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1 (GRE)-luciferase reporter gene activity and mitogen-activated protein kinase phosphatase 1 (MKP-1) a
2 gene expression revealed that Ang II induced mitogen-activated protein kinase phosphatase 1 (MKP-1) e
4 , inactivation and subsequent degradation of mitogen-activated protein kinase phosphatase 1 (MKP-1) i
5 this report, we examined the involvement of mitogen-activated protein kinase phosphatase 1 (MKP-1) i
6 a pro-survival phosphatase, dual specificity mitogen-activated protein kinase phosphatase 1 (MKP-1),
7 We report here the identification of a gene, mitogen-activated protein kinase phosphatase 1 (MKP-1),
8 ed dephosphorylation of ERKs due to elevated mitogen-activated protein kinase phosphatase 1 (MKP-1).
9 vity and expression of phosphatases, such as mitogen-activated protein kinase phosphatase 1 and prote
10 ein scanning, we identified up-regulation of mitogen-activated protein kinase phosphatase 1 as an eff
11 ied the nuclear dual-specificity phosphatase mitogen-activated protein kinase phosphatase 1 as the en
12 pression and a decrease in HDAC activity and mitogen-activated protein kinase phosphatase 1 expressio
13 cantly correlated with dexamethasone-induced mitogen-activated protein kinase phosphatase 1 expressio
14 lular signal-regulated kinase 1/2, increased mitogen-activated protein kinase phosphatase 1 expressio
15 beta-induced CXCL8 release, (2) induction of mitogen-activated protein kinase phosphatase 1 gene expr
16 e suppression of CXCL8 release, induction of mitogen-activated protein kinase phosphatase 1 gene expr
18 -signal-regulated kinases 1/2, a less severe mitogen-activated protein kinase phosphatase 1 increase,
19 lved in the regulation of apoptosis, and the mitogen-activated protein kinase phosphatase 1 that func
22 CD8(+) compared with CD4(+) cells when using mitogen-activated protein kinase phosphatase (1 hour; P
24 sis factor-alpha-induced protein 3 (A20) and mitogen activated protein kinase phosphatase-1 were dete
25 1 was attenuated in cells that overexpressed mitogen activated protein kinase phosphatase-1, indicati
26 cells because it stimulated GC induction of mitogen-activated protein kinase phosphatase-1 (MKP-1) a
27 we have demonstrated that mice deficient in mitogen-activated protein kinase phosphatase-1 (MKP-1) e
29 ious studies identified a potential role for mitogen-activated protein kinase phosphatase-1 (MKP-1) i
33 urther studies reveal that the expression of mitogen-activated protein kinase phosphatase-1 (MKP-1) w
35 eas AMPK inhibition decreased, the levels of mitogen-activated protein kinase phosphatase-1 (MKP-1),
37 The dual-specific phosphatase 1 (DUSP 1), mitogen-activated protein kinase phosphatase-1 (MKP-1),
41 ERK1/2 activation was mediated by repressing mitogen-activated protein kinase phosphatase-1 (MKP-1),
42 radation of the JNK-inactivating phosphatase mitogen-activated protein kinase phosphatase-1 (MKP-1).
43 he mitogen-activated protein kinase pathway, mitogen-activated protein kinase phosphatase-1 (MKP-1).
44 nd GC-inducible protein kinase-1 (SGK-1) and mitogen-activated protein kinase phosphatase-1 (MKP-1).
45 iation of connexin43 with c-Src, ERK1/2, and mitogen-activated protein kinase phosphatase-1 and by de
46 8) mol/L) enhanced FP (10(-9) mol/L)-induced mitogen-activated protein kinase phosphatase-1 mRNA (5.8
47 ibits glucocorticoid induction of the MKP-1 (mitogen-activated protein kinase phosphatase-1) and seru
48 es either by enhanced degradation (i.e., for mitogen-activated protein kinase phosphatase-1) or as sh
49 egative Ras or the ERK-specific phosphatase, mitogen-activated protein kinase phosphatase-1, blocked
51 e 1/2 and phospho-ERK 1/2, and expression of mitogen-activated protein kinase-phosphatase-1 (MKP-1: p
53 We identified the DUSP4 gene, which encodes mitogen-activated protein kinase phosphatase 2 (Mkp2), a
54 de, preopro-urotensin II-related peptide and mitogen-activated protein kinase phosphatase 2 was valid
55 icant differences between insulin-stimulated mitogen-activated protein kinase phosphatase-2, protein-
59 found that 308 keratinocytes highly express mitogen-activated protein kinase phosphatase-3 (MKP-3),
60 ic epithelial cells, we examined the role of mitogen-activated protein kinase phosphatase 5 (MKP5) in
61 with JNK1 outcompetes JNK1 association with mitogen-activated protein kinase phosphatase 5, resultin
62 beta-arrestin-bound ERK1/2 is protected from mitogen-activated protein kinase phosphatase activity.
63 ich was inhibited by cotransfection with the mitogen-activated protein kinase phosphatases CL100 and
65 stream signaling via increased expression of mitogen-activated protein kinase phosphatase MKP-1 in bo
67 rs of dual-specificity phosphatases: CDC25B, mitogen-activated protein kinase phosphatase (MKP)-1 and
68 In this study, we examined the regulation of mitogen-activated protein kinase phosphatase (MKP-1) exp
69 MEK activity but abrogated the expression of mitogen-activated protein kinase phosphatase (MKP-1) nor
70 dual specificity phosphatases referred to as mitogen-activated protein kinase phosphatases (MKP).
75 roteins that contribute to this process, the mitogen-activated protein kinase phosphatases (MKPs), di
76 profiles indicated a rapid induction of the mitogen-activated protein kinase phosphatase (MPK)-1 and
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