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1 s been found previously to interact with the human immunodeficiency virus, type 1 accessory protein V
2 ssociated with BV, such as preterm birth and human immunodeficiency virus type 1 acquisition, are ass
3 41 subunit of the envelope glycoprotein from human immunodeficiency virus type 1, an essential subuni
4                   Different viruses, such as human immunodeficiency virus type 1 and hepatitis C viru
5              Serum analysis was negative for human immunodeficiency virus type 1 and type 2 RNA, and
6         Serum analysis was also negative for human immunodeficiency virus type 1 and type 2 RNA, Vene
7                 The availability of reliable human immunodeficiency virus types 1 and 2 (HIV-1/2) rap
8 ribe the mechanical responses of vancomycin, human immunodeficiency virus type 1 antigens and coagula
9         Among patients with tuberculosis and human immunodeficiency virus type 1, CD4-stratified init
10 CD4(+) T cells was observed in subjects with human immunodeficiency virus type 1 coinfection without
11 opoietic stem/progenitor cells (HSPCs) using human immunodeficiency virus, type 1-derived lentiviral
12                                              Human immunodeficiency virus type 1 drug resistance amon
13                                              Human immunodeficiency virus type 1 enhancer-binding pro
14 lex virus type 2 (HSV-2; herpes) exacerbates human immunodeficiency virus type 1 (HIV) by unclear mec
15 ) alfa plus ribavirin decreases the proviral human immunodeficiency virus type 1 (HIV) DNA level.
16                                  Transmitted human immunodeficiency virus type 1 (HIV) drug resistanc
17                     Intermittent shedding of human immunodeficiency virus type 1 (HIV) in semen occur
18 DSC) expansion and T-cell dysfunction during human immunodeficiency virus type 1 (HIV) infection are
19                                    Untreated human immunodeficiency virus type 1 (HIV) infection is a
20  Progressive T cell depletion during chronic human immunodeficiency virus type 1 (HIV) infection is a
21                            Immune control of human immunodeficiency virus type 1 (HIV) infection is t
22                                              Human immunodeficiency virus type 1 (HIV) infection subs
23 esses may contribute to morbidity in treated human immunodeficiency virus type 1 (HIV) infection.
24 ormed a cohort study involving patients with human immunodeficiency virus type 1 (HIV) infection.
25                        Seventeen mothers had human immunodeficiency virus type 1 (HIV) infection; no
26                               Infection with human immunodeficiency virus type 1 (HIV) is associated
27 nt and understudied topic of cell-associated human immunodeficiency virus Type 1 (HIV) mucosal transm
28  whether distinct populations have differing human immunodeficiency virus type 1 (HIV) neutralizing a
29 cable comorbidities are more prevalent among human immunodeficiency virus type 1 (HIV)-infected indiv
30                                              Human immunodeficiency virus type 1 (HIV)-infected perso
31 s remains a significant cause of death among human immunodeficiency virus type 1 (HIV)-infected perso
32 se mortality among individuals infected with human immunodeficiency virus type 1 (HIV).
33                                          The human immunodeficiency virus type 1 (HIV-1) accessory pr
34 diated cytotoxicity (ADCC), in prevention of human immunodeficiency virus type 1 (HIV-1) acquisition
35  been shown to be preferentially targeted by human immunodeficiency virus type 1 (HIV-1) and are impl
36 ned elusive for important pathogens, such as human immunodeficiency virus type 1 (HIV-1) and herpesvi
37 ion is an efficient mechanism to disseminate human immunodeficiency virus type 1 (HIV-1) and human T
38 reporter genes delivered by vectors based on human immunodeficiency virus type 1 (HIV-1) and Mason-Pf
39                                   Binding of human immunodeficiency virus type 1 (HIV-1) and simian i
40 lpha (AgmTRIM5alpha) potently restricts both human immunodeficiency virus type 1 (HIV-1) and simian i
41 ts the infectivity of retroviruses including human immunodeficiency virus type 1 (HIV-1) and the mobi
42  most therapeutic and vaccine candidates for human immunodeficiency virus type 1 (HIV-1) are evaluate
43               Assembly and maturation of the human immunodeficiency virus type 1 (HIV-1) are governed
44                         Splicing patterns in human immunodeficiency virus type 1 (HIV-1) are maintain
45 s of this regimen on cellular reservoirs for human immunodeficiency virus type 1 (HIV-1) are unknown.
46  2.0), a real-time polymerase chain reaction human immunodeficiency virus type 1 (HIV-1) assay with h
47                                              Human immunodeficiency virus type 1 (HIV-1) assembles as
48 ng proteins incorporated into virions during human immunodeficiency virus type 1 (HIV-1) assembly pro
49                            Here we show that human immunodeficiency virus type 1 (HIV-1) associates w
50  generally suppresses the replication of the human immunodeficiency virus type 1 (HIV-1) but does not
51 its the release of virions of the retrovirus human immunodeficiency virus type 1 (HIV-1) by tethering
52  restriction activity, each was fused to the human immunodeficiency virus type 1 (HIV-1) CA binding p
53                                 Although the human immunodeficiency virus type 1 (HIV-1) CA carboxy-t
54                                              Human immunodeficiency virus type 1 (HIV-1) can efficien
55                                              Human immunodeficiency virus type 1 (HIV-1) can replicat
56                                          The human immunodeficiency virus type 1 (HIV-1) capsid prote
57 of measles and associations with outcome and human immunodeficiency virus type 1 (HIV-1) coinfection,
58 BEC3F (A3F) was highly localized into mature human immunodeficiency virus type 1 (HIV-1) cores and id
59                    Emerging data relating to human immunodeficiency virus type 1 (HIV-1) cure suggest
60 of preventing perinatal transmission (PT) of human immunodeficiency virus type 1 (HIV-1) depends on b
61 ia A who were exposed to but uninfected with human immunodeficiency virus type 1 (HIV-1) did not reve
62                                              Human immunodeficiency virus type 1 (HIV-1) disease is a
63                      HLA strongly influences human immunodeficiency virus type 1 (HIV-1) disease prog
64                        Inflammation in early human immunodeficiency virus type 1 (HIV-1) disease prog
65                     Transcriptionally silent human immunodeficiency virus type 1 (HIV-1) DNA persists
66  a number of years that integration sites of human immunodeficiency virus type 1 (HIV-1) DNA show a p
67                                      Herein, human immunodeficiency virus type 1 (HIV-1) DNA was chos
68                                              Human immunodeficiency virus type 1 (HIV-1) drug resista
69                                   Monitoring human immunodeficiency virus type 1 (HIV-1) drug resista
70                                              Human immunodeficiency virus type 1 (HIV-1) dual infecti
71 inical trial to evaluate the safety and anti-human immunodeficiency virus type 1 (HIV-1) efficacy of
72                                              Human immunodeficiency virus type 1 (HIV-1) entry into c
73 ing conserved CD4-induced (CD4i) epitopes on human immunodeficiency virus type 1 (HIV-1) Env and able
74  reactivity (ER) describes the propensity of human immunodeficiency virus type 1 (HIV-1) Env to chang
75                                          The human immunodeficiency virus type 1 (HIV-1) envelope (En
76  the gp120 subunits of a soluble recombinant human immunodeficiency virus type 1 (HIV-1) envelope (En
77           The extraordinary diversity of the human immunodeficiency virus type 1 (HIV-1) envelope (En
78 y simian immunodeficiency virus (SIV) versus human immunodeficiency virus type 1 (HIV-1) envelope ant
79                   Although antibodies to the human immunodeficiency virus type 1 (HIV-1) envelope gly
80           Recombinant trimeric mimics of the human immunodeficiency virus type 1 (HIV-1) envelope gly
81                                          The human immunodeficiency virus type 1 (HIV-1) envelope gly
82           Helper T-cell epitope dominance in human immunodeficiency virus type 1 (HIV-1) envelope gly
83 s between the gp120 and gp41 subunits of the human immunodeficiency virus type 1 (HIV-1) envelope gly
84                          Antibodies bound to human immunodeficiency virus type 1 (HIV-1) envelope pro
85           Identifying characteristics of the human immunodeficiency virus type 1 (HIV-1) envelope tha
86 used that target nonoverlapping sites on the human immunodeficiency virus type 1 (HIV-1) envelope.
87                The "shock and kill" model of human immunodeficiency virus type 1 (HIV-1) eradication
88 eservoir represents a critical challenge for human immunodeficiency virus type 1 (HIV-1) eradication
89 s maintained in a quiescent state.IMPORTANCE Human immunodeficiency virus type 1 (HIV-1) establishes
90                                              Human immunodeficiency virus type 1 (HIV-1) establishes
91        Transmission lies at the interface of human immunodeficiency virus type 1 (HIV-1) evolution wi
92 es maintain the ability to interact with the human immunodeficiency virus type 1 (HIV-1) Gag precurso
93 an T-cell leukemia virus type 1 (HTLV-1) and human immunodeficiency virus type 1 (HIV-1) Gag proteins
94                                        After human immunodeficiency virus type 1 (HIV-1) gains access
95                         Robust activation of human immunodeficiency virus type 1 (HIV-1) gene express
96 ression of a functional miRNA encoded in the human immunodeficiency virus type 1 (HIV-1) genome.
97 velope glycoprotein (Env) trimers of various human immunodeficiency virus type 1 (HIV-1) genotypes ar
98  (Ab) responses against V1V2 epitopes of the human immunodeficiency virus type 1 (HIV-1) gp120 envelo
99 placing the single VSV glycoprotein (G) with human immunodeficiency virus type 1 (HIV-1) gp160 to cre
100 virtide) and other peptides derived from the human immunodeficiency virus type 1 (HIV-1) gp41 C-termi
101                The hydrophobic groove of the human immunodeficiency virus type 1 (HIV-1) gp41 NHR tri
102                        The life cycle of the human immunodeficiency virus type 1 (HIV-1) has an absol
103                               The origins of human immunodeficiency virus type 1 (HIV-1) have been wi
104                                              Human immunodeficiency virus type 1 (HIV-1) hijacks host
105                                              Human immunodeficiency virus type 1 (HIV-1) impairs majo
106 motif-containing protein 5 (TRIM5) restricts human immunodeficiency virus type 1 (HIV-1) in a species
107 al components of vaccine-induced immunity to human immunodeficiency virus type 1 (HIV-1) in humans an
108 % of genetically linked infections caused by human immunodeficiency virus type 1 (HIV-1) in serodisco
109 iously shown to decrease the accumulation of human immunodeficiency virus type 1 (HIV-1) in the super
110      About 10% of new diagnoses of subtype B human immunodeficiency virus type 1 (HIV-1) in the Unite
111 In the absence of therapy, CXCR4 (X4)-tropic human immunodeficiency virus type 1 (HIV-1) increases ov
112 s chronic infection with RNA viruses such as human immunodeficiency virus type 1 (HIV-1) induces prof
113 ed antiretroviral therapy (cART) in semen of human immunodeficiency virus type 1 (HIV-1) infected men
114 usceptible tuberculosis in a setting of high human immunodeficiency virus type 1 (HIV-1) infection an
115 n are recognized as important in controlling human immunodeficiency virus type 1 (HIV-1) infection an
116 rs in early childhood and is associated with human immunodeficiency virus type 1 (HIV-1) infection an
117 al therapy (cART) administered shortly after human immunodeficiency virus type 1 (HIV-1) infection ca
118                  Guidelines for treatment of human immunodeficiency virus type 1 (HIV-1) infection co
119 iescent proviral genomes that persist during human immunodeficiency virus type 1 (HIV-1) infection de
120                   Research toward a cure for human immunodeficiency virus type 1 (HIV-1) infection ha
121 hylaxis has been shown to reduce the risk of human immunodeficiency virus type 1 (HIV-1) infection in
122                                              Human immunodeficiency virus type 1 (HIV-1) infection in
123                                A hallmark of human immunodeficiency virus type 1 (HIV-1) infection in
124                                              Human immunodeficiency virus type 1 (HIV-1) infection is
125                                      Chronic human immunodeficiency virus type 1 (HIV-1) infection is
126                                              Human immunodeficiency virus type 1 (HIV-1) infection is
127                                              Human immunodeficiency virus type 1 (HIV-1) infection is
128                                              Human immunodeficiency virus type 1 (HIV-1) infection is
129                  The initial phases of acute human immunodeficiency virus type 1 (HIV-1) infection ma
130                                              Human immunodeficiency virus type 1 (HIV-1) infection of
131                                              Human immunodeficiency virus type 1 (HIV-1) infection of
132              A vaccine that protects against human immunodeficiency virus type 1 (HIV-1) infection sh
133 ciency virus (SIV) infection and humans with human immunodeficiency virus type 1 (HIV-1) infection th
134 identified as a late postentry suppressor of human immunodeficiency virus type 1 (HIV-1) infection, i
135  potent anti-retroviral drugs in controlling human immunodeficiency virus type 1 (HIV-1) infection, l
136 rial of preexposure chemoprophylaxis against human immunodeficiency virus type 1 (HIV-1) infection, t
137 al contraceptives regulate susceptibility to human immunodeficiency virus type 1 (HIV-1) infection, t
138 e HCV infection in participants with chronic human immunodeficiency virus type 1 (HIV-1) infection.
139 ophylaxis (PrEP) candidate for prevention of human immunodeficiency virus type 1 (HIV-1) infection.
140 cell-based gene modification therapy against human immunodeficiency virus type 1 (HIV-1) infection.
141  source of type I IFN (IFN-I) in response to human immunodeficiency virus type 1 (HIV-1) infection.
142 ired T-cell function was investigated during human immunodeficiency virus type 1 (HIV-1) infection.
143  interventions to prevent the acquisition of human immunodeficiency virus type 1 (HIV-1) infection.
144 ete roles and functions of DRFs during early human immunodeficiency virus type 1 (HIV-1) infection.
145 of CD4+ T lymphocytes, a major reservoir for human immunodeficiency virus type 1 (HIV-1) infection.
146 ed as prophylaxis can prevent acquisition of human immunodeficiency virus type 1 (HIV-1) infection.
147                              High numbers of human immunodeficiency virus type 1 (HIV-1) infections a
148            Rhesus macaques are used to model human immunodeficiency virus type 1 (HIV-1) infections,
149 timulated genes (ISGs) and potently suppress Human immunodeficiency virus type 1 (HIV-1) infectivity
150 OBEC3G (A3G) is a human enzyme that inhibits human immunodeficiency virus type 1 (HIV-1) infectivity,
151                                          The human immunodeficiency virus type 1 (HIV-1) initiates re
152            Dolutegravir (DTG), a once-daily, human immunodeficiency virus type 1 (HIV-1) integrase in
153 ointestinal mucosa is the primary site where human immunodeficiency virus type 1 (HIV-1) invades, amp
154         The outcome after infection with the human immunodeficiency virus type 1 (HIV-1) is a complex
155 nhibiting infection with diverse variants of human immunodeficiency virus type 1 (HIV-1) is a key, as
156                        The envelope spike of human immunodeficiency virus type 1 (HIV-1) is a target
157                                              Human immunodeficiency virus type 1 (HIV-1) is divided i
158                                              Human immunodeficiency virus type 1 (HIV-1) is pandemic,
159           A hallmark of retroviruses such as human immunodeficiency virus type 1 (HIV-1) is reverse t
160                Reverse transcriptase (RT) of human immunodeficiency virus type 1 (HIV-1) is synthesiz
161                                              Human immunodeficiency virus type 1 (HIV-1) is the resul
162                                              Human immunodeficiency virus type 1 (HIV-1) isolates fro
163            GSK3532795 is a second-generation human immunodeficiency virus type 1 (HIV-1) maturation i
164                                       During human immunodeficiency virus type 1 (HIV-1) maturation,
165       The envelope glycoproteins (Envs) from human immunodeficiency virus type 1 (HIV-1) mediate vira
166       The trimeric envelope glycoproteins of human immunodeficiency virus type 1 (HIV-1) mediate viru
167        Following entry into the target cell, human immunodeficiency virus type 1 (HIV-1) must reverse
168         To establish a productive infection, human immunodeficiency virus type 1 (HIV-1) needs to rev
169                                              Human immunodeficiency virus type 1 (HIV-1) Nef enhances
170                             Minority variant human immunodeficiency virus type 1 (HIV-1) nonnucleosid
171                       Sexual transmission of human immunodeficiency virus type 1 (HIV-1) occurs acros
172                                          The human immunodeficiency virus type 1 (HIV-1) pandemic was
173 impact of CD4+ regulatory T cells (Tregs) on human immunodeficiency virus type 1 (HIV-1) pathogenesis
174                                              Human immunodeficiency virus type 1 (HIV-1) persists in
175 plication to clinically undetectable levels, human immunodeficiency virus type 1 (HIV-1) persists in
176 otyping success rate was evaluated in 12 828 human immunodeficiency virus type 1 (HIV-1) plasma sampl
177 ic visual display, we examine the changes in human immunodeficiency virus type 1 (HIV-1) plasma viral
178   The gp120 portion of the envelope spike on human immunodeficiency virus type 1 (HIV-1) plays a crit
179                    The sequence diversity of human immunodeficiency virus type 1 (HIV-1) presents a f
180                         Soluble forms of the human immunodeficiency virus type 1 (HIV-1) primary rece
181                                Resistance to human immunodeficiency virus type 1 (HIV-1) protease inh
182  Local Alignment Program (LAP) using 115,118 human immunodeficiency virus type 1 (HIV-1) protease, re
183 ic guide RNAs (gRNAs), can excise integrated human immunodeficiency virus type 1 (HIV-1) provirus fro
184        Little is known about the fraction of human immunodeficiency virus type 1 (HIV-1) proviruses t
185                   We sequenced the genome of human immunodeficiency virus type 1 (HIV-1) recovered fr
186                         Among these viruses, human immunodeficiency virus type 1 (HIV-1) remains a ma
187  (HCV) infection in patients coinfected with human immunodeficiency virus type 1 (HIV-1) remains a me
188            Prevention of the transmission of human immunodeficiency virus type 1 (HIV-1) remains a pr
189 is C virus (HCV) in patients coinfected with human immunodeficiency virus type 1 (HIV-1) remains an u
190 (ART) limits proviral reservoirs, a goal for human immunodeficiency virus type 1 (HIV-1) remission st
191  tissues (LTs) are the principal sites where human immunodeficiency virus type 1 (HIV-1) replicates a
192                                              Human immunodeficiency virus type 1 (HIV-1) replication
193 main-containing protein 1 (SAMHD1) restricts human immunodeficiency virus type 1 (HIV-1) replication
194      Alpha interferon (IFN-alpha) suppresses human immunodeficiency virus type 1 (HIV-1) replication
195 ymphoid tissues (LTs) are principal sites of human immunodeficiency virus type 1 (HIV-1) replication,
196 Tregs) suppress T-cell immune activation and human immunodeficiency virus type 1 (HIV-1) replication,
197                         The stability of the human immunodeficiency virus type 1 (HIV-1) reservoir an
198 ciations of CCR5-Delta32 heterozygosity with human immunodeficiency virus type 1 (HIV-1) reservoir si
199                        Limited data exist on human immunodeficiency virus type 1 (HIV-1) resistance i
200                                              Human immunodeficiency virus type 1 (HIV-1) reverse tran
201 t the ribonuclease (RNase) H activity of the human immunodeficiency virus type 1 (HIV-1) reverse tran
202                            All had increased human immunodeficiency virus type 1 (HIV-1) RNA and DNA
203                           We measured plasma human immunodeficiency virus type 1 (HIV-1) RNA levels b
204                                  Full-length human immunodeficiency virus type 1 (HIV-1) RNA serves a
205                        Full-length unspliced human immunodeficiency virus type 1 (HIV-1) RNAs serve d
206 thout condom use among 911 African HSV-2 and human immunodeficiency virus type 1 (HIV-1) serodiscorda
207       The 231-residue capsid (CA) protein of human immunodeficiency virus type 1 (HIV-1) spontaneousl
208 ve reduced effectiveness in the treatment of human immunodeficiency virus type 1 (HIV-1) subtype C in
209                                              Human immunodeficiency virus type 1 (HIV-1) subtype CRF0
210 vir plus 2 nucleos(t)ides for maintenance of human immunodeficiency virus type 1 (HIV-1) suppression.
211  this study was to investigate the effect of human immunodeficiency virus type 1 (HIV-1) Tat on the P
212 s an interferon (IFN)-inducible inhibitor of human immunodeficiency virus type 1 (HIV-1) that acts at
213 ith latency reversing agents (LRAs) enhances human immunodeficiency virus type 1 (HIV-1) transcriptio
214                              The majority of human immunodeficiency virus type 1 (HIV-1) transmission
215 atory state has been hypothesized to enhance human immunodeficiency virus type 1 (HIV-1) transmission
216  affect efavirenz pharmacokinetics, maternal human immunodeficiency virus type 1 (HIV-1) treatment ou
217              It is therefore unclear whether human immunodeficiency virus type 1 (HIV-1) vaccination
218 functional antibodies remains a challenge in human immunodeficiency virus type 1 (HIV-1) vaccine deve
219  design are being developed as immunogens in human immunodeficiency virus type 1 (HIV-1) vaccine deve
220                                 An effective human immunodeficiency virus type 1 (HIV-1) vaccine must
221                The search for an efficacious human immunodeficiency virus type 1 (HIV-1) vaccine rema
222                    Increasing the breadth of human immunodeficiency virus type 1 (HIV-1) vaccine-elic
223 to be an important component of a protective human immunodeficiency virus type 1 (HIV-1) vaccine.
224 mune responses and inflammation to candidate human immunodeficiency virus type 1 (HIV-1) vaccines rep
225                                              Human immunodeficiency virus type 1 (HIV-1) vaccines tha
226                          A limited number of human immunodeficiency virus type 1 (HIV-1) variants ini
227     Recent studies have evaluated cumulative human immunodeficiency virus type 1 (HIV-1) viral load (
228                   A recent study showed that human immunodeficiency virus type 1 (HIV-1) viral protei
229                          Though the steps of human immunodeficiency virus type 1 (HIV-1) virion matur
230 important role in regulating the assembly of human immunodeficiency virus type 1 (HIV-1) virus partic
231 sensitive to the prototypic viral antagonist human immunodeficiency virus type 1 (HIV-1) Vpu.
232 ave shown that infection of cells containing human immunodeficiency virus type 1 (HIV-1) with KSHV le
233  of single-stranded DNA (ssDNA) derived from human immunodeficiency virus type 1 (HIV-1), activated t
234 -cell lymphotropic virus type 1 (HTLV-1) and human immunodeficiency virus type 1 (HIV-1), but arises
235 ergistically induce the expression of latent human immunodeficiency virus type 1 (HIV-1), but studies
236                     Among infants exposed to human immunodeficiency virus type 1 (HIV-1), detection o
237                      Many viruses, including human immunodeficiency virus type 1 (HIV-1), encode a re
238    The surface envelope glycoprotein (SU) of Human immunodeficiency virus type 1 (HIV-1), gp120(SU) p
239 e strong dependence of retroviruses, such as human immunodeficiency virus type 1 (HIV-1), on host cel
240 nd development of three major model systems [human immunodeficiency virus type 1 (HIV-1), Rous sarcom
241 rs based on the env genes of two isolates of human immunodeficiency virus type 1 (HIV-1), specificall
242 ing delayed progression of disease caused by human immunodeficiency virus type 1 (HIV-1), yet a ligan
243 ess to AIDS, in stark contrast to pathogenic human immunodeficiency virus type 1 (HIV-1)-human and SI
244 lated with rotavirus vaccine responses in 68 human immunodeficiency virus type 1 (HIV-1)-infected (an
245 013 that reported virological outcomes among human immunodeficiency virus type 1 (HIV-1)-infected adu
246 a, has long been known to be elevated in the human immunodeficiency virus type 1 (HIV-1)-infected bra
247 ance cytotoxic T lymphocyte (CTL) evasion in human immunodeficiency virus type 1 (HIV-1)-infected CD4
248                                 The decay of human immunodeficiency virus type 1 (HIV-1)-infected cel
249                                       Unlike human immunodeficiency virus type 1 (HIV-1)-infected hum
250 cy virus (SHIV)-infected rhesus macaques and human immunodeficiency virus type 1 (HIV-1)-infected hum
251  alleles (e.g., HLA-B*27) are enriched among human immunodeficiency virus type 1 (HIV-1)-infected ind
252                                              Human immunodeficiency virus type 1 (HIV-1)-infected ind
253 ssion in the central nervous system (CNS) of human immunodeficiency virus type 1 (HIV-1)-infected ind
254 combination antiretroviral therapy (cART) to human immunodeficiency virus type 1 (HIV-1)-infected inf
255                                          All human immunodeficiency virus type 1 (HIV-1)-infected ino
256                                           In human immunodeficiency virus type 1 (HIV-1)-infected peo
257                             The aging of the human immunodeficiency virus type 1 (HIV-1)-infected pop
258 ion of combination antiretroviral therapy to human immunodeficiency virus type 1 (HIV-1)-infected pre
259 -blind, placebo-controlled clinical trial on human immunodeficiency virus type 1 (HIV-1)-infected sub
260                   In this prospective study, human immunodeficiency virus type 1 (HIV-1)-infected sub
261  are reported in uninfected children born to human immunodeficiency virus type 1 (HIV-1)-infected wom
262 at women on PrEP had IgA with higher average human immunodeficiency virus type 1 (HIV-1)-neutralizing
263 ed infectious virions, non-bNAbs and mucosal human immunodeficiency virus type 1 (HIV-1)-positive IgG
264 onal regulatory T cells (Tregs) can suppress human immunodeficiency virus type 1 (HIV-1)-specific imm
265 tion with an anti-PD-L1 antibody may improve human immunodeficiency virus type 1 (HIV-1)-specific imm
266 e control of intracellular pathogens such as human immunodeficiency virus type 1 (HIV-1).
267 this combination in patients coinfected with human immunodeficiency virus type 1 (HIV-1).
268 he reverse transcriptase (RT) complex of the human immunodeficiency virus type 1 (HIV-1).
269 iciency virus (SIVcpz) into pandemic group M human immunodeficiency virus type 1 (HIV-1).
270 TIs) in treatment-naive adults infected with human immunodeficiency virus type 1 (HIV-1).
271 urfaces of cells infected with vpu-deficient human immunodeficiency virus type 1 (HIV-1).
272 ween herpes simplex virus type 2 (HSV-2) and human immunodeficiency virus type 1 (HIV-1).
273 IVgor, which represent the ape precursors of human immunodeficiency virus type 1 (HIV-1).
274                                          The human immunodeficiency virus type 1 (HIV-1)/simian immun
275 ck-and-kill" therapeutic approach to reverse human immunodeficiency virus type-1 (HIV) latency from C
276                                              Human Immunodeficiency Virus type-1 (HIV)-associated neu
277 sease (GUD) in a cohort of women living with human immunodeficiency virus type-1 (HIV-1) in Burkina F
278                                              Human immunodeficiency virus type-1 (HIV-1)-associated n
279  plays a crucial role in the pathogenesis of human immunodeficiency virus type-1 (HIV-1).
280 a wide range of enveloped viruses, including human immunodeficiency virus, type 1 (HIV-1) by directly
281 -1beta is associated with the progression of human immunodeficiency virus, type 1 (HIV-1) disease or
282 brane-proximal external region (MPER) of the human immunodeficiency virus, type 1 (HIV-1) envelope gl
283 e supposed to form a natural barrier against human immunodeficiency virus, type 1 (HIV-1) infection.
284 y contribute to racial disparities in plasma human immunodeficiency virus type 1 : HIV-1) RNA levels
285 e replication of primate lentiviruses (e.g., human immunodeficiency virus type 1 [HIV-1] and simian i
286 or tenofovir/emtricitabine (TDF/FTC) in 1857 human immunodeficiency virus type 1-infected, treatment-
287 for MICA in progression and elite control of human immunodeficiency virus type 1 infection.
288  be fruitful for this scaffold, since strong human immunodeficiency virus type 1 integrase (HIV-1 IN)
289 virus, reovirus, vesicular stomatitis virus, human immunodeficiency virus type 1, or herpes simplex v
290 96-1997, 1998-1999, 2000-2001], and AIDS and human immunodeficiency virus type 1 RNA at baseline) mor
291                      ART-naive subjects with human immunodeficiency virus type 1 RNA load (viral load
292   The role of the adenosine (ADO) pathway in human immunodeficiency virus type 1/simian immunodeficie
293 ized Rous sarcoma virus (RSV) intasomes with human immunodeficiency virus type 1 strand transfer inhi
294  transduction domain of the tat protein from human immunodeficiency virus type 1 (tat-cyclotraxin-B).
295                                  Importance: Human immunodeficiency virus type 1 transmission is esta
296                                       HIV-1 (human immunodeficiency virus type 1) uses its trimeric g
297  the clinical development pathway of an rVSV/human immunodeficiency virus type 1 vaccine.
298                                              Human immunodeficiency virus type 1 Vpr is an accessory
299 at this position and either co-expression of human immunodeficiency virus type 1 vpu or siRNA-mediate
300                                       Unlike human immunodeficiency virus type 1 Vpu protein, which a

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