ライフサイエンスコーパス: MTT

1 MTT [3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazoliu

2 MTT and LDH assays confirmed cytotoxicity of perfluorooc

3 MTT assay of the HPLC fractions identified an active fra

4 MTT assay showed that cell proliferation on denuded AM w

5 MTT assay showed that PEG5K-Fmoc-VE2/DOX exerted signifi

6 MTT assay was performed to quantify mitochondrial stress

7 MTT assays revealed that, of all 30 compounds tested, co

8 MTT assays showed only minor effects of the thioethers a

9 MTT cytotoxicity assay and confocal microscopy images we

10 MTT produced sustained improvement in OR team function,

11 MTT reduction was also significantly inhibited by 1.7 mM

12 MTT was generally inversely related to BF.

13 MTTs in group 2 on days 0 and 2 were significantly longe

14 A total of 4863 MTT debriefings were analyzed.

15 Caspase-3/7, MTT and TUNEL assays elucidated that pEpo-AFPL-TK transf

16 the most active compounds was confirmed in a MTT efficacy assay.

17 to be cytotoxic when assessed by means of a MTT assay against two human prostate cell lines, DuPro a

18 Proliferation was determined with a MTT proliferation assay after exposing goblet cells, whi

19 MTT for the whole kidney (MTT(K) = MTT(A) + MTT(T) + MTT(C)) and fractional MTT of each compartment

20 l MTT of each compartment (MTT(A/K) = MTT(A)/MTT(K), MTT(T/K) = MTT(T)/MTT(K), MTT(C/K) = MTT(C)/MTT(

21 An MTT hyphal damage assay demonstrated significant in vitr

22 We administered an MTT to 12 obese T2D patients and 15 obese nondiabetic (N

23 d in a cellular HDAC inhibition assay and an MTT assay for cytotoxicity.

24 using a modified Boyden chamber assay and an MTT assay, respectively.

25 Cell proliferation was measured by an MTT assay.

26 CPT-L2-BA3 was cytotoxic in an MTT assay for cells transfected with each class of BN re

27 of the delivery system was studied using an MTT assay, and by studying the histology of skin samples

28 (KYSE30) cancer cells were studied using an MTT assay.

29 and to growth factors was evaluated using an MTT assay.

30 EPO protein bioactivity was confirmed via an MTT proliferation bioassay.

31 Cellular toxicity was assessed with an MTT dye reduction assay.

32 a significant difference in baseline BF and MTT values between responders and nonresponders (P < or

33 esults in greater viability (higher BrdU and MTT), more complete basement membrane development at 2 w

34 ary permeability or between vessel count and MTT.

35 ll viability was assessed by cell counts and MTT assay, and apoptosis was measured by nucleosomal deg

36 ll viability were measured using H(2)DCF and MTT assays, respectively.

37 7) than control rats, and lower MTR, DV, and MTT (P = .014, .001, and .010, respectively; alpha = .01

38 There was an increase in F(a), ART, DV, and MTT and a decrease in PV in patients with advanced fibro

39 o compare F(a), F(p), F(t), ART, PV, DV, and MTT between groups.

40 Trypan blue exclusion and MTT assays evaluated TSA cytotoxicity to the cornea.

41 Using trypan blue exclusion and MTT assays, there was no evidence of cell toxicity with

42 n primarily by thioflavin T fluorescence and MTT.

43 here were significant differences in GFR and MTT(K) between the acute dysfunction group (36.4 mL/min

44 and PS values were significantly higher and MTT values were significantly lower (P<.01) with the cur

45 s within the as-synthesized zeolites ITW and MTT that, in conjunction with synchrotron X-ray diffract

46 or as-synthesized siliceous zeolites ITW and MTT.

47 Fn or Fn-fs of 45, 70, 110, or 120 kDa, and MTT conversion was used to measure proliferation and sur

48 LDH and MTT assays demonstrate that peptide 1a is toxic toward S

49 Cell death was assayed by LDH and MTT methods.

50 ondral area with low or no detectable PF and MTT adjacent to the joint surface, which was surrounded

51 PF and MTT were correlated with CCl(4) treatment duration from

52 ll damage determined by both LDH release and MTT reduction assays, dose and time dependently, in both

53 ts (P < .001 and P = .03, respectively), and MTT was shorter on day 2 than on day 0 in group 2 (P = .

54 Nuclear staining and MTT assays following dsRNA stimulation revealed two subp

55 tic resonance (NMR), chemical synthesis, and MTT (3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazoliu

56 cal, hypochlorous acid), cytotoxicity assay (MTT) and quantification of TNF-alpha production in RAW 2

57 -diphenyltetrazolium bromide formazan assay (MTT assay) as a reporter of Abeta-mediated neuronal cell

58 From these preliminary cytotoxicity assays (MTT) we found that C8-propyl-catechin gallate was more a

59 tional effects analyzed using kinase assays, MTT assays were used to assess cell viability as a marke

60 Two cell viability assays, MTT and lactate dehydrogenase (LDH), and an assay measur

61 A cell-based MTT (3-[4,5-dimethylthiazol-2-yl]-2,5-diphenyltetrazoliu

62 MDCT-based MTT and PBF measurements demonstrate globally increased

63 inhibition was evaluated by thiazolyl blue (MTT) conversion.

64 Both MTT and PrestoBlue assays showed higher cell viability a

65 hiazol 2-yl)-2,5-diphenyltetrazolium bromide MTT assays.

66 iazol-2-yl)-2,5-diphenyltetrazolium bromide (MTT) assay and cell cycle analysis showed inhibition of

67 iazol-2-yl)-2,5-diphenyltetrazolium bromide (MTT) assay, flow cytometry with propidium iodide, gene e

68 hiazolyl-2)-2,5-diphenyltetrazolium bromide (MTT) assay.

69 hiazolyl-2)-2,5-diphenyltetrazolium bromide (MTT) assay.

70 iazol-2-yl)-2,5-diphenyltetrazolium bromide (MTT) assay.

71 iazol-2-yl]-2,5-diphenyltetrazolium bromide (MTT) assay.

72 iazol-2-yl)-2,5-diphenyltetrazolium bromide (MTT) assays in bladder transitional cell carcinoma (TCC)

73 iazol-2-yl]-2,5-diphenyltetrazolium bromide (MTT) reduction (mitochondrial function), lactate dehydro

74 azole-2-yl)-2,5-diphenyltetrazolium bromide (MTT) reduction assay, superoxide scavenging activity, re

75 iazol-2-yl)-2,5-diphenyltetrazolium bromide (MTT) reduction assay.

76 iazol-2-yl)-2,5-diphenyltetrazolium bromide (MTT) reduction, lactate dehydrogenase release, and [(3)H

77 iazol-2-yl)-2,5-diphenyltetrazolium bromide (MTT), a substrate commonly used to measure cell viabilit

78 iazol-2-yl)-2,5-diphenyltetrazolium bromide (MTT), and AlamarBlue cell viability assays.

79 iazol-2-yl)-2,5-diphenyltetrazolium bromide (MTT), apoptosis and colony formation), and chelation of

80 iazol-2-yl)-2,5-diphenyltetrazolium bromide (MTT).

81 azol-2-yl)-2,5-diphenyl tetrasodium bromide (MTT) and Alamar Blue cell viability assays.

82 l-2-yl)-2.5-diphenyl-2H-tetrazolium bromide (MTT) and clonal growth assays.

83 azol-2-yl)-2,5-diphenyl tetrazolium bromide (MTT) assay and cell counting, the expression of alpha-SM

84 methylthiazolyldiphenyl-tetrazolium bromide (MTT) assay, Hoechst staining and caspase-3 activation, a

85 azol-2-yl)-2,5-diphenyl tetrazolium bromide (MTT) assay, lactate dehydrogenase (LDH) release assay, H

86 iazol-2-yl)2,5-diphenyl-tetrazolium bromide (MTT) assay.

87 azol-2-yl)-2,5-diphenyl tetrazolium bromide (MTT) assay.

88 methylthiazolyldiphenyl-tetrazolium bromide (MTT) assays and significant tumorigenicity in in vivo al

89 azol-2-yl)-2,5-diphenyl tetrazolium bromide (MTT) assays.

90 azol-2-yl)-2,5-diphenyl tetrazolium bromide (MTT) conversion.

91 azol-2-yl)-2,5-diphenyl-tetrazolium bromide (MTT), and apoptosis assays.

92 azol-2-yl)-2,5-diphenyltetrazolium bromide, (MTT) whereas apoptosis was determined by DNA fragmentati

93 azol-2-yl)-2,5-diphenyltetrazolium bromide] (MTT), glucose-6-phosphate dehydrogenase (G6DP), and calc

94 images, with within-patient CVs for BF, BV, MTT, and PS of 11.2%, 14.4%, 5.5% and 12.1%, respectivel

95 For tumors, BF, BV, MTT, and PS values and reproducibility varied by analyti

96 lammatory, antimicrobial and antioxidant, by MTT, nitric oxide inhibitory assay, agar disc diffusion

97 mbrane matrix and fibronectin was assayed by MTT.

98 The affect of Usp and Imu1-3 was assessed by MTT and Comet assays, infection assays, caspase 3/7 acti

99 ty on PC12 cell lines (viability assessed by MTT assay and intracellular ROS production by DCFH-DA as

100 The cell proliferation rate was assessed by MTT assay and with the electric cell-substrate impedance

101 ell viability in wild-type mice, assessed by MTT assay, was approximately half of that in contralater

102 Viability of RGC-5 cells was assessed by MTT assays.

103 ellular reducing equivalents was assessed by MTT dye reduction and NAD(P)H assays, and cell survival

104 to the aggregation reactions, as assessed by MTT metabolic activity measurements.

105 ity and metabolic activity was calculated by MTT assay.

106 rmal keratinocytes (NHEKs) were conducted by MTT (3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazoliu

107 or = 48 hours in normoxic tissue culture by MTT assay and histology.

108 viability was assessed after 1 to 3 days by MTT assay.

109 umor cell growth in vitro as demonstrated by MTT and clonogenic assays.

110 Cell viability was detected by MTT assay.

111 neuroblastoma SH-SY5Y cells as determined by MTT and LDH release assays.

112 l viability and apoptosis were determined by MTT assay and caspase-3 (DEVDase) activity.

113 ed by cell count, toxicity was determined by MTT assay, and neoplastic transformation was assessed by

114 12 pheochromocytoma cells were determined by MTT assay, while the cell differentiation was evaluated

115 ur human cancer cell lines was determined by MTT assay, yielding dose- and cancer cell line-dependent

116 Cell viability was determined by MTT assay.

117 mercial HEK cell-produced hOPN determined by MTT assay.

118 l lines (A549, CH1, SW480) was determined by MTT assays, yielding IC(50) values of 6-60 muM for three

119 Cell viability was determined by MTT reduction or LDH release assays.

120 in HBVEC treated with Heme was evaluated by MTT and TUNEL assay.

121 Cytotoxicity was evaluated by MTT assay and flow cytometry analysis, while genotoxicit

122 alcein-AM, and cytotoxicity was evaluated by MTT assay.

123 of MCF-7 and SK-BR-3 cells was evaluated by MTT assays.

124 orioallantoic membrane (CAM) was examined by MTT assay, BrdU labeling, cell proliferation assay, cell

125 ced T cell/FLS proliferation was examined by MTT assay.

126 iability and cell death were investigated by MTT, terminal deoxynucleotidyl transferase-mediated dUTP

127 rative activity on human tumor cell lines by MTT assay, for antioxidant potential by DPPH, ABTS and F

128 tions in tumor cell survival, as measured by MTT and crystal violet assays, regardless of IGF1 pre-tr

129 Cell viability was measured by MTT and/or adenosine triphosphate (ATP) content.

130 Cell viability/apoptosis was measured by MTT assay and Annexin V/PI staining , activation related

131 s, and ~45% cancer cell death as measured by MTT assay, when illuminated with 980 nm NIR light.

132 Cell proliferation was measured by MTT assay.

133 Cell growth was measured by MTT assays.

134 peripheral blood mononuclear cells (PBMC) by MTT assay.

135 Tramp-C1 cells affects cell proliferation by MTT assays.

136 Cytoprotection was quantified by MTT assay.

137 Cell growth test by MTT assay showed that induction of Cx50P59A decreased ce

138 herapeutic effect was determined in vitro by MTT assay, [(18)F]fluorodeoxyglucose (FDG)- and [(18)F]f

139 MTT(T/K) = MTT(T)/MTT(K), MTT(C/K) = MTT(C)/MTT(K)).

140 d on the central volume principle (CBF = CBV/MTT) and requires the use of commercially available soft

141 tective/cytotoxic effects upon Caco-2 cells (MTT, cell cycle and reactive oxygen species (ROS)) were

142 as paralleled by a failure to alter cellular MTT reduction.

143 ffect on Abeta-induced decreases in cellular MTT reduction.

144 f fragmented beta2m fibrils rescued cellular MTT reduction.

145 meters were also optimized for colorimetric (MTT [3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazoliu

146 (C)) and fractional MTT of each compartment (MTT(A/K) = MTT(A)/MTT(K), MTT(T/K) = MTT(T)/MTT(K), MTT(

147 (T)), and the collecting system compartment (MTT(C)) were calculated.

148 mpartment (MTT(A)), the tubular compartment (MTT(T)), and the collecting system compartment (MTT(C))

149 of the tracer for the vascular compartment (MTT(A)), the tubular compartment (MTT(T)), and the colle

150 Conversely, MTT was longer with the distributed parameter model than

151 py, ELISA), toxicity assays in cell culture (MTT and lactate dehydrogenase in human SH-SHY5Y cells, m

152 vival, which was detected by flow cytometry, MTT (3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazoliu

153 Cytotoxicity (MTT test) and genotoxicity (micronuclei assay) were not

154 The cytotoxicity (MTT assay) on HaCaT cell line demonstrated high cytocomp

155 alpha neutralisation and rTNFalpha decreased MTT and NFI further.

156 -dimethylthiazol-2-yl)-3,5-diphenylformazan (MTT) to quantitatively determine tissue viability.

157 methylthiazol-2-yl)-2,5-diphenyltetrazolium (MTT) and clonogenic assays]; indolequinones displayed po

158 retrospective and prospective thought (i.e., MTT), here we explored the possibility that the spatiali

159 The Pearson coefficient for blood flow, MTT, K(trans), and PS at the two ROIs demonstrated good

160 One year following MTT, case delays decreased (23% to 10%, P < 0.0001), mea

161 nt LoA were -35% for BF,-43% for BV, 33% for MTT, and -79% for PS.

162 , good (>0.75) for PS, and lower (>0.38) for MTT, irrespective of software version.

163 readers for BF were -33%; for BV, -39%; for MTT, 55%; and for PS, -93%.

164 for BV, or an increase of more than 55% for MTT, could be considered beyond the analysis variability

165 At their EC50 concentrations for MTT reduction, all four APs disrupted cellular ultrastru

166 etween TBMES and osteonecrosis was found for MTT (P = .09) and PF (P = .75) in the surrounding area.

167 dies of structure-directing agents (SDA) for MTT-type zeolites, a large number of amines or quaternar

168 e successfully predicted to be selective for MTT phases.

169 rded versus 200 and 6 x 10(3) cells/well for MTT and fluorescence assays, respectively.

170 ) = MTT(A) + MTT(T) + MTT(C)) and fractional MTT of each compartment (MTT(A/K) = MTT(A)/MTT(K), MTT(T

171 Results from MTT and Wright staining were further supported by the te

172 by decreased ATP and ATP/ADP ratio, impaired MTT conversion and heightened sensitivity to 3-nitroprop

173 liferation of human colon carcinoma cells in MTT and clonogenic assays by arresting cells in G(1).

174 significant reduction in BF and increase in MTT (P < or =.05).

175 L-15 and rTNFalpha, limited the reduction in MTT and nuclear fusion index (NFI) associated with rTNFa

176 Biological evaluation includes MTT and cellular HDAC assays on sensitive and chemoresis

177 conceptualization of time that may influence MTT as well as other temporally relevant cognitive pheno

178 to be highly active in the growth inhibition MTT assay, with GI(50) values in the low nanomolar range

179 OR personal completed a 1-day intensive MTT training.

180 ircumscribed rim of high PF and intermediate MTT, which was only found in joints with osteonecrosis,

181 rounded by a rim of high PF and intermediate MTT.

182 actional MTT of each compartment (MTT(A/K) = MTT(A)/MTT(K), MTT(T/K) = MTT(T)/MTT(K), MTT(C/K) = MTT(

183 MTT(K), MTT(T/K) = MTT(T)/MTT(K), MTT(C/K) = MTT(C)/MTT(K)).

184 rived was MTT for the whole kidney (MTT(K) = MTT(A) + MTT(T) + MTT(C)) and fractional MTT of each com

185 rtment (MTT(A/K) = MTT(A)/MTT(K), MTT(T/K) = MTT(T)/MTT(K), MTT(C/K) = MTT(C)/MTT(K)).

186 ) = MTT(A)/MTT(K), MTT(T/K) = MTT(T)/MTT(K), MTT(C/K) = MTT(C)/MTT(K)).

187 each compartment (MTT(A/K) = MTT(A)/MTT(K), MTT(T/K) = MTT(T)/MTT(K), MTT(C/K) = MTT(C)/MTT(K)).

188 Also derived was MTT for the whole kidney (MTT(K) = MTT(A) + MTT(T) + MTT(C)) and fractional MTT of

189 rest were drawn in areas of high PF and long MTT on each parametric map.

190 w MTT that was surrounded by an area of long MTT and low PF.

191 ubchondral elongated area of high PF and low MTT that was surrounded by an area of long MTT and low P

192 y (EC50 = 1.1 x 10(-9) M vs 1.8 x 10(-11) M, MTT test) in agreement with a reduced binding affinity (

193 18.9 mL/100 mL per minute +/- 11.0 and mean MTT was 213.3 seconds +/- 56.8.

194 y were analysed in methylthiazoltetrazolium (MTT) and trypan blue assays.

195 al respiration was measured using a modified MTT (3-[4,5-dimethylthiazol-2-yl]-2,5-diphenyl-tetrazoli

196 A multidisciplinary MTT committee reviewed and rectified any systems problem

197 rIL-15 increased the thickness of myotubes (MTT) from both age groups to a similar extent.

198 rvival of serum-starved C2C12, HSM, and NCM (MTT, trypan blue) and prevented taxol-induced apoptosis

199 This is the largest case analysis of MTT and one of the few to document an impact of MTT on o

200 conditions necessary for crystallization of MTT phases in borosilicate preparations with some of the

201 hat are selective for the crystallization of MTT-type zeolite phases.

202 and one of the few to document an impact of MTT on objective measures of operating room function and

203 cellular ATP levels, glutamate inhibition of MTT reduction was glucose concentration dependent.

204 toxic to PC12 cells, impairing reduction of MTT and interfering with ERK and Rac signal transduction

205 SY5Y cells, as measured by the reduction of MTT, 3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazoliu

206 Results of MTT- and anchorage-independent growth assays and cell cy

207 ificant regional differences in CBF, CBV, or MTT were found in patients with RR-MS.

208 gh-level debriefing/problem-solving process, MTT can be a foundation for improving OR performance.

209 tions these molecules usually do not produce MTT phases.

210 cantly decreased CBF (P <.005) and prolonged MTT (P <.001) compared with the corresponding region in

211 We propose MTT lesion volume decrease more than 30% 2 hours after t

212 f these materials were evaluated by protein, MTT, and LDH assays, which demonstrate that all the clic

213 APs impaired the ability of HBMECs to reduce MTT which was followed by decreased Trypan blue exclusio

214 Cell viability-related (MTT assay) studies clearly showed that nitrones 1-3 and

215 hree patients with high initial BF and short MTT showed poor response.

216 ample, tumors with initial high BF and short MTT values tended to respond poorly to chemotherapy and

217 Rectal cancer showed higher BF and shorter MTT compared with those of normal rectum (P < or =.05).

218 In contrast, the very similar MTT [3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazoliu

219 ements, microscopic examination and standard MTT viability assays.

220 t erbB receptors, T6-17 and 32D, in standard MTT (3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazoliu

221 the whole kidney (MTT(K) = MTT(A) + MTT(T) + MTT(C)) and fractional MTT of each compartment (MTT(A/K)

222 (MTT(A/K) = MTT(A)/MTT(K), MTT(T/K) = MTT(T)/MTT(K), MTT(C/K) = MTT(C)/MTT(K)).

223 ults using a conventional cytotoxicity test (MTT).

224 logical stimulus, such as a mixed meal test (MTT), has not been determined.

225 using a more traditional methyl tetrazolium (MTT) cytotoxicity assay at selected time points followin

226 The MTT (3-[4,5-dimethylthiazol-2-yl]-2,5-diphenyltetrazodiu

227 The MTT assay confirmed these results for PRO 2000 (4% and 0

228 The MTT assay was confounded by the reduction of the MTT rea

229 The MTT assay was used to test the extract on the effect of

230 The MTT(A/K) was significantly higher in the acute rejection

231 The MTT(T/K) was significantly higher in the ATN group (mean

232 s measured by trypan blue exclusion, and the MTT assay and apoptosis were quantitated by fluorescence

233 analyzed by immunostaining for Ki67 and the MTT assay.

234 cell proliferation-enhancing activity by the MTT assay and anchorage-independent growth in soft agar.

235 th cell lines was normal, as measured by the MTT assay and markers of cytotoxicity, cell cycle, apopt

236 Cell viability was assessed by the MTT assay.

237 ls in 2D, whose toxicity was measured by the MTT assay.

238 prostate cancer cell proliferation from the MTT assay and flow cytometry investigation.

239 In the MTT assay, ExMh showed the highest cytotoxicity, especia

240 l lines Y79 and WERI-Rb1 with the use of the MTT assay, BrdU incorporation assay, flow cytometry, imm

241 lines (CH1, SW480, and A549) by means of the MTT assay, featuring IC(50) values to the low micromolar

242 lines (CH1, SW480 and A549) by means of the MTT colorimetric assay.

243 assay was confounded by the reduction of the MTT reagent by honey's reducing sugars and phenolic comp

244 had lesions of the MD without lesions of the MTT.

245 latin at micromolar concentrations using the MTT and cell invasion assay.

246 gree of cell death was established using the MTT assay.

247 the flavonoids after 24 hours, by using the MTT assay.

248 rmined by quantifying viable cells using the MTT assay.

249 Cell growth was quantified using the MTT assay.

250 e cytotoxic activity was evaluated using the MTT assay.

251 At day 1, using the MTT method, 0.01 nmol/l rapamycin reduced cell viability

252 tion in cell survival as determined with the MTT assay.

253 e level of cell death was monitored with the MTT assay.

254 48-h treatment with FeSO(4), tested with the MTT viability assay.

255 n the one-dimensional pore zeolites with the MTT, TON, and MTW frameworks.

256 te cell survival in many systems; therefore, MTT (1-(4,5-dimethylthiazol-2-yl)-3,5-diphenylformazan)

257 ral blood flow (CBF), and mean transit time (MTT) (referenced to an arterial input function by using

258 wer (>0.30 and >0.39) for mean transit time (MTT) and permeability surface area product (PS), respect

259 Parameter maps for mean transit time (MTT) and plasma flow (PF) were evaluated qualitatively a

260 : hypoperfusion volume on mean transit time (MTT) map decrease >30% from baseline to 2-hour post tPA

261 ribution volume (DV), and mean transit time (MTT) of gadopentetate dimeglumine.

262 ration rate (GFR) and the mean transit time (MTT) of the tracer for the vascular compartment (MTT(A))

263 (BF), blood volume (BV), mean transit time (MTT), and capillary permeability-surface area product we

264 (BF), blood volume (BV), mean transit time (MTT), and permeability-surface area product (PS) for tum

265 (BF), blood volume (BV), mean transit time (MTT), and permeability-surface area product were measure

266 Distribution volume (DV), mean transit time (MTT), and portal fraction (PF) of blood inflow were calc

267 flow (BF), blood volume, mean transit time (MTT), and vascular permeability-surface area product.

268 Blood flow, blood volume, mean transit time (MTT), permeability-surface area product, extraction frac

269 BV), blood flow (BF), and mean transit time (MTT), were calculated at the primary site.

270 ary blood flow (PBF), and mean transit time (MTT).

271 l blood volume (CBV), and mean transit time (MTT).

272 (BF), Blood Volume (BV), Mean Transit Time (MTT)] and permeability parameters [including endothelial

273 cantly shorter than the distant liver tissue MTT at 2.5 mug/mL only (9.7 vs 15.3 sec, P = .0006).

274 antly shorter than the adjacent liver tissue MTT at angiotensin II doses of 2.5 mug/mL (9.7 vs 15.8 s

275 postulated that the mammillo-thalamic tract (MTT)/anterior thalamic nucleus (AN) complex would be cri

276 Medical team training (MTT) has been touted as a way to improve teamwork and pa

277 nd now, this faculty for mental time travel (MTT) is dependent upon an underlying cognitive represent

278 Tumor MTT was significantly shorter than the adjacent liver ti

279 Unlike MTT, WST-1 reagent is reduced extracellularly through tr

280 Using MTT assays of cell proliferation on CEM ALL cells, we fo

281 l lines of NIH/3T3, CCD18-Co and B98-5 using MTT assay.

282 The lutein emulsions were analysed using MTT assay on the gut enterocyte cell line Caco-2 and the

283 ntial function of miR-497 in HUVECs by using MTT and TUNEL assays.

284 proliferation/survival in these cells using MTT, (3)H-thymidine uptake and Annexin-V apoptosis assay

285 d MDCK cells against ADR (demonstrated using MTT (3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazoliu

286 sing human tumor cells was established using MTT assays.

287 ible hepatic toxicity was investigated using MTT assay on HepG2 cells.

288 A (normal epithelial breast cell line) using MTT assay, where they showed highest inhibitory activity

289 re and after treatment with nifurtimox using MTT assays.

290 FEPO's cytotoxicity was tested using MTT (2-(4,5-dimethyl-2-thiazolyl)-3,5-diphenyl-2H-tetraz

291 he pancreatic cancer cell lines tested using MTT and colony formation assays.

292 ox and BSO and evaluated for viability using MTT assays.

293 ponders existed in K(trans), Kep, Ve, Ve_SD, MTT, BV_SD and MTT_SD (P < 0.05).

294 PA treatment by analyzing cell viability via MTT assay, neurosphere formation, and endoplasmic reticu

295 ere confirmed by Apoptag and cell viability (MTT) assays supporting the ability of PDT-BIAS to induce

296 Also derived was MTT for the whole kidney (MTT(K) = MTT(A) + MTT(T) + MTT

297 Cell proliferation was measured with MTT [3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazoliu

298 ls up to 0.25 mg/ml extract as measured with MTT and LDH-leakage assays.

299 Analysis of cell viability with MTT assay showed that activation of BMK1 by CA-MEK5 sign

300 By comparison, for as-synthesized zeolite MTT, F(-) anions reside within the 10-ring channels and