ライフサイエンスコーパス: GLC

1 GLC dynamics were analyzed in single living C2C12 myobla

2 GLC-ITMS can be used successfully to analyze more than 1

3 GLC-MS analysis of the derived taxa-4(5),11(12)-diene, v

4 he kinetic properties of GLC uptake (V1) and GLC consumption (V2) were determined independently and u

5 LC), while after high-temperature annealing, GLC becomes insignificant and trap-limited conduction (T

6 es of common ionic liquids were evaluated as GLC stationary phases.

7 ted acrylamide more efficiently than the ASN-GLC model system during heating at 180 degrees C.

8 in the ASN-HMF model system than in the ASN-GLC model system within 5min at 180 degrees C.

9 ues obtained on the ProteOn's alginate-based GLC chip agreed closer with those from Biacore's flat C1

10 GBCA dose (r = 0.4486; P < .05) and between GLC and liver iron concentration (r = 0.56; P < .05).

11 1 patients had positive correlations between GLC and total GBCA dose (r = 0.4486; P < .05) and betwee

12 ad group was methylated and then analyzed by GLC-CIMS and by the reductive-cleavage method, which rev

13 C-Carbowax, MECC-TTAB/SFC-Carbowax, HPLC-C18/GLC-DB-5, HPLC-PBD/SFC-phenyl, SFC-Carbowax/GLC-DB5, and

14 /GLC-DB-5, HPLC-PBD/SFC-phenyl, SFC-Carbowax/GLC-DB5, and HPLC-phenyl/SFC-phenyl 2-D chromatographic

15 d either the glucose load to the liver (CGMP/GLC; n = 5) or the glucose concentration entering the li

16 ring the last hour of the study in SAL, CGMP/GLC, and CGMP/GCC, respectively.

17 lues derived from gas-liquid chromatography (GLC) are found to be consistently lower than those obtai

18 erature capillary gas-liquid chromatography (GLC) in combination with low voltage (30 eV) electron io

19 graphy (HPLC) and gas-liquid chromatography (GLC) in combination with mass spectrometry were used to

20 matography (SFC), gas-liquid chromatography (GLC), and micellar electrokinetic capillary chromatograp

21 tionary phases in gas-liquid chromatography (GLC).

22 spectroscopic and gas-liquid-chromatography (GLC) data, are consistent with and strongly supportive o

23 and iron and gadolinium liver concentration (GLC).

24 verned by grain-boundary-limited conduction (GLC), while after high-temperature annealing, GLC become

25 min) inhibition of OXPHOS affected cytosolic GLC homeostasis.

26 ct a minimal mathematical model of cytosolic GLC dynamics.

27 ids and makes them very useful as dualnature GLC stationary phases.

28 methods for separation involve ion exchange, GLC, and HPLC (mostly after chemical modification by enz

29 cytosol by glycolytic conversion of glucose (GLC) into pyruvate.

30 up) received a duodenal infusion of glucose (GLC) or saline (SAL), then were fasted from 240-360 min.

31 (ASN) at elevated temperatures with glucose (GLC), and 5-hydroxymethyl-2-furfural (HMF).

32 However, GLC-MS analysis of the taxadiene product derived by enzy

33 chemical and immunological assays, including GLC-MS and matrix-assisted laser desorption ionization t

34 e absence and presence of OXPHOS inhibitors, GLC was consumed at near maximal rates, meaning that GLC

35 In addition, maternal GLC function is required for the paternal embryonic expr

36 Thus, morning GLC primed the liver to extract and store more glucose i

37 cid deletion in the ligand-binding domain of GLC-1, the alpha-subunit of a glutamate-gated chloride c

38 nsertion site, and the molecular identity of GLC is not known.

39 FLII calibration, the kinetic properties of GLC uptake (V1) and GLC consumption (V2) were determined

40 amine therapy had a significant reduction of GLC (from 0.64 mug/g +/- 0.29 to 0.20 mug/g +/- 0.17 [st

41 ious reports, suggesting that LSERs based on GLC-derived partition coefficients will not provide accu

42 ed to a commercial methylphenyl polysiloxane GLC stationary phase.

43 l blood glucose 146 +/- 2 mg/dL) with portal GLC infusion.

44 activation of the enzyme, in dogs receiving GLC compared with those receiving SAL.

45 lamp were markedly greater in dogs receiving GLC compared with those receiving SAL.

46 y of as-deposited Cu2O is due to significant GLC, and the Hall mobility enhancement by high-temperatu

47 hat OXPHOS inhibition increases steady-state GLC uptake and consumption in C2C12 myoblasts.

48 We demonstrated that GLC-ITMS was suitable for analyzing unpooled/underivatiz

49 consumed at near maximal rates, meaning that GLC consumption is rate-limiting under steady-state cond

50 he cause of the significant reduction in the GLC effect.

51 ave been determined by chromatographic (TLC, GLC, and HPLC) and spectral (UV, MS, and 1H NMR) methods

52 The total GLC infusion rate was 14% greater in dogs infused with G

53 P. carinii carinii, were determined by using GLC, MS, and NMR spectroscopy together with the chemical

54 on rate was 14% greater in dogs infused with GLC than in those receiving SAL (AUC360-600min 2,979 +/-