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1 ue from 62 men (46 suicide completers and 16 comparison subjects).
2 endent ADHD sample (452 case subjects, 5,081 comparison subjects).
3 MDD in childhood or adolescence (psychiatric comparison subjects).
4 compared with either psychiatric or noncase comparison subjects.
5 challenge in the dmPFC for the patients and comparison subjects.
6 ents, 19 clinical high-risk patients, and 65 comparison subjects.
7 rontal cortex from schizophrenia and matched comparison subjects.
8 ltreated individuals relative to age-matched comparison subjects.
9 rease was smaller from load N2 to N3 than in comparison subjects.
10 nd they selected 36,588 age- and sex-matched comparison subjects.
11 ) had normal gray matter volumes relative to comparison subjects.
12 ation medication treatments), and 16 healthy comparison subjects.
13 over the whole brain in patients relative to comparison subjects.
14 atched pairs of schizophrenia and unaffected comparison subjects.
15 as 1.67 (95% CI, 1.30-2.17; P<0.001) that of comparison subjects.
16 zed to the frontotemporal cortex relative to comparison subjects.
17 r patients, unaffected siblings, and healthy comparison subjects.
18 s showing greater prefrontal activation than comparison subjects.
19 these associations were observed in healthy comparison subjects.
20 bling pairs), and 46 demographically matched comparison subjects.
21 r, their first-degree relatives, and healthy comparison subjects.
22 hinner in the depressed patients than in the comparison subjects.
23 ed cognitions with those reported by axis II comparison subjects.
24 ared with schizophrenia patients and healthy comparison subjects.
25 ivation within the default-mode network than comparison subjects.
26 lls in 36 matched pairs of SZ and unaffected comparison subjects.
27 gray matter volumes that did not differ from comparison subjects.
28 atients with schizophrenia and 2,342 healthy comparison subjects.
29 e in patients with schizophrenia and healthy comparison subjects.
30 ation medication treatments), and 16 healthy comparison subjects.
31 sant-naive depressed adolescents and healthy comparison subjects.
32 refrontal cortex (BA 46) relative to healthy comparison subjects.
33 rom 14 depressed subjects and 14 age-matched comparison subjects.
34 caine-dependent subjects and matched healthy comparison subjects.
35 and 20 age- and sex-matched healthy sleeper comparison subjects.
36 moderate Alzheimer's disease, and 70 healthy comparison subjects.
37 as compared with the repeat scans of healthy comparison subjects.
38 udied a primary sample of 92 patients and 73 comparison subjects.
39 quencies in 727 children with ADHD and 5,081 comparison subjects.
40 well as symptomatic remission) than axis II comparison subjects.
41 67 (GAD67) in schizophrenia subjects but not comparison subjects.
42 adults with bipolar disorder and in healthy comparison subjects.
43 patients with schizophrenia and 422 healthy comparison subjects.
44 observed to have larger brains than healthy comparison subjects.
45 d) and 23 age- and gender-matched nonsmoking comparison subjects.
46 n with TOF compared with normally developing comparison subjects.
47 op-signal reaction times relative to healthy comparison subjects.
48 second included 22 relatives and 22 matched comparison subjects.
49 th major depression relative to nondepressed comparison subjects.
50 cocaine-dependent women and men relative to comparison subjects.
51 rea during successful inhibition relative to comparison subjects.
52 matched comparison subjects, and 188 general comparison subjects.
53 proportionately worsened relative to that of comparison subjects.
54 essive disorder and age-matched nondepressed comparison subjects.
55 nificantly more common in case subjects than comparison subjects.
56 y in the patients was similar to that of the comparison subjects.
57 tending into the insula, relative to healthy comparison subjects.
58 f 1,006 alcohol-dependent patients and 1,178 comparison subjects.
59 renia but improved performance among healthy comparison subjects.
60 r nonpsychotic healthy siblings, and healthy comparison subjects.
61 ressed patients relative to the nondepressed comparison subjects.
62 es between nonpsychotic siblings and healthy comparison subjects.
63 olescents with bulimia nervosa as in healthy comparison subjects.
64 ssion (unmedicated) and nondepressed elderly comparison subjects.
65 difference between ADHD patients and healthy comparison subjects.
66 cted in bipolar patients relative to healthy comparison subjects.
67 chizophrenia subjects and matched unaffected comparison subjects.
68 igh-risk neonates were similar to the female comparison subjects.
69 significantly more embarrassing than did the comparison subjects.
70 compared with none of 4,155 chromosomes from comparison subjects.
71 eat responses during retaliation relative to comparison subjects.
72 onpsychotic major depression, and 19 healthy comparison subjects.
73 the pulvinar in youths with ADHD relative to comparison subjects.
74 ely, from matched pairs of schizophrenia and comparison subjects.
75 in the DLPFC from schizophrenia and matched comparison subjects.
76 zoaffective disorder patients and 66 healthy comparison subjects.
77 izoaffective disorder and 62 matched healthy comparison subjects.
78 postpartum psychosis and matched postpartum comparison subjects.
79 and clinical depression relative to healthy comparison subjects.
80 ariability was higher in both groups than in comparison subjects.
81 between levels of participants with ADHD and comparison subjects.
82 n patients with schizophrenia and 22 healthy comparison subjects.
83 izophrenia relative to their matched healthy comparison subjects.
84 on were contrasted against 74 never-affected comparison subjects.
85 families with schizophrenia and 249 healthy comparison subjects.
86 25 with unipolar depression, and 37 healthy comparison subjects.
87 schizoaffective disorder and matched normal comparison subjects.
88 ume and amyloid uptake in either patients or comparison subjects.
89 ipolar disorder case subjects and 13 matched comparison subjects.
90 ect-related negativity compared with healthy comparison subjects.
91 lar AMD was 2.57 (95% CI: 1.42-4.64) that of comparison subjects.
92 44 age- and IQ-matched typically developing comparison subjects.
93 olumes in maltreated individuals relative to comparison subjects.
94 at rest was observed in patients relative to comparison subjects.
95 nnectivity in 44 OCD patients and 43 healthy comparison subjects.
96 a or schizoaffective disorder and 31 healthy comparison subjects.
97 th alcohol dependence and 21 matched healthy comparison subjects.
98 g in patients and to schizotypal syndrome in comparison subjects.
102 25 adults and 14 adolescents) and 39 healthy comparison subjects (23 adults and 16 adolescents) match
103 methylated in suicide completers relative to comparison subjects (273 hypermethylated and 93 hypometh
104 =369; first-degree relatives=1072; community comparison subjects=526) in the Consortium on the Geneti
105 ADHD, 78 with subthreshold ADHD, 332 healthy comparison subjects; 55.8% male; average age: 17.0 years
106 a history of childhood maltreatment and 362 comparison subjects (56 children/adolescents and 306 adu
107 derline patients: 78%-99%; range for axis II comparison subjects: 97%-99%) but not recovery (40%-60%
110 S (this infant had also served as an overlap comparison subject and both specimens contained provisio
111 schizophrenia was slower than in any healthy comparison subject and correlated with both positive Pos
113 cond sample with similar frequencies: 46% in comparison subjects and 36% in schizophrenia patients (p
114 tivity in schizophrenia patients and healthy comparison subjects and analyzed the relationship betwee
115 ion in 22 matched pairs of schizophrenia and comparison subjects and by microarray analyses of pooled
116 sorder and schizophrenia relative to healthy comparison subjects and identified putative expression q
117 ex area 9 of 57 schizophrenia and 57 healthy comparison subjects and in antipsychotic-exposed monkeys
118 es specific to sad faces relative to healthy comparison subjects and nonmedicated patients in stable
119 oid patient volunteers compared with healthy comparison subjects and nonparanoid patient volunteers.
120 ectivity during response preparation between comparison subjects and probands and between individuals
121 s with comorbid conduct disorder relative to comparison subjects and relative to those without comorb
123 sion (effect size=1.24) than did the healthy comparison subjects and showed a positive correlation be
124 -compulsive disorder patients, compared with comparison subjects and siblings, showed increased error
125 ection from 19 matched tetrads of unaffected comparison subjects and subjects with SZ, bipolar disord
127 en large samples of OCD patients and healthy comparison subjects and their relation with demographic
128 90 individuals (479 patients and 211 healthy comparison subjects) and adjusted for a range of confoun
129 large duplications in case subjects (none in comparison subjects), and SNPs in the gene had a gene-wi
130 cation sample of 36 patients, 31 age-matched comparison subjects, and 188 general comparison subjects
131 habituation in borderline patients, healthy comparison subjects, and a psychopathological comparison
132 r disorder did not differ significantly from comparison subjects, and both groups performed better th
133 t size: publication year, IQ difference from comparison subjects, and chlorpromazine equivalent daily
134 2 matched pairs of schizophrenia and healthy comparison subjects, and in situ hybridization to assess
135 oss all brain regions than in the respective comparison subjects, and it was lower in the acutely ill
136 hed pairs of subjects with schizophrenia and comparison subjects, and levels of CDC42-PAK-LIMK pathwa
137 e responses was impaired relative to healthy comparison subjects, and patients were more prone to sli
138 19 euthymic bipolar patients and 19 matched comparison subjects, and the second included 22 relative
139 ses relative to their respective age-matched comparison subjects, and there was no evidence of age-re
140 ry cortex of 20 schizophrenia and 20 matched comparison subjects as well as aberrant voltage-gated ca
141 rticipants (51 with Tourette syndrome and 69 comparison subjects) as they either blinked normally or
142 vious history to a higher degree relative to comparison subjects, basing their choice largely on rewa
143 otal hippocampal volume between patients and comparison subjects, but there were no group differences
144 irst compared between depressed patients and comparison subjects by subdivision-wise classical morpho
147 high-risk participants compared with matched comparison subjects (Cohen's d >1.2) and was positively
149 tpatients with schizophrenia, and 36 healthy comparison subjects completed a range of social (facial
151 timulant users (n = 161) and stimulant-naive comparison subjects (CTL) (n = 48) performed a Paper Sci
152 ntly taking antidepressants and nondepressed comparison subjects demonstrated greater total gaze dura
153 adults with OCD and 33 healthy, age-matched comparison subjects during a reward-based learning task
154 2 patients with schizophrenia and 41 healthy comparison subjects during a standard auditory paired-st
155 gnoses with the abused group, and 27 healthy comparison subjects during an individually adjusted trac
156 uals with Tourette's syndrome and 21 healthy comparison subjects during spontaneous or simulated tics
157 of 23 schizophrenia patients and 24 healthy comparison subjects during trials in which they viewed a
163 heir first-degree relatives, and 459 healthy comparison subjects for clinical characterization and de
164 most common haplotype was overrepresented in comparison subjects (frequency, 47%) relative to schizop
165 dditional 2,242 ADHD case subjects and 8,552 comparison subjects from four independent cohorts from t
166 ion (n=73), and schizophrenia (n=56) and 110 comparison subjects from our discovery study who were ge
169 ith generalized social phobia and 16 healthy comparison subjects group-matched on age, gender, and IQ
170 generalized anxiety disorder and 32 healthy comparison subjects group-matched on IQ, gender, and age
171 generalized anxiety disorder and 32 healthy comparison subjects group-matched on IQ, gender, and age
174 gated whether depressed patients and healthy comparison subjects have differences in cortical thickne
175 d bipolar disorder (BP group) and 34 healthy comparison subjects (HC group) received an fMRI scan whi
177 as carried out to determine whether case and comparison subjects in 16 Psychiatric GWAS Consortium (P
178 sed in 20 pairs of schizophrenia and matched comparison subjects in an initial and replication cohort
179 A107 BPND between schizophrenia patients and comparison subjects in any of the brain regions studied
180 nts with borderline personality disorder and comparison subjects in baseline in vivo mu-opioid recept
182 rated brain activation compared with healthy comparison subjects in multiple regions, including the f
183 showed lower fractional anisotropy than the comparison subjects in multiple white matter regions; di
184 tently less accurate and slower than healthy comparison subjects in semantic decisions in which words
185 A methylation between suicide completers and comparison subjects in specific genes have been associat
187 neutral facial expressions than did healthy comparison subjects in the amygdala, orbitofrontal corte
190 tly lower in the cocaine abusers than in the comparison subjects in the limbic striatum (10.0% lower)
191 ere significantly different between case and comparison subjects in the PGC data set; for MGS subject
192 s exhibited less cue-related activation than comparison subjects in the thalamus, anterior cingulate
193 cted relatives did not differ from community comparison subjects in their neurocognitive performance.
194 was 60% steeper in schizophrenia relative to comparison subjects, indicating a significantly faster r
196 Thirty-seven OCD patients and 33 healthy comparison subjects learned to avoid shocks while underg
198 ed to severe childhood abuse, 17 psychiatric comparison subjects matched for psychiatric diagnoses wi
199 ive psychoses made between 1987 and 2003 and comparison subjects matched on sex, date of birth, birth
205 9) and age-, gender-, and IQ-matched healthy comparison subjects (N=173) were included in a quantitat
206 frequency) in children with ADHD (N=896) and comparison subjects (N=2,455) from the IMAGE II Consorti
210 a (n=31) and age-, smoking-, and sex-matched comparison subjects (n=31) participated in one [123I]5-I
211 ssment of At-Risk Mental States, and healthy comparison subjects (N=38) 14 to 29 years of age underwe
213 lings (N=78, 172 scans), and matched healthy comparison subjects (N=79, 198 scans) between the ages o
216 .e., profiles) that were similar to those of comparison subjects on both social and nonsocial cogniti
217 had significantly lower scores than axis II comparison subjects on one mature defense mechanism (sup
218 ., growth that is slower relative to healthy comparison subjects) on tests indexing processing speed,
222 total of 21 patients with OCD and 30 healthy comparison subjects participated in a set of tasks desig
225 ia nervosa, restricting type; and 13 healthy comparison subjects performed a rapid, jittered event-re
226 17 of their siblings, and 37 matched healthy comparison subjects performed a stop-signal task during
227 utpatients with schizophrenia and 23 healthy comparison subjects performed a visual attention task in
228 ents, 17 unaffected siblings, and 37 matched comparison subjects performed a visuospatial n-back task
229 icated schizophrenia patients and 16 healthy comparison subjects performed an action imitation/observ
230 bstance-dependent individuals and 30 healthy comparison subjects played a modified version of the Iow
232 rates among borderline patients than axis II comparison subjects (recurrence: 10%-36% compared with 4
233 right thalamus and brainstem was greater in comparison subjects relative to probands, and cue-relate
234 issl-stained sections from schizophrenia and comparison subjects revealed IFITM mRNA expression in pi
235 as time interval between two fingerprints in comparison, subject's age, and fingerprint image quality
236 r disorders and psychopathic traits, healthy comparison subjects showed a significantly greater incre
238 isorder patients, compared with siblings and comparison subjects, showed increased task-related funct
239 to conflict differently from that of healthy comparison subjects, specifically in frontal regions.
242 ity and interregional causality than healthy comparison subjects throughout all portions of the motor
244 subjects with major depressive disorder and comparison subjects (total subjects, N=102; 49% were wom
245 ETHOD: In matched pairs of schizophrenia and comparison subjects, transcript levels of ARP2/3 complex
247 ed, and 24 were in remission) and 54 healthy comparison subjects underwent an indirect face-emotion p
249 ophrenia patients and 28 age-matched healthy comparison subjects underwent functional "resting state"
250 enia/schizoaffective patients and 13 healthy comparison subjects underwent functional magnetic resona
251 depressed bipolar II patients and 21 healthy comparison subjects underwent functional MRI (fMRI) whil
252 generalized anxiety disorder and 24 healthy comparison subjects underwent functional MRI while perfo
254 nt psychiatric comorbidities, and 12 healthy comparison subjects underwent MRI with a 3-T system.
257 hizophrenia patients and 100 matched healthy comparison subjects underwent structural and resting-sta
259 chronic schizophrenia and 12 matched healthy comparison subjects using [(11)C]IMA107 positron emissio
260 frontal pole between depressed patients and comparison subjects using both uni- and multivariate sta
261 bjects with schizophrenia and nonpsychiatric comparison subjects was analyzed using Western blotting
262 children, hypoactivation in ADHD relative to comparison subjects was observed mostly in systems invol
263 nificant hyperactivation in ADHD relative to comparison subjects was observed predominantly in the de
264 me in alcohol-dependent patients relative to comparison subjects was seen in three regions: the media
265 rderline personality disorder and 72 axis II comparison subjects were assessed during their index adm
267 the 90th percentile of levels observed among comparison subjects were associated with nonaffective ps
268 schizoaffective disorder and 3,611 screened comparison subjects were available for analysis of rare
269 activation between individuals with ADHD and comparison subjects were detected using activation likel
270 hildhood (probands) and 32 carefully matched comparison subjects were followed longitudinally and sca
272 risk for psychosis and a group of 80 healthy comparison subjects were identified and recruited for re
273 s from 20 matched pairs of schizophrenia and comparison subjects were immunolabeled for the vesicular
274 atched pairs of schizophrenia and unaffected comparison subjects were immunolabeled for vGAT, GAD67,
276 pendent patients in treatment and 50 healthy comparison subjects were scanned once using high-resolut
277 who converted to psychosis, and 135 healthy comparison subjects were scanned with magnetic resonance
278 xposure and response prevention, and healthy comparison subjects were tested after a comparable time
280 ents (290 borderline patients and 72 axis II comparison subjects) were assessed at study entry using
282 mong psychotic probands, compared to healthy comparison subjects, were progressively greater from bip
283 ve to both nonpsychotic siblings and healthy comparison subjects, whereas there were no significant v
284 recorded from 45 OCD patients and 39 healthy comparison subjects while performing a flanker task.
285 rospectively confirmed PMDD and asymptomatic comparison subjects while they completed the n-back task
286 urce, state, sex, and age to as many as four comparison subjects who did not use methylphenidate, amp
287 x area 9 of 42 schizophrenia subjects and 42 comparison subjects who had no psychiatric diagnoses in
288 dults and 52 age- and gender-matched healthy comparison subjects who underwent structural MRI, [(18)F
289 dults and 52 age- and gender-matched healthy comparison subjects who underwent structural MRI, [(18)F
290 n participants with PTSD relative to healthy comparison subjects who were either exposed or not expos
292 for more than one adult disorder relative to comparison subjects with no history of childhood psychia
293 from subjects with schizophrenia and matched comparison subjects with no known history of psychiatric
294 with borderline personality disorder and 72 comparison subjects with other axis II disorders were as
295 nts with borderline personality disorder and comparison subjects with other personality disorders and
297 mes higher in ADHD case subjects relative to comparison subjects, with duplications spanning known ge
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