ライフサイエンスコーパス: GPCMV

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1 GPCMV DNA levels in the placenta were reduced from 3.54

2 GPCMV proved to be sensitive to BDCRB (50% inhibitory co

3 Deletion of a GPCMV genome-encoded PKR inhibitor results in a highly a

4 (and its attendant rescuant), generated on a GPCMV bacterial artificial chromosome construct, confirm

5 ious single-cycle (DISC) vaccine strategy, a GPCMV mutant virus was used that lacked the ability to e

6 the guinea pigs with salivary gland-adapted GPCMV.

7 ate (cHPMPC; 35 mg/kg) or placebo 24 h after GPCMV infection.

8 ea pigs were inoculated with guinea pig CMV (GPCMV) during the late second/early third trimester of g

9 (VRPs) expressing GP83, the guinea pig CMV (GPCMV) homolog of the human CMV pp65 phosphoprotein.

10 evaluated for protection against congenital GPCMV infection.

11 ity and plaquing efficiency of cotransfected GPCMV viral DNA was enhanced by GP82.

12 s expressing the guinea pig cytomegalovirus (GPCMV) homologs of the glycoprotein B (gB) and UL83 prot

13 nes encoding the guinea pig cytomegalovirus (GPCMV) homologs of the upper and lower matrix proteins o

14 ainst congenital guinea pig cytomegalovirus (GPCMV) infection was developed, characterized, and teste

15 ainst congenital guinea pig cytomegalovirus (GPCMV) infection.

16 B vaccine in the guinea pig cytomegalovirus (GPCMV) model of congenital infection.

17 valuated using a guinea pig cytomegalovirus (GPCMV) model.

18 ome structure of guinea pig cytomegalovirus (GPCMV) provides a useful model for the study of herpesvi

19 Guinea pig cytomegalovirus (GPCMV) provides a valuable model for congenital cytomega

20 Efficacy was evaluated by immunizing GPCMV-naive guinea pigs twice with either 10(5) or 10(6)

21 nicity by repair of a frameshift mutation in GPCMV gene GP129 using this approach provides a valuable

22 es and CD4(+) and CD8(+) T cell responses in GPCMV-seronegative female guinea pigs.

23 up but significantly decreased the amount of GPCMV in tissues.

24 (50 microM) had no effect on the quantity of GPCMV genomic DNA that was formed in infected cells.

25 as significantly lower, and reduced rates of GPCMV transmission were noted, for dams immunized with g

26 Salivary gland (SG)-passaged stocks of GPCMV are pathogenic, while tissue culture (TC) passage

27 on pup mortality or vertical transmission of GPCMV.

28 enged with 1 x 10(4) plaque-forming units of GPCMV in the third trimester.

29 ing, we investigated the effects of BDCRB on GPCMV.

30 Recombinant GPCMV with a targeted deletion of gp145 (designated Delt

31 owever, a dose of BDCRB sufficient to reduce GPCMV titers by 3 logs (50 microM) had no effect on the

32 P-HA) were bred for pregnancy and subsequent GPCMV challenge during the early third trimester.

33 srupts GP129, which encodes a subunit of the GPCMV pentameric complex (PC) believed to govern viral e

34 Treatment of the GPCMV-infected pregnant dam with 1 dose of cHPMPC improv

35 tivation function of GP82 was not limited to GPCMV, but was also observed for a heterologous virus, h

36 e challenged during pregnancy with wild-type GPCMV (10(5) PFU).

37 cs of the GP83 mutant (vAM409) and wild-type GPCMV indicated that GP83 protein is not required for vi

38 d by comparing the pathogenesis of wild-type GPCMV, vAM409, and a control virus, vAM403, in guinea pi

39 Pregnant dams were challenged with GPCMV subcutaneously during the 3rd trimester.

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