ライフサイエンスコーパス: blood gas

1 pressure, heart rate, and systemic arterial blood gas.

2 recorded at the time of a clinical arterial blood gas.

3 ce marker, was calculated with each arterial blood gas.

4 y, in lung-function laboratories to estimate blood gases.

5 r measurement of arterial and central venous blood gases.

6 daily digital chest radiographs and arterial blood gases.

7 ios and were used to compute global arterial blood gases.

8 usion ratios (V(A)/Q) predictive of arterial blood gases.

9 ary compliance and deterioration in arterial blood gases.

10 ood flow were measured with pulmonary venous blood gases.

11 n intrapulmonary vasodilatation and arterial blood gases.

12 xchange impairment as determined by arterial blood gases.

13 sion did not affect systemic hemodynamics or blood gases.

14 22.7 L. min(-1) under conditions of altered blood gases.

15 ation along with arterial blood pressure and blood gases.

16 lung compliance, inspiratory resistance, and blood gases.

17 collected ventilation variables and arterial blood gases.

18 We conclude that in this range of blood gases: (1) the GG reflex to negative pressure is u

19 catheter (2% to 51%), and determination of a blood gas (24% to 70%).

20 ntion was associated with 128 fewer arterial blood gases, 73 fewer chest radiographs, and 16 fewer RB

21 um erythropoietin levels (76%), and arterial blood gases (75%) were the most frequent tests used in t

22 erature monitoring and intermittent arterial blood gas (ABG) analysis was undertaken.

23 We measured arterial blood gases (ABGs) on air before and 3 mo following LVRS

24 crossover format, and the patients' arterial blood gases (ABGs) were measured at baseline and at inte

25 ir pulmonary function tests (PFTs), arterial blood gases (ABGs), and respiratory muscle strength as e

26 The authors explored associations between blood gas abnormalities in more than 1,000 preterm infan

27 of the subjects, with separate analyses for blood gas abnormalities on multiple days and for partial

28 ssels but did not cause alveolar flooding or blood gas abnormalities.

29 hemorrhagic shock, we measured hemodynamics, blood gases, acid-base status, metabolism, organ functio

30 e obtained concerning demographics, arterial blood gas, Acute Physiology and Chronic Health Evaluatio

31 alveolar dead space fraction (first arterial blood gas after intubation) (per 0.1 unit increase: odds

32 ra vigilance should be applied in monitoring blood gases after delayed sternal closure to assess clin

33 phragm energy expenditure (effort), arterial blood gases, airway pressure, tidal volume and its coeff

34 nts, lung injury was assessed by analysis of blood gases, alveolar permeability, lung histology, AFC,

35 The results of arterial blood gas analyses at t = 4 minutes and t = 13 minutes (

36 Hemodynamic measurements and blood gas analyses were obtained using Swan-Ganz and art

37 In total, 295,079 arterial blood gas analyses, including the PaO2, between July 201

38 Arterial blood gas analysis (PaO2, PaCO2), peak airway pressure (

39 Gas exchange was assessed by arterial blood gas analysis after ventilation with each gas mixtu

40 Gas exchange was evaluated by blood gas analysis and multiple inert gas elimination te

41 24 hours prior to ICU arrival, and arterial blood gas analysis performed within 24 hours following I

42 arrest preceding PICU admission and arterial blood gas analysis taken within 1 hour of PICU admission

43 and compared with PaCO2 values when arterial blood gas analysis was performed.

44 ion, pulmonary function testing and arterial blood gas analysis, and echocardiographic, imaging, and

45 tal sinus vein for collection of samples for blood gas analysis.

46 may decrease the need for repeated arterial blood gas analysis.

47 re measured and blood samples were drawn for blood gas analysis.

48 nnula was taken every half-hour for arterial blood gas analysis.

49 e values determined by standard intermittent blood gas analysis.

50 re measured and blood samples were drawn for blood gas analysis.

51 son-Hasselbalch equation and reported on the blood gas analysis.

52 A total of 409 patients with arterial blood gases analyzed at least once and with a complete s

53 validated through concurrent, blind, ex situ blood gas analyzer (BGA) measurements.

54 As Na(+)(direct) measurements on a blood gas analyzer are not influenced by the total prote

55 arterial oxygen levels were measured with a blood gas analyzer.

56 s were analyzed in duplicate on two separate blood gas analyzers and CO-oximeters.

57 Blood gas analyzers and core laboratory chemistry analyz

58 However, as blood gas analyzers are not available at all clinical wa

59 The SenDx 100 portable blood gas and electrolyte analyzer is a simple and easy

60 body fluid homeostasis, indicated by similar blood gas and electrolyte concentrations in urine and bl

61 nd corresponding arterial and central venous blood gas and lactate measurements were made.

62 Concurrent arterial blood gas and lactate measurements were taken.

63 t maintain stable haemodynamics, have normal blood gas and oxygenation parameters and maintain patenc

64 ondary outcomes included changes in arterial blood gas and respiratory parameters, weaning duration,

65 Blood gas and tissue pH regulation depend on the ability

66 Rapid decreases in blood gases and a slower reduction in blood glucose sugg

67 Blood gases and biochemistry were monitored to assess or

68 Arterial blood gases and electrolytes were measured before and af

69 for continuous intravascular measurements of blood gases and electrolytes.

70 Arterial blood gases and hydrogen ion concentrations were measure

71 ized carboxyhemoglobin and improved arterial blood gases and intrapulmonary vasodilatation, reflectin

72 ET using cycle ergometry and ramp protocols; blood gases and lactate concentrations were measured eve

73 Consequences of CSA, including altered blood gases and neurohormonal activation, could result i

74 Arterial blood gases and pH were measured every 30 mins for the f

75 axic breathing pattern with markedly altered blood gases and pH, and pathological responses to challe

76 gy and other aspects of physiology including blood gases and respiration, the physiology and biomecha

77 positive study results with normal arterial blood gases and therefore do not fulfill criteria for HP

78 tter were studied for evaluation of arterial blood gases and validation of the grading method for pre

79 Seven days post-CBD, blood gases and ventilation in 21% O2 were normal, where

80 Arterial blood gases and ventilator settings were monitored every

81 ere not accompanied by marked alterations in blood gases and were abolished by vagotomy or atropine.

82 approximately 85% of maximum while arterial blood gases and work of breathing were assessed.

83 nert gas measurements) and 10 (hemodynamics, blood gases) and 20 (hemodynamics, blood gases, inert ga

84 entral venous pressure, heart rate, arterial blood gas, and pulse oximetric saturation were recorded.

85 Arterial blood gas, and systemic and pulmonary hemodynamics were

86 y function tests, Brasfield scores, arterial blood gases, and age were correlated with lung pathology

87 scles while monitoring respiration, arterial blood gases, and blood glucose in mice exposed to 8% O2

88 mpared with baseline in heart rate, arterial blood gases, and blood pressure, but serum nitrite conce

89 xercise testing with measurement of arterial blood gases, and bronchoalveolar lavage (BAL).

90 rial pressure, heart rate, systemic arterial blood gases, and cardiac output and index.

91 tion tests, including lung volumes, arterial blood gases, and chest radiographs were also monitored.

92 pulmonary functions, lung volumes, arterial blood gases, and chest radiographs were done daily.

93 sessed by pulmonary function tests, arterial blood gases, and chest X-rays, but the correlation with

94 Data on vital signs, electrolytes, arterial blood gases, and coagulation were collected before and a

95 Ventilator parameters, arterial blood gases, and derived oxygenation and ventilation ind

96 invasive monitoring to measure hemodynamics, blood gases, and gas exchange during exercise.

97 iographs, pulmonary function tests, arterial blood gases, and grading of pulmonary symptoms.

98 arterial pressure, cardiac output, arterial blood gases, and lactate were measured concurrently with

99 nography, pulmonary function tests, arterial blood gases, and left ventricular ejection fraction were

100 xide concentration in the exhaled gas (ENO), blood gases, and mean arterial pressure were measured ev

101 measurements, ventilatory settings, arterial blood gases, and methemoglobin were recorded at each stu

102 Pulmonary hemodynamics, arterial blood gases, and plasma concentrations of arachidonate m

103 Systemic and pulmonary hemodynamics, blood gases, and plasma nitrate were assessed.

104 n systemic and cerebral venous hemodynamics, blood gases, and prostanoid (prostaglandin E2, 6-ketopro

105 ng injury in terms of respiratory mechanics, blood gases, and pulmonary edema.

106 ro-oculography, minute ventilation, arterial blood gases, and serum theophylline levels.

107 gh (cough spikes), to arterialized capillary blood gases, and to inspiratory muscle strength.

108 therapists recorded demographic information, blood gases, and ventilator type and settings, and they

109 asurements of baseline vital signs, arterial blood gases, and ventilatory settings.

110 Arterial blood gases are critical in regulation of cerebral blood

111 indices of cerebral oxygenation to arterial blood gases are not well defined.

112 or the fetus, although the roles of arterial blood gases are recognized to be critical in the regulat

113 ary vasoconstriction may help to explain why blood gases are within physiologic ranges for a certain

114 Basal and hourly hemodynamics and blood gases (arterial and venous) under steady state res

115 Tonometric Pco2, arterial blood gases, arterial and portal venous lactates, and po

116 ther rat strains.We measured ventilation and blood gases at rest (eupnoea) and during hypoxia (FIO2 =

117 reduced as expected, whereas ventilation and blood gases at rest under normoxia were normal.

118 , excessive lung distension directly affects blood-gas barrier and lung vascular permeability.

119 a history suggestive of lung bleeding alters blood-gas barrier function resulting in higher concentra

120 The blood-gas barrier is able to maintain its extreme thinne

121 How the blood-gas barrier is regulated to be extremely thin but

122 The pulmonary blood-gas barrier needs to satisfy two conflicting requi

123 The strength of the blood-gas barrier on the thin side is attributable to th

124 apillary transfer is sensitive to changes in blood-gas barrier thickness of approximately 5 microm.

125 ibrosis, and edema, which cause an increased blood-gas barrier thickness, impair the efficiency of th

126 lmonary circulation and diffusion across the blood-gas barrier.

127 gs were collected (age, smoking history, and blood gas before lung harvesting).

128 ure, brain temperature, cerebral blood flow, blood gases, blood pressure, and pH.

129 ies have historically focused on normalizing blood gases but new research suggests that a higher PCO2

130 e, but no effects on pulmonary hemodynamics, blood gases, cardiac output, or lung water accumulation.

131 Prostacyclin does not mediate blood gas changes, alterations of pulmonary hemodynamics

132 We assessed the effects of blood gas changes, within the range encountered during m

133 minutes of each phase, we measured arterial blood gases, changes in end-expiratory lung volume of no

134 ith the baseline period, unadjusted arterial blood gas, chest radiograph, and RBC utilization in the

135 ider financial incentives targeting arterial blood gas, chest radiograph, and RBC utilization.

136 designed to decrease the avoidable arterial blood gases, chest radiographs, and RBC utilization on u

137 utcome was the number of orders for arterial blood gases, chest radiographs, and RBCs per patient.

138 score were noted before and during capillary blood gas collection to assess discomfort associated wit

139 ncremental cycle exercise test with arterial blood gas collection.

140 ssive cycle exercise test with capillary (c) blood gas collection.

141 scopy in neonates on intraoperative arterial blood gases, compared with open surgery.

142 measured in 13 subjects for eight different blood gas conditions, with the end-tidal partial pressur

143 Changes in blood gases conformed to predictions of a computer lung

144 well characterized because of challenges in blood gas control and limited availability of validated

145 tasets comprising hemodynamics, calorimetry, blood gases, cytokines, and cardiac and renal function w

146 We analyzed arterial blood gas data during 0 to 24 hours after the return of

147 th inhalation anesthesia, and improvement of blood gas data relative to spontaneous respiration.

148 r, in some practice settings, daily arterial blood gas data required to calculate the respiratory com

149 blood pressure monitoring and for assessing blood gas data.

150 Every blood gas derangement (hypoxemia, hyperoxemia, hypocapni

151 Findings suggest that individual blood gas derangements do not increase brain damage risk

152 for tracking unanticipated events related to blood gas deterioration.

153 Hemodynamics, arterial blood gas determination, alveolar permeability, wet-to-d

154 ially to 0.4+/-0.1 at the time of the second blood gas determination, thus permitting greater concent

155 se testing, including rest and peak exercise blood gas determinations, on 21 consecutive patients bef

156 A Student's t-test on pre- to post-blood gas differences showed a significantly lower PetCO

157 ratory rate, oxygen saturation, and arterial blood gases do not measure dyspnoea.

158 The effect of thoracoscopy on blood gases during repair of EA/TEF in neonates requires

159 found between NKCC2 +/+ and +/- mice in BP, blood gas, electrolytes, creatinine, plasma renin concen

160 utput by thermodilution, arterial and venous blood gases; electrolytes; lactate; base excess; oxygen

161 dicating that the altered fetal hormonal and blood gas environment around the spontaneous onset of la

162 Arterial blood gases estimated from the PET-based V(A)/Q distribu

163 o lung epithelial cells is essential for air-blood gas exchange.

164 We measured arterial blood gases, FRC, Rrs, and Crs in supine and prone posit

165 We studied 1,034 arterial blood gases from 703 patients; 650 arterial blood gases

166 erial blood samples taken for measurement of blood gases, glucose and lactate and plasma adrenaline,

167 en at appropriate intervals for biophysical (blood gases, glucose, lactate) and endocrine (catecholam

168 en at appropriate intervals for biophysical (blood gases, glucose, lactate) and endocrine (catecholam

169 Blood gas, hemodynamic, and gastric tonometric data were

170 Arterial and venous blood gases, hemodynamics, and pulmonary mechanics were

171 maintained alveolar ventilation and arterial blood gas homeostasis but at the expense of earlier dyna

172 tress in the dental office may help maintain blood gas homeostasis.

173 functional residual capacity increased, and blood gas improved until reaching the flat portion of th

174 Heart and respiratory rates and blood gases improved similarly for patients in both mask

175 Animals were ventilated to maintain arterial blood gases in a normal range (i.e., pH of 7.35 to 7.45,

176 formed direct field measurements of arterial blood gases in climbers breathing ambient air on Mount E

177 in MAP increased, despite similar changes in blood gases in response to umbilical cord occlusion, ove

178 e minimal level that would maintain arterial blood gases in the following ranges: pH 7.35-7.45, PaCO2

179 easurements were taken before (hemodynamics, blood gases, inert gas measurements) and 10 (hemodynamic

180 dynamics, blood gases) and 20 (hemodynamics, blood gases, inert gas measurements) minutes after induc

181 We analyzed matched blood samples for blood gas, inflammatory cytokine concentration, cystatin

182 the end of each phase, we measured arterial blood gases, inspiratory effort, and work of breathing b

183 animals in which no significant hemodynamic, blood gas, lactate, microcirculatory, and tissue Pco2 ab

184 l and is refractory to modest alterations of blood gas levels of CO2 and O2.

185 es included serial pulmonary function tests, blood gases, lung compliance, computed tomography (CT) i

186 Pulmonary and systemic hemodynamics, blood gases, lung pressures, subpleural blood flow (lase

187 ia after out-of-hospital cardiac arrest, two blood gas management strategies are used regarding the P

188 Measurement of blood gases, mean arterial blood pressure, functional ca

189 oxyglucose, tissue myeloperoxidase, arterial blood gases, mean arterial pressure, and lung tissue pro

190 for CPO, p =.0125), obtained fewer arterial blood gas measurements (2.7 +/- 1.2 for IPO vs. 4.1 +/-

191 ly ventilated patients may not have arterial blood gas measurements available at relevant timepoints.

192 Blood gas measurements were collected on healthy lifetim

193 Arterial blood gas measurements were performed immediately before

194 dings, all values from preoperative arterial blood gas measurements, and BAS procedure data.

195 Arterial blood gas measurements, intubation rate, days of mechani

196 Radial artery catheterization, arterial blood gas measurements, mechanical ventilation, vasopres

197 e was assessed from symptom scores, arterial blood gas measurements, pulmonary function testing, and

198 regard to oxygen weaning and use of arterial blood gas measurements.

199 CGRP antagonism did not alter basal arterial blood gas, metabolic, cardiovascular or endocrine status

200 Arterial samples were taken for blood gases, metabolic status and hormone analyses.

201 Perioperative POCT includes arterial blood gas monitoring, chemistry, co-oximetry panels, par

202 ry is increasingly substituting for arterial blood gas monitoring, noninvasive surrogate markers for

203 nitoring was done simultaneous with arterial blood gas monitoring.

204 in 30 minutes from LUS, using transcutaneous blood gas monitoring.

205 ed on the concurrent availability of routine blood gas Na(+)(direct) as well as core laboratory Na(+)

206 isolated remaining CB to maintain normal CB blood gases (normoxic, normocapnic perfusate), to inhibi

207 unit arrival, and postresuscitation arterial blood gas obtained.

208 lung disease by failure to maintain desired blood gases on the maximum ventilatory settings, 4 mL/kg

209 The CGRP antagonist did not alter basal blood gas or cardiovascular status in the fetus.

210 increases in cerebral blood flow, changes in blood gases or brain temperature, or rat strain; (3) the

211 eractions are mediated either via changes in blood gases or by brainstem neuronal connections, but th

212 sham CBD had no effect on resting breathing, blood gases or chemoreflexes (P >0.05).

213 anges in vital signs, electrolytes, arterial blood gases, or coagulation parameters.

214 sure monitoring, measurement of mixed venous blood gases, or monitoring of cardiac output by oxygen c

215 total of 70 simultaneous pulse oximeter and blood gas pair samples.

216 Each patient's initial pulse oximeter/blood gas pair was used in the statistical analysis.

217 gs, peak inspiratory pressures, and arterial blood gases (Pao2, Paco2, pH, and oxygen saturation).

218 Arterial blood gas parameters, clinical symptoms, health-related

219 Arterial blood gas parameters, serum electrolytes, and urine elec

220 EOV to invasive hemodynamic measurements and blood gases performed during exercise.

221 ated with NIPPV demonstrated higher arterial blood gas pH (p < .001), lower PaCO2 (p < .05), and a lo

222 re monitored continuously and fetal arterial blood gases, pH and metabolites were measured at predete

223 traction ratio, plasma lactate, hemoglobin), blood gases, pH, and hematocrit were made before fractur

224 tantially modify arterial pressure, arterial blood gases, pH, hematocrit, plasma glucose and rectal t

225 gs were adjusted to obtain standard arterial blood gases: pH of 7.35 to 7.45; PaCO2 of 35 to 40 torr

226 Arterial blood gases predicted from PET data correlated well with

227 Blood gas profiles were determined at each level of hype

228 Simultaneous blood gas, pulse oximetry, and ventilator settings were

229 xcluded from comparison, leaving 21 arterial blood gas/pulse oximeter pairs for analysis.

230 in the equations allows better prediction of blood gas reference values at sea level and at altitudes

231 tant role for the carotid bodies in eupnoeic blood gas regulation, (2) suggest that the carotid bodie

232 Cardiac nerve blockade exaggerated the fetal blood gas response to haemorrhage somewhat but did not s

233 Severity criteria often depend on arterial blood gas results.

234 Blood gas samples from the pulmonary artery and both int

235 Blood gas samples were drawn at 0, 4 and 13 minutes.

236 eplaced left carotid artery catheters, acute blood gas samples were taken 1 to 24 hours after gavage

237 However, chest radiography and arterial blood gas sampling seem useful while acute spirometry do

238 One thousand one hundred ninety blood gas, SpO2, and ventilator settings from 137 patien

239 moval, probes were tested in room air and in blood gas standard calibration solutions.

240 In the pilot study, left-over blood gas syringes were collected for further laboratory

241 judged by volume of colloid given, number of blood gases taken, and by measurement taken from cranial

242 dynamic variables, systemic and mixed venous blood gas tensions and oxygenation, arterial lactate con

243 vidence in humans that forcibly altering the blood gas tensions during repeated periods of exercise a

244 minimising any deviations from normal in the blood gas tensions, as sensed by the chemoreceptors.

245 Arterial blood gas tensions, hemodynamic variables, and the oxyge

246 Arterial blood gas testing, chest radiographs, and RBC transfusio

247 r the maintenance of physiological levels of blood gases through the regulation of breathing.

248 consistently <14 cm H2O with normal arterial blood gases throughout the observation period.

249 blood was removed at 0, 2, 4, and 5 hrs for blood gases, tumor necrosis factor (TNF)-alpha, nitric o

250 distress syndrome survival, laboratory use, blood gases use, radiograph use, and appropriate use of

251 s of variance using these same pre- and post blood gas values confirmed the significant decrease in P

252 The authors compared infants with blood gas values in the highest or lowest quintile for g

253 priori based on PaO(2) on the first arterial blood gas values obtained in the ICU.

254 LD-CBF, cortical tP(O2), and sagittal sinus blood gas values to P(a,O2).

255 After each experiment, blood gas values were measured, and retinas were isolate

256 tory parameters, hemodynamic parameters, and blood gas values were measured.

257 Arterial and venous blood gas values, glucose, and cardiac output were colle

258 volume aimed in part at normalizing arterial blood gas values.

259 cols designed to optimize lung inflation and blood gas values.

260 globin concentration, oxygen saturation, and blood gas values.

261 O2), heart rate, cardiac output and arterial blood gas variables at peak exercise on a cycle ergomete

262 Arterial blood gas variables included Pa(O(2)), Pa(CO(2)), pH, an

263 ures, cardiac output, urine output, arterial blood gases, ventilation:perfusion ratio (VA/Q), and hem

264 A baseline arterial blood gas was obtained on noninvasive passive therapy an

265 A baseline arterial blood gas was obtained on noninvasive therapy and 4 mins

266 on pressure support ventilation, an arterial blood gas was obtained, V(D)/V(T) was calculated, and th

267 A normal baseline for arterial blood gases was achieved by adjusting the inspiratory/ex

268 ND INTERVENTIONS: Whole-lung CT and arterial blood gases were acquired simultaneously in 77 patients

269 Before and after arterial blood gases were also obtained.

270 rterial oxygen content, Hb, lactate, pH, and blood gases were analyzed in blood samples.

271 blood gases from 703 patients; 650 arterial blood gases were associated with SpO2 less than or equal

272 phic and clinical data and room-air arterial blood gases were collected and analyzed.

273 -CPR for 15 minutes, and arterial and venous blood gases were collected at baseline and minutes 5, 10

274 nges in arterial blood pressure and arterial blood gases were comparable at any given level of inspir

275 Physiologic parameters and arterial blood gases were continuously monitored.

276 In 5 dogs, blood gases were determined at baseline and at 2 min of

277 Arterial blood gases were drawn at baseline (sea level and at alt

278 were drawn every 6 hrs for 72 hrs, arterial blood gases were drawn every 12 hrs for 72 hrs, and both

279 Arterial blood gases were drawn every 15 mins, and the ventilator

280 Cerebral venous blood gases were drawn from a jugular bulb venous cathet

281 90 mins, simultaneous arterial and capillary blood gases were drawn.

282 Arterial and deep venous blood gases were measured and oxygen consumption (VO2) w

283 iac outputs, filling pressures, and arterial blood gases were measured at 1-minute intervals during e

284 Arterial and mixed venous blood gases were measured at baseline, 1 min after cardi

285 nerve discharge, end-tidal CO2, and arterial blood gases were measured before during and after hypoxi

286 usion pressure, cardiac output, and arterial blood gases were measured before, 1 min after, and then

287 Oxygen delivery and blood gases were measured during both conditions.

288 Arterial blood gases were measured every 30 minutes intraoperativ

289 rial pressures, cardiac output, and arterial blood gases were measured following drug instillation.

290 Cardiopulmonary variables and blood gases were measured serially.

291 Arterial blood gases were measured, and ventilator settings were

292 Flows in these vessels and arterial blood gases were measured.

293 Respiratory pattern variables and capillary blood gases were not significantly modified between expe

294 At those settings, blood gases were pH 7.31 +/- 0.06, PaCO2 was 58 +/- 21 m

295 res, mean blood pressure, plasma glucose and blood gases were similar among groups.

296 with the nuclear lumen while respiratory and blood gases were stabilized.

297 rst minute of CPR, arterial and mixed venous blood gases were superior in the 3 experimental groups c

298 Blood gases were within acceptable limits during TLV.

299 Arterial blood gases were within the normal range and effective a

300 of ventilated TBI patients who had arterial blood gases within 24 h of admission to the ICU at 61 US