ライフサイエンスコーパス: methylmercury

1 toxic solutes (e.g., monomeric aluminum and methylmercury).

2 s bioavailability and thus the production of methylmercury.

3 are consuming fish heavily contaminated with methylmercury.

4 ment of the fluorescence quenching caused by methylmercury.

5 ich these microorganisms produce and degrade methylmercury.

6 sediments containing coal ash was present as methylmercury.

7 particularly if the metal is in the form of methylmercury.

8 consumption of fish high in PUFAs and low in methylmercury.

9 ffect of PUFA on MI risk was counteracted by methylmercury.

10 uptake that leads to the formation of toxic methylmercury.

11 highly prone to form inorganic mercury from methylmercury.

12 dria within the astrocytes to the effects of methylmercury.

13 ansform reactive ionic mercury to neurotoxic methylmercury.

14 ict the conversion of Hg(II) to bioavailable methylmercury.

15 al moderating effect of prenatal exposure to methylmercury.

16 have sulfate-reducing bacteria that produce methylmercury.

17 ry and convert the oxidized Hg to neurotoxic methylmercury.

18 t can be subsequently transformed into toxic methylmercury.

19 organisms is a key step in the production of methylmercury, a biomagnifiable toxin.

20 re a complex process, with the production of methylmercury, a potent human neurotoxin, repeatedly dem

21 Humans are exposed to methylmercury, a well-established neurotoxin, through fi

22 The pollutant methylmercury accumulates within lean tissues of birds a

23 to controls at PND60, 47 days after the last methylmercury administration.

24 ock, 6-10% of total mercury was converted to methylmercury after one day.

25 A detection limit of 5.9 nM methylmercury and a repeatability expressed as a relativ

26 method is described for the determination of methylmercury and butyltin compounds in marine sediment

27 ME-GC-MIP-AES method, enrichment factors for methylmercury and butyltin compounds of 50-100 were achi

28 e materials (CRMs) with certified values for methylmercury and butyltin compounds.

29 e found between carapace length and mercury, methylmercury and cadmium concentrations, and between fa

30 oil and cardiovascular risk, (2) effects of methylmercury and fish oil on early neurodevelopment, (3

31 lean tissue during flight which may mobilize methylmercury and increase circulating levels of this ne

32 Hydrophobicity of methylmercury and its ability to facilitate a nonradiati

33 0.1, and 0.6 parts per million (ppm) dietary methylmercury and measured changes in blood total mercur

34 fish consumption have frequently focused on methylmercury and omega-3 fatty acids, not persistent po

35 r nutrients, but because of contamination by methylmercury and other toxicants, higher fish intake of

36 Uniform depth distributions of methylmercury and oxygen encountered in the poorly strat

37 PCBs may account for the adverse effects of methylmercury and the degree to which co-exposure to doc

38 tudies incorporating information on both the methylmercury and the docosahexaenoic acid contained wit

39 ent matrixes were possible at 1-2 microg/kg (methylmercury) and 10-100 ng/kg (butyltins).

40 ty of environmental factors such as alcohol, methylmercury, and maternal seizure activates HSF1 in ce

41 blood (1999-2004) predominantly represented methylmercury, and urine (1999-2002) represented inorgan

42 nhibition of cystine uptake in astrocytes by methylmercury appears to be due to actions on the System

43 omponent of the mercury cycle that maintains methylmercury at low concentrations in natural waters.

44 red to express merBpe may be used to degrade methylmercury at polluted sites and sequester Hg(II) for

45 Methylmercury, at low levels generally considered safe,

46 For example, methylmercury bioaccessibility decreased significantly a

47 Methylmercury bioaccessibility varied between 10 (octopu

48 Methylmercury bioaccumulation in zooplankton is higher t

49 al of total mercury by 600 nmol m(-2) and of methylmercury by 214 nmol m(-2) in the Gotland Deep, pro

50 events and human activities and converts to methylmercury by anaerobic organisms.

51 tion, thus affecting the production of toxic methylmercury by PCA cells.

52 The production of methylmercury by some bacteria is a key first step in th

53 Here we show for the first time that methylmercury can be produced in particles sinking throu

54 sent at low concentrations in the substrate, methylmercury can biomagnify to concentrations that pois

55 er, depended on water constituents that bind methylmercury cations.

56 We investigated microbial methylmercury (CH(3)Hg) production in sediments from the

57 l mesocosms and measured the uptake of toxic methylmercury (CH3 200Hg+) and inorganic 201Hg2+ by biot

58 Methylmercury (CH3Hg(+)) is the common form of organic m

59 d is present in either inorganic [Hg(II)] or methylmercury (CH3Hg) form.

60 y in foods, in inorganic form [Hg(II)] or as methylmercury (CH3Hg), can have adverse effects.

61 Inorganic Hg(II) and methylmercury ([CH3Hg(II)](+)) are commonly complexed wi

62 , which was approximately 3 times higher for methylmercury chloride than for thimerosal.

63 for the three species mercury(II) chloride, methylmercury chloride, and thimerosal after intoxicatio

64 In contrast, methylmercury-chloride complexes, which are dominant in

65 ansformations likely contributed to the high methylmercury concentration found in settling particles.

66 y (0.236+/-0.1001mg/kg(-1)), and the highest methylmercury concentration was found at the Labe - Obri

67 thers, we detected significantly higher mean methylmercury concentrations and higher proportions of s

68 Methylmercury concentrations decreased by 77% in underly

69 of total mercury concentrations to estimate methylmercury concentrations in bird eggs.

70 Methylmercury concentrations in clams and snails also de

71 south arm decreased by approximately 81% and methylmercury concentrations in deep waters decreased by

72 mercury in bird eggs have actually measured methylmercury concentrations in eggs.

73 centrations in eggs are a reliable proxy for methylmercury concentrations in eggs.

74 Total mercury and methylmercury concentrations in surface waters were decr

75 eviously implied connection between elevated methylmercury concentrations in the deep brine layer and

76 high-spatial-resolution depiction of surface methylmercury concentrations in this area.

77 ent geochemical data suggested that elevated methylmercury concentrations occurred in regions where n

78 expressing both genes grow on 50-fold higher methylmercury concentrations than wild-type plants and u

79 and settling particles were not significant, methylmercury concentrations were about ten-fold greater

80 Methylmercury concentrations were determined by gas chro

81 cana) and Forster's terns (Sterna forsteri), methylmercury concentrations were highly correlated (R(2

82 Total mercury and methylmercury concentrations were measured in the settli

83 stratification, exacerbating high biological methylmercury concentrations.

84 r study and that co-exposure to nutrients in methylmercury-contaminated fish may have obscured and/or

85 for AIE-based fluorescence imaging study on methylmercury-contaminated live cells and zebrafish for

86 sed in mercury monitoring programs to assess methylmercury contamination and toxicity to birds.

87 f this work was to determine the mercury and methylmercury content in muscle tissue of chub (Leuciscu

88 Similarly, methylmercury content in the Emory and Clinch River sedi

89 Methylmercury cycling in the Pacific Ocean has garnered

90 Methylmercury demethylation rate constants (kd) were of

91 ral cortical astrocytes were pretreated with methylmercury, either 1 microM for 24 h, or 10 microM fo

92 r day is a scientifically justified level of methylmercury exposure for maternal-fetal pairs.

93 nic release of mercury into the environment, methylmercury exposure from fish consumption is a pathwa

94 imental neurocognitive effects from prenatal methylmercury exposure from maternal fish consumption du

95 Their prenatal methylmercury exposure had been assessed from the mercur

96 Although prenatal methylmercury exposure has been linked to poorer intelle

97 may predate clinical disease by years; thus, methylmercury exposure may be relevant to future autoimm

98 We developed equations to estimate methylmercury exposure of piscivorous birds and sport fi

99 However, prenatal methylmercury exposure seemed to attenuate these positiv

100 Clinically evident neurologic damage from methylmercury exposure was well described following pois

101 In the group with lower prenatal methylmercury exposure, a 1 SD increase in VO2Max was as

102 in delta(13)C over time and delta(15)N with methylmercury exposure, year remained a significant inde

103 was observed in the group with high prenatal methylmercury exposure.

104 e compared groups with low and high prenatal methylmercury exposure.

105 Better models included filtered water methylmercury (FMeHg) or unfiltered water methylmercury

106 oil on early neurodevelopment, (3) risks of methylmercury for cardiovascular and neurologic outcomes

107 ether the percentage of total mercury in the methylmercury form differed among species.

108 The mean percentage of total mercury in the methylmercury form in eggs was 97% for American avocets

109 The percentage of total mercury in the methylmercury form ranged from 63% to 116% among individ

110 The net potential for methylmercury formation, assessed by the ratio between t

111 above the maximum allowable limit with toxic methylmercury found as the dominant mercury species with

112 Exposure to methylmercury from fish consumption has been linked to a

113 In practice, contamination by methylmercury from fish consumption is assessed by measu

114 Exposure to methylmercury from fish has been associated with increas

115 Methylmercury from seafood, ethylmercury used as a bacte

116 e morphology, genes, or proteins involved in methylmercury generation.

117 (13)C]lactate was decreased in the 10 microM methylmercury group.

118 H(sebenzim(Me)) also reacts with methylmercury halides, but the reaction is accompanied b

119 thylation of inorganic mercury to neurotoxic methylmercury has been attributed to the activity of ana

120 ) as optical nanoprobes for the detection of methylmercury has been developed.

121 rovide a basis for assessing and remediating methylmercury hotspots in the environment.

122 combination of cadmium chloride (CdCl2) and methylmercury (II) chloride (CH3HgCl) (0, 0.125, 0.5, or

123 Health effects of low-level methylmercury in adults are not clearly established; met

124 A first step toward bioaccumulation of methylmercury in aquatic food webs is the methylation of

125 Elevated levels of neurotoxic methylmercury in Arctic food-webs pose health risks for

126 hey may play a key role in the production of methylmercury in biofilms.

127 t step in the production and accumulation of methylmercury in biota.

128 o examine the relationship between total and methylmercury in eggs of two species, and (2) reviewing

129 e SPME-GC-MIP-AES method was used to measure methylmercury in four marine tissue CRMs ranging from oy

130 rcury, we compared the elimination of [203Hg]methylmercury in GGT-deficient mice with that in wild-ty

131 er techniques to assign certified values for methylmercury in oyster, mussel, and fish tissue CRMs.

132 spill, with a specific focus on mercury and methylmercury in sediments.

133 The production of the neurotoxic methylmercury in the environment is partly controlled by

134 that cause the production and degradation of methylmercury in the environment is ultimately needed to

135 layer in the accumulation and persistence of methylmercury in the Great Salt Lake.

136 the bioaccumulation of the potent neurotoxin methylmercury in the marine food web.

137 vity of anaerobic bacteria, the formation of methylmercury in the oxic water column of marine ecosyst

138 ic sea ice can harbour a microbial source of methylmercury in the Southern Ocean.

139 The production of methylmercury in these areas is a concern because this n

140 The extent of exposure to methylmercury in US women of reproductive age is not kno

141 atalase (1000 U/ml) significantly attenuated methylmercury-induced inhibition of 3H-aspartate uptake,

142 nitrone (PBN), or catalase to attenuate the methylmercury-induced inhibition of aspartate uptake.

143 t oxidation of the transporter might mediate methylmercury-induced inhibition of glutamate transport.

144 This study suggests that methylmercury-induced overproduction of H2O2 is a mechan

145 pected from reservoir creation will increase methylmercury inputs to the estuary by 25-200%, overwhel

146 important and changing source of mercury and methylmercury into the Arctic Ocean marine ecosystem.

147 Methylmercury is a global pollutant of aquatic ecosystem

148 Methylmercury is a highly toxic, organic derivative foun

149 Methylmercury is a potent neurotoxin produced in natural

150 Methylmercury is an environmental pollutant that biomagn

151 Methylmercury is an environmental toxicant that biomagni

152 sediments where NOM can be reduced and toxic methylmercury is formed.

153 Our findings indicate that methylmercury is mobilized from lean tissues during prot

154 Methylmercury is one of the more toxic forms of mercury

155 Methylmercury is the environmental form of neurotoxic me

156 son collections coincided with uniformly low methylmercury levels along the river downstream from the

157 gnificant independent covariate with feather methylmercury levels among the albatrosses.

158 he Pacific basin exhibit temporal changes in methylmercury levels consistent with historical global a

159 Methylmercury levels declined gradually to 200 km downst

160 Methylmercury levels in larvae, however, were much less

161 In general, the methylmercury levels in plankton and fishes downstream f

162 We investigated the seasonal variation of methylmercury levels in the Balbina reservoir and how th

163 ratification of the reservoir influenced the methylmercury levels in the reservoir and in the river d

164 Higher methylmercury levels observed 200-250 km downstream from

165 fied, and anoxic hypolimnion water with high methylmercury levels was exported downstream.

166 e dramatic mercury loss from deep waters and methylmercury loss from underlying sediment in response

167 DHA appears beneficial for, and low-level methylmercury may adversely affect, early neurodevelopme

168 rcury in adults are not clearly established; methylmercury may modestly decrease the cardiovascular b

169 To assess if methylmercury-mediated inhibition of 3H-aspartate transp

170 Methylmercury (MeHg(+) ) is one of the most potent neuro

171 sted for removal of mercury species [Hg(2+), methylmercury (MeHg(+)), ethylmercury (EtHg(+)), and phe

172 tate uptake were studied as indices of acute methylmercury (MeHg) (5 microM) cytotoxicity.

173 In this study, the biodilution hypothesis of methylmercury (MeHg) accumulation was examined in a Hg-c

174 m, respectively, to study the effect on the methylmercury (MeHg) accumulation.

175 Methylmercury (MeHg) affects wildlife and human health m

176 Human exposure to methylmercury (MeHg) and elemental mercury vapor (Hg(0)(

177 stem-scale study examining the production of methylmercury (MeHg) and greenhouse gases from reservoir

178 posed here to compare toxicity mechanisms of methylmercury (MeHg) and inorganic mercury (iHg) in musc

179 atographic method was developed to determine methylmercury (MeHg) and inorganic mercury (iHg) levels

180 The toxic effects of methylmercury (MeHg) are attributable, at least in part,

181 enyls (PCBs), organochlorine pesticides, and methylmercury (MeHg) are environmentally persistent with

182 orption data especially for mercury (Hg) and methylmercury (MeHg) at the low porewater concentrations

183 Exposure to methylmercury (MeHg) before birth can adversely affect c

184 divalent mercury (Hg(II)) is transformed to methylmercury (MeHg) by anaerobic microbes.

185 The production of methylmercury (MeHg) by anaerobic microorganisms depends

186 ct of the seasonal inundation of wetlands on methylmercury (MeHg) concentration dynamics in the Amazo

187 er delta to quantify total mercury (THg) and methylmercury (MeHg) concentrations and export.

188 position, along with total mercury (THg) and methylmercury (MeHg) concentrations and fluxes, to decre

189 Methylmercury (MeHg) concentrations can increase by 1000

190 Rapid growth could significantly reduce methylmercury (MeHg) concentrations in aquatic organisms

191 To better understand the source of elevated methylmercury (MeHg) concentrations in Gulf of Mexico (G

192 We present a case study comparing metrics of methylmercury (MeHg) contamination for four undeveloped

193 a: Anisoptera: Gomphidae) as biosentinels of methylmercury (MeHg) contamination.

194 Methylmercury (MeHg) determinations in hake, its food-ch

195 isease (FMD), which is caused by exposure to methylmercury (MeHg) during development, many neurons ar

196 s-independent fractionation (MDF and MIF) of methylmercury (MeHg) during trophic transfer into fish.

197 t produce a dramatic acceleration of urinary methylmercury (MeHg) excretion in poisoned animals, but

198 Developmental methylmercury (MeHg) exposure alters dopamine neurotrans

199 Seafood consumption is the primary route of methylmercury (MeHg) exposure for most populations.

200 disturbances are hallmarks of developmental methylmercury (MeHg) exposure, but the molecular mechani

201 limination of inorganic mercury [Hg(II)] and methylmercury (MeHg) in a marine fish, Terapon jarbua.

202 Elevated concentrations of methylmercury (MeHg) in freshwater ecosystems are of maj

203 s of high concentrations of mercury (Hg) and methylmercury (MeHg) in mangroves, in conjunction with t

204 Net formation of methylmercury (MeHg) in sediments is known to be affecte

205 Rice is known to accumulate methylmercury (MeHg) in the rice grains.

206 d dietary fish consumption, from which daily methylmercury (MeHg) intake was estimated.

207 Methylmercury (MeHg) is a known neuro-toxicant.

208 Methylmercury (MeHg) is a neurotoxic compound that threa

209 Methylmercury (MeHg) is a potent neurotoxin that has bee

210 Methylmercury (MeHg) is a potent neurotoxin that is biom

211 Methylmercury (MeHg) is an established neurotoxicant of

212 Fluvial methylmercury (MeHg) is attributed to methylation in up-

213 Methylmercury (MeHg) is highly neurotoxic with an appare

214 production of the bioaccumulative neurotoxin methylmercury (MeHg) is stimulated in newly flooded soil

215 Methylmercury (MeHg) is the only species of mercury (Hg)

216 The neurotoxicity of high levels of methylmercury (MeHg) is well established both in humans

217 on the fatty acid profile, mercury (Hg), and methylmercury (MeHg) levels of salmon was studied.

218 Methylmercury (MeHg) may affect fetal growth; however, p

219 A suggested mechanism of methylmercury (MeHg) neurotoxicity in brain involves the

220 f the environmental cerebellar neurotoxicant methylmercury (MeHg) on spontaneous IPSCs (sIPSCs) of bo

221 Methylmercury (MeHg) production by ND132 increased linea

222 gions where sloughed Cladophora accumulates, methylmercury (MeHg) production is enhanced.

223 lack of estimation and comparison of the net methylmercury (MeHg) production or degradation in these

224 the resulting short and long-term changes in methylmercury (MeHg) production.

225 atic systems and accumulated as highly toxic methylmercury (MeHg) represents a threat to wildlife and

226 Inorganic Hg is readily converted to toxic methylmercury (MeHg) that bioaccumulates in aquatic food

227 To understand the biochemistry of methylmercury (MeHg) that leads to the formation of merc

228 Maternal transfer of methylmercury (MeHg) to eggs is an important exposure pa

229 tion exists on their potential as sources of methylmercury (MeHg) to freshwaters.

230 ells over-expressing MT-I to withstand acute methylmercury (MeHg) treatment was measured by the relea

231 proposed to investigate inorganic (iHg) and methylmercury (MeHg) trophic transfer and fate in a mode

232 urrent frequency caused by the neurotoxicant methylmercury (MeHg) was examined in Purkinje cells of c

233 ion, and magnitude of hydrological fluxes of methylmercury (MeHg), a bioavailable Hg species of ecolo

234 Mercury (Hg) is of particular interest as methylmercury (MeHg), a neurotoxin which bioaccumulates

235 fate-reducing bacterium capable of producing methylmercury (MeHg), a potent human neurotoxin.

236 educing bacterium (SRB) capable of producing methylmercury (MeHg), a potent human neurotoxin.

237 portant link between the global contaminant, methylmercury (MeHg), and human exposure through consump

238 cosystems are contaminated with highly toxic methylmercury (MeHg), but the specific sources and pathw

239 ed in the environment, and its organic form, methylmercury (MeHg), can extensively bioaccumulate and

240 dology for the simultaneous determination of methylmercury (MeHg), ethylmercury (EtHg), and inorganic

241 ations of both total Hg (THg) and especially methylmercury (MeHg), the species of Hg having the highe

242 tu amendments for remediation of mercury and methylmercury (MeHg), using a study design that combined

243 The formation of methylmercury (MeHg), which is biomagnified in aquatic f

244 al into the highly bioaccumulated neurotoxin methylmercury (MeHg).

245 es leading to significant bioaccumulation of methylmercury (MeHg).

246 onstituents and often display high levels of methylmercury (MeHg).

247 AOSR) of Canada contain elevated loadings of methylmercury (MeHg; a neurotoxin that biomagnifies thro

248 The sources of methylmercury (MeHg; the toxic form of mercury that is b

249 atmospheric pollutants, epilimnetic aqueous methylmercury (MeHgaq) and mercury in small yellow perch

250 Mercury (Hg) methylation and methylmercury (MMHg) demethylation activity of periphyto

251 dy of a method for the determination of mono methylmercury (MMHg) in foodstuffs of marine origin by g

252 lue mussel, killifish, eider) to investigate methylmercury (MMHg) sources and exposure pathways in fi

253 ion energy of the carbon-mercury bond on the methylmercury molecule6-7 and subsequently increased rea

254 The effect of methylmercury on glutamate metabolism was studied by (13

255 d astrocytes and neurons, and the effects of methylmercury on this transport were assessed.

256 traction techniques commonly used to extract methylmercury or mercury species from various matrixes h

257 potential to transform inorganic mercury to methylmercury, or vice versa, during sample preparation

258 Chronic exposure to low levels of methylmercury (organic) and inorganic mercury is common,

259 5 years of age were estimated for chemicals (methylmercury, organophosphate pesticides, lead) and a v

260 able for only three environmental chemicals (methylmercury, organophosphate pesticides, lead), the re

261 Our results explain methylmercury photodecomposition rates that are relative

262 icals as developmental neurotoxicants: lead, methylmercury, polychlorinated biphenyls, arsenic, and t

263 The neurotoxic heavy metal methylmercury potently and specifically inhibits the tra

264 Elevated human exposure to methylmercury primarily results from consumption of estu

265 orthern ecosystems and reservoir flooding on methylmercury production and bioaccumulation through a c

266 ing bacteria are responsible for the rate of methylmercury production and thus bioaccumulation in mar

267 However, net methylmercury production in cultures exposed to nanopart

268 y from the wastes but also the potential for methylmercury production in receiving waters.

269 s offer new insights into the role of DOM in methylmercury production in the environment.

270 Methylmercury production rates are generally related to

271 o risk-benefit model on the basis of data on methylmercury, PUFA, and MI risk has yet been presented.

272 at studies on bioaccumulation should measure methylmercury rather than total mercury when using museu

273 be how exposure to both marine n-3 PUFAs and methylmercury relates to MI risk by using data from Finl

274 levels ( approximately 40,000 ng . g(-1)) of methylmercury relative to prior time points, suggesting

275 be 0.2 and 0.1 ng g(-1) for mercuric ion and methylmercury, respectively.

276 s characterized for the Hg-C protonolysis of methylmercury rule out the direct protonation mechanism

277 benthic sediment was thought to be the main methylmercury source for coastal fish.

278 and modeling show that currently the largest methylmercury source is production in oxic surface seawa

279 vations in freshwater lakes) applied only to methylmercury species bound to organic sulfur-containing

280 ects of prenatal fish consumption as well as methylmercury suggest there are benefits from prenatal f

281 may experience greater surges in circulating methylmercury than demonstrated here as a result of thei

282 ons may also generate a higher proportion of methylmercury than more oligotrophic environments.

283 total mercury and, for a subset of samples, methylmercury (the bioaccumulated form of mercury) in mu

284 because fish are the primary source of toxic methylmercury to humans.

285 Some published methods converted methylmercury to inorganic mercury approximately 100% (i

286 enhanced transfer of accumulated mercury and methylmercury to the planktonic food chain and finally t

287 males, because breeding females can depurate methylmercury to their eggs.

288 thus decreases in GSH synthesis may increase methylmercury toxicity.

289 tion-based methods induced very little or no methylmercury transformation to inorganic mercury.

290 er methylmercury (FMeHg) or unfiltered water methylmercury (UMeHg), whereas filtered total mercury di

291 stantially to models explaining the observed methylmercury variation.

292 ish can also be used to estimate the loss of methylmercury via photoreduction in aquatic ecosystems.

293 A filter-passing methylmercury vs DOC relationship was also developed usi

294 Exposure to methylmercury was associated with increased risk of MI,

295 In other methods, as much as 45% of methylmercury was converted to inorganic mercury during

296 Exposure to methylmercury was shown to decrease neural stem cell pop

297 role of GGT in the whole-body disposition of methylmercury, we compared the elimination of [203Hg]met

298 humans and wildlife is the net production of methylmercury, which occurs mainly in reducing zones in

299 dual Hg species (inorganic Hg, ehtylmercury, methylmercury) with inductively coupled plasma mass spec

300 ve been caused by production of bioavailable methylmercury within those wetlands.

301 een assay is achieved for quick detection of methylmercury without the use of tedious sample preparat