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1 tion and was responsible for the majority of zymogenic activity.
2 report crystal structures of legumain in the zymogenic and fully activated form in complex with diffe
3 e corpus including surface pit, mucous neck, zymogenic, and parietal cells expressed Shh.
4 ction of NHE2, which is expressed in mucous, zymogenic, and parietal cells, we prepared mice with a n
5 are progenitors for mucus neck, parietal and zymogenic, but not for pit or enterochromaffin-like cell
6  cellular and genetic pathways that underlie zymogenic cell (ZC) differentiation are poorly understoo
7 lls and a depletion of their neck and mature zymogenic cell descendants, and (ii) an approximately 2-
8 tiated GEP descendant lineages (parietal and zymogenic cell).
9 ck cells (NCs) to digestive enzyme-secreting zymogenic cells (ZCs) involves an increase in rough endo
10                                              Zymogenic cells (ZCs), acid-producing parietal cells (PC
11 l secretory apparatus established by gastric zymogenic cells as they differentiate from their progeni
12 gastric corpus is altered, with parietal and zymogenic cells reduced markedly in number.
13  was the earliest quantifiable aberration in zymogenic cells undergoing transition to a precancerous
14 nd pepsinogen and intrinsic factor-producing zymogenic cells) is accurately controlled, despite marke
15                               Unlike pit and zymogenic cells, parietal cells complete their different
16 g stem cells or differentiated, post-mitotic zymogenic chief cells in the gland base.
17 ch drives the expansion of AMCase-expressing zymogenic chief cells that facilitate chitin digestion.
18 atients with adenocarcinoma, we found normal zymogenic chief cells that were transitioning into SPEM
19 stric parietal cells (PCs) and metaplasia of zymogenic chief cells within 3 days.
20 progenitor of the digestive enzyme secreting zymogenic (chief) cell (ZC).
21 e, specialized secretory vesicles in gastric zymogenic (chief) cells (ZCs) as they differentiate from
22                     Adult zymogen-producing (zymogenic) chief cells (ZCs) in the mammalian gastric gl
23 he role of bulk solvent in transitioning its zymogenic dimeric form to an activated monomeric form.
24 erine protease-1 (MASP-1) and MASP-3 contain zymogenic FD (pro-FD), and it is becoming evident that M
25 lization (first calcium binding site) of its zymogenic form and the possible modes of deactivation (t
26 and a thorough kinetic characterization of a zymogenic form of the enzyme were used to investigate th
27                           The binding of the zymogenic form of urokinase-type plasminogen activator (
28 nderstand the key structural elements in the zymogenic form that participates in the activation proce
29          MMPs are produced in their inactive zymogenic forms, which are subsequently proteolytically
30 undant plasma protein that exists in various zymogenic forms.
31 ing about the detailed transformation of the zymogenic FXIII to its activated form especially in the
32 sults provide the structural determinants of zymogenic inhibition of RgpB by way of a novel inhibitor
33 e mislocalized PCs did not maintain adjacent zymogenic lineage cells in the progenitor state, demonst
34  block in the differentiation program of the zymogenic lineage with an accumulation of pre-neck cells
35 f PCs led to failed patterning of the entire zymogenic lineage: progenitors showed premature expressi
36 cells that express markers of mucus neck and zymogenic lineages, and activation of proliferation.
37 inal differentiation programs of the pit and zymogenic lineages.
38 nts are not active in members of the pit and zymogenic lineages.
39  well as committed precursors of the pit and zymogenic lineages.
40                        In spite of this, the zymogenic nature of some of the molecules and the presen
41 ty required trypsin activation, suggesting a zymogenic nature.
42                    Prourokinase (proUK) is a zymogenic plasminogen activator that at pharmacological
43 more, we report the first X-ray structure of zymogenic prepro-SleC at 2.1 angstrom resolution.
44  disparity can be exploited to create a more zymogenic pro-urokinase (lower intrinsic catalytic activ
45 ain K-mediated proteolytic processing of the zymogenic proform of KLK13.
46 ring their biocompatibility and retained its zymogenic properties until converted by thrombin into an
47 ammed cell death, is executed by a family of zymogenic proteases known as caspases, which cleave an a