ライフサイエンスコーパス: GC

1 GC-IMS was used to detect the volatile compound profile

2 GC-QTOF-MS extracts were run in negative chemical ioniza

3 GC/MS analysis of the volatiles identified common consti

4 0 (19.25%) O&P examinations, 47/204 (23.04%) GC-EIA, and 249/1,229 (20.26%) STCUL were ordered after

5 designed sequence containing a mixture of 14 GC pairs and 11 AU pairs resists condensation relative t

6 A 36.6% GC was found in prophages in contrast to 39% in H. pylor

7 wever, clinical situations are emulated by a GC intervention initiated in the midst of rampant inflam

8 dent phosphorylation sites is required for a GC-A conformation capable of transmitting the hormone bi

9 to-onion puree has been investigated using a GC-MS fingerprinting approach.

10 with a retention time locked method using a GC-QTOF-MS pesticide library (containing exact mass frag

11 es, clusters of proliferating B cells with a GC-like phenotype can be generated in the organoids at c

12 Recently, the genuine leptin gene with a GC-rich ( 70%) repetitive-sequence content was identifie

13 of systemic energy metabolism, and aberrant GC action is linked to metabolic dysfunctions.

14 Although self-ligands directly affect GC B cell responses, the loss of Mer in dendritic cells

15 mutation drives GC content lower in already GC-poor regions, and using our precise context-dependent

16 Although GC's involvement in encoding the chemical identity and h

17 ectrodes (ISEs), the surfaces of Pt, Au, and GC electrodes were coated with 0.1, 1.0, 2.0, and 4.0 mu

18 effects despite large variations in BOD5 and GC/MS profiles.

19 suggest that polymorphisms in the CYP2R1 and GC gene may contribute to variation in baseline serum 25

20 Results from adsorption-desorption and GC headspace analyses showed that these MOFs could encap

21 atile compounds were analyzed using HPLC and GC-MS.

22 then allowed excellent separation (by LC and GC methods) of carbazole, dibenzothiophene, fluorenones,

23 xamined the interaction between p38 MAPK and GC-A signaling.

24 Thanks to GC-O and GC-MS analysis, cooked fruit notes were identified as 3-

25 at late times for maintenance of the Tfh and GC B cells.

26 he POLD1-R689W are GC-->TA transversions and GC-->AT transitions, with transversions showing a strong

27 were analysed using spectrophotometry-UV and GC-MS-SPME, respectively.

28 In a sparsely connected model of MCs and GCs, we found first that interglomerular synchrony was g

29 y synchronizing interactions between MCs and GCs.

30 Applying GC-MS-olfactometry and aroma extract dilution analysis (

31 of mutations induced by the POLD1-R689W are GC-->TA transversions and GC-->AT transitions, with tran

32 ngle measurements over PEDOT(PSS)-coated Au, GC, and Pt electrode surfaces.

33 cin complex 1-dependent canonical autophagy, GC B cell autophagy occurred predominantly through a non

34 duction, thereby tipping the balance between GC formation and degradation.

35 But when pol32 or pif1 mutants block BIR, GC increases 3-fold, indicating that the steps blocked b

36 aling by interaction with the membrane bound GC receptor, followed by stimulation of beta-catenin and

37 n the acid-catalyzed process and analysis by GC-IRMS gives a smaller than expected value of 1.022 for

38 vents and its extracts were characterized by GC-FID and HPLC-UV.

39 ve compounds of leaves were characterized by GC-Olfactometry (GC-O) and Aroma Extract Dilution Analys

40 o assess the volatile and fixed compounds by GC-MS and UHPLC-QTOF-MS.

41 aroma active compounds also distinguished by GC-O.

42 Analysis of fractions by GC x GC/MS also allowed a series of thioxanones to be id

43 rvone and linalool chemotypes, identified by GC-MS analyses of the essential oils.

44 Molar sums of PFASs obtained by GC-MS, LC-MS/MS, and precursors were compared to total f

45 ion of AAI and airway hyperresponsiveness by GCs.

46 d apoptosis-related genes in gastric cancer (GC) and adjacent mucosa with atrophic gastritis or intes

47 Gastric cancer (GC) has a poor prognosis with wide variation in survival

48 ers, but the role of FPR2 in gastric cancer (GC) has not yet been elucidated.

49 sa associated with increased gastric cancer (GC) risk.

50 areas, with more ectopic SC and germ cells (GC) than DBP-FW treatment; DBP-LW induces no dysgenesis.

51 ancy, and the ability of mouse glioma cells (GC) to be cultured under stem cell conditions as compare

52 ream inputs to PCs-excitatory granule cells (GCs) and inhibitory molecular layer interneurons-in proc

53 s receive inputs from dentate granule cells (GCs) and project back to GCs locally, contralaterally, a

54 etween mitral cells (MCs) and granule cells (GCs) can generate synchronized oscillations in the roden

55 m interactions with GABAergic granule cells (GCs), yet the incidence of MC-GC connections is very low

56 erous functions executed by granulosa cells (GCs) in ovarian physiology, the role of multifunctional

57 s) flanking two dumbbell-shaped guard cells (GCs)-is linked to improved stomatal physiology.

58 d CXCL-8(+) DLBCL from both germinal center (GC) and non-GC subtypes.

59 ciency enhanced CD4 TFH and germinal center (GC) B cell numbers in naive mice and hastened islet allo

60 impaired the acquisition of germinal center (GC) B cell phenotype, plasma cell generation, and virus-

61 elper T (Tfh) cells promote germinal center (GC) B cell survival and proliferation and guide their di

62 e activated B cell-like and germinal center (GC) B cell-like subtypes of diffuse large B cell lymphom

63 inhibiting proliferation of germinal center (GC) B cells.

64 toward antigen in activated germinal center (GC) B cells.

65 we show that among B cells, germinal center (GC) cells exhibited the highest rate of autophagy during

66 The germinal center (GC) reaction begins with a diverse and expanded group of

67 tion of viral reservoirs in germinal center (GC) T follicular helper (Tfh) cells.

68 s, many of which arise from germinal center (GC)-experienced B cells.

69 s successfully competed in germinal centers (GC), underwent extensive somatic hypermutation, and diff

70 nating in the formation of germinal centers (GC).

71 quired for B cells to form germinal centers (GC).

72 nd associates with ectopic germinal centers (GCs) development and inflammation in the thymus.

73 lupus is the formation of germinal centers (GCs) in lymphoid tissues where self-reactive B cells exp

74 a cells (PCs) derived from germinal centers (GCs) secrete the high-affinity antibodies required for l

75 ular pathway gives rise to germinal centers (GCs) that can take weeks to fully develop.

76 body production through the germinal centre (GC) response is a pivotal process in adaptive immunity.

77 yethylene glycol (PEG) with glycol chitosan (GC).

78 ehensive two-dimensional gas chromatography (GC x GC) retention times can be used to predict 26 relev

79 f olive oils relative to gas chromatography (GC) and (1)H NMR.

80 ith a polar or non-polar gas chromatography (GC) column coupled to ion mobility spectrometry (IMS) ha

81 used in combination with gas chromatography (GC) coupled to hybrid quadrupole time-of-flight (QTOF) m

82 ment within NP that is distinct from classic GC-mediated mechanisms.

83 cytoskeleton remodeling in the growth cone (GC) during axon outgrowth and pathfinding.

84 Compared to conventional GC-FID, the signal-to-noise ratio has been increased by

85 They are engaged by converging GC-inputs and provide dendritic inhibition to the DG cir

86 Functional analyses of cultured GC from these tumors showed that cells of NSC-like origi

87 RhoGC is a rhodopsin (Rho)-guanylyl cyclase (GC) gene fusion molecule that is central to zoospore pho

88 terase (PDE6) and retinal guanylyl cyclases (GCs), and mutations in genes that disrupt cGMP homeostas

89 Nine polymorphisms in VDR, CYP24A, CYP27B1, GC, and RXRA were analyzed as effect modifiers of 25(OH)

90 the stomatal regulator AtMUTE, which defines GC precursor fate in Arabidopsis The novel role of BdMUT

91 ures of DCs usually differ for gene density, GC content, housekeeping genes, and recombination freque

92 isotype-switched ASC requires CD19-dependent GC formation in CLN.

93 auses strand- and sequence-context-dependent GC --> AT transitions.

94 aphy coupled with electron capture detector (GC-ECD), and validated for screening and quantification

95 n SLB-IL111 was evaluated based on different GC oven temperature programs.

96 file, which correlates with the differential GC content between adjacent introns and exons.

97 ion suppressed T follicular differentiation, GC B cell frequency, and class switching of GC B cells t

98 omic distances, showing that mutation drives GC content lower in already GC-poor regions, and using o

99 11beta-HSD1 shapes endogenous GC action and is immunomodulatory.

100 ton, and phase imaging techniques to enhance GC contrast.

101 expression in B cells substantially enhanced GC B cell responses and anti-Plasmodium Ab production.

102 s a double-negative feedback loop, enhancing GC-regulated transcription to synergistically kill even

103 The mechanisms that connect excess GC to tissue inflammation remain unknown.

104 recoveries were in the range of 92-103% for GC-MS/MS and 108-117% for GC-HRMS.

105 nge of 92-103% for GC-MS/MS and 108-117% for GC-HRMS.

106 However, accounting for GC content bias in ChIP-seq is challenging because the b

107 rea, whereas downregulation is essential for GC formation.

108 aphy-tandem mass spectrometry (GC-MS/MS) for GC-amenable pesticides; (ii) hydrophilic interaction liq

109 15.9 mum) for MRW, 38 mum (+/- 3.4 mum) for GC-IPLT, and 38 mum (+/- 4.2 mum) for cpRNFLT.

110 No differences were observed for GC signaling or other drug metabolism/transport-related

111 roduct of the SMARCA2 gene) are required for GC-regulated expression of the blocked genes but not for

112 as 0.1 per thousand and 0.2 per thousand for GC-IRMS.

113 2 rescued this effect, suggesting a role for GCs in promoting c-src-mediated proteosomal degradation

114 Glucocorticoid (GC)-induced ocular hypertension (OHT) is a serious adver

115 Glucocorticoid (GC)-refractory acute rejection (AR) is a risk factor for

116 The mechanisms driving glucocorticoid (GC) insensitivity in patients with severe asthma are sti

117 t al. describe mechanisms of glucocorticoid (GC) downregulation of wound healing by interaction with

118 Glucocorticoids (GCs) are important regulators of systemic energy metabol

119 Glucocorticoids (GCs; referred to clinically as corticosteroids) are ster

120 Although glucocorticoids (GCs) are a mainstay in the clinical management of asthma

121 spite the widespread use of glucocorticoids (GCs), their anti-inflammatory effects are not understood

122 The synthetic glucocorticoids (GCs) dexamethasone, mometasone furoate, and triamcinolon

123 7-kb DNA contigs with an exceptionally high GC content, each containing a long inverted repeat with

124 s of interest tend to be more common in high GC-content regions, which confounds real biological sign

125 The study results confirmed that NA008 High GC reference material is fit for the purpose of being us

126 ry study, conducted using blinded NA008 High GC reference material to assess reproducibility among se

127 g global sequence features, such as the high GC content in nucleosome-rich regions.

128 We found significantly higher GC content in smaller eccDNAs (<500 bp) than the larger

129 Transgenes with higher GC content exhibited increased transcript and protein ac

130 However, GCs are considered to be immune privileged for antiviral

131 fingerprints (obtained from an off-line HPLC-GC-FID system) for the quantification of extra virgin ol

132 HS-GC results showed that human saliva strongly decreased t

133 chemical screen in zebrafish that identifies GCs as activators of hypoxia-inducible factors (HIFs) in

134 eration short hairpin RNA screen to identify GC-regulated "effector" genes that contribute to cell de

135 ic gastritis or intestinal metaplasia (AG/IM GC+), as well as in atrophic gastritis or intestinal met

136 aplasia mucosa of patients without GC (AG/IM GC-) and in control biopsy samples of non-transformed ga

137 nflammatory functions of GR and help improve GC-based therapy.

138 mitochondrial homeostasis and alterations in GC and antibody-secreting cells.

139 found that the levels of FPR2 expression in GC were positively correlated with invasion depth, lymph

140 uential deletion of individual glutamates in GC-A-8E progressively increased the Km Double Ala substi

141 We also found that retained introns, high in GC content, served as substrates for the formation of tr

142 ucleotide polymorphisms of genes involved in GC signaling (GR, GLCCI1) and drug metabolism and transp

143 Despite the marked reduction of GC-A mRNA in GC-A KO podocytes to 1% of the control level, Podo-GC-A

144 Km 23- to 70-fold but the same mutations in GC-A-8E only increased the Km 8-fold, consistent with on

145 probabilistic description of peak overlap in GC-MS separations to determine the probability of obtain

146 particular: structural variants, variants in GC-rich regions, which have significantly improved cover

147 n years, whereas there was more variation in GC incidence in patients with IM (0.38 to 17.08 per 1000

148 Moreover, RNAi-mediated knockdown of YY1 in GC cells significantly decreased ATP6V1A mRNA and protei

149 Cs under conditions where ACs accumulated in GCs of Sle1bMer(-/-) mice.

150 did not exhibit increased AC accumulation in GCs compared with B6.Sle1b mice, indicating that Mer imm

151 d class-switching of autoreactive B cells in GCs under conditions where ACs accumulated in GCs of Sle

152 mechanisms by which ATP6V1A overexpressed in GCs, we investigated the relationship between transcript

153 Patch clamp recordings in GCs reveal that movement is accompanied by changes in mo

154 e properties of microbial genomes, including GC content and genome size, are known to vary widely amo

155 nts on vitamin D synthetic pathway including GC (rs4588, rs7041, rs22020, rs2282679), CYP2R1 (rs20607

156 rs the potential to spatially map individual GCs to underlying amacrine, bipolar, horizontal, photore

157 uency, affinity, and avidity for interclonal GC competition and indicate that germline-targeting immu

158 ent kinase-9 (CDK9), which is recruited into GC-induced GR:GRIP1:CDK9 hetero-complexes, producing dis

159 he diversity of B cell clones recruited into GCs are unclear.

160 One aspect of gene variation is GC content, which differs across species and is bimodal

161 read lengths than conventional methods, less GC bias, and the ability to read DNA base modifications.

162 HS-SPME and LLE-GC/MS analyses revealed that metabolism of the compounds

163 Adding more glutamates to make GC-A-9E or GC-A-10E had little effect on activity, and s

164 granule cells (GCs), yet the incidence of MC-GC connections is very low, around 4%.

165 esult from the presence of basally migrating GC and a weakened basal lamina, whereas GC migration was

166 In advanced glaucoma, more GC-IPL tissue remains above the measurement floor compar

167 J passenger allele system to assay, in mouse GC B cells, sequence-intrinsic SHM-targeting rates of nu

168 ctrometry (LC-MS) and gas chromatography-MS (GC-MS) data relative to labelling experiments.

169 tes measured by ultra-high performance LC-MS/GC-MS and retinol concentration (from HPLC) using linear

170 We present a new GC-specific MAKER annotation protocol to predict new and

171 ation products were analyzed by UV/vis, NMR, GC-MS, and EPR.

172 Pharmacologic long-term treatment with a NO-GC stimulator altered auditory nerve responses but did n

173 Interestingly, NO-GC stimulation exacerbated the loss of auditory nerve re

174 DLBCL from both germinal center (GC) and non-GC subtypes.

175 These studies revealed that the novel GC/rGO-Nf@Ag6 sensor electrode could be a potential cand

176 bottleneck has been our inability to observe GCs and their degeneration in the living human eye.

177 n between Mer and TLRs in the development of GC responses and autoimmunity.

178 ion is considered the main driving factor of GC development.

179 all, our results highlight the importance of GC formation in TLS during tumor development and treatme

180 vel, simple, and reproducible mouse model of GC-induced OHT.

181 /beta-catenin-mediated anti-proliferation of GC cells, which is different from the canonical SOCE/Ca(

182 a wide variation in annual incidence rate of GC from premalignant lesions.

183 The incidence rate of GC in patients with GA ranged from 0.53 to 15.24 per 100

184 Despite the marked reduction of GC-A mRNA in GC-A KO podocytes to 1% of the control leve

185 ccumulation and prolonged tumor retention of GC-PEG-PpIX were realized after intravenous injection, w

186 ished to June 2016 investigating the risk of GC in individuals with GA or IM.

187 Although selection of GC B cells is triggered by antigen-dependent signals del

188 tified, in which there were eight studies of GC incidence in GA and nine in IM cohorts (two articles

189 GC B cell frequency, and class switching of GC B cells to IgG1.

190 d B cell tolerance and reduced the amount of GCs and pathogenic autoantibody.

191 Moreover, increased apoptosis of GCs and follicular atresia, reduced ovulation rate, and

192 may facilitate the temporal coordination of GCs with activity patterns governed by the medial septum

193 idea of how to improve the safety profile of GCs, recent studies have investigated the complex mechan

194 y on GC layer somas, including projection of GCs onto photoreceptors and identification of the primar

195 eaves were characterized by GC-Olfactometry (GC-O) and Aroma Extract Dilution Analysis (AEDA): volati

196 We perform morphometry on GC layer somas, including projection of GCs onto photore

197 y lymphoid organs, they may join the ongoing GC response.

198 ns that mediate feed-forward inhibition onto GCs.

199 Optimized GC-qMS parameters (dwell time 70 ms, 2 most abundant ion

200 ngs and the genital tract with the optimized GC-coated LPN adjuvant upon nasal immunization of mice w

201 Adding more glutamates to make GC-A-9E or GC-A-10E had little effect on activity, and sequential d

202 tate replication through telomeres and other GC-rich regions.

203 ssion of the blocked genes but not for other GC-regulated genes.

204 orting conventional therapies and overcoming GC resistance in pediatric T-ALL patients.

205 O podocytes to 1% of the control level, Podo-GC-A KO mice and control littermates did not differ in B

206 However, only Podo-GC-A KO mice developed massive albuminuria (controls: 35

207 ements for B7 and CD40 expression in primary GC responses to vaccine immunization with protein antige

208 oreceptors and identification of the primary GC subtypes, even beneath nerve fibers.

209 HT) is a serious adverse effect of prolonged GC therapy that can lead to iatrogenic glaucoma and perm

210 rap gas chromatography-mass spectrometry (PT-GC-MS).

211 hromatography/flame ionisation detection (Py-GC/FID), pyrolysis-gas chromatography/mass spectrometry

212 compositions (C, H, N, O, S), Py-GC/FID, Py-GC/MS and SEM imaging reveal extensive degradation of th

213 etection in selected ion monitoring mode (py-GC-SIM-MS).

214 Elemental compositions (C, H, N, O, S), Py-GC/FID, Py-GC/MS and SEM imaging reveal extensive degrad

215 sis-gas chromatography/mass spectrometry (Py-GC/MS) and scanning electron microscopy (SEM).

216 d the results compared with the quantitative GC-MS data.

217 d immunomodulatory functions of Mer regulate GC responses to prevent the development of autoimmunity.

218 edominately singly scattered light-to reveal GCs has led to a focus on multiply-scattered, fluorescen

219 carries substantial information that reveals GC somas, axons, and other retinal neurons and permits t

220 The RuNPs/GC based DPV technique could be used to determine the co

221 the electroreduction of UO2(2+) by the RuNPs/GC was studied using density functional theory calculati

222 f the surface was identified as a saturable, GC concentration-dependent increase in particle size and

223 /Cryptosporidium enzyme immunoassay screens (GC-EIA) performed for patients hospitalized >3 days.

224 C1-5 alkyl homologues were easily separated (GC x GC/MS), allowing high-quality mass spectra (EI) to

225 li is well studied, it is unknown how single GC neurons process olfactory stimuli emanating from the

226 ectrode recordings to investigate how single GC neurons respond to intraorally delivered tastants and

227 follicular helper T cells, antigen-specific GC B cells, and high-affinity class-switched antibody pr

228 e difficulty of quantifying antigen-specific GC Tfh cells and the difficulty of tracking GC in human

229 and Gas Chromatography - Mass Spectrometery (GC-MS) together with (13)C stable isotope-labelled gluco

230 nductively coupled plasma mass spectrometry (GC-ICPMS) measurement conditions are employed for the th

231 Gas chromatography-mass spectrometry (GC-MS) analysis revealed the presence of 38 compounds wi

232 chromatography coupled to mass spectrometry (GC-MS) analysis.

233 Gas chromatography-mass spectrometry (GC-MS) and liquid chromatography-tandem mass spectrometr

234 robust gas chromatography-mass spectrometry (GC-MS) method for the simultaneous determination of prop

235 Using gas chromatography mass spectrometry (GC-MS), we identified compounds typically associated wit

236 zed by gas chromatography-mass spectrometry (GC-MS).

237 gas chromatography-tandem mass spectrometry (GC-MS/MS) for GC-amenable pesticides; (ii) hydrophilic i

238 ses by gas chromatography/mass spectrometry (GC/MS), provided important insights into Braque's unusua

239 as chromatography-ion mobility spectrometry (GC-IMS) to differentiate lactic acid bacteria (LAB) thro

240 nds by gas chromatography-mass spectroscopy (GC-MS) was also performed.

241 le organic compound production using HS-SPME-GC/MS analysis.

242 mined using ICP-OES and volatiles using SPME-GC/MS.

243 compared with other measurements, suggesting GC-IPL thickness is the better candidate for detecting p

244 leotides activate P2Y6 receptors to suppress GC growth through a novel SOCE/Ca(2+)/beta-catenin-media

245 e-infused, and high salt-fed (ALDO) systemic GC-A KO mice with enhanced phosphorylation of p38 mitoge

246 nt metabolites were quantified in a targeted GC-MS approach.

247 uniquely suited to normalizing non-targeted GC/MS metabolomics data due to explicit accommodation of

248 awing accurate conclusions from non-targeted GC/MS metabolomics data.

249 We found that GC neurons could either be unimodal, responding exclusiv

250 Contrary to our expectation, we found that GC receptor (GR) expression in immune cells was dispensa

251 We find that GCs limit the expression of Von Hippel Lindau protein (p

252 The GC coating of the surface was identified as a saturable,

253 The GC microelectrodes have more than 70% wider electrochemi

254 The GC-MS component co-elution was overcome by GCxGC-qMS.

255 The GC/N-CDs electrode shows the limit of detection of 13x10

256 The GC/rGO-Nf@Ag6 electrode exhibited an excellent electroch

257 in maximal mechanistic information about the GC coating of the LPNs.

258 sed with ICOS on Tfh cells in and around the GC, and ICOS-ICOSL interactions were similarly crucial a

259 s, and 651 novel genes were predicted by the GC-specific MAKER protocol.

260 cription factor E2F1 induces EZH2 during the GC reaction.

261 differentially increased MT dynamics in the GC with more MT growth in the distal than the proximal r

262 the colocalization of UNC5C and TUBB3 in the GC.

263 llicular helper T (TFH) cells can induce the GC response to self-antigens, subsequently leading to au

264 cooled through forced convection inside the GC oven within the time frame of a single modulation per

265 gly, when encountering plasma membranes, the GC-PEG-PpIX NPs can disassemble and stably attach to pla

266 s that have impeded our understanding of the GC and Tfh-cell processes involved in bnAb generation, i

267 n the distal than the proximal region of the GC during repulsion, and knockdown of either UNC5C or TU

268 spectrum, causing further enhancement of the GC to AT bias characteristic of organisms with normal MM

269 hysiological energy metabolism depend on the GC receptor (GR) in adipocytes remains unclear.

270 Deletion of Cdkn1a rescues the GC reaction in Ezh2 (-/-) mice.

271 corticosterone promote HSC migration via the GC receptor Nr3c1-dependent signaling and upregulation o

272 lls showed extensive binding of Smad2/3/4 to GC-rich cis-regulatory elements.

273 er, the precise contribution of apoptosis to GC biology and selection is not well defined.

274 at PEDOT-PSS adhered significantly better to GC than Pt, and allowed drastic reduction of electrode s

275 mit of detection of 0.2 ng/mL, comparable to GC/MS.

276 Thanks to GC-O and GC-MS analysis, cooked fruit notes were identif

277 ate the transcriptional response of B-ALL to GCs with a next-generation short hairpin RNA screen to i

278 tate granule cells (GCs) and project back to GCs locally, contralaterally, and along the longitudinal

279 a chemokine receptor required for homing to GCs) and expand in lymph nodes (LNs) following pathogeni

280 GC Tfh cells and the difficulty of tracking GC in human and non-human primate vaccine studies.

281 The HS-Trap GC-FID method was optimized for the parameters: thermost

282 l clones into ongoing immunization-triggered GC responses.

283 ce of ATP were similar to those of wild-type GC-B assayed in the presence of ATP.

284 s also a valuable resource for understanding GC biology and the mechanistic details of GR-regulated t

285 ed, giving an alternative to the widely used GC (FID/MS) methodologies.

286 c) > 5.05%, HbA1c < 4.92%] and assayed using GC-MS, chromatograms were analyzed using MetaboliteDetec

287 7 vitamin D and calcium pathway genes (VDR, GC, DHCR7, CYP2R1, CYP27B1, CYP24A1, and CASR) modify th

288 ting GC and a weakened basal lamina, whereas GC migration was minimal in DBP-FW animals.

289 ubset is sufficient for inducing cGVHD while GC is dispensable.

290 inhibition of GCase and was associated with GC storage, tissue inflammation and proinflammatory cyto

291 ase MicroExtraction (HS-SPME), combined with GC-MS, to an aqueous extract obtained by homogenization

292 ive to a panel of cancer drugs compared with GC of a more differentiated origin.

293 composition by means of HS-SPME coupled with GC-MS; ii) assess the polyphenolic content by UHPLC mass

294 0 months of follow-up, patients treated with GC care had a higher risk of bleeding hospitalization (h

295 stinal metaplasia mucosa of patients without GC (AG/IM GC-) and in control biopsy samples of non-tran

296 and either Thr-500, Ser-510 or Thr-513 in WT-GC-A increased the Km 23- to 70-fold but the same mutati

297 s by as much as 80% but failed to inhibit WT-GC-B.

298 ive two-dimensional gas chromatography (GC x GC) retention times can be used to predict 26 relevant p

299 Analysis of fractions by GC x GC/MS also allowed a series of thioxanones to be identif

300 alkyl homologues were easily separated (GC x GC/MS), allowing high-quality mass spectra (EI) to be ob