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1 n, even when grafted directly into the first branchial arch.
2 the same spatial location within the fourth branchial arch.
3 arise in soft tissues derived from the first branchial arch.
4 eveloping anterior pituitary gland and first branchial arch.
5 a specific rhombomere and its corresponding branchial arch.
6 icles, the pharyngeal endoderm and the first branchial arch.
7 neural crest cells migrating into the third branchial arch.
8 but not mesenchymal expression in the first branchial arch.
9 oid arch take on the properties of the first branchial arch.
10 lates Msx1 expression in the growing, distal branchial arch.
11 interact with Hoxa-2 and Dlx-2 in the second branchial arch.
12 the epithelium of the non-odontogenic second branchial arch.
13 cells from r7 are observed within the third branchial arch.
14 rate along normal pathways toward the second branchial arch.
15 crest cells migrate primarily to the fourth branchial arch.
16 the depleted hindbrain region and the third branchial arch.
17 a, but not by neural crest within the second branchial arch.
18 ut not in the maxillary process of the first branchial arch.
19 chial arch, but not in the limb or the first branchial arch.
20 mb bud or maintenance in the first or second branchial arch.
21 nto a distal-to-proximal invasion of the 2nd branchial arch.
22 downstream genes in each domain of the first branchial arch.
23 and proximal/distal domains within the first branchial arch.
24 NC cells in the proximal domain of the first branchial arch.
25 Tcf21, and Msc in the first, but not second, branchial arch.
26 ade the branchial arches, especially the 2nd branchial arch.
27 itx2 mutant descendents moved into the first branchial arch.
28 y pit, and mandibular component of the first branchial arch.
29 tube and navigate over long distances to the branchial arches.
30 on, are all active in the oral epithelium or branchial arches.
31 ls and are derived from the first and second branchial arches.
32 and is required for dHAND expression in the branchial arches.
33 d hyomandibular and reduced third and fourth branchial arches.
34 control expression of dHAND in the heart and branchial arches.
35 also causes apoptosis of neural crest in the branchial arches.
36 ral crest cell migration into the developing branchial arches.
37 with defects of NCCs from the first to third branchial arches.
38 y elements that control transcription in the branchial arches.
39 of the neural tube, and neural crest derived branchial arches.
40 anglia, to their targets, the muscles of the branchial arches.
41 ndbrain neural crest cell migration into the branchial arches.
42 ence at the dorsal midline to entry into the branchial arches.
43 t cell migration streams bound for different branchial arches.
44 d close to the top of the clefts between the branchial arches.
45 ain, eyes, somites, ventral blood island and branchial arches.
46 m cells migrate to fill the core of specific branchial arches.
47 ld faster doubling time after populating the branchial arches.
48 er it simply populates the nearest available branchial arches.
49 nes regulate proximodistal patterning of the branchial arches.
50 ximal regions of the murine first and second branchial arches.
51 ell populations and targets them to specific branchial arches.
52 neurons, in the genital tubercle, and in the branchial arches.
53 cations, with at least five operating in the branchial arches.
54 embryonic vasculature and development of the branchial arches.
55 ugh different microenvironments and into the branchial arches.
56 ry routes to precise targets in the head and branchial arches.
57 ding open neural tubes and reductions in the branchial arches.
58 exploit neural crest streams to populate the branchial arches.
59 stinct functions in mandibular versus caudal branchial arches.
60 d expression pattern in developing visceral (branchial) arches.
61 laterally from their hyoid and gill-bearing (branchial) arches.
62 the rhombomere 4 (r4) migratory stream into branchial arch 2 (ba2), is due to chemoattraction throug
63 or absence of neural crest cells traversing branchial arches 3, 4 and 6, and entering the cardiac ou
64 e traced from the occipital neural tube, via branchial arches 3, 4 and 6, into the aortic sac and aor
65 apped binding of Meis, Pbx, and Hoxa2 in the branchial arches, a series of segments in the developing
66 signals in the vicinity of the hindbrain and branchial arches act on migrating myogenic cells to infl
67 gh muscles derived from the first and second branchial arches also share a clonal relationship with d
69 reduction of Hoxa-3 expression in the third branchial arch and corresponding deficits in third arch-
73 ts including cleft face (involving the first branchial arch and frontonasal processes), incomplete he
75 ral crest cells that migrate into the second branchial arch and is essential for proper patterning of
76 within the mandibular component of the first branchial arch and later in the primordia of middle ear-
81 presumptive secondary heart field within the branchial arch and splanchnic mesoderm that contributes
82 sumptive secondary heart field, derived from branchial arch and splanchnic mesoderm, patterns the for
87 expression in epaxial and hypaxial somites, branchial arches and central nervous system, and argued
88 sively expressed in cranial placodes, in the branchial arches and CNS and in complementary or overlap
90 is expressed in the epithelial layer of the branchial arches and encodes for the endothelin-1 (ET-1)
91 in the ectodermal surfaces of the limb buds, branchial arches and epidermal appendages, which are all
93 radation-1-like gene, expressed in embryonic branchial arches and in the conotruncus, appears to play
96 erted from their migration pathways into the branchial arches and instead migrated around the otic ve
97 ression disrupts morphogenesis of the caudal branchial arches and leads to a failure to correctly ela
99 e skeletal muscle precursors of the myotome, branchial arches and limbs as well as in the developing
100 pressed in midgestation mouse embryos in the branchial arches and limbs, consistent with the human ph
103 rk sheds light on the homology of vertebrate branchial arches and supports their common origin at the
104 2 isoforms have interchangeable functions in branchial arches and that Pitx2 target pathways respond
105 l elements derived from the first and second branchial arches and that there are heterogeneous popula
108 os exhibited an increased number of somites, branchial arches and the presence of forelimb buds; howe
109 l crest cells are directed in streams to the branchial arches and to the forelimb of the developing q
110 enotypes that are both more severe (head and branchial arches) and less severe (allantois growth) tha
112 res 4 and 5, the selective loss of the 2(nd) branchial arch, and the loss of most, but not all, 2(nd)
113 and the mandible bone derive from the first branchial arch, and their development depends upon the c
114 i3 mutants are able to migrate, populate the branchial arches, and display some elements of correct p
115 nt to the even-numbered rhombomeres into the branchial arches, and each stream contains contributions
116 In the olfactory pathway, as in the limbs, branchial arches, and heart, mesenchymal/epithelial indu
118 dition, foxi1 is expressed in the developing branchial arches, and jaw formation is disrupted in hear
119 notochord, somites, heart, pronephric ducts, branchial arches, and jaw muscles in embryos and larvae.
123 ofound abnormalities in the first and second branchial arches, and the early remodeling of blood vess
124 and Hand2 transcription factor genes in the branchial arches, and we identify a branchial arch-speci
125 n factors required to initiate myogenesis in branchial arch- and somite-derived muscles are known, bu
126 f the neural-crest-derived components of the branchial arch are expressed in dHAND-null embryos, sugg
127 es and of the maxillary process of the first branchial arch are integral to lip and primary palate de
129 erge from the neural tube or en route to the branchial arches, areas where cell-cell interactions typ
131 ilure to establish the initial complement of branchial arch arteries in the caudal pharyngeal region.
132 -31, shows that the initial formation of the branchial arch arteries is not disturbed in ETA-/- or EC
133 cts that includes abnormal patterning of the branchial arch arteries, double-outlet right ventricle,
134 rentiation of smooth muscle cells within the branchial arch arteries, which are derived from the neur
139 allele resulted in abnormal morphogenesis of branchial-arch arteries (BAAs) and defective OFT septati
140 n mice impairs asymmetric remodelling of the branchial arch artery (BAA) system, resulting in randomi
141 ayed micropthalmia and the loss of the first branchial arch, as detected by the expression of pax-6,
142 itiation of MyoD expression in limb buds and branchial arches, as enhancer deletion delayed MyoD acti
143 prior to fusion, and also in the developing branchial arches at the times of critical morphogenetic
146 yclopia, as well as alterations in the first branchial arch (BA1) leading to lack of jaw (agnathia).
151 undetectable in the mesenchyme of dHAND-null branchial arches but unaffected in the limb bud, consist
152 ndbrain and proximal mesenchyme of the first branchial arch, but did not involve loss of expression o
153 t signaling in the epaxial somite and second branchial arch, but not in the limb or the first branchi
154 organisation of structures derived from the branchial arches, but that exposure to increasingly nove
155 the development of the embryonic pharyngeal (branchial) arches, but its effects on innervation of the
156 2 is also expressed in Xenopus neural folds, branchial arches, cardiac outflow tract, inflow tract, a
157 y fail to form neurons in cranial ganglia or branchial arch cartilage, illustrating that they are at
160 of Ph/Ph cells to PDGF-A treatment of normal branchial arch cells in vitro with recombinant PDGF-AA s
161 it would in normal development, resulting in branchial arches containing mixed cell populations not o
164 disorder characterized by the association of branchial arch defects, hearing loss, and renal anomalie
165 , including external ear anomalies, abnormal branchial arch derivatives, heart malformations, diaphra
166 while partial disruption leads to defects of branchial arch derivatives, similar to those seen in CAT
169 ssing their differentiation, while a further branchial arch derived signal, namely Bmp7, is an overal
171 ET-1/ECE-1/ETA pathway results in defects in branchial arch- derived craniofacial tissues, as well as
172 s of neural crest-derived tissues, including branchial arch-derived craniofacial structures, aortic a
173 utant mice are not viable and display severe branchial arch-derived facial skeleton defects, includin
175 stablishing a baseline molecular bauplan for branchial arch-derived jaw development and further valid
177 ssed by myogenic cells that migrate into the branchial arches despite its expression in migrating pre
178 in derivatives of neurogenic placodes and in branchial arches, despite the fact that cephalochordates
179 In mouse embryos, mutation of Prdm1 affects branchial arch development and leads to persistent trunc
180 To determine the potential role of dHAND in branchial arch development and to assess the role of the
181 1 reveals that it is essential for posterior branchial arch development, although the mandibular arch
188 te host peripheral structures, including the branchial arches, dorsal root and sympathetic ganglia.
189 expressed in the differentiating somites and branchial arches during embryogenesis and is skeletal mu
191 later in development and is characterized by branchial arch dysplasia and aberrant segmental boundari
192 elates with a delay in expression of Fgf8 in branchial arch ectoderm and a failure of neural crest ce
196 polarity is first established in mandibular branchial arch ectomesenchymal cells by a signal, Fgf-8,
197 riants most likely perturb the patterning of branchial arches, either through excessive activity (und
199 Endothelin-1 (ET-1) signaling regulates the branchial arch enhancer and is required for dHAND expres
205 neural crest cells fail to fully invade the branchial arches, especially the 2nd branchial arch.
207 ealed that geniculate axons were repelled by branchial arch explants that were previously shown to be
208 mesenchymal cells, cell migration from Ph/Ph branchial arch explants was compared to migration from n
209 e Hand2 allele or deleting the ventrolateral branchial arch expression of Hand2 led to a novel phenot
211 comparing protein binding to these sites in branchial arch extracts from endothelin receptor A (Ednr
212 r element that drives Myf5 expression in the branchial arches from 9.5 days post-coitum and show that
213 lar pathway results in growth failure of the branchial arches from apoptosis, while partial disruptio
217 that HAND2 is involved in development of the branchial arches, heart, limb, vasculature, and nervous
218 s, including the craniofacial skeleton, ear, branchial arches, heart, lungs, diaphragm, gut, kidneys,
220 there is a tight link between the resulting branchial arch Hox code and a particular skeletal morpho
221 resulting in the persistence of an abnormal branchial arch Hox code and extensive defects in the hyo
225 ventolateral regions of the first and second branchial arches in these mutant mice, but expression wa
226 and defects in the derivatives of the first branchial arch, including cleft palate, suggesting a pro
227 T-1) mutant embryos, dHAND expression in the branchial arches is down-regulated, implicating it as a
228 reveals that crest does not migrate into the branchial arch it would have colonised in normal develop
230 Overexpression of Hoxa2 in the chick first branchial arch leads to a transformation of first arch c
231 hed during development and patterning of the branchial arches may set up signals that the neural plat
232 ocyte Growth Factor, although a component of branchial arch-mediated growth promotion and chemoattrac
235 c sac mesothelium and in left splanchnic and branchial arch mesoderm near the junction of the aortic
238 ecule is expressed in ventral splanchnic and branchial-arch mesoderm and outflow-tract (OFT) myocardi
241 rs Myf5, MyoD and myogenin were expressed in branchial arch muscle, but at comparatively late stages
242 ted to the lateral rectus and proximal first branchial arch muscles; many also contributed to the dor
243 ing has been implicated in somite, limb, and branchial arch myogenesis but the mechanisms and roles a
244 llary and mandibular components of the first branchial arch, nasal processes, eyelid, midbrain, medul
253 shown to function as brain, olfactory bulb, branchial arch, otic vesicle and fin enhancers, recapitu
254 showed comparable expression patterns in the branchial arch, otic vesicle, forebrain and/or limb at e
255 the mesenchyme and endothelial cells of the branchial arches, outflow tract, and heart suggest that
256 mus alters Hoxa2 expression and consequently branchial arch patterning, demonstrating that neural cre
257 heir nested expression patterns in the first branchial arch (primordium for jaw) and mutant phenotype
258 -less gene family, is expressed in the first branchial arch, prior to the initiation of tooth develop
259 erentiation of the cartilage elements in the branchial arches, rather than during crest migration, im
261 supports internal to the gills--the visceral branchial arches--represents one of the key events in ea
262 position of origin after migration into the branchial arches, resulting in skeletal abnormalities.
263 so lacked the early defects in growth of the branchial arches seen in Hand2 null embryos and survived
264 es mix along migration routes and within the branchial arches, separate groups of premigratory neural
267 mal lineage, including craniofacial regions, branchial arches, somites, vibrissal and hair follicles,
268 late mesoderm cells did not migrate into the branchial arches, somitic cells were capable of migratin
270 s in the branchial arches, and we identify a branchial arch-specific enhancer in the Dlx5/6 locus, wh
271 onistic signals from the neural tube and the branchial arches specify extraocular versus branchiomeri
272 reduction in head size after 1 day, loss of branchial arch structures after 2 days, and embryos with
275 d defective angiogenesis of the yolk sac and branchial arches, stunted development and embryo wasting
276 s from adjoining rhombomeres within a single branchial arch support the notion that the pattern of hi
277 ed region contributes to a greater number of branchial arches than it would in normal development, re
278 ebrate face develop from transient embryonic branchial arches that are populated by cranial neural cr
279 he establishment of signaling centers in the branchial arches that are required for neural crest surv
280 re heterogeneous populations of cells in the branchial arches that rely on different cis-regulatory e
281 the chondrogenic regions of first and second branchial arches, the appendicular skeleton, and the der
282 f the somites, the ventromedial pathway, the branchial arches, the gut, the sensory ganglia, and the
283 a from both human fetal ear and mouse second branchial arch tissue confirmed that genes located among
285 d from the neural tube that migrate into the branchial arches to generate the distinctive bone, conne
287 mbryo, including paraxial mesoderm, somites, branchial arches, vibrissae, developing central nervous
288 tic activity during NC cell migration to the branchial arches was altered when premigratory cells wer
289 al cues in driving neural crest cells to the branchial arches, we isochronically transplanted small s
291 ent, whereas muscles derived from the second branchial arch were merely distorted in Pitx2 mutants at
292 ll-derived skeletal structures of the second branchial arch were morphologically transformed into ele
293 om locations close to the hindbrain into the branchial arches where they undergo muscle differentiati
294 migrate from hindbrain segments to specific branchial arches, where they differentiate into distinct
295 d crest-derived prechondrocytes in posterior branchial arches, whereas a third paralogue is expressed
296 xpressed in the distal (ventral) zone of the branchial arches, whereas the Hand2 expression domain ex
297 C development in the third, fourth and sixth branchial arches, while the bone malformations present i
299 maintain a spatially-ordered invasion of the branchial arches with differences in cell proliferation
300 in the head, optic vesicles, spinal cord and branchial arches with weaker expression in the somites.
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