ライフサイエンスコーパス: hypercortisolism

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1 tropic hormone, indicating primary (adrenal) hypercortisolism.

2 mpact of clinical conditions associated with hypercortisolism.

3 ipates in PRKAR1A-mediated tumorigenesis and hypercortisolism.

4 g to high bone mass despite hypogonadism and hypercortisolism.

5 thways that each are driven and sustained by hypercortisolism.

6 iately high, considering the degree of their hypercortisolism.

7 rophin-releasing hormone expression, chronic hypercortisolism, adrenocortical hyperplasia, glucose in

8 The phenomena of basal hypercortisolism and of dexamethasone resistance have lo

9 glucocorticoid sensitivity in the absence of hypercortisolism and to the pathogenesis of AIDS.

10 e development of adrenal cortex hyperplasia, hypercortisolism, and spleen atrophy, which was attenuat

11 ngal infections that arise in the setting of hypercortisolism, and the ways in which glucocorticoids

12 lectomy for symptomatic relief of persistent hypercortisolism appears to be an effective treatment op

13 Thus, the hypercortisolism associated with bilateral macronodular

14 as well as metabolic disturbances mimicking hypercortisolism caused by Cushing disease.

15 ential diagnosis and treatment of endogenous hypercortisolism--Cushing's syndrome.

16 Despite their hypercortisolism, depressed patients had normal levels o

17 The primary treatment of Cushing disease (hypercortisolism due to ACTH-producing adenomas, which i

18 eatment of Cushing's disease, being cured of hypercortisolism for a minimum of 10 years at study entr

19 Psychologic stress and clinical hypercortisolism have been related to direct effects on

20 iseases (such as primary hyperaldosteronism, hypercortisolism, hyperandrogenism, or hyperestrogenism)

21 itive impairments associated with endogenous hypercortisolism in humans.

22 l implication of expanding the definition of hypercortisolism in patient populations with compromised

23 nt of a buffalo hump cannot be attributed to hypercortisolism in these eight men.

24 e cause in approximately 65% of the cases of hypercortisolism) is adenoma resection and medical thera

25 rm effects and comorbidities associated with hypercortisolism need ongoing care.

26 unction, hyperparathyroidism, acromegaly and hypercortisolism on bone.

27 It is proposed that the negative impact of hypercortisolism on neurocognitive function mediates thi

28 isolation were associated in our study with hypercortisolism or feedback resistance.

29 t detectable in 40 patients with subclinical hypercortisolism or in 82 patients with other adrenal tu

30 quately treated hyperandrogenism, iatrogenic hypercortisolism, or both.

31 ho had received curative treatment and whose hypercortisolism remained in remission for more than 10

32 cians who treat patients with ACC and severe hypercortisolism should recognize that uncontrolled horm

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