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1 ydrazine has a reactivity similar to that of methylamine.
2 isible transmittance) due to dissociation of methylamine.
3 his enzyme is not essential for oxidation of methylamine.
4  times, including the first monoarylation of methylamine.
5  after treatment with a lysosomal inhibitor, methylamine.
6 ely 50% of each cofactor form at 0.8 or 2 mM methylamine.
7 ved with sodium periodate in the presence of methylamine.
8 ted a 1000-fold increase in the Km value for methylamine.
9  fld1 host by selection on plates containing methylamine.
10 adily penetrate germinated spores, including methylamine.
11 rting methyl-Arg to citrulline and releasing methylamine.
12 , none is required for growth on methanol or methylamine.
13 n was irradiated (365 nm) in the presence of methylamine.
14 dergo conformational change by reaction with methylamine.
15 e by other microorganisms not directly using methylamine.
16 al environment that assimilate nitrogen from methylamine.
17 sialic acids by covalent derivatization with methylamine.
18 cid into the corresponding N-protected alpha-methylamine.
19 ld during the growth of M. extorquens PA1 on methylamine.
20 tance of yeast to the toxic transport analog methylamine.
21 otrophs and non-methylotrophs, to metabolize methylamine.
22 yl-dH(4)MPT accumulation, enhances growth on methylamine.
23 own pathways by which methane is formed from methylamines.
24 thylamine:CoM methyl transfer from all three methylamines.
25 ved; and 6) mutual counteraction by urea and methylamines.
26 genesis in Methanosarcina barkeri growing on methylamines.
27 transferase specific for methanogenesis from methylamines.
28 tant generally is reduced by urea and/or the methylamines.
29  80 degrees C, or <1 h in ammonium hydroxide/methylamine (1:1) (AMA) at 80 degrees C).
30 ed an increase in growth factors for glyoxal-methylamine (19% by vol) and methylglyoxal-methylamine (
31 h a homologous series of amines or alcohols (methylamine, 2-methyl-2-aminopropane, methanol, or 2-met
32                Dansylcadaverine (20 microM), methylamine (20 mM), and bacitracin (2 mg/ml) prevented
33 ) and three isomeric 15,16-bisnorpimarenyl-N-methylamines (26a-c) were synthesized and evaluated as a
34 g [ZnTe] slabs and terminal hydrazine (2) or methylamine (3) molecules.
35       Unexpectedly, N-benzyl-N-cyclopropyl-N-methylamine (4) was found not to inactivate P450 and not
36 l-methylamine (19% by vol) and methylglyoxal-methylamine (8% by vol) aerosol, indicating that unusual
37 l permeability to the ammonia analog [(14) C]methylamine (+80%, P < 0.05).
38 n base treatment, this second species formed methylamine, a breakdown product characteristic of symme
39  also, for the first time, hepcidin bound to methylamine-activated alpha2M (alpha2M-MA).
40 iments on glycolaldehyde- and hydroxyacetone-methylamine aerosol found that the aerosol particles wer
41                   Glyoxal- and methylglyoxal-methylamine aerosol particles shattered in Raman microsc
42  these growth factors to immobilized alpha2M-methylamine (alpha2M-MA).
43                    The utilization of (15) N methylamine also led to the release of (15) N ammonium t
44  four different amines: N,N-dimethylamine, N-methylamine, ammonia, and morpholine.
45 -rice showed an excellent gas-sensitivity to methylamine among the four natural pigments sensitized f
46 nted for by interactions between the preQ(1) methylamine and base G5 of the aptamer.
47  covalent incorporation of (14)C from [(14)C]methylamine and benzylamine into PSII subunits has been
48 cleaved with the small-molecule nucleophiles methylamine and histamine, but when Spy0125 was mechanic
49 ce is evident under conditions of saturating methylamine and oxygen with D319E.
50  The D319E mutant catalyzes the oxidation of methylamine and phenethylamine, but not that of benzylam
51                                              Methylamine and phenylethylamine were not determined in
52            Labeling of CP47, D2, and D1 with methylamine and phenylhydrazine approached a one-to-one
53 epend on ion concentration, pH, the specific methylamine and substrate, and identity of even a single
54 but the BL group had reduced urine levels of methylamines and aromatic amino acids metabolites.
55 clusion that inhibition by dansylcadaverine, methylamine, and bacitracin is not due to an alkalinizat
56 nd indoxyl sulfate, but levels of hippurate, methylamine, and dimethylamine were not significantly lo
57 ith similarity to the methanogenic methanol, methylamine, and methanethiol methyltransferases and to
58 yl)furfuryl alcohol, N,N-dimethylthiophene-2-methylamine, and N,N-dimethylbenzylamine.
59 osine, arginine dipeptides, gamma-D-Glu-Gly, methylamine, and others.
60 ses initiate methanogenesis from the various methylamines, and these enzymes are encoded by genes wit
61                      The protein contents in methylamine- and methanol-grown cells showed a significa
62                                              Methylamine- and pyridine-treated films are also p-type.
63 ormyl-4-methylphenol and bis(2-aminoethyl)-N-methylamine) are described.
64 y = N,N-bis(2-pyridylmethyl)-N-bis(2-pyridyl)methylamine) are investigated in comparison to those of
65 hydrazine) and [ZnTe(ma)] (3; ma = MeNH(2) = methylamine) are two-dimensional (2-D) layered structure
66 AAmRe(O)(X)] DAAm = N,N-bis(2-arylaminoethyl)methylamine; aryl = C6F5 (X = Me, 1, COCH3, 2, Cl, 3) as
67 hylotrophs that have to switch between using methylamine as a carbon and energy source or just a nitr
68 n order to use the NMG pathway for growth on methylamine as a carbon and energy source.
69 lamide synthetase is essential for growth on methylamine as a carbon source but not as a nitrogen sou
70 s that can grow on one-carbon compounds, use methylamine as a carbon source.
71 thylotrophic microorganisms may also utilize methylamine as a nitrogen source, but little is known ab
72  pastoris on methanol as a carbon source and methylamine as a nitrogen source.
73 only when cells are grown in the presence of methylamine as a sole carbon source and is repressed by
74  source and certain alkylated amines such as methylamine as nitrogen sources.
75 with ammonium sulfate as nitrogen source) or methylamine as sole nitrogen source (with glucose as car
76 ables rapid growth on high concentrations of methylamine as the primary carbon and energy substrate,
77 hway plays a pivotal role during growth with methylamine as the sole nitrogen source, which we demons
78 grow on reduced single-carbon compounds like methylamine as the sole source of carbon and energy.
79 se complex when the enzyme is incubated with methylamine as the substrate.
80 he primary C-H bonds in linear- and branched-methylamines, as well as secondary C-H bonds in higher d
81              The structures of the molecules methylamine-borane, MeH(2)N.BH(3), and dimethylamine-bor
82 talytic dehydrocoupling/dehydrogenation of N-methylamine-borane, MeNH(2).BH(3) (7), to yield the solu
83   The synthesis and biological activity of a methylamine-bridged enkephalin analogue (MABE) is presen
84 port an association of LysRS1 with growth on methylamine, but not an essential role for LysRS1/LysRS2
85 carbonyl cyanide m-chlorophenylhydrazone and methylamine, but not by spermine, consistent with an act
86 pt levels were also high in media with CO or methylamines, but much lower with methanol.
87 erent substrates (72, 190, and 162 s(-1) for methylamine, butylamine, and benzylamine, respectively).
88 observed, respectively, for the reactions of methylamine, butylamine, and benzylamine.
89  counteraction of urea effects on enzymes by methylamines can depend on ion concentration, pH, the sp
90 we report the effects of 1, 3-cyclohexanebis(methylamine) (CBM) on secretion in vivo, a compound chos
91                                              Methylamine (CH(3)NH(2))-mediated deacylation has previo
92            We report herein the discovery of methylamine (CH3NH2) induced defect-healing (MIDH) of CH
93  vitro ATP-dependent reductive activation of methylamine:CoM methyl transfer from all three methylami
94                 SIP experiments using (15) N methylamine combined with metagenomics and metaproteomic
95                           After cooling, the methylamine complex is re-formed, returning the absorber
96  layer-composed of a metal halide perovskite-methylamine complex-from a transparent state (68% visibl
97 e, but not other common C1 compounds such as methylamine, could support growth.
98 ny of the persisting organisms play roles in methylamine cycling, ultimately supporting methanogenesi
99 nding character of the adjacent C9-O2 to the methylamine (Cys69 backbone).
100                                              Methylamine dehydrogenase (MADH) and amicyanin form a ph
101 tentials for the oxidized/reduced couples of methylamine dehydrogenase (MADH) and aromatic amine dehy
102 pared with the complex of the TTQ-containing methylamine dehydrogenase (MADH) and the cupredoxin amic
103 s for methylamine oxidation: the periplasmic methylamine dehydrogenase (MaDH) and the cytoplasmic N-m
104 tryptophylquinone (TTQ) in substrate-reduced methylamine dehydrogenase (MADH) by amicyanin is known t
105                                              Methylamine dehydrogenase (MADH) catalyzes the oxidative
106                                              Methylamine dehydrogenase (MADH) contains the protein-de
107                          The biosynthesis of methylamine dehydrogenase (MADH) from Paracoccus denitri
108                          The biosynthesis of methylamine dehydrogenase (MADH) from Paracoccus denitri
109                                              Methylamine dehydrogenase (MADH) has been immobilized in
110 factor tryptophan tryptophylquinone (TTQ) in methylamine dehydrogenase (MADH) involves the post-trans
111                                              Methylamine dehydrogenase (MADH) is a tryptophan tryptop
112                                              Methylamine dehydrogenase (MADH) is a tryptophan tryptop
113                     Paracoccus denitrificans methylamine dehydrogenase (MADH) is an enzyme containing
114 yptophan tryptophylquinone (TTQ) cofactor of methylamine dehydrogenase (MADH) is covalently modified
115                                              Methylamine dehydrogenase (MADH) may be immobilized in a
116                                              Methylamine dehydrogenase (MADH) possesses an alpha(2)be
117                          The biosynthesis of methylamine dehydrogenase (MADH) requires formation of s
118 in that mediates electron transfer (ET) from methylamine dehydrogenase (MADH) to cytochrome c-551i.
119 he diheme enzyme MauG and different forms of methylamine dehydrogenase (MADH) were subjected to kinet
120            The scheme is based on the use of methylamine dehydrogenase (MADH) which converts primary
121                             The quinoprotein methylamine dehydrogenase (MADH), type I copper protein
122    Contrary to the TTQ-containing subunit of methylamine dehydrogenase (MADH), which is catalytically
123 yptophan tryptophylquinone (TTQ) cofactor of methylamine dehydrogenase (MADH).
124 inone (TTQ), the protein-derived cofactor of methylamine dehydrogenase (MADH).
125 tural electron acceptor for the quinoprotein methylamine dehydrogenase (MADH).
126 tural electron acceptor for the quinoprotein methylamine dehydrogenase (MADH).
127 activity and the spectroscopic properties of methylamine dehydrogenase (MADH).
128 nal modification of the precursor protein of methylamine dehydrogenase (preMADH) to complete biosynth
129 ional modification of a precursor protein of methylamine dehydrogenase (preMADH) to complete the bios
130 ation of a biosynthetic precursor protein of methylamine dehydrogenase (PreMADH) with partially synth
131 rent from those for the related quinoprotein methylamine dehydrogenase and its associated redox prote
132 ee-dimensional structure of the quinoprotein methylamine dehydrogenase from Paracoccus denitrificans
133 ese data in which the reduction of Cu(2+) by methylamine dehydrogenase is a true ET reaction while th
134 talyzes posttranslational modifications of a methylamine dehydrogenase precursor protein to generate
135 -studied aerobic methylotroph, a periplasmic methylamine dehydrogenase that catalyzes the primary oxi
136 on-transfer reaction from the quinol form of methylamine dehydrogenase to amicyanin.
137 ectron transfer (ET) reactions from O-quinol methylamine dehydrogenase to oxidized native and mutant
138 tophylquinone (TTQ), the prosthetic group of methylamine dehydrogenase, is formed by post-translation
139 or the ET reactions from another TTQ enzyme, methylamine dehydrogenase, to amicyanin.
140  related to M. extorquens AM1 but is lacking methylamine dehydrogenase, to dissect the genetics and p
141  pyruvate:ferredoxin oxidoreductase, and the methylamine dehydrogenase-amicyanin complex.
142                                   Within the methylamine dehydrogenase-amicyanin-cytochrome c-551i co
143 tryptophan tryptophylquinone cofactor within methylamine dehydrogenase.
144 n tryptophylquinone, the prosthetic group of methylamine dehydrogenase.
145 es, methylotrophy is enabled by methanol and methylamine dehydrogenases and their specific electron t
146                             Experiments with methylamine demonstrated that ozonation converts methyla
147 n of Tmm and DMS oxidation in R. pomeroyi is methylamine-dependent and regulated at the post-transcri
148 ergy barrier for the ET from copper to heme, methylamine-dependent reduction of heme by the three-pro
149 try of IPA/NO, we prepared the corresponding methylamine derivative as a sodium salt that was highly
150                           Interestingly, its methylamine derivative, 49, displayed good enzyme inhibi
151 played positive chemotactic responses toward methylamine, dimethylamine, and trimethylamine but did n
152                                 We find that methylamine, dimethylamine, and trimethylamine undergo m
153 small numbers of water molecules to ammonia, methylamine, dimethylamine, and trimethylamine, and thei
154 eration is found to increase the basicity of methylamine, dimethylamine, benzylamine, and N,N-dimethy
155 eration is found to increase the basicity of methylamine, dimethylamine, benzylamine, N,N-dimethylani
156  reports have shown that the organic cations methylamine, dimethylamine, ethylamine, and trimethylami
157 tion of the omega-bromo groups with ammonia, methylamine, dimethylamine, or trimethylamine provided p
158 tal structures have also been determined for methylamine-, dimethylamine-, and trimethylamine-borane,
159  Whereas the poorly nucleophilic ammonia and methylamine do not react, hydroxylamine, methoxylamine,
160  values are affected by the degree of excess methylamine employed.
161  (Arg), and several primary amines including methylamine, ethylamine, n-propylamine, n-butylamine, an
162  from uric acid, glucose, ammonia, caffeine, methylamine, ethylenediamine, hydroxylamine, n-butylamin
163 f homologous nucleophiles (e.g., ammonia and methylamine) facilitates recognition and identification
164 (13)C-labeled glucose by derivatization with methylamine followed by multiple reaction monitoring sca
165 onothio-1,3-diketones with alpha-substituted methylamines, followed by their alpha-nitrosation with s
166 ines as functional groups (e.g., secondary N-methylamines) formed chloropicrin at high yields, likely
167            The usual explanation is that the methylamines found in the renal medulla, namely glycerop
168 t a very mild synthesis of N-protected alpha-methylamines from the corresponding amino acids.
169 ssay conditions (pH and salt concentration), methylamines (glycerophosphocholine, betaine, and trimet
170 terium extorquens AM1, MaDH is essential for methylamine growth, but the NMG pathway has no known phy
171 ne > pyridine > triethylamine > ethylamine > methylamine &gt; diethylamine > tert-butylamine > ammonia.
172 2M, but with the same relative reactivity of methylamine &gt; ethylamine > ammonia.
173 and N,N-bis(2-pyridylmethyl)-N-bis(2-pyridyl)methylamine, has been proposed to attack C-H bonds by an
174 ylglyoxal and the primary amines glycine and methylamine have been determined.
175                            However, urea and methylamines have the similar (not counteracting) effect
176 ich prevents acidification of endosomes) and methylamine HCl (which neutralizes acidification of endo
177 t (4-bromo-3,6-dimethoxybenzocyclobuten-1-yl)methylamine hydrobromide (TCB-2) enhanced C-fiber-evoked
178 s (4-bromo-3,6-dimethoxybenzocyclobuten-1-yl)methylamine hydrobromide (TCB-2).
179 st 4-bromo-3,6-dimethoxybenzocyclobuten-1-yl)methylamine hydrobromide, binds less to the mGlu2 promot
180 5,5-bis-(4-fluorophenyl)tetrahydrofuran-2-yl]methylamine hydrochloride, NCC1048) in a model of hypoxi
181 respect to nucleophilic addition to ammonia, methylamine, hydroxylamine, methoxylamine, and hydrazine
182        Increasing vesicular pH with internal methylamine in hair cells also abolished the transient b
183 rom the dark reactions of methylglyoxal with methylamine in simulated evaporated cloud droplets.
184        In HPAO-2, an inflated (D)k(cat)/K(m)(methylamine) in relation to (D)k(cat)/K(m)(benzylamine)
185 C3), we coupled the free sulfhydryl group of methylamine-inactivated C3 to a thiolSepharose matrix.
186                                       [(14)C]Methylamine incorporation showed the k(cat)/K(m)((app))
187                                 The K(m) for methylamine increases from 9 microM to 15 mM.
188                                              Methylamine-induced thin-film transformation at room-tem
189 ion in the adenine base to incorporate [(3)H]methylamine into the synthesis of [(3)H]MRS2279 to high
190               In HPAO-1, the k(cat)/K(m) for methylamine is 330-fold greater than for benzylamine, wh
191                                  kcat/Km for methylamine is found to be 80-fold reduced compared to t
192              Archaeal methane formation from methylamines is initiated by distinct methyltransferases
193 is(2-[2-(N',N'-4-dimethylamino)pyridyl]ethyl)methylamine) is described, to model aspects of the chemi
194 (N4Py: N,N-bis(2-pyridylmethyl)bis(2-pyridyl)methylamine), is the least basic oxidant.
195 ve results acquired from peptide mapping and methylamine labeling.
196 The method employs neutron encoded (NeuCode) methylamine labels ((13)C or (15)N enriched) that are af
197 nd the second a bis-tert-butyl-substituted N-methylamine ligand.
198 -N-(2,2,2-trifluoroethylsulfonyl)-pyrid-3-yl methylamine (LY487379) is a selective positive allosteri
199 id (OxA) on NPF from the reaction of MSA and methylamine (MA) at 1 atm and 294 K in the presence and
200 rimethylamine (TMA), dimethylamine (DMA) and methylamine (MA) in fish.
201            RhAG also conferred resistance to methylamine (MA), a toxic analog of ammonium, and expres
202 h trimethylamine (TMA), dimethylamine (DMA), methylamine (MA), and ammonia over the range of 21-28 de
203  with the primary and secondary alkylamines: methylamine (MA), dimethylamine (DMA), and ethylamine (E
204 th the radioactive analog of ammonium [(14)C]methylamine (MA), had an apparent EC(50) of 1.6 mm and a
205  mtaCB3 fusions was delayed, suggesting that methylamines may repress their expression.
206 No measurable amounts of cadaverine (CAD) or methylamine (MEA) were found, showing no spoilage sympto
207 (tfepma = (bis[bis(trifluoroethoxy)phosphino]methylamine, MeN(P[OCH2CF3]2)2), have been prepared by t
208 l2CN(t)Bu (9) (dfpma = bis(difluorophosphino)methylamine, MeN(PF2)2).
209 lized and the effects of Etn, MeEtn, Me2Etn, methylamine (MeNH2), and dimethylamine (Me2NH) were stud
210 hylated ethanolamines (MeEtn and Me2Etn) and methylamines (MeNH2, Me2NH) were used as Etn models that
211 type of DeltappylT reveals the deficiency in methylamine metabolism expected of a Methanosarcina spec
212 ysine derivative encoded by the UAG codon in methylamine methyltransferase genes of Methanosarcina ba
213                                         Each methylamine methyltransferase methylates a cognate corri
214  is encoded by a single amber (UAG) codon in methylamine methyltransferase transcripts.
215                  Genes encoding methanogenic methylamine methyltransferases all contain an in-frame a
216 vealed only four protein families, including methylamine methyltransferases and transposases.
217 have been characterized previously, a set of methylamine methyltransferases in which Pyl is assumed t
218                                         Only methylamine methyltransferases matched the Pyl trait and
219 codon in genes encoding proteins such as the methylamine methyltransferases present in some Archaea a
220 etry in A. arabaticum proteins including two methylamine methyltransferases.
221 rtion of pyrrolysine into the active site of methylamine methyltransferases.
222  were comparable with those determined using methylamine-modified alpha(2)M, suggesting that higher-o
223 n the presence of excess substrate, the next methylamine molecule initiates a nucleophilic attack of
224 pe, the DeltappylT strain cannot grow on any methylamine, nor use monomethylamine as sole nitrogen so
225 anomalous basicity effect of ammonia and the methylamines on going from the gas phase to aqueous solu
226 d two analogs of ammonium (hydroxylamine and methylamine) on ANR activity in soil slurries.
227 atial arrangement, namely to the interior of methylamine or ammonia-treated alpha(2)M and to the exte
228  ectoderm cultured in the presence of either methylamine or ammonium chloride.
229  prepared from nitrophenyl carbamate 14a and methylamine or directly from 5-aminoimidazole-4-carboxam
230  Aminolyses of these isoquinolinediones with methylamine or ethanolamine produced the isoquinolinedio
231 he compound 3 reacts at normal pressure with methylamine or ethylamine, forming N-alkylpyridinium sal
232                                 In contrast, methylamine or the inhibitor of macroautophagy, 3-methyl
233      In carbonyl compound reactions with AS, methylamine, or AS/glycine mixtures, product absorbance
234 ature experiments, regardless of whether AS, methylamine, or glycine was present.
235 pheric aldehydes with ammonium sulfate (AS), methylamine, or glycine.
236 lly or chemically in solutions of hydrazine, methylamine, or pyridine to produce electronically coupl
237 Native MADH exhibits a strong preference for methylamine over longer carbon chain amines.
238                               kcat for D319E methylamine oxidase is reduced 200-fold compared to that
239                    Profiles for WT-catalyzed methylamine oxidation and Y305A-catalyzed ethylamine oxi
240 mutation had no effect on the parameters for methylamine oxidation by MADH, but significantly affecte
241 ection of proteins for the N-methylglutamate methylamine oxidation pathway that appears to be auxilia
242 omparable, while profiles of Y305A-catalyzed methylamine oxidation suggest the pH-dependent build-up
243 ination of the steady state species of D319E methylamine oxidation, in combination with the kinetic d
244 actor to yield a mature enzyme competent for methylamine oxidation.
245 , such as the N-methylglutamate pathway, for methylamine oxidation.
246 cally relevant N-methylglutamate pathway for methylamine oxidation.
247 cterium extorquens AM1 encode two routes for methylamine oxidation: the periplasmic methylamine dehyd
248 hotoactive layers rapidly decompose yielding methylamine, PbI2 , and I2 as products.
249 c amine permeases and may therefore encode a methylamine permease.
250                         Ammonium transporter/methylamine permease/rhesus (AMT/Mep/Rh) transporters ar
251 led that aniline and aliphatic amines (e.g., methylamine) prefer to attack C8 of intermediate 4a, aff
252                          Methanogenesis from methylamines, probably stemming from degradation of bact
253 ing at approximately 40% RH for all aldehyde-methylamine products.
254 shown that an mtdB mutant is able to grow on methylamine, providing a system to study the role of Mtd
255 ns,trans,trans-[Pt(N3 )2 (OH)2 (MA)(Py)] (MA=methylamine, Py=pyridine).
256 t linearly with increasing concentrations of methylamine (r=0.931).
257                                              Methylamine reduction at acidic or neutral pH has reveal
258                                     However, methylamine reduction at pH 8.5 has revealed a copper-li
259           The studies with hydroxylamine and methylamine showed that both of these ammonium analogs i
260 hyltransferase activity, including MtbA, the methylamine-specific CoM methylase and the pyl operon re
261 acetivorans C2A encode putative methanol- or methylamine-specific MT2 enzymes.
262 comes significantly depleted compared to the methylamine starting material.
263 ion of Km of wild-type enzyme by urea and/or methylamines that is partially additive, whereas at the
264 omotropic weak bases (NH4Cl, chloroquine, or methylamine) that increased lysosomal pH and sensitized
265                         For t-butylamine and methylamine, the amount of labeling increased when PSII
266 es in aquatic habitats on the utilization of methylamine, the simplest methylated amine, have mainly
267 pid immobility in and the slow permeation of methylamine through the inner membrane of dormant spores
268 sintegration, either by serum treatment with methylamine to block C4 and C3 split product binding or
269 itrogen during its two-electron reduction by methylamine to form an aminoquinol (N-quinol).
270 MADH) catalyzes the oxidative deamination of methylamine to formaldehyde and ammonia.
271 cterium extorquens AM1 oxidizes methanol and methylamine to formaldehyde and subsequently to formate,
272 nase that catalyzes the primary oxidation of methylamine to formaldehyde has been examined in great d
273 ylamine demonstrated that ozonation converts methylamine to nitromethane at approximately 100% yield.
274 ion and (b) S(N)2 substitution reaction with methylamine to provide diamine 14 with inversion of conf
275 laterally separated by 120 A, whereas in the methylamine-transformed alpha2M, the epitopes are at the
276                    Hypochlorite oxidation of methylamine-treated alpha2M (alpha2M*), an analogue of t
277                 The most significant drop in methylamine uptake was seen for the ump2 and the ump1ump
278  while ump1 disruption only slightly reduced methylamine uptake.
279 tory and assimilatory modules suggested that methylamine use via the N-methylglutamate pathway requir
280 B-like and sox-like genes play a key role in methylamine utilization and encode N-methylglutamate syn
281 ded by the amber codon in genes required for methylamine utilization by members of the Methanosarcina
282 rrectly assembled MADH, eight genes from the methylamine utilization gene cluster of P. denitrificans
283 lete genome of a model obligate methanol and methylamine utilizer, Methylobacillus flagellatus (strai
284                     The draft genomes of two methylamine utilizers were obtained and their metabolism
285 teomics facilitated identification of active methylamine-utilizing Alpha- and Gammaproteobacteria.
286 re firstly exposed to atmospheres containing methylamine vapours with concentrations over the range 2
287 ibition of ANR activity by hydroxylamine and methylamine was due to formation of the glutamine analog
288 btained and their metabolism with respect to methylamine was examined.
289 yano-induced cleavage of cysteinyl proteins, methylamine was found to be superior to ammonia for cyan
290         With FXIIIa or tTG catalysis, [(14)C]methylamine was incorporated into Q2A-alpha(2)AP, indica
291 participating in coenzyme M methylation with methylamines, was not inhibited by dimethylsulfide and d
292 he improved NPF descriptions for ammonia and methylamines, we placed special focus on the potential t
293 series of N-substituted (2-phenylcyclopropyl)methylamines were designed and synthesized, with the aim
294 l reacted (presumably on chamber walls) with methylamine with a rate constant k = (9 +/- 2) x 10(-17)
295  Both forms are rapidly reduced by substrate methylamine with a rate constant of 199 s(-1) but behave
296 mann-type coupling reactions of methanol and methylamine with iodobenzene by beta-diketone- and 1,10-
297 uctive amination of an epoxy aldehyde with N-methylamine with subsequent intramolecular oxirane ring
298 ethanosarcina spp. begin methanogenesis from methylamines with methyltransferases made via the transl
299 ion showed highest reporter gene activity on methylamines with much lower expression on CO or methano
300 f lysK or lysS grew normally on methanol and methylamines with wild-type levels of monomethylamine me
301 mational change upon thiol ester cleavage by methylamine, with the presence of crosslinks correlating

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