ライフサイエンスコーパス: HIV-associated nephropathy

1 haracterized by a proliferative phenotype in HIV-associated nephropathy.

2 loss of podocytes are important features of HIV-associated nephropathy.

3 thological changes leading to proteinuria in HIV-associated nephropathy.

4 ts with CKD, irrespective of the presence of HIV-associated nephropathy.

5 f HIV-infected patients who have evidence of HIV-associated nephropathy.

6 s been shown to change the course of classic HIV-associated nephropathy.

7 s, immune-mediated glomerulonephritides, and HIV-associated nephropathy.

8 alleles are at very high risk for developing HIV-associated nephropathy.

9 renal functional and pathologic outcomes of HIV-associated nephropathy.

10 n of renal epithelium in the pathogenesis of HIV-associated nephropathy.

11 A in renal epithelial cells of patients with HIV-associated nephropathy.

12 sy is essential to differentiate HCV-GD from HIV-associated nephropathy.

13 nal disease that can be easily confused with HIV-associated nephropathy.

14 athogenesis of human immunodeficiency virus (HIV)-associated nephropathy.

15 6 patients with CKD (96.7%; 38 patients with HIV-associated nephropathy, 39 patients with HIV-positiv

16 Overall, 79% of patients with HIV-associated nephropathy and 2% of population controls

17 role of APOL1 variants in 120 patients with HIV-associated nephropathy and CKD and 108 controls from

18 APOL1 variants are associated with HIV-associated nephropathy and FSGS in African Americans

19 The rise in the number of patients with HIV-associated nephropathy and HIV-infection with end-st

20 ding focal and segmental glomerulosclerosis, HIV-associated nephropathy and hypertensive nephrosclero

21 ell as in kidney biopsies from patients with HIV-associated nephropathy and idiopathic focal segmenta

22 explain much of the increased risk for FSGS, HIV-associated nephropathy, and hypertension-attributed

23 interval, 18 to 912; P<0.001) of developing HIV-associated nephropathy compared with HIV-positive co

24 rker Ki-67 in collapsing idiopathic FSGS and HIV-associated nephropathy compared with minimal change

25 HIV-associated nephropathy has a characteristic patholog

26 for improved renal survival of patients with HIV-associated nephropathy has become more realistic wit

27 The development of HIV-associated nephropathy has been definitively linked

28 Historically, HIV-associated nephropathy has been the predominant caus

29 fect of fosinopril, 10 mg by mouth daily, in HIV-associated nephropathy (HIV-AN).

30 n-embedded renal biopsies from patients with HIV-associated nephropathy (HIVAN) (n = 13), HIV-associa

31 c focal segmental glomerulosclerosis (FSGS), HIV-associated nephropathy (HIVAN) and end-stage kidney

32 well as in renal biopsies from patients with HIV-associated nephropathy (HIVAN) and FSGS.

33 The glomerular lesions of HIV-associated nephropathy (HIVAN) are associated with t

34 tubular epithelial cells from patients with HIV-associated nephropathy (HIVAN) express HIV-1 transcr

35 HIV-associated nephropathy (HIVAN) is a major cause of H

36 HIV-associated nephropathy (HIVAN) is a progressive glom

37 HIV-associated nephropathy (HIVAN) is characterized by c

38 Renal parenchymal injury in HIV-associated nephropathy (HIVAN) is characterized by e

39 The collapsing glomerulopathy of HIV-associated nephropathy (HIVAN) is characterized by p

40 HIV-associated nephropathy (HIVAN) is now the third lead

41 troviral therapy, kidney diseases other than HIV-associated nephropathy (HIVAN) predominate in HIV-in

42 hology in chronic kidney diseases, including HIV-associated nephropathy (HIVAN) that ultimately progr

43 y, we hypothesized that HIV-1-induced occult HIV-associated nephropathy (HIVAN) would become apparent

44 , focal segmental glomerulosclerosis (FSGS), HIV-associated nephropathy (HIVAN), and hypertensive nep

45 on is a prominent histopathologic feature of HIV-associated nephropathy (HIVAN), but its pathogenesis

46 The classic kidney disease of HIV infection, HIV-associated nephropathy (HIVAN), is an aggressive for

47 focal segmental glomerulosclerosis (FSGS) of HIV-associated nephropathy (HIVAN), podocytes exhibit a

48 genesis of several renal diseases, including HIV-associated nephropathy (HIVAN), the most common caus

49 y has a critical role in the pathogenesis of HIV-associated nephropathy (HIVAN).

50 -nine-fold higher odds (95% CI 13 to 68) for HIV-associated nephropathy (HIVAN).

51 resembling those in human disease, including HIV-associated nephropathy (HIVAN).

52 important components of the pathogenesis of HIV-associated nephropathy (HIVAN).

53 apid decline in kidney function characterize HIV-associated nephropathy (HIVAN).

54 odocytes in the collapsing glomerulopathy of HIV-associated nephropathy (HIVAN).

55 to the pathogenesis of glomerular disease in HIV-associated nephropathy (HIVAN).

56 a central role in the glomerular disease of HIV-associated nephropathy (HIVAN).

57 IV- I develop renal disease resembling human HIV-associated nephropathy (HIVAN).

58 oinfection, cardiovascular disease risk, and HIV-associated nephropathy increasingly prompt earlier t

59 Human immunodeficiency virus (HIV)-associated nephropathy is a significant cause of mo

60 HIV-associated nephropathy is a clinicopathologic entity

61 HIV-associated nephropathy is a unique pattern of sclero

62 HIV-associated nephropathy is characterized by renal pod

63 HIV-associated nephropathy is infrequently cited as a co

64 to have focal segmental glomerulosclerosis, HIV-associated nephropathy, or ESRD, prospective studies

65 erosis (FSGS), including idiopathic FSGS and HIV-associated nephropathy, podocytes undergo characteri

66 observed in the kidneys of a mouse model of HIV-associated nephropathy (Tg26 mice).

67 As a result, HIV-associated nephropathy, the classic HIV-driven kidne

68 In animal models of HIV-associated nephropathy, the expression of HIV regula

69 contrast, in collapsing idiopathic FSGS and HIV-associated nephropathy, there was disappearance of a

70 suggests a shift in the etiology of CKD from HIV-associated nephropathy toward other etiologies.

71 Typical lesions of human HIV-associated nephropathy were undetectable.

72 ely, however, that by the end of the decade, HIV-associated nephropathy will be the third leading cau