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1                                              PP chelates Mn(III) produced by the enzyme and subsequen
2                                              PP DCs augment IgA production by integrin alphavbeta8-me
3                                              PP detectors process signals produced by each particle a
4                                              PP IgA class switching requires innate lymphoid cells, w
5                                              PP plays a multifaceted role, slowing the reaction by co
6                                              PP prepared with Irgafos 168 led to a significantly stro
7                                              PP requiring hospitalization significantly decreased in
8                                              PP TC development across juvenile, transition, and adult
9                                              PP TCs with extensive wall ingrowths are ubiquitous in m
10                                              PP type M cells are thought to be dependent on cytokines
11                                              PP was analyzed as a continuous and categorical (i.e., b
12                                              PP was associated with reduced risk of neutropenia-relat
13                                              PP, mean arterial pressure, cardiac output, SVR, and asc
14                                              PP, VmaxAo, SVC, and IVC could be measured in 78.5%, 78.
15 ropenia-related hospitalization for TC (2.0% PP; 7.1% no PP; adjusted odds ratio [AOR], 0.29; 95% CI,
16 glycoprotein 2 as a marker for PDX1+/NKX6-1+ PPs.
17  deficits in different frequency bands in 25 PP-MS patients (12 M, mean age 50.9 +/- 10.5 years) and
18 ], 0.29; 95% CI, 0.22 to 0.39) and TCH (1.3% PP; 7.1% no PP; AOR, 0.19; 95% CI, 0.12 to 0.30), but no
19 ies are required for between 14 (pH) and 32 (PP) years to distinguish a climate change trend from nat
20  the presence of genes for both CPD and (6-4)PP photolyases, as well as genes for nucleotide excision
21                                     The (6-4)PPs are detected as early as 5 min after UV treatment, w
22 trate that Arabidopsis removes CPDs and (6-4)PPs by a dual-incision mechanism that is essentially ide
23 esolved UV damage maps of both CPDs and (6-4)PPs by HS-Damage-seq and compared them to the complement
24 -4)pyrimidine-pyrimidone photoproducts [(6-4)PPs] (at 5 and 20 min and 1, 2, and 4 h) in normal human
25 imidine-pyrimidone (6-4) photoproducts [(6-4)PPs] from UV-irradiated cellular and naked DNA revealed
26 imidine (6-4) pyrimidone photoproducts [(6-4)PPs] that must be repaired for the survival of the plant
27 ts covalently bound 4'-phosphopantetheine (4-PP)-conjugated acyl chain to support maximal cysteine de
28  an obligate subunit but also exploits the 4-PP-conjugated acyl chain to coordinate mitochondrial fat
29 6 no PP); 2,292 patients received TCH (1,444 PP; 848 no PP); and 1,638 patients received AC (857 PP;
30 e [ITT: Delta = -2.0% (95% CI: -7.6%, 3.6%); PP: -1.9% (95% CI: -5.3%, 1.4%)], weight gain [Delta = -
31 .19; 95% CI, 0.12 to 0.30), but not AC (4.7% PP; 3.8% no PP; AOR, 1.21; 95% CI, 0.75 to 1.93) regimen
32 TT: Delta = -20.8% (95% CI: -29.9%, -11.7%); PP: -17.2% (95% CI: -25.6%, -8.7%)].
33 W-GS combinations of 91kDa+80kDa+78kDa+74kDa PPs showed very high grain hardness index (97-100).
34 A total of 4,815 patients received TC (2,849 PP; 1,966 no PP); 2,292 patients received TCH (1,444 PP;
35  no PP); and 1,638 patients received AC (857 PP; 781 no PP) regimen.
36 1 PHQ-9 points [95% CI -1.3 to 1.5], p=0.89; PP: CBT 7.9 PHQ-9 points [7.3]; BA 7.8 [6.5], mean diffe
37 ule membrane major glycoprotein 2 (GP2) as a PP-specific cell surface marker.
38 yorelaxant peptide (SMP) was identified as a PP/OK-type neuropeptide in the starfish Patiria pectinif
39  (CPET), which relies on data collected by a PP detector to reconstruct the 3D distribution of a radi
40                               Furthermore, a PP/OK-type neuropeptide (starfish myorelaxant peptide, S
41 ctronic ground-state calculations indicate a PP multiple bond character in which the terminal phospho
42 that PP-IP4 partially compensates for absent PP-IP5/IP7 in ipk1Delta grown under this condition.
43                Finally, we show how to adapt PP-GLM estimation procedures to guarantee model stabilit
44 4/pRB pathway may no longer be accurate; all PPs are potentially critical targets of CDK-cyclins in m
45 of the CST complex is its primase-Pol alpha (PP) stimulatory activity.
46 ntly greater than that of IVC (P = 0.02) and PP (P = 0.01).
47                                   The AP and PP diets each reduced liver fat by 36%-48% within 6 week
48 ders a selective defect in POLA2-binding and PP stimulation, indicating that these activities are cri
49 ication group (e.g., central systolic BP and PP root-mean-square-errors of 3.3 and 4.2 mm Hg versus 6
50 and diabetes were expressed in the delta and PP/gamma cell types.
51  identified distinct alpha, beta, delta, and PP/gamma cell-type signatures.
52  a significantly strong cytotoxic effect and PP prepared with Irganox 1076 induced a dose-dependant e
53 l cellular regulatory functions of InsPs and PP-InsPs (including telomere maintenance, phosphate sens
54 ty (IP5 and IP6) kinase producing PP-IP4 and PP-IP5/IP7.
55 ylmorphine and hydromorphone for the ITT and PP analyses were reported.
56 rresponding figures on day 42 in the ITT and PP populations were 90.48% and 91.34%, respectively.
57 d a greater diagnostic accuracy than IVC and PP, but its measurement requires transesophageal echocar
58                   Mean arterial pressure and PP were continuously recorded and cardiac output and sys
59 significantly associated with higher SBP and PP in males.
60 monal insulin+, glucagon+, somatostatin+ and PP+ cells and glucose-responsive synchronized calcium si
61 C-terminal regulatory amyloid motif known as PP.
62 d inhibits neuronal activity with attenuated PP-DG synapse plasticity, leading to hippocampus-depende
63 esional skin from psoriasis patients before (PP) and after treatment (PT) with adalimumab and in norm
64  may be substantially compensated by benthic PP, which increased by up to 190%.
65 lankton may have positive effects on benthic PP at the ecosystem scale.
66 f 971 lncRNAs between PP and NN, 157 between PP and PT, and 377 between PT and NN.
67 rformed to determine the association between PP and cardiovascular outcomes, including cardiovascular
68 fferential expression of 971 lncRNAs between PP and NN, 157 between PP and PT, and 377 between PT and
69 ing differentially expressed lncRNAs between PP and NN, we identified a molecular lncRNA signature th
70 ectively potentiate Y4R agonism initiated by PP was confirmed in mouse descending colon mucosa prepar
71 P) amplification (ratio of radial to central PP).
72                                  Complicated PP rates also decreased (P < .0001).
73                                  Complicated PP rates decreased steadily in 2011-2014.
74                               In conclusion, PP-IP5/IP7, not IP6, is essential for fungal virulence.
75                  STP incubated with Corolase PP for 1h (STP-C1) had the most potent angiotensin conve
76 e to obtain for the first time with Corolase PP soy hydrolysates with potential antioxidant and ACE i
77 der two temperature conditions with Corolase PP was studied, assessing the impact of hydrolysis on po
78         FE 204038 dose-dependently decreased PP, did not modify mean arterial pressure, and increased
79                               RVXB decreases PP in two rat models of cirrhosis.
80  to phenotypically characterize hPSC-derived PPs and distinguish these cells from non-PP populations
81 rovide a stability framework for data-driven PP-GLMs and shed new light on the stochastic dynamics of
82           Benthic contributions to ecosystem PP are rarely measured in large aquatic systems, but our
83                                       Egeghy PP, Sheldon LS, Isaacs KK, Ozkaynak H, Goldsmith M-R, Wa
84                                 Encapsulated PPs, despite their reduced antifungal activity, could be
85 antifungal activity of free and encapsulated PPs.
86 lis var. Energo genotypes (polyploid Energo [PP-E]; 2n = 4x = 76) with variation in the green pixel-b
87 all samples were obtained by MAE as follows: PP (18.92+/-0.11), ML (15.19+/-0.11), HL (12.69+/-0.16),
88                                          For PP females, the relative risk was 3.6 (95% confidence in
89                                          For PP mixed with AMO-LDH-OCNT hybrids to produce a composit
90 0% in each group (for AP diet P < .0002; for PP diet P < .0002); decrease in FGF21 correlated with lo
91 % within 6 weeks (for AP diet P = .0002; for PP diet P = .001).
92 isk 9.3 [95% confidence interval 3.3-26] for PP and 9.7 [95% confidence interval 3.2-29.6] for second
93                 ICD therapy is indicated for PP in postpubertal males and in females >/= 30 years wit
94 at implantation 43 years), 66 (97%) were for PP.
95 at implantation 31 years), 61 (76%) were for PP; of 68 females (median age at implantation 43 years),
96                                         Free PPs revealed remarkable antifungal properties with isoeu
97                                Isolates from PP overexpressed more ompA than those from colonized pat
98                          Among isolates from PP, those from bacteremic patients overexpressed nonsign
99  precursor N-acetyl-D-galactosamine (GalNAc)-PP-Und (2 weeks) and (ii) enzymatic extension of the pre
100  and NMR) and deprotection, generates GalNAc-PP-Und.
101  and N-acetylperosamine (PerNAc) onto GalNAc-PP-Und to form the intact RU-PP-Und in a stepwise manner
102 itional intensity function (CIF) for general PP-GLMs.
103 ltration were significantly reduced in group PP relative to group PR, and the loss of collagen matrix
104 eater collagen deposition was noted in group PP relative to groups PR and PH at 4 and 14 days after s
105  periodontitis plus hydrogel with PTB (group PP).
106            Specifically, in nonlinear Hawkes PP-GLMs, the CIF is expressed as a function of the previ
107                                      Hepatic PP-IX accumulation may lead to end-stage liver disease.
108 data were divided into low, middle, and high PP amplification groups.
109                                       Higher PP categories had greater odds for low VA compared with
110 while showing similar accuracy in the higher PP amplification groups.
111 Here, we review current understanding of how PPs foster B-cell encounters with antigen, how they favo
112 , discuss how they have helped to illuminate PP-InsP structure and function, and highlight opportunit
113   Sebaceous glands were markedly atrophic in PP versus nonlesional psoriatic skin (91% average reduct
114 autotrophic structure with no net decline in PP?
115 results point to wall ingrowth deposition in PP TCs being under control of the genetic program regula
116 ve regulators of wall ingrowth deposition in PP TCs.
117 e of a widespread cognitive deterioration in PP-MS patients, with main involvement of visuo-spatial a
118 dule N5 genes were strongly downregulated in PP versus normal skin (fold change < 0.25, 44.4-fold) an
119 l activity underlie cognitive dysfunction in PP-MS, and that CLs possibly play a role in variability
120 lely of module N5 genes, whose expression in PP versus normal skin was inversely correlated with the
121 orphometry identified elongated follicles in PP versus nonlesional psoriatic skin (average 1.7 vs. 1.
122 es, and suggest that loss of visible hair in PP skin may result from abnormal sebaceous gland functio
123      We also observed fewer visible hairs in PP skin, compared with uninvolved nonlesional psoriatic
124                                 Melanomas in PP;Trp53-deficient mice lacked either Ras or Braf mutati
125 ided therapy achieved a greater reduction in PP, which may have contributed to reduce the risk of reb
126  decreased differentiation gene signature in PP/PN-KCs that had not been identified by pre-genomic st
127           On univariate analysis, increasing PP quartile was associated with worse outcomes (p < 0.05
128 s suggest that inflammatory cytokine-induced PP type M cells may be a useful correlate of chronic int
129                                  SFB induced PP Tfh cell differentiation by limiting the access of in
130 phate, UDP-glucose, and Delta(2)-isopentenyl-PP play similar roles in using group transfer chemistry
131 uses excess production of protoporphyrin IX (PP-IX), the final biosynthetic precursor to heme.
132 y monolayer KC cultures grown from lesional (PP) and non-lesional (PN) biopsies of psoriasis patients
133 P with greater accuracy than GTFs in the low PP amplification group (e.g., central systolic BP and PP
134                                  Lox and Lox-PP can reenter the cells where Lox promotes cell prolife
135 ; however, the oncosuppressive action of Lox-PP was not found to prevail.
136  active enzyme (Lox) and the propeptide (Lox-PP).
137 cell proliferation and invasion, whereas Lox-PP acts as tumor suppressor because of its Ras recision
138 cal persistence of MAC-LD was defined as MAC-PP exceeding 1 year, in contrast with the negative-conve
139  factors had a 4.5-fold increased OR for MAC-PP relative to the comparison group.
140 , and positive AFS had an OR of 17.7 for MAC-PP, and those with >/=2 of the factors had a 4.5-fold in
141 f persistent culture-positivity for MAC (MAC-PP) and its impact on radiographic deterioration in MAC-
142                        The predictors of MAC-PP are low BMI, NB pattern, and high AFS grade; if these
143                Independent predictors of MAC-PP were low body mass index (BMI), radiographic nodular-
144                               Other than MAC-PP, the highest AFS grade and no anti-MAC treatment were
145 tients with MAC-LD, 75 (60%) were in the MAC-PP group; these patients had a higher proportion of radi
146 stant N-glycans originating from Man5GlcNAc2-PP-dolichol transferred by TbSTT3A, and endoglycosidase
147 ed to translocate the glycolipid Man5GlcNAc2-PP-dolichol (needed to synthesize N-glycan precursors) a
148 itive N-glycans originating from Man9GlcNAc2-PP-dolichol transferred by TbSTT3B.
149 138 +/- 19/79 +/- 11 mm Hg, rendering a mean PP of 49 +/- 16 mm Hg.
150     Point process generalized linear models (PP-GLMs) provide an important statistical framework for
151  but analysis of KC cultures identified more PP- and PN-decreased DEGs.
152 east milk samples had >50% detection for MP, PP, and EP.
153 ted hospitalization for TC (2.0% PP; 7.1% no PP; adjusted odds ratio [AOR], 0.29; 95% CI, 0.22 to 0.3
154  CI, 0.22 to 0.39) and TCH (1.3% PP; 7.1% no PP; AOR, 0.19; 95% CI, 0.12 to 0.30), but not AC (4.7% P
155 d 1,638 patients received AC (857 PP; 781 no PP) regimen.
156  0.12 to 0.30), but not AC (4.7% PP; 3.8% no PP; AOR, 1.21; 95% CI, 0.75 to 1.93) regimens.
157 ,292 patients received TCH (1,444 PP; 848 no PP); and 1,638 patients received AC (857 PP; 781 no PP)
158 815 patients received TC (2,849 PP; 1,966 no PP); 2,292 patients received TCH (1,444 PP; 848 no PP);
159 ved PPs and distinguish these cells from non-PP populations during differentiation.
160  occurring in systemic lymphoid tissues, not PPs.
161 13 serotypes 19A, 3, 7F, and 1 caused 80% of PP.
162 stent with this conclusion, the abundance of PP TCs with extensive wall ingrowths varied across roset
163  contrasts with the myoexcitatory actions of PP/OK-type neuropeptides in protostomian invertebrates.
164                                  Analysis of PP as a continuous variable yielded similar results.
165 e still responsible for half of the cases of PP in 2011-2014.
166 nalysis that revealed distinct clustering of PP samples from NN.
167                               Development of PP TCs involves extensive deposition of wall ingrowths a
168                   However, the efficiency of PP generation in vitro is highly variable, negatively im
169 FB, by driving differentiation and egress of PP Tfh cells into systemic sites, boosted systemic Tfh c
170                    Retro-orbital infusion of PP-IX or the iron chelator deferoxamine mesylate (DFO),
171                          The intersection of PP-downregulated and sebaceous hyperplasia-upregulated g
172 nthetic CO2 uptake was detected in leaves of PP-E plants with increased chlorophyll and carotenoid co
173 vulinic acid (ALA), generates high levels of PP-IX in zebrafish larvae.
174 n the prevalence of low VA and the levels of PP.
175 implications for the molecular mechanisms of PP, STN1 and STN1-related molecular pathology.
176                           The performance of PP with theDrosophiladata suggests that staNMF provides
177                     Hospitalization rates of PP decreased from 53.6 to 23.3 per 100000 admissions pos
178 bitory-excitatory transition in the roles of PP/OK-type neuropeptides as regulators of muscle activit
179                 However, exome sequencing of PP;Trp53 KO melanomas failed to reveal any additional re
180           In the substrate-free structure of PP, this domain is positioned so as to block nucleic aci
181 hod was demonstrated by the determination of PPs in GPEs of different red grape varieties.
182 control of GPEs, this is the first report of PPs content for Bonarda and Aspirant Bouchet GPE, so the
183 R6 to access the subepithelial dome (SED) of PPs.
184 s in biological media.1-3 The versatility of PPs stem from innovative synthetic strategies that have
185  of the V1a -AVP receptor partial agonist on PP was associated with an improvement in urine volume an
186 carried out to evaluate the effect of CDs on PPs aqueous solubility.
187 AP (rich in meat and dairy foods; n = 18) or PP (mainly legume protein; n = 19) without calorie restr
188  C. neoformans: ipk1Delta produced no IP6 or PP-IP5/IP7 and, in contrast to ipk1Deltakcs1Delta, accum
189  lag period between induction of total PP or PP/Trp53 KO and melanoma development indicates that addi
190    Those with incomplete 4-year follow-up or PP data (final n = 45,087) were excluded.
191            However, there are also two other PPs, p107 and p130, which, when phosphorylated by CDK-cy
192 -dimensional morphometric analysis of paired PP and nonlesional psoriatic skin biopsies.
193 ll ingrowth deposition in phloem parenchyma (PP) transfer cells (TCs) in leaf veins of Arabidopsis (A
194 ficantly increased numbers of Peyer's patch (PP) phenotype M cells were induced at the peak of inflam
195 s model, we demonstrated that Peyer's patch (PP) Tfh cells were essential for gut commensal segmented
196                             Peyer's patches (PPs) are unique compared to other secondary lymphoid tis
197 marily occurs in intestinal Peyer's patches (PPs).
198 tiretroviral treatment (progressor patients [PP]).
199 nd, staNMF identified 21 principal patterns (PP).
200 resentation ofDrosophilaexpression patterns, PP are comparable to a fate map generated experimentally
201 ), 4-tert-butylphenol (TBP), 4-pentylphenol (PP), 4-hexylphenol (HP), 4-tert-octylphenol (TOP), 4-n-o
202                               Pedal peptide (PP) and orcokinin (OK) are related neuropeptides that we
203                    Molluscan pedal peptides (PPs) and arthropod orcokinins (OKs) are prototypes of a
204 ifungal activities of some phenylpropanoids (PPs) were investigated.
205  clinical aspects of pneumococcal pneumonia (PP) in children has not been reported.
206 ic or nonbacteremic patients with pneumonia (PP) caused by A. baumannii (13 from the unicenter and 23
207 ed via aqueous precipitation polymerization (PP) of di(ethylene glycol) methyl ether methacrylate (ME
208 dant delta and gamma/pancreatic polypeptide (PP) cells.
209  its cognate ligand, pancreatic polypeptide (PP), are involved in the regulation of energy expenditur
210 d quality control of multiclass polyphenols (PPs).
211                              Polyphenylenes (PPs) represent a class of conjugated polymers that have
212         Polyethylene (PE) and polypropylene (PP) represent approximately 92% of total plastic product
213 ibes the printing of an inert polypropylene (PP) architecture with the concurrent printing of soft ma
214 cs such as polyethylene (PE), polypropylene (PP), polystyrene (PS), and polyethylene terephthalate (P
215  This approach was applied to polypropylene (PP) films prepared with a selection of additives.
216 (ArPPLN1a-e) derived from the SMP precursor (PP-like neuropeptide precursor 1; ArPPLNP1) in the starf
217 stemic administration of the heme precursors PP-IX or ALA + DFO into zebrafish larvae provides a new
218       However, results from the prespecified PP population suggest that enhancing remyelination in th
219 VXB significantly decreased portal pressure (PP) in both models of cirrhosis without changes in porta
220 heir usefulness in reducing portal pressure (PP) in this condition.
221 the hemodynamic response of portal pressure (PP) to drug therapy accurately stratifies the risk of va
222 or each 15 mmHg increment in pulse pressure (PP) (p < 0.001).
223 y not adapt to variations in pulse pressure (PP) amplification (ratio of radial to central PP).
224 4; each 1-mm Hg increment in pulse pressure (PP) was associated with 1.6% (95% CI: 0.7-2.5%), 0.8% (9
225 re wave pulsatility (central pulse pressure [PP], reflected pressure wave, and reservoir pressure) at
226  medication use, and mean arterial pressure, PP quartile was still associated with all outcomes excep
227  follow-up 8.5 years) after ICD for primary (PP) and secondary prophylaxis in males and females, dete
228 e continuous data, e.g. particle-processing (PP) detectors, has the potential to eliminate null funct
229 Exogenously infused or endogenously produced PP-IX accumulates preferentially in the liver of zebrafi
230 l specificity (IP5 and IP6) kinase producing PP-IP4 and PP-IP5/IP7.
231 cosystem function and on primary production (PP) in particular.
232  oxygen concentration or primary production (PP).
233 on of DCF and other pharmaceutical products (PPs) with a KF Freundlich constant of 1.7 L g(-1) and no
234  applications of GP2.Pancreatic progenitors (PPs) can be derived from human pluripotent stem cells in
235    PDX1(+)/NKX6-1(+) pancreatic progenitors (PPs) give rise to endocrine cells both in vitro and in v
236 lity of purified human STN1 alone to promote PP activity in vitro.
237                         Primary prophylaxis (PP) was defined as G-CSF administration within 5 days of
238 eveloped to distinguish between the proposed PP-InsP signal transduction mechanisms: protein binding,
239 zed for methylparaben (MP), n-propylparaben (PP), ethylparaben (EP), butylparaben (BP), isobutylparab
240 igh in animal protein (AP) vs plant protein (PP), which differ in levels of methionine and BCAAs, in
241 way via its influence on the pocket protein (PP) pRb.
242   Intention-to-treat (ITT) and per-protocol (PP) analyses showed noninferiority of SMS-RUTF compared
243 n-to-treat (ITT); prespecified per-protocol (PP) analyses were also done.
244 Both intent-to-treat (ITT) and per-protocol (PP) analyses were conducted.
245                         In the per-protocol (PP) analysis, adjusted-ACPR was achieved in 100% (165/16
246 8 in intent-to-treat (ITT) and per-protocol (PP) populations was 92.86% and 92.46% and 99.25% and 99.
247 east eight treatment sessions (per protocol [PP]).
248     Similar to patients with protoporphyria, PP-IX is excreted through the biliary system.
249  second oxidation step, using pyrophosphate (PP) as a Mn(III) trap.
250 horylation of IP3 to inositol pyrophosphate, PP-IP5/IP7, which is essential for virulence of Cryptoco
251                 The inositol pyrophosphates (PP-InsPs) are a unique group of intracellular messengers
252 sphates (InsPs) and inositol pyrophosphates (PP-InsPs).
253 , accumulated IP5 and its pyrophosphorylated PP-IP4 derivative.
254 Ac) onto GalNAc-PP-Und to form the intact RU-PP-Und in a stepwise manner.
255 hesis of Escherichia coli (serotype O157) RU-PP-Und.
256 tic extension of the precursor to produce RU-PP-Und (2 weeks).
257 epeating unit undecaprenyl pyrophosphate (RU-PP-Und), the key building block in this pathway.
258 precursors: an SMP-type precursor (A. rubens PP-like neuropeptide precursor 1; ArPPLNP1) and a second
259 s in primary-progressive multiple sclerosis (PP-MS) and to explore how they are affected by the prese
260 orrelation networks were constructed for six PP that span along the embryonic anterior-posterior axis
261 nscriptome study of lesional psoriatic skin (PP) versus normal skin, we found a coexpressed gene modu
262 on GPE showed the highest content of studied PPs (9804.2 mug g(-1)GPE) followed by Bonarda GPE (7302.
263 the solubility and photostability of studied PPs (by 2 to 17-fold and 2 to 44-fold, respectively).
264  international cohort of high-risk subjects, PP, a readily available hemodynamic parameter, is associ
265 espite passing common goodness-of-fit tests, PP-GLMs estimated from data are often unstable, leading
266 ranscriptome sequence data has revealed that PP/OK-type neuropeptides also occur in a deuterostomian
267 ll mutants except ipk1Delta, suggesting that PP-IP4 partially compensates for absent PP-IP5/IP7 in ip
268                                          The PP motif is similar to the RHIM amyloid motif directing
269                                          The PP-E plants developed wider leaves with thicker midrib a
270 we observed a strong correlation between the PP-stimulatory activity of STN1 variants and their abili
271 sttranslational modification mediated by the PP-InsPs.
272 Nod-like receptor, both of which display the PP motif.
273                  These studies establish the PP SED as a niche supporting DC-B cell interactions need
274 iority of hydromorphone was confirmed in the PP analysis (-1.44; 90% CI, -3.22 to 0.27) for street he
275 up, whereas 135 (61%) were assessable in the PP population in the BA group compared with 151 (69%) in
276 ed opicinumab and 36 received placebo in the PP population.
277                  Genetic perturbation of the PP-InsP biosynthetic network indicates a central role fo
278 is of the putative mutation signature of the PP;Trp53 KO melanomas suggests that melanocytes are prim
279   Our findings not only demonstrate that the PP-InsPs provide a central line of communication between
280                         Finally, we used the PP to generate local transcription factor regulatory net
281 ficantly higher in subjects on the AP vs the PP diet.
282                                        These PP TCs potentially facilitate phloem loading by enhancin
283            In sum, deregulation of all three PPs appears central to neoplastic progression for melano
284 ingly, melanocyte-specific loss of all three PPs facilitated melanoma development (median age of onse
285 s (KOs) of various combinations of the three PPs were generated.
286            Cryptococcal Kcs1 converts IP6 to PP-IP5/IP7, but the kinase converting IP5 to IP6 is unkn
287                    Antigens are delivered to PPs by specialized microfold (M) epithelial cells and th
288    The lag period between induction of total PP or PP/Trp53 KO and melanoma development indicates tha
289 om the starfish Asterias rubens revealed two PP/OK-type precursors: an SMP-type precursor (A. rubens
290 oles of neuropeptides derived from these two PP/OK-type precursors in starfish.
291 her approach, and pulse pressure variations (PP) were recorded with the patient in the semirecumbent
292 itated by the total synthesis of the various PP-InsP isomers and by the development of new methods th
293 ver the state of the art research on various PPs related to the relationship between their structure
294 geneous cohort of HIC patients but also when PP were included in the analysis.
295 e aim of this study was to determine whether PP is associated with major adverse cardiovascular outco
296 usceptibility, and outcomes of children with PP 4 years before and 4 years after the introduction of
297 adult leaves, an observation consistent with PP TC development reflecting vegetative phase change (VP
298                 A total of 377 patients with PP requiring hospitalization were identified.
299 of 40%, greater than the PHRR reduction with PP/20 wt% AMO-LDH (31%).
300      We identified patients </=18 years with PP at 8 children's hospitals in the United States (2006-

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