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1 inct age groups (childhood, adolescence, and young adulthood).
2 valuation of FASD in childhood as well as in young adulthood.
3 n sibling loss during childhood and death in young adulthood.
4 uced myopia, particularly in adolescence and young adulthood.
5 ntal and mental health outcomes persist into young adulthood.
6 and young adulthood as well as opioid use in young adulthood.
7 ges in dental caries from early childhood to young adulthood.
8 rly childhood, but also into adolescence and young adulthood.
9 ff enough in youth to alter BP levels during young adulthood.
10 n childhood and DSM-5 diagnostic criteria in young adulthood.
11 e increases substantially in adolescence and young adulthood.
12 nificant precursors of crime and violence in young adulthood.
13 demic performance throughout adolescence and young adulthood.
14 permits protein expression and survival into young adulthood.
15 me physical activity in late adolescence and young adulthood.
16 levels of depressive symptoms, persist into young adulthood.
17 Reactions to insect venom were seen more in young adulthood.
18 an important determinant of LTL, at least by young adulthood.
19 slipidemia and markers of atherosclerosis in young adulthood.
20 ngly healthy level of MVPA from childhood to young adulthood.
21 ompetence (eg, academic or interpersonal) in young adulthood.
22 stress, which often emerge in adolescence or young adulthood.
23 ril 1, 2002, through January 31, 2006) until young adulthood.
24 e development of asthma through childhood to young adulthood.
25 s transition to a relatively stable state by young adulthood.
26 onic disease might emerge decades earlier in young adulthood.
27 sk of developing depression and psychosis in young adulthood.
28 ipation of monetary reward from childhood to young adulthood.
29 ring adolescence compared with childhood and young adulthood.
30 annabis use are wide ranging and extend into young adulthood.
31 sseria meningitidis peaks in adolescence and young adulthood.
32 ed to juvenile monkeys upregulates SERT into young adulthood.
33 ges during adolescence persist or recover in young adulthood.
34 ce exposed to CAPs, and it persisted through young adulthood.
35 ys on a long-term ~30% restricted diet since young adulthood.
36 s that FFA is associated with IR starting in young adulthood.
37 age 9 to 10 for approximately ten years into young adulthood.
38 ood, and internalizing symptoms, assessed in young adulthood.
39 prospectively followed up from childhood to young adulthood.
40 nction plays a major part in adolescence and young adulthood.
41 severity was observed across adolescence and young adulthood.
42 e exacerbated high-frequency hearing loss in young adulthood.
43 caused behavioral deficits that manifest in young adulthood.
44 veloping bipolar disorder in adolescence and young adulthood.
45 d reduced quality of life in adolescence and young adulthood.
46 urse of self-harm from middle adolescence to young adulthood.
47 isk of Hodgkin lymphoma in childhood through young adulthood.
48 ancy (<3 months) with any outcome measure in young adulthood.
49 ns during memory retrieval also develop into young adulthood.
50 cy was associated with insulin resistance in young adulthood.
51 stems in the human brain from childhood into young adulthood.
52 t periodontal outcomes in adolescence and in young adulthood.
53 chronic illness are reaching adolescence and young adulthood.
54 h increased mortality in early childhood and young adulthood.
55 d elevated blood pressure in adolescence and young adulthood.
56 in girls with and without ADHD followed into young adulthood.
57 at ages 9, 11, and 13 and followed them into young adulthood.
58 des (>or=1.70 mmol/L [>or=150 mg/dL]) during young adulthood.
59 inate between different BMI status groups in young adulthood.
60 urance coverage for survivorship services in young adulthood.
61 nd to have subsequent experiences of rape by young adulthood.
62 have a stable renal and hepatic situation in young adulthood.
63 e throughout the school years, persisting to young adulthood.
64 e generally continuing to have low intake in young adulthood.
65 g any behavior of despair in adolescence and young adulthood.
66 expectedly large burden of mental illness in young adulthood.
67 hreat extinction learning in adolescence and young adulthood.
68 with BA requiring LT during adolescence and young adulthood.
69 attenuated cerebrovascular function, during young adulthood.
70 terns of social mobility from adolescence to young adulthood.
71 ey during the transition from adolescence to young adulthood.
72 may be present in individuals with OSA since young adulthood.
73 ps during the transition from adolescence to young adulthood.
74 tly predicted preclinical atherosclerosis in young adulthood.
75 2 weeks of gestation and HF in childhood and young adulthood.
76 overweight or obesity during adolescence and young adulthood.
77 he induction of EAE during the age window of young adulthood.
78 te, tended to decline during adolescence and young adulthood.
79 upwardly mobile minorities is compromised in young adulthood.
80 h in early childhood and body composition in young adulthood.
81 r (OUD) frequently begins in adolescence and young adulthood.
82 t is often discovered during adolescence and young adulthood.
83 avior that often develops in adolescence and young adulthood.
84 on-based reciprocity from early childhood to young adulthood.
85 sity of grandchildren during adolescence and young adulthood.
86 among women who performed shift work during young adulthood.
87 n concentration through late adolescence and young-adulthood.
90 73 participants were assessed 3 times during young adulthood (19, 21, and 24-26 years of age, for a t
91 ion, or a suicide attempt), were assessed in young adulthood (19, 21, and 24-26 years) by use of stru
92 ctively examined the relationships of BMI in young adulthood (21 years of age) and adult obesity traj
97 ly during the transition from adolescence to young adulthood, a period characterised by a sharp rise
98 for body mass index (BMI) in adolescence and young adulthood, a period of high risk for weight gain,
99 were calculated to compare dietary intake in young adulthood across quartiles of adolescent intake.
101 re consistently obese during adolescence and young adulthood (adjusted odds ratio = 1.97, 95% confide
102 set in childhood (age 18 years or before) or young adulthood (age 19-45 years) was associated with al
103 ds ratios (OR) for overweight and obesity in young adulthood (age 20-29) were calculated using logist
104 positive effects by reducing adolescent and young adulthood (age 25) behaviors that precede these de
106 across four distinct epochs of adulthood: 1) young adulthood (ages 18-25 years), adulthood (ages 26-4
108 , 0.95-1.03; P = .64) and then reappeared in young adulthood (ages 18-36 years: aHR, 0.96; 95% CI, 0.
111 ths) for symptoms of DMDD and three times in young adulthood (ages 19, 21, and 24-26 years; 3,215 obs
113 s maternal educational immunity such that by young adulthood, all immune cells responding to a foster
115 and behavioral problems from adolescence to young adulthood among individuals with and without food
116 smoking during pregnancy and tobacco use in young adulthood, an association that was largely concent
117 s shown in this paper and elsewhere, between young adulthood and >70 y of age the effective clonal di
119 es that evaluated the associations of BMI in young adulthood and adult weight trajectory with mortali
121 intake of fruits and vegetables (F/V) during young adulthood and coronary atherosclerosis later in li
122 and parietal cortices during adolescence and young adulthood and discrepantly accelerated age-related
123 ng heritability from early childhood through young adulthood and females exhibiting decreasing herita
125 evaluate the association between early life, young adulthood and later adulthood SEP with each inflam
126 link between regional aerobic glycolysis in young adulthood and later development of Alzheimer patho
127 n immune-cell gene regulation that emerge by young adulthood and may help explain social disparities
128 er number of ideal cardiovascular metrics in young adulthood and middle age were independently associ
129 nce of airborne allergen sensitization up to young adulthood and risk factors for early and late onse
130 een cumulative exposure to hyperlipidemia in young adulthood and subsequent CHD risk (adjusted hazard
131 se HBsAg seroprevalence in childhood through young adulthood and subsequently reduce the risk of PLC
132 , produce distinct phenotypes in mice during young adulthood and suggest sex-based differences in the
133 different phenotypic clusters persisted into young adulthood and that these clinical differences were
134 at human cognitive function improves through young adulthood and then declines across the later life
135 gratification improves between childhood and young adulthood and this improvement is predicted by the
136 pressure (BP) trajectories from childhood to young adulthood and to examine whether this relation is
138 -onset depression and development of T2DM in young adulthood and whether the association differs by s
139 ing mortality (during infancy and throughout young adulthood) and psychiatric (psychotic or bipolar d
140 n institution (n=67 at ages 6 years; n=50 at young adulthood) and UK controls (n=52 at age 6 years; n
141 rth: 10-19 years (adolescence), 20-24 years (young adulthood), and 25 years or older (adulthood).
142 s that affect development in adolescence and young adulthood, and influence educational attainment an
143 onnectivity during an attention bias task in young adulthood, and internalizing symptoms, assessed in
144 sed over a wide age range from birth through young adulthood, and older age at diagnosis is associate
145 may be tightly linked during adolescence and young adulthood, and reduced DMN connectivity may reflec
146 worsen as pediatric patients transition into young adulthood, and that SES does not explain all of th
147 in reporting their weight as they move into young adulthood, and we examined differences in self-rep
149 ther lower energy and higher PUFA intakes in young adulthood are associated with shorter LTL in cross
150 1 and increases in ICAM-1 over the course of young adulthood are associated with worse indices of LV
151 mal levels of LDL and HDL cholesterol during young adulthood are independently associated with corona
153 on and hazardous drinking in adolescence and young adulthood as well as opioid use in young adulthood
154 15, 2.99) adolescent girls who were obese by young adulthood, as well as young women who were consist
155 Because bone mass accrual is completed by young adulthood, assessing stimulant effects on bone den
156 public schools and were resurveyed twice in young adulthood at mean +/- SD ages 25.3 +/- 1.5 and 31.
157 is syndrome developed during adolescence and young adulthood because a considerable period from the s
159 between childhood body-mass index (BMI) and young adulthood BMI status in a longitudinal cohort.
161 may relate to aspects of physical fitness in young adulthood, but replication of these findings is re
162 was recently shown that increasing age after young adulthood can cause hypothalamic microinflammation
165 r at five points in adolescence and three in young adulthood, commencing at a mean age of 15.5 years
167 smaller effect on BMI during adolescence and young adulthood compared with older adults (P < 0.05).
168 displayed a greater increase in BP levels in young adulthood compared with their counterparts without
169 ent and tracking of lung function throughout young adulthood could be used to provide an early warnin
170 ted that depressive symptoms were highest in young adulthood, decreased across middle adulthood, and
171 However, it is unclear if low RF burden in young adulthood decreases the quantitative burden and qu
172 h was associated with increased mortality in young adulthood even among individuals born late preterm
173 eparation examined during postnatal life and young adulthood exhibited enhanced hippocampal neurogene
174 in childhood and healthier lifestyles during young adulthood explained most of the association betwee
177 The mortality risk was 2-fold higher through young adulthood for persons with ASD than for persons wi
179 additional adjustment for maximum weight in young adulthood further attenuated this association (mul
183 ho lost a parent to suicide in childhood and young adulthood had earlier onset of hospitalization for
184 n institution (n=98 at ages 6 years; n=72 at young adulthood) had persistently higher rates than UK c
185 nd UK controls (n=52 at age 6 years; n=39 at young adulthood) had similarly low levels of symptoms ac
186 ss index (BMI; weight (kg)/height (m)(2)) in young adulthood have been associated with glioma risk, b
187 ancer diagnosed during adolescence and early young adulthood have not been characterised independentl
188 al obesity and hypo-alpha-lipoproteinemia in young adulthood, hypertension and hypertriglyceridemia i
189 lipoproteinemia, and hypertriglyceridemia in young adulthood, hypertension in middle age, and diabete
190 cation are maintained across adolescence and young adulthood, implicating developmentally dissociable
191 of achieving the developmental milestones of young adulthood, important work remains to be done in th
195 ted were at increased risk for depression in young adulthood in models adjusted for sex and family ha
197 y matter density increases from childhood to young adulthood, in contrast with gray matter volume and
199 is strongly associated with hypertension in young adulthood, including an increased risk among those
200 ons that commonly manifest in adolescence or young adulthood, including neuropsychiatric disorders; a
201 e-like patterns of excessive drinking during young adulthood increase the propensity for alcohol use
202 unburns, particularly during adolescence and young adulthood, increase the risk of developing skin ca
203 Cumulative exposure to hyperlipidemia in young adulthood increases the subsequent risk of CHD in
205 course cohort studies spanning childhood and young adulthood, integrated with prospective biospecimen
206 the association of body mass index (BMI) in young adulthood, into middle age, and through late life
209 sence of cardiovascular risk factors (RF) in young adulthood is associated with a lower risk for card
210 ze that higher intake of these B vitamins in young adulthood is associated with better cognition late
213 ther adopting a healthy lifestyle throughout young adulthood is associated with the presence of the l
215 ndings suggest socioeconomic disadvantage in young adulthood is independently associated with later l
216 Maintaining a healthy lifestyle throughout young adulthood is strongly associated with a low cardio
217 t (kg)/height (m)(2)) and premature death in young adulthood is very limited, especially for specific
218 nol but not continuous ethanol or water from young adulthood leads to an increase in DOP-R activity i
220 has been shown to improve vWM performance in young adulthood, little is known on whether a similar en
221 findings suggest that dietary consumption in young adulthood may affect cognitive function in midlife
222 or at least delaying the onset of obesity in young adulthood may lower the risk of developing atheros
223 ciated with similarity in weight change from young adulthood (mean age: 20 y) to middle age (mean age
225 S were assessed at 3 life epochs (childhood, young adulthood, midlife) via questionnaire (2001-2002)
229 and the frequent emergence in their teens or young adulthood of myelodysplastic syndrome (MDS) and ac
230 he symptomatic and neuroanatomic effects, in young adulthood, of exposure to peer verbal abuse during
232 sease (CVD), but effects of exposures during young adulthood on later life CVD risk have not been wel
233 tial consequences of asthma in childhood and young adulthood on lung structure in older adults have n
234 Participants who reported being obese in young adulthood only or in both young and middle adultho
235 ding which blood pressure (BP) components in young adulthood optimally determine cardiovascular disea
239 years (p=0.17), and then marked increases by young adulthood (p=0.0005), with similar effects seen fo
241 in cognitive performance in adolescence and young adulthood, particularly in verbal ability, is asso
243 termined the extent to which lipid levels in young adulthood predict future lipid trajectories and as
245 ical, and health outcomes in adolescence and young adulthood; role transitions, and later health and
247 natures of myocardial and vascular health in young adulthood specify known/novel pathways of metaboli
248 nal age, not only in childhood but also into young adulthood, suggesting that growth factor pathways
250 er predict risk of overweight and obesity in young adulthood than can standards that are currently us
251 r likelihood of having ongoing disorder into young adulthood than did those without such a background
252 tly more likely to develop severe obesity in young adulthood than normal-weight or overweight adolesc
253 vior changes that increase mortality through young adulthood that are stronger than the protective ef
254 f sudden death in childhood, adolescence and young adulthood that presents during fetal life, but is
255 ne patients with longitudinal follow-up into young adulthood that were identified in the internationa
256 djustment for modifiable risk factors during young adulthood, the disparity was no longer statistical
257 individuals progressed through childhood and young adulthood, they occurred at similar rates for indi
259 in increasing the risk of hypertension from young adulthood through middle age and into late life.
261 f the DOP-R in high ethanol consumption from young adulthood through to late adulthood by measuring D
262 h observations spanning the life course from young adulthood to later life, and imputed risk factor t
263 defined by the American Heart Association in young adulthood to middle age and cognitive function in
264 le age, and achieving increased fitness from young adulthood to middle age are associated with less d
266 cumulative exposure to BP over 25 years from young adulthood to middle age is associated with incipie
267 oung adulthood, less decline in fitness from young adulthood to middle age, and achieving increased f
270 e association of cumulative BP exposure from young adulthood to midlife with gait and cognitive funct
271 ), we documented profound aging effects from young adulthood to old age (18-83 y) on neurocognitive a
274 eported famine exposure during childhood and young adulthood was associated with an increased type 2
275 cores.Higher intake of B vitamins throughout young adulthood was associated with better cognitive fun
278 al cohort study, higher intake of F/V during young adulthood was associated with lower odds of preval
284 ght better predict overweight and obesity in young adulthood, we examined the association between chi
285 ring who lost a parent during adolescence or young adulthood were at greatest risk within 1 to 2 year
286 year life course; mean non-HDL-C measured in young adulthood were highly predictive of levels later i
287 ange their properties during late puberty to young adulthood, when bone growth and accrual decelerate
288 ntiation of muscarinic excitation emerges in young adulthood, when executive function typically reach
289 risk for albuminuria and eGFRcys decline in young adulthood, whereas disparities between low-risk bl
290 ship networks are linked to risk for SV into young adulthood, which suggests that network-focused SV
291 omyelitis (EAE) during adolescence and early young adulthood, while an increase in immunological tole
292 al, socioeconomic, and behavioral factors in young adulthood with the observed racial disparity in di
293 ract most often diagnosed in adolescence and young adulthood, with a rising incidence in pediatric po
294 adolescence reported no further self-harm in young adulthood, with a stronger continuity in girls (13
295 idence of acne occurs during adolescence and young adulthood, with approximately 80% of persons aged
296 rder symptoms in childhood, adolescence, and young adulthood, with depression and anxiety becoming mo
297 ntally dynamic from middle childhood through young adulthood, with males and females displaying diffe
298 m engagement in physical activity throughout young adulthood, with stronger associations on concurren
299 methylation (DNAm) in inflammatory genes in young adulthood would be predicted by early life nutriti
300 clerosis risk and change in risk assessed in young adulthood years before subclinical atherosclerosis