ライフサイエンスコーパス: ABC

1 ABC exporters pump substrates across the membrane by cou

2 ABC transporters are polytopic membrane proteins that ut

3 ABC transporters comprise a large and ubiquitous family

4 ABC transporters form one of the largest protein superfa

5 ABC-bleeding score also yielded a higher c-index score i

6 ABC-stroke score achieved C indices of 0.65 with both hs

7 isorder (age 18-45 years; full scale IQ >70; ABC-Irritability subscale 13).

8 induction regimen of 3 drugs (DTG/abacavir [ABC]/3TC), followed by DTG + 3TC maintenance if virologi

9 CD154 (CD40L)-deficient mice did not accrue ABCs, confirming that they arise primarily through T-dep

10 Analysis of shikimic acid accumulation, ABC-transporter gene expression, and cell death were use

11 In addition, ABC-DLBCL frequently displays focal copy number gains af

12 Although ABC-DLBCL has been associated with NF-kappaB activation,

13 biosynthetic enzymes (FatM and RAM2) and an ABC transporter (STR) that are required for symbiosis an

14 al observations raise the possibility for an ABC-DLBCL-specific resistance mechanism that is directed

15 and its interacting protein AtSTAR1 form an ABC transporter complex in the tonoplast.

16 heteroditopic monomers that then generate an ABC sequence-controlled supramolecular terpolymer.

17 However, a mutation in an ABC transporter gene (ABCC2) is linked to Cry1Ac resista

18 and ibrutinib led to tumor regression in an ABC-DLBCL mouse model.

19 ABCA7 is an ABC transporter expressed on the plasma membrane, and ac

20 MacB is an ABC transporter that collaborates with the MacA adaptor

21 R implicated the potential role of MDR49, an ABC transporter, in DDT resistance, however, to date the

22 rted an unusual case in which the loss of an ABC transporter in Candida albicans, orf19.4531 (previou

23 ria that elaborate a capsule dependent on an ABC-type transporter for surface localization.

24 MacB represents an ABC transporter in pathogenic microorganisms with unique

25 This process requires an ABC transporter in the inner envelope membrane with thre

26 We also establish that an ABC transporter of unknown function, YadH, together with

27 cells resulted in differentiation toward an ABC-like phenotype.

28 detected from both sampling schemes using an ABC algorithm.

29 otes type III EBV latency in B cells with an ABC or naive phenotype by enhancing EBNA2 activation of

30 otein, MlaD, forms a ring associated with an ABC transporter complex in the inner membrane.

31 yeast oligomycin resistance 1 gene (YOR1, an ABC transporter) of Saccharomyces cerevisiae phenocopies

32 ively; ABC-bleeding vs HAS-BLED p<0.0001 and ABC-bleeding vs ORBIT p=0.0008).

33 ORBIT; ABC-bleeding vs HAS-BLED p<0.0001 and ABC-bleeding vs ORBIT p=0.0016).

34 , and reduced ABC subfamily G member 5/8 and ABC subfamily B member 11 transporter expression in comp

35 to calculated interlayer distances in AB and ABC alpha-graphityne (3.255 and 3.206 A vs 3.266 and 3.2

36 ET2 is expressed in normal naive B cells and ABC type lymphomas.

37 trality test and Bayesian analysis (EBSP and ABC).

38 ypermutation (SHM) in the ASC (effector) and ABC (memory) lineages.

39 lly combined venetoclax treatment of MCL and ABC-DLBCL xenografts with a pretargeted RIT (PRIT) syste

40 via periplasmic-binding proteins (PBPs) and ABC-transporters.

41 alterations in oxidative phosphorylation and ABC transporters, suggesting energy accumulation and inc

42 e examples of neurotransmitter receptors and ABC transporters with the dual CARC/CRAC motifs are pres

43 major classes of drug transporters, SLC and ABC, in resting human blood neutrophils.

44 We observed that mutated MYD88 WM and ABC DLBCL cell lines, as well as primary WM cells show e

45 growth and survival of mutated MYD88 WM and ABC DLBCL cells, and is a direct target of ibrutinib.

46 rapeutic development in MYD88-mutated WM and ABC DLBCL, and possibly other diseases driven by mutated

47 aneously analyzing antigen-specific ASCs and ABCs in human blood after vaccination against influenza

48 Most of the AR genes were characterized as ABC efflux pumps, RND efflux pumps, and tetracycline MFS

49 l pathways under stressed condition, such as ABC transporters, two-component systems, and carbohydrat

50 a Drosophila multidrug resistant-associated ABC transporter.

51 be regarded as a model system for asymmetric ABC exporters in general, and for TAP in particular.

52 nical and degenerate ATP sites in asymmetric ABC proteins, and of the gating defects caused by two co

53 y models have been developed using bacterial ABC transporters as templates but these have low sequenc

54 idate the recently developed biomarker-based ABC (age, biomarkers [high-sensitivity troponin and N-te

55 The biomarker-based ABC-stroke score was well calibrated and consistently pe

56 ABCC6 (ATP binding cassette transporter C6) ABC transporter are associated with pseudoxanthoma elast

57 t observation of amorphous barium carbonate (ABC), which transforms into a previously unknown barium

58 ABCD2 are peroxisomal ATP-binding cassette (ABC) half-transporters of fatty acyl-CoAs with both dist

59 , encoding distinctive ATP binding cassette (ABC) proteins associated with iron(III)-yersiniabactin i

60 MacB, a member of the ATP-binding cassette (ABC) superfamily.

61 ance (MDR) mediated by ATP-binding cassette (ABC) transport proteins remains a major problem in the c

62 overexpression of the ATP-binding cassette (ABC) transport proteins, like ABCG2, triggering active e

63 rithm to proteins from ATP-binding cassette (ABC) transporter complexes, and obtain accurate predicti

64 processing (TAP) is an ATP-binding cassette (ABC) transporter essential to cellular immunity against

65 enosine triphosphate (ATP)-binding cassette (ABC) transporter expressed at the canalicular membrane o

66 brosis, belongs to the ATP-binding cassette (ABC) transporter family and works as a channel for small

67 annel evolved from the ATP-binding cassette (ABC) transporter family.

68 BP) associated with an ATP binding cassette (ABC) transporter from the probiotic Bifidobacterium anim

69 accept cholesterol via ATP-binding cassette (ABC) transporter G1, an impaired ability of HDL3 to supp

70 ranscriptome involving ATP binding cassette (ABC) transporter genes.

71 terize a non-canonical ATP-binding cassette (ABC) transporter in Escherichia coli that provides energ

72 ntly proposed that the ATP-binding cassette (ABC) transporter Mdr49 functions in the embryonic mesode

73 The ATP-binding cassette (ABC) transporter multidrug resistance protein 1 (MRP1/AB

74 res membrane-localized ATP-binding cassette (ABC) transporter PENETRATION (PEN) 3.

75 s PENETRATION 3 (PEN3) ATP binding cassette (ABC) transporter plays a role in defense against numerou

76 t of the two-component ATP-binding cassette (ABC) transporter TarGH, which exports WTA precursors to

77 nce protein MRP1 is an ATP-binding cassette (ABC) transporter that confers resistance to many antican

78 regulator (CFTR) is an ATP-binding cassette (ABC) transporter that uniquely functions as an ion chann

79 ida adenosine triphosphate-binding cassette (ABC) transporter, PhABCG1, we demonstrate that passage o

80 s study focuses on the ATP-binding cassette (ABC) transporter-dependent pathway, where glycans are co

81 Lan, which encodes an ATP-binding cassette (ABC) transporter.

82 des (O-PS) involves an ATP-binding cassette (ABC) transporter.

83 re orchestrated by the ATP-binding cassette (ABC) transporters and the organic solute carrier family

84 eless be controlled by ATP-binding cassette (ABC) transporters expressed at the blood-brain barrier.

85 ATP-binding cassette (ABC) transporters help export various substrates across

86 trate translocation by ATP-binding cassette (ABC) transporters involves coupling of ATP binding and h

87 rug efflux mediated by ATP-binding cassette (ABC) transporters is one of the major MDR mechanisms.

88 ATP binding cassette (ABC) transporters of the exporter class harness the ener

89 ATP binding cassette (ABC) transporters play critical roles in maintaining ste

90 The ATP-binding cassette (ABC) transporters represent a superfamily of proteins th

91 of adenosine triphosphate-binding cassette (ABC) transporters, ABCA1 and ABCG1.

92 butyrate biosynthesis, ATP-binding cassette (ABC) transporters, flagella assembly and bacterial chemo

93 A member 1 (NR4A1) and ATP-binding cassette (ABC) transporters, influenced the function and different

94 is in part mediated by ATP-binding cassette (ABC) transporters.

95 environments by using ATP-binding cassette (ABC) transporters.

96 d ubiquitous family of ATP-binding cassette (ABC) transporters.

97 stinguishes the aggressive activated B cell (ABC) diffuse large B-cell lymphoma (DLBCL) subtype from

98 d type III latency with an activated B cell (ABC) phenotype.

99 ) is repressed by FOXP1 in activated B-cell (ABC) and germinal center B-cell (GCB) DLBCL cell lines w

100 ell malignancies including activated B-cell (ABC) diffuse large B-cell lymphoma (DLBCL).

101 els representing high-risk activated B-cell (ABC) DLBCL, while no response was observed in a third AB

102 naling is a feature of the activated B-cell (ABC) subtype and is triggered by autocrine production of

103 tion by ibrutinib, and the activated B-cell (ABC) subtype.

104 ll lymphoma (DLBCL) of the activated B-cell (ABC) type, a molecular subtype characterized by adverse

105 The activated B-cell (ABC)-like subgroup resembles post-GC plasmablasts underg

106 ge B-cell (GCB-DLBCL), and activated B-cell (ABC-DLBCL) lymphomas.

107 (FOXP1) in the aggressive, activated B-cell (ABC-DLBCL) subtype.

108 ences with respect to COO (activated B-cell [ABC]-like DLBCL v germinal center B-cell [GCB]-like DLBC

109 origin and nature of age-associated B cells (ABCs) in mice are poorly understood.

110 onal features-termed Age-associated B cells (ABCs)-has recently been identified in both mice and huma

111 s, which we have called 'activated B cells' (ABCs), that were distinct from ASCs and were committed t

112 t to the follicular and marginal zone cells, ABCs displayed significant somatic hypermutation.

113 Chondroitinase ABC injection during chronic stroke without additional t

114 last growth factor (aFGF) and chondroitinase ABC (ChABC).

115 inal injections of the enzyme chondroitinase ABC) augments rewiring of circuits connecting the brain

116 via intraspinal injections of chondroitinase ABC.

117 crosslinking conditions) and chondroitinase-ABC concentrations (mimicking glycosaminoglycan depletio

118 Analyzer of Bioresource Citation (ABC) is a data mining tool extracting strain related pub

119 lipids associated with the Escherichia coli ABC transporter McjD, which translocates the antibacteri

120 The Mce systems are complex ABC transporters that are encoded by different numbers o

121 e that incorporates added benign complexity (ABC), which improves activity 10-fold, and is now access

122 ence using Approximate Bayesian Computation (ABC) and Markov Chain Monte Carlo (MCMC) methods.

123 l, with an approximate Bayesian computation (ABC) framework for parameter estimation of neighborhood

124 within an Approximate Bayesian Computation (ABC) inference scheme, and suggest that parameters simul

125 Approximate Bayesian computation (ABC) methods provide an elaborate approach to Bayesian i

126 d based on approximate Bayesian computation (ABC) to discriminate among three well-studied network mo

127 (SDM) and approximate Bayesian computation (ABC) to explore the changes in population size and distr

128 thin the heart of cohesin's highly conserved ABC-like ATPase machinery and find that both ATPase site

129 unction that are not present in conventional ABC transporters.

130 prepared from amphiphilic block copolymers (ABCs) have found numerous applications in pharmaceutical

131 dentified the previously identified ctrABCD (ABC transporter) operon, a lipA (kpsC)-like gene, a lipB

132 uman tumors, including aneurysmal bone cyst (ABC), and the related benign lesion nodular fasciitis.

133 he producing bacteria that utilize dedicated ABC transporters to provide self-immunity.

134 essed in trans with the mutant CBM-deficient ABC transporter.

135 NF-kappaB and selectively kill BCR-dependent ABC DLBCL lines, supporting their clinical evaluation in

136 We detected ABCs in humans after infection with Ebola virus or influ

137 ype of MWW and can be viewed as a disordered ABC-type stacking of MWW-layers.

138 y, most tumors with p100 signaling displayed ABC phenotype and harbored ABC-associated mutations in g

139 DLBCL) compared with activated B-cell DLBCL (ABC-DLBCL) and predict poor outcomes.

140 to germinal center (GC) B-cell (GCB) DLBCL, ABC DLBCL cell lines expressed high levels of the AP-1 f

141 In activated B cell DLBCLs (ABC-DLBCLs), a class of DLBCLs that respond poorly to cu

142 hanges as previously proposed for multi-drug ABC exporters.

143 (4) standard of care: 3-drug regimen of DTG/ABC/3TC.

144 e profiles indicated distinct roles for each ABC transporter in root exudation.

145 ) operons which have been proposed to encode ABC-like import systems.

146 ing the structure and mechanism of an entire ABC transporter superfamily and the many diverse functio

147 er prevalence of typical Western type (EPIYA ABC) and variant AB'C type (EPIYT B) CagA.

148 t enables substrate transfer in a eukaryotic ABC exporter.

149 evelopment, and supports the roles of "faded ABC model" mechanism and miRNA-targets regulations under

150 These are the first ABC transporter inhibitors shown to block ATPase activit

151 Current alternating access models for ABC exporters including the multidrug and Lipid A transp

152 omeostasis to improve treatment outcomes for ABC-DLBCLs.

153 f pathogens provide the signals required for ABC differentiation.

154 the T-bet transcription factor, a marker for ABCs, were generated after multiple cell divisions from

155 ee-component synthesis of the functionalized ABC ring system of the Aspidosperma alkaloid jerantinine

156 h atrophy of basal ganglia and cerebellum (H-ABC).

157 ffects of TUBB4A mutations responsible for H-ABC (p.Asp249Asn), DYT4 (p.Arg2Gly), a severe combined p

158 The H-ABC mutation (p.Asp249Asn) that exhibits a combined neur

159 gnaling displayed ABC phenotype and harbored ABC-associated mutations in genes such as MYD88 and PIM1

160 Health, Aging, and Body Composition (Health ABC) (n = 1015) study.

161 processing (TAP) is a 150 kDa heterodimeric ABC transport complex that selects peptides for export i

162 DEER measurements on the heterodimeric ABC exporter TM287/288 from Thermotoga maritima, which c

163 pregulated surface expression of HLA-DR, HLA-ABC, CD40, CD80, CD83, and CD86, induced naive alphabeta

164 fferentiating heterodimeric from homodimeric ABC exporters.

165 rameter in the mechanism of this homodimeric ABC transporter.

166 Forty-eight human ABC transporters have been identified in the genome, and

167 vitro and in vivo, as well as primary human ABC-DLBCL specimens ex vivo.

168 Using the energy of ATP hydrolysis, ABC transporters catalyze the trans-membrane transport o

169 ATP binding by Type I ABC transporters (importers of amino acids, sugars, pept

170 ng the broadest IgE sensitization stage (ie, ABC) had significantly higher risk of mite-related AR an

171 To determine what Igs ABCs express, we sequenced VH and Vkappa rearranged gene

172 In Type II ABC transporters (importers of trace elements, e.g. vita

173 We propose that other Type II ABC transporters likely share the fundamentals of this m

174 mes Atlas of Biosynthetic gene Clusters (IMG-ABC).

175 w tools and features add to the value of IMG-ABC's vast body of BC data, facilitating their in-depth

176 Here, we describe an update of IMG-ABC, which includes ClusterScout, a tool for targeted id

177 ription factor activator protein-1 (AP-1) in ABC DLBCL.

178 As a result, Dox-induced cytotoxicity in ABC-DLBCLs is often dependent on oxidative stress, rathe

179 D79B mutations are substantially enriched in ABC-DLBCL compared with germinal center B-cell DLBCL.

180 ong-range conformational couplings, e.g., in ABC exporters or other ATP-driven molecular machines.

181 ated the expression of nearly 3,000 genes in ABC DLBCL cells, and the chromatin surrounding many of t

182 sequence, ATF3 expression was much higher in ABC vs GCB DLBCL cell lines.

183 e approach, encoding low-chi interactions in ABC bottlebrush triblock terpolymers (chiAC [Formula: se

184 Additionally, silencing p100 in ABC-DLBCL cells resulted in a GCB-like phenotype, with s

185 ) inhibitor AZD8835 showed marked potency in ABC DLBCL models, whereas the protein kinase B (AKT) inh

186 of normal B cells, suggesting p100's role in ABC phenotype development.

187 steps of astaxanthin biosynthesis, including ABC transporters, cytochrome P450 enzymes, and an acyltr

188 der to assess the contribution of individual ABC transporters to root exudation, we performed an LC-M

189 th CHOP to trigger oxidative stress and kill ABC-DLBCL cells in preclinical models.

190 B-cell like (GCB) and activated B-cell like (ABC).

191 Activated B-cell-like (ABC) and germinal center B-cell-like diffuse large B-cel

192 ll-like (GCB) and the activated B-cell-like (ABC) DLBCLs.

193 ng, a hallmark of the activated B cell-like (ABC) subtype of diffuse large B cell lymphoma (DLBCL), e

194 idence suggests they are components of lipid ABC transporters.

195 ce protein (BCRP, ABCG2) are the three major ABC transport proteins conferring resistance to many str

196 iply effective inhibitors of the three major ABC transporters.

197 Many ABC transporter genes were regulated in the bacteria att

198 implication in the regulation of metastable ABC transporters and other plasma membrane proteins acti

199 A modified ABC-bleeding score using alternative biomarkers (haemato

200 te-coupled conformational cycle of the mouse ABC efflux transporter P-glycoprotein (Pgp; also known a

201 The multidrug ABC transporter, aadE, bacA, acrB, tetM, tetW, vanR and

202 ilico experiments revealed that MYD88-mutant ABC-DLBCL cells in particular display an actionable addi

203 apture the transformation of nanoparticulate ABC into gortatowskite crystals, highly anisotropic shee

204 The O12 ABC transporter also binds its cognate O-PS via a CBM, a

205 P hydrolysis and defined the architecture of ABC exporters, a detailed structural dynamic understandi

206 PRI over time when paired with a backbone of ABC/3TC: 16% per 5 years (95% confidence interval [CI],

207 ic and immunophenotypical characteristics of ABC-DLBCL.

208 nistic divergence within the efflux class of ABC transporters.

209 ndirect FOXP1 transcriptional enforcement of ABC-DLBCL hallmarks, including the classical NF-kappaB a

210 f tumors, which recapitulate key features of ABC and nodular fasciitis; however, the identity of USP6

211 nt activity on the stability and function of ABC transporters or any other enzyme.

212 important element controlling the growth of ABC DLBCL.

213 in event-free survival (hazard ratio [HR] of ABC-like disease v GCB-like disease, 1.0; 95% CI, 0.6 to

214 Compared with earlier implementations of ABC model choice, the ABC RF approach offers several pot

215 Broad-spectrum inhibitors of ABC transport proteins can be of great use in cancers th

216 the compound, suggesting the involvement of ABC transporters in the uptake or intracellular accumula

217 fuse distribution, respectively, in lines of ABC and GCB subtypes.

218 oach led to impaired growth of a majority of ABC DLBCL cell lines.

219 nerated a novel autochthonous mouse model of ABC-DLBCL that could be used as a preclinical platform f

220 Specifically, in cell line models of ABC-DLBCL, Dox is often enriched in the cytoplasm away f

221 nsis and exposed to MLF showed modulation of ABC and solute liquid carrier transporters gene transcri

222 hat reside in the highly conserved motifs of ABC transporters, involved in ATP binding.

223 A large number of ABC and SLC transpoters exist; however, only a small num

224 educed 5hmC levels in comparison to those of ABC type lines.

225 therapeutic approaches for the treatment of ABC-DLBCL.

226 rter activity, as excretion of BR depends on ABC and SLC transporters.

227 a examined the effects of PL inactivation on ABC renewal in the same rats.

228 Only ABC/3TC-containing regimens were associated with an incr

229 kinase signaling in mutated MYD88 WM and/or ABC DLBCL cells.

230 and a backbone containing either TDF/FTC or ABC/3TC.

231 for HAS-BLED vs 0.68 [0.65-0.70] for ORBIT; ABC-bleeding vs HAS-BLED p<0.0001 and ABC-bleeding vs OR

232 a feature distinguishing CFTR from all other ABC transporters is the helix-loop transition in transme

233 Our ABC framework used both topological and distance-based t

234 Finally, we applied our ABC inference on two different outbreaks from the Swedis

235 epared from polystyrene-poly(ethylene oxide) ABCs.

236 propose a novel mechanism for toxic peptide ABC exporters that only requires the transient opening o

237 (SDP1) triacylglycerol lipase or PEROXISOMAL ABC TRANSPORTER 1 (PXA1), here we show that TAG is a key

238 the interaction between the Yersinia pestis ABC heme importer (HmuUV) and its partner substrate-bind

239 In this key position, ABC transporters can mediate multidrug resistance in can

240 rs might be most effective in poor prognosis ABC-DLBCL, which shows higher levels of IL10RA, JAK2, an

241 n Ankyrin-like protein, IKI3 family protein, ABC transporter G family and pentatricopeptide repeat pr

242 These observations show that quiescent ABCs are Ag-experienced cells that accumulate during T c

243 increased hepatic inflammation, and reduced ABC subfamily G member 5/8 and ABC subfamily B member 11

244 .59-0.63] vs 0.65 [0.62-0.67], respectively; ABC-bleeding vs HAS-BLED p<0.0001 and ABC-bleeding vs OR

245 he ATPase component of a putative riboflavin ABC transport system.

246 Scc2 (Nipbl) stimulates cohesin's ABC-like ATPase and is essential for loading cohesin ont

247 er enzymes that use ATP as an energy source, ABC transporters are notorious for having high levels of

248 minant Bacteroides that lack glycan-specific ABC-transporters.

249 bilities may be poorly evaluated by standard ABC techniques.

250 ged across the known lifetime of the stands (ABC = average biomass change).

251 Furthermore, FX1 suppressed ABC-DLBCL cells in vitro and in vivo, as well as primary

252 ibution introduces main-chain supramolecular ABC and ABB'A block copolymers sustained by orthogonal m

253 dged in the transmembrane bundle of the SUR1-ABC core connected to the first nucleotide binding domai

254 The SUR1-ABC core is found in an unusual inward-facing conformati

255 d by the Active Bacterial Core surveillance (ABCs) of the Centers for Disease Control and Prevention

256 ate their therapeutic efficacy for targeting ABC-DLBCL.

257 It is well documented that ABC-DLBCL cases have a significantly poorer survival res

258 Although it is established that ABC half-transporters have at least to dimerize to gener

259 These findings indicate that ABC-DLBCL is a BCL6-dependent disease that can be target

260 ze of datasets and complexity of models that ABC can handle.

261 ter deliberate immunization, suggesting that ABCs have undergone mild stimulation from endogenous Ags

262 The ABC model is widely used as a genetic framework for unde

263 The ABC score should be considered an improved decision supp

264 The ABC transporter McjD exports the antibacterial peptide M

265 The ABC-bleeding score (age, biomarkers [GDF-15, cTnT-hs, an

266 The ABC-bleeding score performed better than HAS-BLED and OR

267 endent NRAMP family transporter MntH and the ABC transporter MntABCD.

268 ter, schizokinen transporter (SchT), and the ABC-type transport system FhuBCD.

269 erent drug efflux capability mediated by the ABC transporter ABCG2 using the side population assay, a

270 sol:membrane interface, before export by the ABC transporter.

271 of patients at higher risk estimated by the ABC-CHD (Age, Biomarkers, Clinical-Coronary Heart Diseas

272 ier implementations of ABC model choice, the ABC RF approach offers several potential improvements: (

273 uman pancreatic KATP channel, containing the ABC transporter SUR1 and the inward-rectifier K(+) chann

274 nent of a wider acetylation signature in the ABC subgroup of diffuse large B-cell lymphoma, and one o

275 d separately, the outcome of patients in the ABC subgroup was inferior to that of patients in the GCB

276 mechanism of conformational coupling in the ABC transporter BtuCD-F, which imports vitamin B12 acros

277 t manipulation of miR-181a expression in the ABC-like DLBCL genetic background may result in a signif

278 and to NF-kappaB signaling dependency in the ABC-like DLBCL subgroup.

279 or growth rate and prolonged survival in the ABC-like DLBCL xenografts compared with the GCB-like DLB

280 on of NF-kappaB activation and growth of the ABC subtype of DLBCL cell lines in vitro at high concent

281 y network orchestrates the production of the ABC transporter BceAB, the UPP phosphatase BcrC and the

282 with PstS, the periplasmic component of the ABC transporter complex (PstSACB) involved in phosphate

283 These findings suggest that the ABC-mediated uptake of raffinose provides an important c

284 Thus, B cells with the ABC phenotype and transcriptional signature appear durin

285 ith nonDE within the GCB, but not within the ABC subgroup.

286 As I shall explain in this Primer, the 'ABC model' is a simple and satisfying explanation for ho

287 L, while no response was observed in a third ABC model, and intermediate responses were observed in t

288 s, which define the crucial function of this ABC transporter in human immunity and health.

289 xplain the wide functional diversity of this ABC transporter subfamily.

290 With this ABC we infer recombination rate, mutation rate, and reco

291 This "ABC-CHD" model had high discriminatory ability for CV de

292 ical strigolactones lack the fused tricyclic ABC-ring system commonly present in canonical-type strig

293 lete 3D structural information on the unique ABC ion channel, CFTR, hinders elucidation of its functi

294 or the proliferation of otherwise-unmodified ABC-DLBCL lines.

295 For the denominator, we used ABCs catchment area population estimates from the 2011 t

296 nes were present, and genes encoding various ABC transporters, glutamate synthase and CO oxidation we

297 dentified conserved pathways associated with ABC-DLBCL pathology.

298 g their clinical evaluation in patients with ABC DLBCL.

299 edicts treatment outcome among patients with ABC-DLBCL, but not patients with GCB-DLBCL.

300 inferior outcome compared with patients with ABC-like DLBCL without DE (5-year PFS rate, 39% [95% CI,