencing (scRNA-seq) of yeast cells do not match the throughput and relative sim
145 ined the range of proteins that aggregate in yeast cells during normal growth and after exposure to s
147 w that both vegetative and pheromone-treated yeast cells exhibit discrete and asynchronous Ca(2+) bur
150 the transcriptomes of >2,000 single fission yeast cells exposed to various environmental conditions
155 alogs to thiophosphorylate its substrates in yeast cell extracts as well as when produced as recombin
157 molecules, have been proposed to explain how yeast cells filter fluctuations and detect shallow gradi
158 terized by emergence of a germ tube from the yeast cell followed by mold-like growth of branching hyp
161 nction of pH and protein phase separation in yeast cells for pH values close to the isoelectric point
165 for the SWI/SNF complex in the transition of yeast cells from fermentative to respiratory modes of me
170 rformed a genome-wide expression analysis in yeast cells grown in the presence or absence of the drug
171 we identified 377 IVC-associated proteins in yeast cells grown under steady-state low-glucose conditi
172 responding eIF2gamma-I318M mutation impaired yeast cell growth and derepressed GCN4 expression, an in
175 selected differently in haploid and diploid yeast cells: haploid cells bud in an axial manner, while
178 n early after telomerase inactivation (ETI), yeast cells have accelerated mother cell aging and mildl
179 y responded to sphingolipid, suggesting that yeast cells have, in addition to Orm phosphorylation, an
180 oilseed rape in the microsomal fractions of yeast cells heterologously expressing these enzymes.
181 the budding pattern and pre-mRNA splicing in yeast cells; however, no Bud13p homologs have been ident
183 HIF-1alpha creates a hostile environment for yeast cells in human macrophages by interrupting the abi
184 s, namely (i) the chromosome interactions of yeast cells in quiescence and in exponential growth, and
185 e measurement of ac-DEP lateral migration of yeast cells in solutions with different electrical condu
187 of Mediator subunits in wild-type and mutant yeast cells in which RNA polymerase II promoter escape i
188 function is required for sterol secretion in yeast cells, indicating that members of this superfamily
190 ions, the time-integrated mEos3.2 signal per yeast cell is similar in live cells and fixed cells imag
193 yeast expression system and discovered that yeast cells lacking endogenous potassium channels could
197 to cellular toxicity and cell cycle delay in yeast cells lacking PSH1, but not in cells lacking UBR1,
198 rnover as Cse4 degradation is compromised in yeast cells lacking RCY1 Excessive Cse4 accumulation in
200 d cell death caused by DNA damage in budding yeast cells lacking the Rad53 checkpoint protein kinase
202 nt in tension over multiple isogenic budding yeast cell lines by genetically altering the magnitude o
204 and formation of a shmoo-like morphology in yeast cells, lower pheromone doses elicit elongated cell
205 purified a high quantity of mRNA from crude yeast cell lysate compared to a phenol/chloroform extrac
206 closporine A with cyclophilin A protein in a yeast cell lysate is successfully detected and quantifie
208 ancement was also shown on a separation of a yeast cell metabolite extract, where the enhanced TIC fo
210 mimetic FUS reduces aggregation in human and yeast cell models, and can ameliorate FUS-associated cyt
213 by both plasmolysed (PYC) and nonplasmolysed yeast cell (NPYC) and stability of thymoquinone and bioa
215 mely, the geometrical effect of the dividing yeast cell on the diffusion of protein aggregates, and t
216 at constitutive membrane anchoring of GIV in yeast cells or rapid membrane translocation in mammalian
220 ryotype-specific dosage effects in aneuploid yeast-cell populations with random and diverse chromosom
222 1R) ) in the FRB domain of Tor2 that renders yeast cells rapamycin resistant and temperature sensitiv
223 show that upon growth at higher temperature, yeast cells relax the retention of DNA circles, which ac
224 r fit, our model quantitatively predicts the yeast cell response to pheromone gradient providing an i
225 Nitrogen replenishment of nitrogen-starved yeast cells resulted in substantial transcriptome change
227 (B. cereus, E. coli, and S. enterica) and a yeast cell (S. cerevisiae), ranging in size from 1 to 6.
228 , cancer cells (MCF-7, MDA-435 and CD34(+)), yeast cells (saccharomyces cerevisiae, listeria innocua
230 report comprehensive ribosome profiling of a yeast cell size series from the time of cell birth, to i
235 designs and assessment of their stability on yeast cell surface, detailed biophysical characterizatio
240 (GET) pathway was described in mammalian and yeast cells that serve as a blueprint of TA protein inse
242 , promotes the symmetric division of fission yeast cells through spatial control of cytokinesis.
246 he predicted 2nd WD40 propeller was shown in yeast cells to bind Vernalization 5 (VRN5), which contai
249 Transient exposure to lactic acid caused yeast cells to heritably circumvent glucose repression.
250 tion localization microscopy of live fission yeast cells to improve the spatial resolution to approxi
251 scence microscopy techniques in live budding yeast cells to investigate how Mex67 facilitates mRNA ex
253 novative single-molecule imaging approach in yeast cells to measure chromatin association of individu
254 CSLAs from different species, we programmed yeast cells to produce an HM backbone composed exclusive
255 possibilities by using clonal populations of yeast cells to quantify the inherent relationships betwe
256 llular traffic of the Chs3 protein, allowing yeast cells to regulate morphogenesis, depending on envi
257 a stress-induced survival strategy, allowing yeast cells to save energy, protect proteins from degrad
259 and mathematical modeling in single fission yeast cells to uncover the precise molecular mechanisms
261 d chromosomal loci during interphase in live yeast cells together with polymer models of chromatin ch
262 t MAb 4D1 binds to and recognizes conidia to yeast cells' transition inside of a human monocyte-like
263 de enzymes produced by SSF were utilised for yeast cell treatment leading to simultaneous release of
265 popular green-to-red PCFP mEos3.2 in fission yeast cells under a wide range of imaging conditions.
266 t, we systematically monitored the growth of yeast cells under various frequencies of oscillating osm
267 shown that in response to pheromone, budding yeast cells undergo a rise of cytosolic Ca(2+) that is m
269 osed to a high dose of mating pheromone, the yeast cell undergoes growth arrest and forms a shmoo-lik
272 im32 proteins are essential for viability of yeast cells upon treatment with the redox mediators gall
273 his work, we measured LacI binding in living yeast cells using a fluorescent repressor operator syste
275 was found in glycolytic oscillations in real yeast cells, verifying that chronotaxicity could be used
276 n1, a DNA/RNA helicase that is essential for yeast cell viability and homologous to human senataxin,
279 sently, commercial MOS is being derived from yeast cell wall mannan and is widely used as prebiotic i
282 tact yeast cells and plasmolyzed yeast, i.e. yeast cell wall particles (YCWPs), of Saccharomyces cere
284 f purified mannoproteins (MP), isolated from yeast cell walls upon the enzymatic treatment, revealed
286 bility of the CBD to bind to the surfaces of yeast cells was found to be unperturbed by this modifica
288 er to monitor Fus3 and Kss1 activity in live yeast cells, we demonstrate that overall mating MAPK act
289 wild-type levels of mcm10-m2,3,4 in budding yeast cells, we observed a severe growth defect and a su
290 from both wild-type and hydroxyurea-treated yeast cells, we show that our model is more accurate tha
291 longating transcript sequencing (NET-seq) to yeast cells, we show that Xrn1 functions mainly as a tra
293 ntial to aid the enrichment of low-abundance yeast cells when filler volume fractions approximately 1
298 of live and dead yeast cells as well as the yeast cells with targeted diameter and dielectric proper
299 rowth and DNA replication defects in budding yeast cells, with diminished DDK phosphorylation of Mcm2
300 improved the ability to metabolize xylose of yeast cells without adaptive evolution, suggesting that