ライフサイエンスコーパス: arginine

1 er transport rate of indospicine compared to arginine.

2 abolite 2-aminopimelic acid in comparison to arginine.

3 , is known as the Sakaguchi test and targets arginine.

4 ted by nitric oxide-dependent depletion of l-arginine.

5 n various ASS-deficient cancers by depleting arginine.

6 enile fish fed with graded levels of dietary arginine.

7 an enzyme that metabolizes l-citrulline to l-arginine.

8 predicted and confirmed an integral role of arginine(172), located in the 2nd extracellular loop, in

9 nd was not affected adversely by mutation of arginine(172).

10 ZNF326 is symmetrically dimethylated at arginine 175 (R175) and this modification is lost in a P

11 ygous TGM1 genotype had a mutation at either arginine 307 or 315, providing evidence that mutations a

12 Among these, arginines 35, 36 and 60 were the most frequently altered

13 oxide synthase inhibitor l-N(G)-monomethyl-l-arginine acetate (l-NMMA) into both femoral arteries rev

14 lts in a dramatic decrease in methylation of arginines adjacent to M6CK-phosphorylated amino acids.

15 Substitutions of the invariant arginine anchor residue in GAG result in global redistri

16 Disrupting the 'arginine anchor' on CENP-C for the nucleosomal acidic pa

17 t coincide with the preference for P2 and P7 arginine anchors.

18 Decreases in arginine and citrulline and increases in ADMA were obser

19 Arginine and Glutamate-Rich protein 1 (ARGLU1) is a prot

20 r example, methylated derivatives of lysine, arginine and glutamine are readily accessible.

21 tained by diet and endogenous synthesis from arginine and glycine.

22 Most nNOS inhibitors mimic l-arginine and have poor bioavailability.

23 is: sphingolipid metabolism (P=6.6x10(-5) ), arginine and proline metabolism (P=1.12x10(-7) ), glycer

24 pathway analysis, compounds associated with arginine and proline metabolism were found to be the mos

25 ers, making cells dependent on extracellular arginine and targetable by the arginine-degrading enzyme

26 Through coincubation with supplemental arginine and the arginine antagonist canavanine, we show

27 iation between consumption of the amino acid arginine and the rate of adverse birth outcomes using da

28 Consumption of amino acids such as arginine and tryptophan by immunoregulatory macrophages

29 R579W) and one that deletes three pathogenic arginines, and explored phenotypes of these lines alongs

30 ncubation with supplemental arginine and the arginine antagonist canavanine, we show that arginine av

31 rameters of pancreatic injury in mice with L-arginine AP.

32 s) residues of the hD4R are substituted with arginine (Arg) residues, cellular hD4R protein levels in

33 hism in ZC3HC1 (rs11556924), resulting in an arginine (Arg) to histidine (His) substitution in its en

34 Here, we show that L-arginine (Arg) uptake through the host cell's SLC7A2-enc

35 the key enzyme that controls biosynthesis of arginine (Arg).

36 hism substitution from glutamine (Gln, Q) to arginine (Arg, R) at codon 460 of the purinergic P2X7 re

37 tonometry (EndoPAT), and plasma levels of l-arginine, arginase-1, and asymmetric dimethylarginine we

38 We find enrichment of glycine, valine, and arginine as both individual amino acids and as a part of

39 ctively, and 0.78 (95% CI: 0.64 to 0.97) for arginine at 1 month.

40 iated with the toothed whales or Odontoceti (arginine at 156) and baleen whales or Mysticeti (glutami

41 hat replaced the lysine at codon 110 and the arginine at codon 111 with alanine codons failed to repl

42 A2*05 allotype prefers the basic amino acid arginine at the second position of the peptide, and hydr

43 arginine antagonist canavanine, we show that arginine availability is a determinant of GBS cytotoxici

44 Can1 mutants altered in the arginine-binding site or a cytosolic tripeptide sequence

45 RNA sequencing and qRT-PCR revealed that arginine biosynthesis genes (argR, argB, argC, argG, arg

46 conclude that by promoting the expression of arginine biosynthetic genes, especially argB gene, the c

47 tic stability by substituting the N-terminal arginine by norarginine.

48 First, we investigated the uptake of arginine by the cells of the biofilm as well as the meta

49 -Valentine leukocidin (PVL), SCCmec IVa, the arginine catabolic mobile element (ACME), and a specific

50 cumulation that results from the shunting of arginine catabolism into alternative nitrogen repositori

51 the biofilms, indicating active uptake, and arginine catabolites citrulline, ornithine, and putresci

52 ctions for specificity were also enriched in arginine CDR mutations, but these antibodies possessed s

53 eal unique hydrophobic environments near the arginine CDR mutations.

54 are governed by the relative contribution of arginine CDR residues to the overall antibody affinity.

55 Significant increases in urea, arginine, citrulline, asymmetric and symmetric dimethyla

56 Plasma levels of arginine, citrulline, ornithine, asymmetric dimethylargi

57 ne substitution enhances the stability of an arginine-containing collagen peptide and provide a struc

58 lexes with a central binding cavity for twin-arginine-containing signal peptides.

59 lated properties studied, relative lysine to arginine content was found to be higher in CH1 and CL th

60 Suppression of arginine-creatine metabolism by CKMT1-directed shRNAs or

61 phosphocreatine-mediated reactivation of the arginine-creatine pathway.

62 a thioether, or an extra amine group such as arginine, cysteine, lysine, methionine, and tryptophan h

63 extracellular arginine and targetable by the arginine-degrading enzyme pegylated arginine deiminase (

64 Arginine deiminase (ADI) inhibits growth in various ASS-

65 Purpose Pegylated arginine deiminase (ADI-PEG 20) depletes essential amino

66 e by the arginine-degrading enzyme pegylated arginine deiminase (ADI-PEG 20).

67 ole of the arginine-lowering agent pegylated arginine deiminase (ADI-PEG20) has not been evaluated in

68 cristatus and P. gingivalis, and identified arginine deiminase (ArcA) of S. cristatus as the signali

69 (SAPP) derived from Streptococcus cristatus arginine deiminase (ArcA) was able to repress the expres

70 Protein arginine deiminase 2 (PAD2) plays a key role in the onse

71 ) to citrulline with recombinant polypeptide arginine deiminase 4 (PAD4) abolished ADAMDEC1-catalyzed

72 Peptidyl arginine deiminase 4 (PAD4), which binds proteins and ci

73 racellular trap formation involving peptidyl arginine deiminase 4.

74 enter phase 2 randomized clinical trial, the Arginine Deiminase and Mesothelioma (ADAM) study, was co

75 d decrease in the expression of genes in the arginine deiminase pathway during stringent response act

76 Peptidyl arginine deiminases have been shown to be hyperactive in

77 onal oxygenase activities, acting as both an arginine demethylase and a lysyl-hydroxylase.

78 JMJD6 functions directly or indirectly as an arginine demethylase of G3BP1 that promotes SG formation

79 sponse activation and increased virulence is arginine dependent.

80 d warrants further clinical investigation in arginine-dependent cancers.

81 Randomization 2:1 to arginine deprivation (ADI-PEG20, 36.8 mg/m2, weekly intr

82 d with ASS1 immunohistochemistry, and longer arginine deprivation correlated with improved PFS.

83 Preclinical studies show that arginine deprivation is synthetically lethal in arginino

84 aled the significance of macrophage-mediated arginine deprivation to T cells.

85 In this trial, arginine deprivation with ADI-PEG20 improved PFS in pati

86 After 2 days of arginine deprivation, mTORC1 activity declined paralleli

87 Arginine derived biomolecules were found to involve in m

88 in the CCT domain, corresponding to an NF-YA arginine directly involved in CCAAT recognition, abolish

89 2-aminopimelic acid were more cytotoxic than arginine, displaying the highest toxicity in HepG2 liver

90 substitution of APP C-terminal lysines with arginine disrupts APP ubiquitination and that an increas

91 ells in G1, and this effect is mediated by l-arginine elimination rather than metabolite generation.

92 n, and inhibition of NO by N(G)-monomethyl-L-arginine enhanced intracellular survival of Mtb.

93 The beneficial effects of preoperative arginine-enriched dietary supplements (AES) are highly c

94 e after 6, 20, or 48 h of preincubation with arginine exhibited a recovery to higher pH.

95 This is the first report of an arginine finger observed in a septin and suggests that C

96 etween Hda monomers occludes the active site arginine finger, blocking its access to DnaA.

97 rs; (ii) mutational analysis reveals a trans-arginine finger, R158, indispensable for ATP hydrolysis;

98 mpatible with the location of the identified arginine finger.

99 mutations, suggesting that over-reliance on arginine for affinity leads to reduced specificity.

100 On the other hand, arginine formed free Amadori product, free imidazolinone

101 However, the full repertoire of arginine GlcNAcylation induced by pathogen-delivered Nle

102 e derivatives, model systems of guanidine or arginine/glucose or (13)[C-6]-glucose were heated in aqu

103 ptor, generally alphavbeta6, via a conserved arginine-glycine-aspartic acid (RGD) motif in the expose

104 nt conformation beta5/beta3 integrins and an arginine-glycine-aspartic acid (RGD) sequence in the fir

105 ro by cell attachment to the high density of arginine-glycine-aspartic acid tripeptide present in DAP

106 e designed to mimic the tripeptide sequence (arginine-glycine-aspartic acid) of the natural ligands;

107 for a series of AMPA channels with different arginine/glycine (R/G) editing and flip/flop status.

108 One such effector is the arginine glycosyltransferase NleB1 (NleB(CR) in C. roden

109 ature and pH but also by additives including arginine, guanidine, methionine, and thiocyanate.

110 ed interest is the mutation Histidine 147 to Arginine (H147R) in human TRiC subunit 5 (CCT5), which h

111 or the MARylation of glutamate/aspartate and arginine have been identified, the respective enzymes wi

112 to react with a small group of amino acids (arginine, histidine, lysine, phenylalanine, tyrosine, an

113 gR, argB, argC, argG, argH and argJ) and two arginine/histidine permease genes (SSA_1568 and SSA_1569

114 Overall, we show that arginine improved pH homeostasis through a remodeling of

115 replaced mainly by cysteine, tryptophan and arginine in a codon-specific manner.

116 to give ethylene is promoted by binding of l-arginine in a nonoxidized conformation and of 2-oxogluta

117 single amino acid at position 112, which is arginine in apoE4 and cysteine in apoE3.

118 There is evidence that the use of arginine in dentifrices helps protect against caries.

119 as a significantly higher concentration than arginine in the fingerprint content of both males and fe

120 stigate the reactivity of free guanidine and arginine in the formation of imidazolinone derivatives,

121 osphine complex incorporating the amino acid arginine in the outer coordination sphere was immobilize

122 erine protease with a narrow specificity for arginine in the P1 position, which catalyzes the first e

123 Citrulline and arginine increased significantly in HD compared to contr

124 tinuous and categorical associations between arginine intake and adverse birth outcomes.

125 The continuous associations of arginine intake with preterm birth before 37 weeks and w

126 al form of arginase (ARG2), which hydrolyzes arginine into ornithine and urea, is induced upon obesit

127 r ATP hydrolysis; (iii) the location of this arginine is conserved with the HerA/FtsK ATPase superfam

128 Targeting arginine is safe and warrants further clinical investiga

129 neurons, whereas mutagenesis of lysine 68 to arginine (K68R), mimicking deacetylation, increased acti

130 Arginine kinase (AK), which is a member of the phosphage

131 Among them, invertebrate tropomyosin, arginine kinase, myosin light chain, sarcoplasmic calciu

132 re, the generated acidic H2 O2 can oxidize l-Arginine (l-Arg) into NO for enhanced gas therapy.

133 esult in substitution of leucine-115 with an arginine (L115R) or deletion of the neighbouring leucine

134 Lys(129) mutation to glutamate or arginine led to misfolded protein that was degraded in v

135 lalanine, the latter of a size comparable to arginine, led to spectacular reductions of apparent Na(+

136 wed a serum amino acid signature composed of arginine, leucine/isoleucine, phenylalanine, tyrosine, v

137 These results indicate that 1.62% dietary arginine level improves glycolysis and fatty acid synthe

138 sulin signaling pathway, while 2.70% dietary arginine level led to inhibition of these genes.

139 ith the control group (0.87%), 2.31% dietary arginine level resulted in the upregulation of the relat

140 Furthermore, 2.70% dietary arginine level significantly lowered GLUT2 and increased

141 1.62% dietary arginine level significantly upregulated ACC, FAS and G6

142 s by increasing GK mRNA level; 2.70% dietary arginine level upregulated gluconeogenesis and resulted

143 1.62% dietary arginine level upregulated glycolysis by increasing GK m

144 The role of the arginine-lowering agent pegylated arginine deiminase (AD

145 Arginine, lysine, isoleucine, leucine, methionine, pheny

146 hook, and RK clusters (clusters enriched for arginine-lysine residues).

147 anced NO activity, because N(G)-monomethyl-l-arginine markedly blunted the flow response to obestatin

148 lline (800, 1600 mum) rescued NOx when the l-arginine media concentration was 25 mum but failed to do

149 Under reduced l-arginine media conditions, HBECs treated with l-citrulli

150 nitric oxide synthase with N(omega) -nitro-l-arginine methyl ester.

151 ings reveal the importance of PRMT5-mediated arginine methylation during DSB repair pathway choice th

152 controlling about 85% of the total cellular arginine methylation in proteins.

153 regulating DNA repair; however, the role of arginine methylation in this process is poorly understoo

154 Arginine methylation is a common posttranslational modif

155 and mass spectrometry, we show that elevated arginine methylation of SRSF5 and lower phosphorylation

156 (PRMT5) complexed with MEP50/WDR77 catalyzes arginine methylation on histones and other proteins.

157 Arginine methylation on histones is a central player in

158 rginine methyltransferases (PRMTs) introduce arginine methylation, a post-translational modification

159 scription by attenuating the effect of local arginine methylation.

160 Protein arginine methyltransferase (PRMT) activity has been impl

161 However, a conclusive role for the protein arginine methyltransferase (PRMT) enzymes that catalyze

162 sferase 1 (CARM1) is a member of the protein arginine methyltransferase (PRMT) family and methylates

163 Coactivator associated arginine methyltransferase 1 (CARM1) is a member of the

164 PRMT4 or coactivator-associated arginine methyltransferase 1 (CARM1) is a propitious tar

165 Coactivator-associated arginine methyltransferase 1 (CARM1) is a protein methyl

166 Protein arginine methyltransferase 1 (PRMT1) is an essential enz

167 d receptor coactivator 1 (SRC1), and protein arginine methyltransferase 1 (PRMT1) only modestly incre

168 EYA1 physically interacts with protein arginine methyltransferase 1, which methylates EYA1 at t

169 tase-transcription activator EYA1 by protein arginine methyltransferase 1: mechanistic, functional, a

170 requires assembly factors united in protein arginine methyltransferase 5 (PRMT5) and survival motor

171 Protein arginine methyltransferase 5 (PRMT5) complexed with MEP5

172 Protein arginine methyltransferase 5 (PRMT5) is an emerging epig

173 kinases (M6CKs) bind subunits of the protein arginine methyltransferase 5 (PRMT5) molecular complex t

174 Protein arginine methyltransferase 7 (PRMT7) catalyzes the intro

175 Here we use mice lacking protein arginine methyltransferase 8 (PRMT8) in the brain to exa

176 recruitment of CARM1 not only adds a protein arginine methyltransferase activity to the ER-coactivato

177 Protein arginine methyltransferase enzyme 5 (PRMT5) regulates ma

178 h an shRNA screen, we identified the protein arginine methyltransferase Prmt1 as a vulnerable interve

179 Here we identify the arginine methyltransferase PRMT5 as a key regulator of h

180 of Cancer Cell, Braun et al. report that the arginine methyltransferase PRMT5 is critical for tumor c

181 The type II arginine methyltransferase PRMT5 is responsible for the

182 ly modulates enzymatic activity of a protein arginine methyltransferase vital to abiotic stress toler

183 CARM1 is an arginine methyltransferase with diverse histone and non-

184 ng flagellar dynamics, we focused on protein arginine methyltransferases (PRMTs) 1, 3, 5, and 10.

185 Protein arginine methyltransferases (PRMTs) introduce arginine m

186 ification in eukaryotes catalyzed by protein arginine methyltransferases (PRMTs) that are typically t

187 rination activity more than any other single arginine mutation in yeast.

188 Single arginine mutations eliminated the fast interactions with

189 These findings demonstrate that arginine mutations in antibody CDRs display context-depe

190 ereas the less specific antibodies contained arginine mutations in more hydrophilic regions.

191 nine, we generated single, double and triple arginine mutations in RTA.

192 The more specific antibodies contained arginine mutations in the most hydrophobic portions of t

193 antibodies were much less dependent on their arginine mutations, suggesting that over-reliance on arg

194 Indospicine, a non-proteinogenic analogue of arginine, occurs only in Indigofera plant species and ac

195 Substitution of the active site binding arginine of CP2 to N-varepsilon-trimethyl-lysine or meth

196 ic arrangement and number of the lysines and arginines of the PBD vary among the lipins.

197 fferent variants or isoforms of tropomyosin, arginine or creatine kinase, glyceraldehyde-3-phosphate

198 We also found no effects with alanine, arginine, or a mixture of both amino acids.

199 activity against the substrate N-benzoyl-DL-arginine p-nitroanilide, is proposed as an alternative t

200 tively with arginase-1 and positively with l-arginine (P = .001).

201 membrane translocation, we synthesized seven arginine placement variants of LRLLRWC and compared thei

202 Arginine play numerous roles in several biological proce

203 The toxic proline:arginine (PRn) poly-dipeptide encoded by the (GGGGCC)n r

204 Here we report that serine-arginine protein kinase 2 (SRPK2) phosphorylates delta-s

205 re collected for 16S sequencing and targeted arginine quantification, and supernatants were prepared

206 Here, we show that a single Arginine (R) to Glycine (G) mutation at position 76 in t

207 We previously showed that the amino acid arginine (R) within the sequence SRL (amino acids 445 to

208 g the genomically encoded glutamine (Q) with arginine (R); thus this editing site is referred to as t

209 tides including those containing an amidated arginine(R)-phenylalanine(F) motif at their C-termini (R

210 tween two short highly charged peptides-deca-arginine (R10) and deca-lysine (K10).

211 Glutamate E153 on the E-loop and arginine R210 on the adjacent subunit's backbone alpha-h

212 One is the presence of an arginine R696 in the middle of the TM helix.

213 SLC38A9 mediates the transport, in an arginine-regulated fashion, of many essential amino acid

214 The results show that these arginines relay critical information between the PCNA-bi

215 Activation of mTORC1 by arginine requires SLC38A9, a poorly understood lysosomal

216 at mediate assembly, via interaction with an arginine residue at a similar register to these aspartic

217 e by substituting the highly conserved first arginine residue in transmembrane segment 4 (domain 1),

218 at the introduction of a single lipid-facing arginine residue near the middle of the beta barrel of t

219 e N terminus as well as a strictly conserved arginine residue toward the C terminus of ORF52 play cri

220 tion of cathepsin G or citrullination of the arginine residue within an LC3-interacting region motif

221 formed by stacking of the side chains of two arginine residues and by salt bridges formed between the

222 Proteins with methyl arginine residues are also enriched at the tip and base

223 electrostatics; within this code lysine and arginine residues are non-equivalent and prenyl chain le

224 Arginine residues form a positively charged patch to bin

225 CR) in C. rodentium) that modifies conserved arginine residues in death domain-containing host protei

226 e that forms adducts on cysteine, lysine and arginine residues of proteins, thereby affecting their f

227 introduction of monomethylation marks at the arginine residues of substrate proteins.

228 target genes via symmetric dimethylation of arginine residues on histones H4R3, H3R8 and H2AR3.

229 lly cleave proteins C-terminal to lysine and arginine residues prior to LCMS/MS analysis of the resul

230 In particular, we identified several arginine residues that interact with the polyanionic sub

231 found that EHEC NleB1 glycosylated two GAPDH arginine residues, Arg(197) and Arg(200), and that these

232 Here, we demonstrate that 2 conserved arginine residues, R304 and R306, of EYA1 are essential

233 genetic modifications by methylating histone arginine residues.

234 N-varepsilon-trimethyl-lysine or methylated arginine results in cyclic peptide substrates, indicatin

235 lls adjust their metabolism when deprived of arginine revealed the significance of macrophage-mediate

236 SR (serine-arginine)-rich proteins influence multiple steps in mRNA

237 eracts with and hydroxylates multiple serine/arginine-rich (SR) proteins and SR related proteins, inc

238 We now identify an active role for arginine-rich domains in these phase separations.

239 Expression of arginine-rich DPRs in cells induced spontaneous stress g

240 Short arginine-rich proteins called protamines mediate the nea

241 that binds with high affinity to serine- and arginine-rich splicing factor 2 (SRSF2), a crucial prote

242 r reader protein, in conjunction with serine/arginine-rich splicing factor 3 (SRSF3) and SRSF10.

243 or shuttling specific cargoes such as serine/arginine-rich splicing factors from the cytoplasm to the

244 The versatile functions of SR (serine/arginine-rich) proteins in pre-mRNA splicing and process

245 precursor proteins possess a conserved twin-arginine (RR) motif in their signal peptides that is inv

246 vation by cholesterol independently from its arginine-sensing function.

247 CFIm activator functions are mediated by the arginine-serine repeat (RS) domains of CFIm68/59, which

248 Thus, through SLC38A9, arginine serves as a lysosomal messenger that couples mT

249 ux into the urea cycle and infusion of (15)N-arginine shows that Arg2 loss causes significant ammonia

250 small contribution of charge at a conserved arginine side chain previously suggested to form a salt

251 Variants of TP2 with shorter and longer arginine side-chain analogs translocate slower than TP2.

252 unction as the primary docking site for twin-arginine signal peptides.

253 ation of caspase proteases and inhibition of arginine-specific proteases.

254 both an ITIM mutation and either alanine or arginine substitutions had reduced titers and small plaq

255 ompared to that of the wild-type gBcyt while arginine substitutions had wild-type-like fusion levels

256 In contrast, arginine substitutions resulted in syncytia with only 2-

257 Alanine and arginine substitutions were used to determine if the pos

258 o Tat signal peptides with inactivating twin arginine substitutions.

259 rporation of (13) C/(15) N-lysine and (13) C-arginine such that each peptide derived from trypsin dig

260 est was reversible at any point by exogenous arginine, suggesting starved T cells remain poised await

261 ASS1 catalyzes the rate-limiting step of arginine synthesis in urea cycle and citrulline-nitric o

262 We observed specific up-regulation of the arginine synthesis pathway associated with overexpressio

263 cinate synthetase 1 (ASS1), a key enzyme for arginine synthesis, occurs in many cancers, making cells

264 pecificity is achieved by a cluster of three arginines that accommodates the terminal carboxyl group

265 s complex task, here we focused on conserved arginines that might play a central coordinating role in

266 tase 1 (ASS1) -negative tumors by converting arginine to citrulline and ammonia.

267 the H(+),K(+)-ATPase, the ability of the M8 arginine to donate an internal cation binding at the thi

268 unlike JM22, uses a tryptophan instead of an arginine to fill a critical notch between GIL and the HL

269 hesis: the transfer of an amidino group from arginine to glycine to form ornithine and guanidinoaceta

270 interface, one monomer supplied a catalytic arginine to the opposing subunit, greatly accelerating t

271 (the gene encoding the LiaS protein with an arginine-to-glycine change at position 135 [liaS(R135G)]

272 An arginine-to-histidine replacement at residue 435 in the

273 an populations and differs from PRDM9a by an arginine-to-serine change (R764S) in ZF9 and by replacem

274 The twin-arginine translocase (Tat) transports folded proteins ac

275 Twin-arginine translocation (Tat) systems transport folded pr

276 t surface display (YSD) screening and a twin-arginine translocation pathway selection.

277 hat transport-elicited ubiquitylation of the arginine transporter Can1 is promoted by transition to a

278 hift in the microbial community structure in arginine-treated biofilms as well as increased species d

279 ate the mechanistic and ecological effect of arginine treatment on the oral microbiome and its regula

280 ycin treatment, suggesting that the enhanced arginine uptake mediates AS II-induced wound healing.

281 ughout the vasculature, where it regulates L-arginine uptake.

282 activated via the TORC1 kinase complex upon arginine uptake.

283 Arginase-II (Arg-II), the type-II L-arginine-ureahydrolase, is highly expressed in pancreas.

284 Some DRY Arginine variants correlate with smell preferences in su

285 Receptor 2 suggested that a single lysine - arginine variation at the extracellular face of the rece

286 unction indicated that although the lysine - arginine variation between human and mouse orthologs had

287 In response to 1-desamino-8-D-arginine vasopressin (DDAVP), peak VWF:Ag levels exceede

288 sponsible for the clearance of misfolded pro-arginine vasopressin (proAVP) in the ER.

289 Arginine vasopressin colocalized with EGFP more often in

290 otropin-releasing hormone and do not express arginine vasopressin or oxytocin.

291 Expression of arginine vasopressin receptor 1a (Avpr1a) and oxytocin r

292 dogenous NO production from the amino acid l-arginine, via nitric oxide synthase (NOS) enzymes, resea

293 Arginine was detected within the cells of the biofilms,

294 estigate the individual contribution of each arginine, we generated single, double and triple arginin

295 Dietary intakes of arginine were assessed using repeated 24-hour recalls th

296 T3R3 hexamers loaded with both serotonin and arginine were obtained.

297 to K305 in human NHA2 has been replaced with arginine, which is a mutation that makes NapA electroneu

298 Some of the PC studies also included arginine, which may be abundant in insulin granules upon

299 and proline, glycine, glutamate, lysine and arginine, which were all consumed significantly faster.

300 KCl and the addition of flavor enhancers (l-arginine, yeast and oregano extract) on probiotic Prato