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1 d N297D/S298A-IYG optimally drove tumor cell phagocytosis.
2 ecific, as opposed to general alterations in phagocytosis.
3 rform the critical physiological function of phagocytosis.
4 of interrelationships between chemotaxis and phagocytosis.
5 s essential for effector functions including phagocytosis.
6   This caused a disruption of Fc-binding and phagocytosis.
7 FcgammaRIIIA dimers were the main drivers of phagocytosis.
8 e in the optical force arises independent of phagocytosis.
9 ot resting macrophages following silica bead phagocytosis.
10 LMO1 preferentially regulated LC3-associated phagocytosis.
11 ncentrations and deterioration in neutrophil phagocytosis.
12 ctor 1-alpha and was essential for increased phagocytosis.
13 d many strategies to resist opsonization and phagocytosis.
14 y out functions that are analogous to animal phagocytosis.
15 eta-amyloid (Abeta) clearance independent of phagocytosis.
16 toskeletal processes required for macrophage phagocytosis.
17 etal remodeling as a way to block macrophage phagocytosis.
18  undergo complex, multiphasic changes during phagocytosis.
19 (POS) through a multistep process resembling phagocytosis.
20 n gliomas by internalizing the virus through phagocytosis.
21 lar material internalized by endocytosis and phagocytosis.
22 f neutrophils under conditions that preclude phagocytosis.
23  C5 split product needed for upregulation of phagocytosis.
24  The antibodies raised were found to promote phagocytosis.
25 tor-signaling is not essential for bacterial phagocytosis.
26 ts were limited to Fcgamma receptor-mediated phagocytosis.
27 ut also to detect and retain bacteria during phagocytosis.
28 tions in macrophages, namely TNF release and phagocytosis.
29 le expression, and capacity for professional phagocytosis.
30 o alter host actin dynamics and thus cripple phagocytosis.
31 by cancer cells directly protected them from phagocytosis.
32 ors and by up-regulating monocyte/macrophage phagocytosis.
33 ed the ability of RAW 264.7 cells to perform phagocytosis.
34 thus promotes hyper-inflammation and weakens phagocytosis.
35 creased survival in whole human blood due to phagocytosis.
36 irming that trogocytosis occurs, rather than phagocytosis.
37 , transgene-expressing cells from microglial phagocytosis.
38 ling, reactive oxygen species production and phagocytosis.
39  in cooperation and independently to mediate phagocytosis.
40 vation including nitric oxide production and phagocytosis.
41 e or reactive oxygen species production, and phagocytosis.
42 ed siRNA, using glucan particles taken up by phagocytosis.
43 inflammation and antibody-dependent cellular phagocytosis.
44 ular cytotoxicity, and Ab-dependent cellular phagocytosis.
45  CaMK4 recapitulated the observed defects in phagocytosis.
46 m pathways, and inhibited cell migration and phagocytosis.
47 and to discriminate between trogocytosis and phagocytosis.
48 ing proliferation, survival, clustering, and phagocytosis.
49 rotein alpha (SIRPalpha) controls macrophage phagocytosis.
50 bp gene, as a strong regulator of microglial phagocytosis.
51                                        After phagocytosis, A. fumigatus conidia rapidly escaped from
52 eads to cellular effector functions, such as phagocytosis, Ab-dependent cellular cytotoxicity, and cy
53                                              Phagocytosis activity of blood-borne macrophages decreas
54                                      Opsonic phagocytosis activity was strongly associated (hazard ra
55 t 4, 12, and 18 months of age for macrophage phagocytosis activity, ocular histological changes, and
56 3, CD4bs, and gp41 for Ab-dependent cellular phagocytosis (ADCP) activity, implicated in protective i
57 ses by enhancing antibody-dependent cellular phagocytosis (ADCP) in xenograft models.
58             Antibody-dependent cell-mediated phagocytosis (ADCP) was rarely detected during AIM (4 of
59 ether they can mediate Ab-dependent cellular phagocytosis (ADCP), which is an important element of an
60  cord microglia lacking ABCD1 are primed for phagocytosis, affecting neurons within an altered metabo
61                                              Phagocytosis after myocardial infarction (MI) is a prere
62 cytes and keratinocytes were used to examine phagocytosis and bacterial invasion, respectively.
63     DJ-1(-/-) macrophages exhibited enhanced phagocytosis and bactericidal activity in vitro, and ado
64 ranulation of primary granules and inhibited phagocytosis and bactericidal activity of neutrophils, w
65  and Alzheimer disease (AD) are defective in phagocytosis and degradation amyloid beta1-42 (Abeta1-42
66              C1q opsonization also increased phagocytosis and efferocytosis in macrophage foam cells.
67                                   Inhibiting phagocytosis and endosomal acidification in BMDCs or moD
68                                 However, CR3 phagocytosis and fibronectin haptotaxis, both integrin-d
69 ible roles of JN in RPE molecular transport, phagocytosis and formation of outer blood-retinal barrie
70 Yop proteins into the cytoplasm that prevent phagocytosis and generation of proinflammatory cytokines
71 I) are defective in amyloid-beta1-42 (Abeta) phagocytosis and have low resistance to apoptosis by Abe
72 decrease in the macrophage capacity for NTHi phagocytosis and increased nasopharyngeal bacterial load
73 ossibility that platelets enhance macrophage phagocytosis and intracellular killing of S. aureus In t
74 es further defined the connection between OS phagocytosis and ketogenesis in wild-type mice and mice
75 pathogens have developed strategies to evade phagocytosis and killing by neutrophils.
76                                              Phagocytosis and killing of NTHi by macrophages were eva
77 on pathogenic mechanism involving macrophage phagocytosis and microglial synaptic pruning, and raises
78 ount of consideration of the contribution of phagocytosis and other host defenses in the research for
79                   Monocytes were studied for phagocytosis and oxidative burst.
80 eticulin, the mBiNE stimulated HER2-targeted phagocytosis and produced durable antitumour immune resp
81           Host mechanisms controlling fungal phagocytosis and replication remain obscure.
82 d, following a therapeutic dosage, activates phagocytosis and resolution signals in type 2 diabetes.
83 dogenous diabetic overexpression, activating phagocytosis and resolution signals.
84  show multimodal dynamics of PtdIns4P during phagocytosis and suggest that the phosphoinositide plays
85 ing processes we describe are LC3-associated phagocytosis and targeting by autophagy proteins, both o
86 s as ones that participate in B. burgdorferi phagocytosis and the resulting cytokine activation.
87 te M1-M2 phenotype that is optimal for Abeta phagocytosis and the stabilization of cognitive decline.
88  mechanisms by which these receptors mediate phagocytosis and to identify signaling pathways activate
89 rther show that specific cellular processes (phagocytosis and transendothelial migration) are enriche
90  Indeed, three antibodies displayed cellular phagocytosis and/or antibody-dependent cell-mediated cyt
91 tes play a central role in pathogen sensing, phagocytosis, and antigen presentation and consist of mu
92 tworks involved in lamellipodial protrusion, phagocytosis, and cell adhesion.
93  immune system involved in the opsonization, phagocytosis, and destruction of microorganisms infectin
94                         Macrophage motility, phagocytosis, and differentiation in vivo are thus coupl
95 ) demonstrate decreased autophagy, augmented phagocytosis, and enhanced scavenger receptor (macrophag
96            Complement-mediated opsonization, phagocytosis, and immune stimulation are critical proces
97 onal survival, outgrowth, synaptogenesis and phagocytosis, and induce the death of neurons and oligod
98 urther enhances MFGE8 expression, aggravates phagocytosis, and leads to neuronal injury.
99 , upregulated molecular pathways involved in phagocytosis, and prevented the renal function decline a
100 lism as well as augmented size, granularity, phagocytosis, and respiratory burst.
101 epithelial macrophage projections, efficient phagocytosis, and stabilized enteroid barrier function r
102 ebbishields fused with immune cells to evade phagocytosis, and the resultant hybrid cells exhibited i
103  by necroptotic cells drives recognition and phagocytosis, and this may limit the inflammatory respon
104 n, antibody-dependent cellular cytotoxicity, phagocytosis, and virion capture).
105 species (ROS) production, degranulation, and phagocytosis are normal in the absence of STIM2, suggest
106 ts ligand Gas6, astrocytic genes involved in phagocytosis, are upregulated after acute sleep deprivat
107      MiR-142(-/-) neutrophils showed altered phagocytosis as a consequence of chemotactic behavior, i
108  EhRab35 is involved in the initial stage of phagocytosis as well as in the phagolysosomal biogenesis
109         WGA-Fc opsonization increased fungal phagocytosis, as well augmented the antifungal functions
110 infected erythrocytes (IE); however, current phagocytosis assays use IE collected from infected indiv
111 onfirmed by immunoblot analyses and in vitro phagocytosis assays.
112                    We further found that the phagocytosis-associated protein MERTK was significantly
113       Lack of P2X7 receptor function reduced phagocytosis at all ages compared to wild-type mice.
114 y cytokines, miRNAs, and functions including phagocytosis, autophagy, and cell metabolism.
115  involvement of skin barrier and endocytosis/phagocytosis/autophagy, in addition to known innate and
116 rescence lifetimes would correlate well with phagocytosis because phagosomes become acidified and the
117 e T3S protein PcrV did not enhance bacterial phagocytosis but did enhance killing of the few bacteria
118                  We hypothesized that myelin phagocytosis by astrocytes is an early event during lesi
119 We find that androgen regulates Sertoli cell phagocytosis by controlling expression of miR-471-5p and
120 exhibit delays in two processes that require phagocytosis by glial cells, the immune cells in the bra
121 le MER), a critical biomarker of compromised phagocytosis by innate macrophages.
122 omplement on the bacterial surface, promoted phagocytosis by macrophages and neutrophils, and protect
123 ed protein response and improve amyloid-beta phagocytosis by macrophages of patients with mild cognit
124                                              Phagocytosis by macrophages plays a critical role in can
125 esis of this study was that active Abeta1-42 phagocytosis by macrophages prevents brain amyloidosis a
126 ng Pf phage, P. aeruginosa was less prone to phagocytosis by macrophages than bacteria not producing
127 al-3 to bind and opsonize the live cells for phagocytosis by microglia.
128 to CD47-mediated protection of exosomes from phagocytosis by monocytes and macrophages.
129 eath of the neutrophils and their subsequent phagocytosis by monocytes and macrophages.
130 diator, resolvin D1, in vitro increase Abeta phagocytosis by Mvarphis of patients with MCI.
131 on of neutrophils in the nerve, and elevated phagocytosis by neutrophils.
132 sed survival compared with WT bacteria after phagocytosis by neutrophils.
133 riments, we could not detect any significant phagocytosis by purified PMNs of anti-CD20-opsonized CLL
134 und that MARCO was involved in C. neoformans phagocytosis by resident pulmonary macrophages and DC.
135       Both phenotypes are linked to impaired phagocytosis by specialized phagocytes: Sertoli cells an
136  Pharmacological inhibition of ATG-dependent phagocytosis by the cardiac glycoside neriifolin, an inh
137 h serum components and undergoes substantial phagocytosis by the reticuloendothelial system, causing
138 ut not A53T pMac, show significantly reduced phagocytosis capability and this can be phenocopied by a
139 CDCC) and complement-dependent cell-mediated phagocytosis (CDCP) by immunological effector molecules
140 rives evasion of chromosomal instability and phagocytosis checkpoints by apoptotic cancer stem cells.
141 oid cells, including inflammatory responses, phagocytosis, chemokine secretion, and proangiogenic act
142 nd MEK/ERK pathways in the regulation of RPE phagocytosis, confirmed by immunoblot analyses and in vi
143                                   Failure of phagocytosis could be induced experimentally by incubati
144 zation, antibodies mediate functions such as phagocytosis, cytotoxicity, and maintenance of immune ho
145                       Because LC3-associated phagocytosis delivers cargo for degradation, the contrib
146 new phagocyte functions after apoptotic cell phagocytosis demonstrates the enormity of ways to mediat
147 hare mechanistic pathways for chemotaxis and phagocytosis; Dictyostelium chemotax toward bacteria and
148 k underscore the physiologic significance of phagocytosis during tissue injury.
149 ovel linkage between genomic instability and phagocytosis evasion that is coordinated by the blebbish
150 ability, blebbishield emergency program, and phagocytosis evasion to offer poor prognosis.
151 ximal epitope, whereas Ab-dependent cellular phagocytosis favored an epitope positioned further away.
152 zheimer's disease and MCI, possess effective phagocytosis for Abeta and protect homeostasis of the br
153 oideum This social amoeba kills bacteria via phagocytosis for nutrient acquisition at its single-cell
154 ne-rod dystrophy, while interfering with the phagocytosis function of RPE associated with down-regula
155 ange of actin-dependent functions, including phagocytosis, granule exocytosis, and migration.
156 As a result, understanding the mechanisms of phagocytosis has been an area of great interest in the f
157 ctions of CD36 in parasite sequestration and phagocytosis have been clearly defined, less is known ab
158 receptors that participate in B. burgdorferi phagocytosis have been reported, including the scavenger
159                                          RPE phagocytosis helps maintain the viability of photorecept
160 e defined the core signature of macrophages, phagocytosis imprinted a distinct antiinflammatory profi
161 ere we report a primitive form of artificial phagocytosis in a binary community of synthetic protocel
162 K) cell activity and neutrophil and monocyte phagocytosis in a concentration- and species-dependent m
163 ABCD1 in microglial activity toward neuronal phagocytosis in cell culture.
164 ll pathogenicity, and identify ATG-dependent phagocytosis in DCs as a key regulator in driving autoim
165 ired protein enzymes allowing the control of phagocytosis in macrophages.
166  to an increase in the initial efficiency of phagocytosis in milk.
167 enhancing clearance of debris via macrophage phagocytosis in multiple tumor types.
168 al role of NLRP3 in modulating autophagy and phagocytosis in neutrophils and suggest that therapies s
169 should be targeted to modulate autophagy and phagocytosis in neutrophils to control bacterial burden
170 solution markers CD163+CD206], and Abeta1-42 phagocytosis in patients initially diagnosed as having M
171 tween plant and animal PCD is the absence of phagocytosis in plants.
172 bined results indicated that early stages of phagocytosis in the RPE are mainly characterized by pron
173         The regulatory mechanisms underlying phagocytosis in the RPE are not fully understood, althou
174 ssion, and augmented mAb-mediated tumor cell phagocytosis in vitro However, only STINGa reversed the
175 ve engorge-and-accumulate processes in vivo, phagocytosis in vitro inhibited macrophage migration thr
176                                    Abeta1-42 phagocytosis increased in both ApoE groups (P = 0.03 in
177 ses of signaling-networks for chemotaxis and phagocytosis indicate that chemoattractant receptor-sign
178 s many LPS responses, including endocytosis, phagocytosis, inflammation, and pyroptosis.
179  RNA-like ER kinase (PERK) expression, Abeta phagocytosis, intermediate M1-M2 Mvarphi type, and a Min
180 gingivalis (Pg) capsule enables evasion from phagocytosis, invasion of keratinocytes, and bacterial s
181                                        Thus, phagocytosis is a source of macrophage heterogeneity tha
182 breakdown, indicating that astroglial myelin phagocytosis is an early and prominent feature.
183                                              Phagocytosis is an essential process for the proliferati
184                            Antibody-mediated phagocytosis is an important immune effector mechanism a
185 ical model, we postulate that if the rate of phagocytosis is great enough, for acute, normally self-l
186                                         Cell phagocytosis is impaired in type 2 diabetes and requires
187 /Syk/ROS/NLRP3 pathway during L. amazonensis phagocytosis is important for macrophage restriction of
188 s sense apoptotic cell death and discuss how phagocytosis is integrated with environmental cues to dr
189                                              Phagocytosis is normally inhibited by binding of cell su
190 in classical autophagy, while LC3-associated phagocytosis is ULK1 independent.
191 ytic receptor(s) responsible for tumour cell phagocytosis is(are) largely unknown.
192                  It has been shown to hinder phagocytosis, is retained in tumors, and aids in cellula
193 how that Sertoli cells employ LC3-associated phagocytosis (LAP) by recruiting autophagy member protei
194                               LC3-associated phagocytosis (LAP) is a novel form of non-canonical auto
195 e of apoptotic germ cells via LC3-associated phagocytosis (LAP).
196 phagy called "Light-chain 3 (LC3)-associated phagocytosis" (LAP), lacked such defects.
197 l-trafficking pathway called "LC3-associated phagocytosis" (LAP).
198 ella from polymyxins, mediates resistance to phagocytosis, limits the activation of inflammatory resp
199 ese DE genes showed enrichment for ribosome, phagocytosis, lysosome, proteasome, oxidative phosphoryl
200  phagocytosing Exserohilum Despite a lack of phagocytosis, macrophage production of tumor necrosis fa
201                                   Astrocytic phagocytosis, mainly of presynaptic components of large
202  al. show that the inflammatory response and phagocytosis mediated by the interaction between protect
203 tracellular signaling pathways that increase phagocytosis-mediated bacterial clearance, survival, and
204                      One of these functions, phagocytosis, mediates the natural disposal of billions
205 es, chemokines, inflammatory regulators, and phagocytosis mediators, are involved in prion pathogenes
206 g array of genes, including genes related to phagocytosis, metabolism, and retinal disease in humans.
207 mbrane protrusions, were first implicated in phagocytosis more than 100 years ago, but little is stil
208                   In contrast, inhibition of phagocytosis nearly completely blocks NET release to bot
209 viously described function of CD47 in normal phagocytosis of aging red blood cells and results report
210 nflammatory genes and consistently depressed phagocytosis of amyloid-beta1-42 (Abeta) by monocytes an
211 Ns) have previously been reported to mediate phagocytosis of anti-CD20-opsonized B cells from patient
212 PMNs mediate mostly trogocytosis rather than phagocytosis of anti-CD20-opsonized CLL B cells, and we
213 eptor antagonist polyinosinic acid inhibited phagocytosis of apoptotic cells by CD138(+) MPhi.
214                                              Phagocytosis of apoptotic cells is thus coupled to speci
215 icient dendritic cells displayed hyperactive phagocytosis of apoptotic cells, which stimulated excess
216 y reprogramming of macrophages following the phagocytosis of apoptotic cells.
217  neurodegenerative microglia phenotype after phagocytosis of apoptotic neurons.
218 oreover, aging decreases alveolar macrophage phagocytosis of apoptotic neutrophils, downregulates the
219 that is required for reporter activation and phagocytosis of axonal debris.
220 ase II, and this enzymatic activity inhibits phagocytosis of B. pertussis in vitro.
221 itivity to bacterial infection and decreased phagocytosis of bacteria by systemic immune cells.
222                    Ex vivo airway neutrophil phagocytosis of bacteria was reduced in patients with in
223 hat Fmr1 plays a cell-autonomous role in the phagocytosis of bacteria.
224 significantly enhanced peritoneal macrophage phagocytosis of both methicillin-resistant S. aureus and
225  molecule able to mediate the engulfment and phagocytosis of C. albicans cells by human immune cells
226 aused cell death of cancer cells followed by phagocytosis of cell debris by MPhi.
227 D47 is a cell surface molecule that inhibits phagocytosis of cells that express it by binding to its
228 /8 activation would be predicted to increase phagocytosis of circulating ICs, while disarming their i
229 nated off-target antibody-dependent monocyte phagocytosis of cynomolgus monkey platelets, and cynomol
230                                              Phagocytosis of daily shed photoreceptor outer segments
231 ow-derived Cd36 was essential for both early phagocytosis of dying cardiomyocytes and for smaller inf
232 photoreceptor cell layer, proliferation, and phagocytosis of dying cells.
233 leukocyte activation, without affecting host phagocytosis of E. coli RA101295 treatment reduced plasm
234 hich are involved in iron metabolism and the phagocytosis of erythrocytes and blood-borne pathogens a
235                FlnA deletion does not affect phagocytosis of Escherichia coli or Staphylococcus aureu
236                                   Neutrophil phagocytosis of F. nucleatum ssp. polymorphum was signif
237                                   Neutrophil phagocytosis of F. nucleatum strains and neutrophil apop
238                                              Phagocytosis of filamentous bacteria occurs through tubu
239  cell-specific receptors, we determined that phagocytosis of haematopoietic tumour cells during SIRPa
240  that macrophages are much more efficient at phagocytosis of haematopoietic tumour cells, compared wi
241 t of FcgRIIA, shifting neutrophils away from phagocytosis of ICs toward the programmed form of necros
242 ngly, productive viral replication decreased phagocytosis of IgG-opsonized bioparticles and Fc recept
243                          These findings link phagocytosis of injured oligodendrocytes, a pathological
244 ide presentation to CD4(+) T cells following phagocytosis of injured, phosphatidylserine-exposing oli
245 ctivation, which was accompanied by enhanced phagocytosis of Klebsiella GPVI-depleted mice showed inc
246  data show that PTC124 was able to reinstate phagocytosis of labeled photoreceptor outer segments at
247  Mtb aggregates caused macrophage death, and phagocytosis of large aggregates was more cytotoxic than
248 RvD2-DRV2 interaction significantly enhanced phagocytosis of live Escherichia coli, an action depende
249  Myeloid receptor CD36 is required for early phagocytosis of myocardial infarcts and induction of Nr4
250   This suggested that CERKL may regulate the phagocytosis of OSs by the retinal pigment epithelium (R
251 complement system in the recognition and the phagocytosis of PapMV nanoparticles and identified an un
252  of phagocytic receptors and showed enhanced phagocytosis of parasite-infected erythrocytes than thos
253 f this finding, CD19-DE was found to enhance phagocytosis of patient-derived ALL blasts by human macr
254 ron-like) cells, and it increased microglial phagocytosis of PC12 cells or primary neurons, which was
255                       LPS-induced microglial phagocytosis of PC12 was prevented by small interfering
256 , and exhibited marked functional defects in phagocytosis of photoreceptor outer segments.
257 ent with lactadherin significantly increased phagocytosis of platelets by approximately 2-fold, dimin
258                             Most strikingly, phagocytosis of POS by cultured RPE cells was almost com
259                                  LPS-induced phagocytosis of primary neurons by primary microglia was
260 ndolysosomes against damage initiated by the phagocytosis of silica beads.
261 EL-induced PTX3 promoted the association and phagocytosis of Staphylococcus aureus into macrophages.
262 GNIFICANCE STATEMENT We find that astrocytic phagocytosis of synaptic elements, mostly of presynaptic
263 icroglial activation and enhanced microglial phagocytosis of synaptic elements, without obvious signs
264                                              Phagocytosis of the Lyme disease-causing pathogen Borrel
265                                     Notably, phagocytosis of the pathogen activates the host autophag
266  of either MHC class I or LILRB1 potentiated phagocytosis of tumor cells both in vitro and in vivo, w
267               MERTK has an essential role in phagocytosis, one of the major functions of the RPE.
268 2/3 complex is not a general requirement for phagocytosis or chemotaxis but is a critical driver of i
269  of Mertk (-/-) mice it fails to enhance RPE phagocytosis or prevent photoreceptor degeneration.
270                                     Blocking phagocytosis or specific phagocytic receptors may allevi
271 otype, which expressed molecules involved in phagocytosis, oxidative injury, antigen presentation and
272  as well as evidence of defective macrophage phagocytosis (p = 0.029).
273 ish-derived omega-3 emulsion increased Abeta phagocytosis, PERK expression, and UPR RNA signature, an
274 ory and have impaired Pseudomonas aeruginosa phagocytosis, phenocopying CF MPhis.
275 Cd36 deficiency led to reduced expression of phagocytosis receptor Mertk and nuclear receptor Nr4a1 i
276 s and mice with AMN, upregulation of several phagocytosis-related markers, such as MFGE8 and TREM2, p
277 radation of bacteria also attenuated further phagocytosis, resulting in elevated bacterial load.
278 eless able to observe inhibition of platelet phagocytosis several days after hexameric-Fc dosing.
279 r 2 (HER2) expressed by cancer cells and pro-phagocytosis signalling mediated by calreticulin, the mB
280 tors such as IgG1, IgG2, IgM, IL-6 and PMPhi phagocytosis, stimulation of secretion of IL-10.
281 s and to mediate antibody-dependent cellular phagocytosis, suggesting a range of anti-HIV immunologic
282                      A mathematical model of phagocytosis suggests that hFcgammaRI and hFcgammaRIIIA
283  IL-2 following initiation of actin-mediated phagocytosis that leads to Src and Syk kinase activation
284         These events promote a rapid form of phagocytosis that produces an "immunologically silent" c
285 EM2 may regulate microglial inflammation and phagocytosis through coupling to the adaptor protein TYR
286 he Yops, YopO (also known as YpkA) obstructs phagocytosis through disrupting actin filament regulatio
287 ophages to maintain respiratory burst during phagocytosis via enhancing mitochondrial complex-II meta
288 abling Gal-3 binding to PC12 cells and their phagocytosis via MerTK.
289                                              Phagocytosis was augmented by IgG opsonization, and inhi
290                                              Phagocytosis was circadianally regulated and mediated by
291                                              Phagocytosis was enhanced in the presence of BEV + RES c
292 ation of PA in macrophages during frustrated phagocytosis was examined using these PA sensors and was
293 ost SLAM receptor functions, SLAMF7-mediated phagocytosis was independent of signalling lymphocyte ac
294        In this study, we examined whether OS phagocytosis was linked to ketogenesis.
295                            However, monocyte phagocytosis was normal and oxidative burst was augmente
296 rmed Golgi-derived vesicles recruited during phagocytosis were secretory vesicles as their recruitmen
297     In the latter mixed infection, Fn evaded phagocytosis, whereas in the capsulated mixed infection
298 ygen species (ROS) production, as well as by phagocytosis, which sequesters pathogens within phagosom
299                  We found that shortly after phagocytosis, wild-type (WT) L. monocytogenes escaped fr
300 he processes of spreading onto surfaces, and phagocytosis within 100 s of stimulation.

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