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1 modulatory receptors, is mutated in X-linked lymphoproliferative disease.
2 lects HVL patients at risk of posttransplant lymphoproliferative disease.
3 -4-deficient mice develop fatal, early onset lymphoproliferative disease.
4  viral loads and emergence of EBV-associated lymphoproliferative disease.
5 f adult T-cell leukemia (ATL), an aggressive lymphoproliferative disease.
6 host defect led to a specific and consistent lymphoproliferative disease.
7 e of cytomegalovirus disease and no cases of lymphoproliferative disease.
8            We had no cases of posttransplant lymphoproliferative disease.
9  the human genetic immunodeficiency X-linked lymphoproliferative disease.
10  Epstein-Barr virus-positive post-transplant lymphoproliferative disease.
11 homa, Hodgkin's lymphoma, and posttransplant lymphoproliferative disease.
12 nfections are associated with development of lymphoproliferative disease.
13 ted protein), the protein absent in X-linked lymphoproliferative disease.
14 TNF-family receptor result in autoimmune and lymphoproliferative disease.
15 th of immune control in limiting LCV-induced lymphoproliferative disease.
16 ed, but there was no evidence of LCV-induced lymphoproliferative disease.
17 pus erythematosus that also have features of lymphoproliferative disease.
18 ciated with hypertension, hyperlipidemia, or lymphoproliferative disease.
19 reated before HSCT because of EBV-associated lymphoproliferative disease.
20  and pathological spectrum of posttransplant lymphoproliferative disease.
21 rowth and the monitoring for post-transplant lymphoproliferative disease.
22 viously reported as a form of posttransplant lymphoproliferative disease.
23 entially fatal EBV-associated posttransplant lymphoproliferative disease.
24 s, and early stem cells and induces a lethal lymphoproliferative disease.
25 eased risk for development of posttransplant lymphoproliferative disease.
26 many are at increased risk of virus-driven B-lymphoproliferative disease.
27 tient in each group developed posttransplant lymphoproliferative disease.
28 atopoiesis, and bone marrow fibrosis, but no lymphoproliferative disease.
29  at risk for Epstein-Barr virus (EBV)-driven lymphoproliferative disease.
30 e targeted in T cell therapy of EBV-driven B-lymphoproliferative disease.
31 ferentiation into effector cells and induced lymphoproliferative disease.
32 lar to those found in patients with X-linked lymphoproliferative disease.
33 e of the noncanonical NF-kappaB pathway in B lymphoproliferative disease.
34 om macroglobulinemia is a distinct low-grade lymphoproliferative disease.
35 ostic information for the development of EBV lymphoproliferative diseases.
36 mphocytes and is responsible for a number of lymphoproliferative diseases.
37 llularly could be useful in the treatment of lymphoproliferative diseases.
38  well-tolerated treatment for autoimmune and lymphoproliferative diseases.
39 cus on the role of TWIST2 in CLL and related lymphoproliferative diseases.
40 etent people, EBV causes several cancers and lymphoproliferative diseases.
41 in B cells, leading to HCV-associated B-cell lymphoproliferative diseases.
42 ses infectious mononucleosis and can lead to lymphoproliferative diseases.
43 ed to the development of both KS and several lymphoproliferative diseases.
44 lue to the etiology of HCV-associated B-cell lymphoproliferative diseases.
45 nt recipients are at risk for development of lymphoproliferative diseases.
46 ation and express B220 as seen in autoimmune lymphoproliferative diseases.
47 he role of EBV in the pathogenesis of T-cell lymphoproliferative diseases.
48 plays a role in the chemotherapy of indolent lymphoproliferative diseases.
49 can potentially be targeted in autoimmune or lymphoproliferative diseases.
50 s and provide protection from EBV-associated lymphoproliferative diseases.
51  of Kaposi's sarcoma (KS) and several B-cell lymphoproliferative diseases.
52  in clinical development for treating B-cell lymphoproliferative diseases.
53 lticentric Castleman disease (MCD) and other lymphoproliferative diseases.
54 r the potential development of MCD and other lymphoproliferative diseases.
55 etiologic agent of Kaposi's sarcoma (KS) and lymphoproliferative diseases.
56                                     X-linked lymphoproliferative disease 1 (XLP1), due to mutations i
57 ped hematologic malignancies (posttransplant lymphoproliferative diseases, 18; Hodgkin disease, 2; an
58 e causal gene responsible for XLP2 (X-linked lymphoproliferative Disease 2).
59 ral infections (2, HIV=1, parvovirus=1), and lymphoproliferative disease (2).
60 nce of approximately 80% (49% splenic B-cell lymphoproliferative disease, 28% lymphoma).
61 drome/myeloproliferative neoplasm (19%), and lymphoproliferative diseases (30%).
62 s the genetic locus responsible for X-linked lymphoproliferative disease, a fatal immunodeficiency.
63       Mutations in SH2D1A cause the X-linked lymphoproliferative disease, a primary immunodeficiency.
64 AM-associated protein (SAP) lead to X-linked lymphoproliferative disease, a rare but fatal immunodefi
65 lines to treat EBV-associated posttransplant lymphoproliferative disease, a response rate of 52% was
66 I, 1.03-3.10; P = 0.038) and post-transplant lymphoproliferative disease (adjusted HR, 2.72; 95% CI,
67  is a complex retrovirus associated with the lymphoproliferative disease adult T-cell leukemia/lympho
68 y immunogenic tumors such as post-transplant lymphoproliferative disease, although resistance occurre
69 ls contributes to the pathophysiology of the lymphoproliferative disease and AHA in IL-2-deficient mi
70                         These mice developed lymphoproliferative disease and autoimmunity and died pr
71   This mechanism probably contributed to the lymphoproliferative disease and autoimmunity of miR-17-9
72 e discuss this unique form of posttransplant lymphoproliferative disease and briefly review the clini
73 ontaneously mounting a severe autoaggressive lymphoproliferative disease and can modulate immune resp
74 onset of a variety of pathologies, including lymphoproliferative disease and cancers.
75 H2D1A, the gene that is aberrant in X-linked lymphoproliferative disease and familial hemophagocytic
76 ase but not in EBV-associated posttransplant lymphoproliferative disease and Hodgkin disease.
77 mal in 14 of 16 patients (88%) with X-linked lymphoproliferative disease and in 8 of 14 patients (57%
78                            In human X-linked lymphoproliferative disease and its gene-targeted mouse
79 r virus (EBV) often induces extensive B-cell lymphoproliferative disease and lymphoma.
80 f in vivo immune control over posttransplant lymphoproliferative disease and lymphomas, our investiga
81 s adoptive immunotherapy against EBV-related lymphoproliferative disease and many other EBV-associate
82 n-Barr virus (EBV)-associated posttransplant lymphoproliferative disease and melanoma.
83                         These mice develop a lymphoproliferative disease and occasional transformatio
84 ometimes fatal syndromes, including X-linked lymphoproliferative disease and severe cases of common v
85 h are well-known for their ability to induce lymphoproliferative disease and to establish latency in
86 Epstein-Barr virus-associated posttransplant lymphoproliferative disease and was characterized by the
87  Burkitt lymphoma, nasopharyngeal carcinoma, lymphoproliferative disease and, though still debated, b
88 hotropic herpesvirus strongly linked to both lymphoproliferative diseases and Kaposi's sarcoma.
89 rus (KSHV) is involved in the development of lymphoproliferative diseases and Kaposi's sarcoma.
90 ated herpesvirus (KSHV), are associated with lymphoproliferative diseases and several human tumors.
91 ociation of hepatitis C virus infection with lymphoproliferative diseases and, newly, with cholangioc
92  cardiac allografts is limited by rejection, lymphoproliferative disease, and coronary vasculopathy.
93 odies to EBV, EBV RNA in lymph nodes, T-cell lymphoproliferative disease, and hemophagocytic lymphohi
94  stromal lymphopoietin levels, milder B-cell lymphoproliferative disease, and improved survival in Ms
95 th everolimus and hemolytic uremic syndrome, lymphoproliferative disease, and proteinuria, and higher
96 erative syndromes, hemophagocytic disorders, lymphoproliferative diseases, and novel differential dia
97 2%, avascular necrosis; 0.2%, posttransplant lymphoproliferative disease; and 0%, polyomavirus.
98 isk of adverse events such as posttransplant lymphoproliferative disease are discussed.
99                                              Lymphoproliferative diseases are characterized by massiv
100 % of patients with autoimmune, allergic, and lymphoproliferative diseases are refractory to glucocort
101  oncogenesis of Epstein-Barr virus in B-cell lymphoproliferative disease arising in immunosuppressed
102 ponse rates in patients with post-transplant lymphoproliferative disease as well as EBV-positive lymp
103 lls are frequently observed in several other lymphoproliferative diseases as well.
104 ciated with the development of lymphomas and lymphoproliferative diseases, as well as several other t
105  is pathologically similar to posttransplant lymphoproliferative disease associated with Epstein-Barr
106 umvent experimental limitations due to fatal lymphoproliferative disease associated with genetic abla
107 atched related donor, the risk of developing lymphoproliferative disease associated with the Epstein-
108 ed sufficient insulin until the mice died of lymphoproliferative disease at 12 months age.
109 echanisms responsible for the induction of a lymphoproliferative disease, atypical lymphoid hyperplas
110 aland black (NZB) mice with autoimmune and B lymphoproliferative disease (B-LPD) are a model for huma
111 cally decreased risks of posttransplantation lymphoproliferative disease but is followed by a prolong
112 Epstein-Barr virus (EBV) posttransplantation lymphoproliferative disease, but the extent of immune de
113                               Posttransplant lymphoproliferative disease can be treated by the infusi
114 ce demonstrated a role for Erk activation in lymphoproliferative disease caused by the LAT knock-in m
115 g Pten in Treg cells developed an autoimmune-lymphoproliferative disease characterized by excessive T
116 ced-stage PSC was 18.7%, with posttransplant lymphoproliferative diseases, colorectal cancer, and ren
117                          Dianzani autoimmune lymphoproliferative disease (DALD) has a similar phenoty
118 c/prolymphocytic leukemia (T-CLL/T-PLL) is a lymphoproliferative disease derived from immunocompetent
119 ism by which HTLV-1, a virus associated with lymphoproliferative disease, dysregulates common T-cell
120                Epstein-Barr virus-associated lymphoproliferative disease (EBV-LPD) is a potentially l
121 plantation recipients with biopsy-proven EBV-lymphoproliferative disease (EBV-LPD).
122 te activity can lead to life-threatening EBV lymphoproliferative disease (EBV-PTLD).
123 ologically against inflammatory, immune, and lymphoproliferative diseases for more than 50 years.
124 xp3 leads to development of fatal autoimmune lymphoproliferative disease; furthermore, ectopic Foxp3
125  that depend upon expression of the X-linked lymphoproliferative disease gene product, SH2D1A/DSHP/SA
126 nd congenic Fas ligand-deficient generalized lymphoproliferative disease (gld) mice were exposed to 7
127 us lymphoproliferation (lpr) and generalized lymphoproliferative disease (gld) mutations exhibit abno
128 teen patients with infections/posttransplant lymphoproliferative disease had a mean ATP of 48 ng/ml.
129                  Treatment of posttransplant lymphoproliferative disease has been augmented by the us
130 graft-versus-host disease, or posttransplant lymphoproliferative disease has been observed.
131 arr virus (EBV)-positive posttransplantation lymphoproliferative disease has suggested that such lesi
132 nstrated to promote Treg cell suppression of lymphoproliferative diseases, has an unexpected function
133 r tumors and various forms of posttransplant lymphoproliferative disease have occurred after mTOR con
134 n cancer, Kaposi sarcoma, and posttransplant lymphoproliferative disease have standardized incidence
135 onucleosis or its fatal equivalent, X-linked lymphoproliferative disease; (ii) EBV infection in a ran
136  Marek's disease virus (MDV) causes an acute lymphoproliferative disease in chickens, resulting in T
137 as a surprise in this issue of Immunity that lymphoproliferative disease in Foxo3a-deficient mice may
138  in T cell development and leads to a severe lymphoproliferative disease in homozygous knock-in mice.
139 is associated with an increased incidence of lymphoproliferative disease in immunocompromised hosts.
140 n either inhibit or enhance the induction of lymphoproliferative disease in immunocompromised mice.
141  prophylaxis and treatment of EBV-associated lymphoproliferative disease in immunocompromised patient
142  human malignancies including posttransplant lymphoproliferative disease in immunosuppressed patients
143 ayed the onset and decreased the severity of lymphoproliferative disease in LAT knock-in mice.
144  products might suppress MAPK activation and lymphoproliferative disease in LAT mutant mice.
145 reduced expression is a major determinant of lymphoproliferative disease in mice and humans; moreover
146    We report here the development of a CD30+ lymphoproliferative disease in mice lacking the proapopt
147 1 (PLC-gamma1)-binding site leads to a fatal lymphoproliferative disease in mice.
148  and has been reported to be associated with lymphoproliferative disease in mice.
149              The incidence of posttransplant lymphoproliferative disease in patients receiving siroli
150 eutic efficacy of these drugs for EBV-driven lymphoproliferative disease in patients.
151 e effective for the inhibition of EBV-driven lymphoproliferative disease in SCID mice than chemothera
152 d strongly inhibit the growth of EBV-induced lymphoproliferative disease in SCID mice.
153 lignancy in mouse models, and development of lymphoproliferative disease in three individuals with se
154 e and effective prophylaxis or treatment for lymphoproliferative disease in transplantation recipient
155 elayed the development of fatal EBV-positive lymphoproliferative disease in vivo.
156 mechanistic insight into the pathogenesis of lymphoproliferative disease in XLP.
157 ells or isolated CD14 cells induced lymphoma/lymphoproliferative diseases in 74% (20 of 27) of inject
158       Parasites in the genus Theileria cause lymphoproliferative diseases in cattle, resulting in eno
159 is to educate the reader about two cutaneous lymphoproliferative diseases in childhood: pityriasis li
160  of human lymphoid and epithelial tumors and lymphoproliferative diseases in immunocompromised patien
161 tion disrupts T-cell homeostasis and induces lymphoproliferative diseases in mice.
162 i (Saimiriine herpesvirus-2) causes lethal T lymphoproliferative diseases in the susceptible species
163 ases of GI involvement by an indolent T-cell lymphoproliferative disease, including 6 men and 4 women
164 esviral infection is closely associated with lymphoproliferative diseases, including B cell lymphomas
165 opment of Kaposi's sarcoma and several other lymphoproliferative diseases, including primary effusion
166 O mice developed lupus-like autoimmunity and lymphoproliferative disease, indicating that ubiquitin l
167                    Kaposi's sarcoma (KS) and lymphoproliferative diseases induced by KS-associated he
168             Epstein-Barr virus (EBV)-induced lymphoproliferative disease is an important complication
169                                     X-linked lymphoproliferative disease is caused by mutations affec
170  cell therapy for EBV-driven post-transplant lymphoproliferative disease is stimulating efforts to ta
171      Lymphadenopathy in autoimmune and other lymphoproliferative diseases is in part characterized by
172                  The role of angiogenesis in lymphoproliferative diseases is not well established.
173 stein-Barr virus (EBV)-driven posttransplant lymphoproliferative disease, is an important goal in cli
174 velopment of three major human neoplastic or lymphoproliferative diseases: Kaposi's sarcoma (KS), pri
175                    Individuals with X-linked lymphoproliferative disease lack invariant natural kille
176 V in the pathogenesis of EBV-positive T-cell lymphoproliferative diseases largely unresolved.
177      Scurfy mice develop CD4 T-cell-mediated lymphoproliferative disease leading to death within 4 we
178 ne function; however, treated mice developed lymphoproliferative disease, likely due to viral-promote
179 different centers to prevent or treat EBV(+) lymphoproliferative disease (LPD) arising after hematopo
180 on of these mice (~5%) develop a spontaneous lymphoproliferative disease (LPD) that results in dramat
181 tion are defective in promoting EBV-mediated lymphoproliferative disease (LPD).
182 iates with the development of EBV-associated lymphoproliferative disease (LPD).
183 ymorphic extranodal or (3) polymorphic nodal lymphoproliferative disease (LPD); and (4) diffuse large
184                          Immunophenotypes in lymphoproliferative diseases (LPD) are prognostically si
185                                              Lymphoproliferative diseases (LPDs) associated with the
186  that are associated with the development of lymphoproliferative diseases, lymphomas, as well as othe
187 pment; however, they rapidly develop a fatal lymphoproliferative disease marked by the uncontrolled e
188 -125b are associated with the development of lymphoproliferative disease, marked by expansion of CD8(
189 ciated with Kaposi sarcoma (KS) and 2 B cell lymphoproliferative diseases, namely primary effusion ly
190                                Patients with lymphoproliferative diseases not in CR (except Hodgkin l
191                        This acute and lethal lymphoproliferative disease occurs after a prolonged asy
192                    Marek's disease (MD) is a lymphoproliferative disease of chickens caused by the on
193                    Marek's disease (MD) is a lymphoproliferative disease of chickens induced by a her
194                           Marek's disease, a lymphoproliferative disease of chickens, is caused by an
195              The natural killer (NK) type of lymphoproliferative disease of granular lymphocytes (LDG
196                                              Lymphoproliferative disease of granular T lymphocyte (T-
197 d cell aplasia (PRCA) can be associated with lymphoproliferative disease of granular T lymphocytes (T
198                                   Chronic NK lymphoproliferative disease of large granular lymphocyte
199 anular lymphocyte (LGL) leukemia is a clonal lymphoproliferative disease of mature T and natural kill
200 Adult T-cell leukemia (ATL) is an aggressive lymphoproliferative disease of poor clinical prognosis a
201 ces malignant catarrhal fever (MCF), a fatal lymphoproliferative disease of ruminants, including catt
202  B-cell-tropic virus associated with various lymphoproliferative diseases of both B-cell and non-B-ce
203 osely related protozoan parasites that cause lymphoproliferative diseases of cattle.
204        These mice do not develop spontaneous lymphoproliferative disease or EAE and are resistant to
205 l confounders or a history of posttransplant lymphoproliferative disease or Epstein-Barr virus; altho
206 duced in T cells from patients with X-linked lymphoproliferative disease or normal T cells transfecte
207 t increase in infection, posttransplantation lymphoproliferative disease, or other side effects, othe
208 cleosis, hemophagocytic lymphohistiocytosis, lymphoproliferative disease, organomegaly, and/or malign
209 cystis carinii pneumonia, and no evidence of lymphoproliferative disease over 1 year.
210                     Furthermore, in X-linked lymphoproliferative disease patients, SAP deficiency red
211                      Primary CD30+ cutaneous lymphoproliferative disease (PCLPD) is a spectrum of ind
212 ene expression contributes to EBV-associated lymphoproliferative disease, potentially through inducti
213 rcoma and is also associated with two B-cell lymphoproliferative diseases, primary effusion lymphoma
214  had no previous diagnosis of posttransplant lymphoproliferative disease (PT-LPD), while nine had rec
215  describes the development of posttransplant lymphoproliferative disease (PTLD) after allogeneic SpTx
216 es such varied conditions as post-transplant lymphoproliferative disease (PTLD) and Burkitt's lymphom
217 sodes of rejection as well as posttransplant lymphoproliferative disease (PTLD) and graft-versus-host
218  more than 1000 copies/mL and posttransplant lymphoproliferative disease (PTLD) developed in 2 of the
219 city (p = 0.014) and previous posttransplant lymphoproliferative disease (PTLD) diagnosis (p = 0.006)
220 -Barr virus (EBV)-associated post-transplant lymphoproliferative disease (PTLD) encompasses a histolo
221 nsplant Epstein-Barr virus-associated B-cell lymphoproliferative disease (PTLD) has a higher incidenc
222           PURPOSE Adult post-transplantation lymphoproliferative disease (PTLD) has a reported 3-year
223  the incidence and hazard for posttransplant lymphoproliferative disease (PTLD) in a study of 3170 pe
224                               Posttransplant lymphoproliferative disease (PTLD) is a common Epstein-B
225  virus (EBV)-associated post-transplantation lymphoproliferative disease (PTLD) is a common, often fa
226 Epstein-Barr virus-associated posttransplant lymphoproliferative disease (PTLD) is a life-threatening
227                               Posttransplant lymphoproliferative disease (PTLD) is a major complicati
228                          Posttransplantation lymphoproliferative disease (PTLD) is a major complicati
229                               Posttransplant lymphoproliferative disease (PTLD) is a serious complica
230                               Posttransplant lymphoproliferative disease (PTLD) is a serious complica
231                               Posttransplant lymphoproliferative disease (PTLD) is a serious complica
232                          Posttransplantation lymphoproliferative disease (PTLD) is an often Epstein-B
233 development of EBV-associated posttransplant lymphoproliferative disease (PTLD) is the basis for stra
234 tients with EBV-positive posttransplantation lymphoproliferative disease (PTLD) on the basis of the b
235           Optimal therapy for posttransplant lymphoproliferative disease (PTLD) remains problematic.
236         Optimal management of posttransplant lymphoproliferative disease (PTLD) remains to be defined
237 irus (EBV) is associated with posttransplant lymphoproliferative disease (PTLD), and EBV load measure
238 ding Burkitt's lymphoma (BL), posttransplant lymphoproliferative disease (PTLD), nasopharyngeal carci
239 o several diseases, including posttransplant lymphoproliferative disease (PTLD), which involves very
240  risk for EBV-associated posttransplantation lymphoproliferative disease (PTLD).
241 months after implantation for posttransplant lymphoproliferative disease (PTLD).
242 ein Barr virus (EBV)-positive posttransplant lymphoproliferative disease (PTLD).
243 alovirus (CMV) infection, and posttransplant lymphoproliferative disease (PTLD).
244  and/or gastrointestinal (GI) posttransplant lymphoproliferative disease (PTLD).
245 central nervous system (PCNS) posttransplant lymphoproliferative disease (PTLD).
246 ant recipients are at risk of posttransplant lymphoproliferative disease (PTLD).
247  infections and trigger post-transplantation lymphoproliferative disease (PTLD).
248 everal interventions can cure posttransplant lymphoproliferative disease (PTLD); a sequential approac
249                          Posttransplantation lymphoproliferative diseases (PTLD) are mainly Epstein-B
250 tein-Barr virus (EBV)(+) posttransplantation lymphoproliferative diseases (PTLD) in cord blood transp
251 y, and EBV-related morbidity (posttransplant lymphoproliferative disease [PTLD] or symptomatic EBV in
252 , and Epstein-Barr virus) and posttransplant lymphoproliferative disease remained low.
253 eficiency with a loss of Ripk3 gives rise to lymphoproliferative disease reminiscent of lpr or gld mi
254              We analyzed the virological and lymphoproliferative disease response (LDR) of 46 patient
255 characterized by the progression of a T-cell lymphoproliferative disease (restricted to BM and lympho
256 ribed patients with immune dysregulation and lymphoproliferative disease resulting from 2 different g
257 and encoded by Foxp3) causes a rapidly fatal lymphoproliferative disease, similar to that seen in mic
258 c T-lymphocyte epitopes in 25 posttransplant lymphoproliferative disease specimens from 19 patients.
259 one of the patients developed posttransplant lymphoproliferative disease, successfully treated with a
260 entiviruses cause an incurable, progressive, lymphoproliferative disease that affects millions of ani
261  granular lymphocytic leukaemia (T-LGL) is a lymphoproliferative disease that presents with immune-me
262 tive for CAEBV, even in patients with active lymphoproliferative disease that was unresponsive to che
263       Hairy cell leukemia (HCL) is a chronic lymphoproliferative disease, the cause of which is unkno
264 xposure to these medications and the risk of lymphoproliferative disease, the known predisposition of
265      In type III latency, exemplified by EBV lymphoproliferative diseases, the full range of these vi
266 unopathology of IM in boys with the X-linked lymphoproliferative disease trait, and as a chronic acti
267 Barr virus strains present in posttransplant lymphoproliferative disease tumors.
268 en), poor compliance (three), posttransplant lymphoproliferative disease, (two), hepatocellular carci
269 aturally infects rhesus macaques and induces lymphoproliferative diseases under experimental conditio
270                     Sixty-five patients with lymphoproliferative diseases underwent sibling (n = 57)
271 t HTLV-1, an etiologic agent associated with lymphoproliferative diseases, uses a conserved accessory
272 ity allogeneic stem cell transplantation for lymphoproliferative diseases using a BEAM-alemtuzumab pr
273                   EBV-related posttransplant lymphoproliferative disease was not observed up to 12 mo
274 ne (1.7%) case of polymorphic posttransplant lymphoproliferative disease was seen, which regressed wi
275 y determine the role of Erk in this delay of lymphoproliferative disease, we also bred a transgenic,
276 aired in T cells from patients with X-linked lymphoproliferative disease, which lack SLAM-associated
277             The boy developed posttransplant lymphoproliferative disease, which resolved after treatm
278 igh-risk recipients developed EBV-associated lymphoproliferative disease, while 2 of the subjects had
279 h adult T-cell leukemia (ATL), an aggressive lymphoproliferative disease with a dismal prognosis.
280      Chronic lymphocytic leukemia (CLL) is a lymphoproliferative disease with a highly variable outco
281 ed homeostatic expansion, which manifests as lymphoproliferative disease with autoantibody production
282 s-like and one Burkitt's-like posttransplant lymphoproliferative disease) with restricted viral gene
283                                     X-linked lymphoproliferative disease (XLP) is a congenital immuno
284                                     X-linked lymphoproliferative disease (XLP) is a primary immunodef
285                                     X-linked lymphoproliferative disease (XLP) is a rare congenital i
286                                     X-linked lymphoproliferative disease (XLP) is a rare immune disor
287        One of the manifestations of X-linked lymphoproliferative disease (XLP) is progressive agammag
288                       Patients with X-linked lymphoproliferative disease (XLP) lack such class-switch
289 C4 mutations, is described to cause X-linked lymphoproliferative disease (XLP) phenotypes.
290  (SAP), is altered in patients with X-linked lymphoproliferative disease (XLP), a primary immunodefic
291           The human genetic disease X-linked lymphoproliferative disease (XLP), which is caused by mu
292 s with the primary immunodeficiency X-linked lymphoproliferative disease (XLP), which is caused by mu
293          Seventeen individuals with X-linked lymphoproliferative disease (XLP), who harbored germline
294          T cells from patients with X-linked lymphoproliferative disease (XLP), who lack functional S
295 ients with the clinical syndrome of X-linked lymphoproliferative disease (XLP).
296 ible for the human genetic disorder X-linked lymphoproliferative disease (XLP).
297 ls from SAP-deficient patients with X-linked lymphoproliferative disease (XLP).
298                                     X-linked lymphoproliferative disease (XLP1) arises from mutations
299 ergistic signalling is defective in X-linked lymphoproliferative disease (XLP1) NK cells entailing 2B
300                                     X-linked lymphoproliferative disease (XLP1), described in the mid

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