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1 ndard diagnostic methods (ImmunoCAP and skin prick test).
2 , and to be careful about negative result of prick test.
3 ants with allergic mothers positive for skin prick test.
4 ISA inhibition, basophil activation and skin prick test.
5 = 0.02), and with similar findings for skin prick tests.
6 tient's allergic triggers or performing skin prick tests.
7 thma Study completed questionnaires and skin prick tests.
8 Atopy was assessed by using skin prick tests.
9 Atopy was assessed using skin prick tests.
10 erved pasteurized raw egg challenge and skin prick tests.
11 atic reintroduction analysis but not by skin-prick tests.
12 6 years by specific IgE assessments and skin prick tests.
13 l examinations including serum IgEs and skin prick tests.
14 ne challenge tests, questionnaires, and skin prick tests.
15 ophils and elicit positive responses in skin prick tests.
16 tients assigned to immediate or delayed skin prick testing.
17 tatus were determined by spirometry and skin prick testing.
18 rat basophil leukaemia cell line and by skin prick testing.
19 th IL-31 and NaCl (negative control) by skin prick testing.
20 attend research clinics and consent to skin-prick testing.
21 erwent a clinical examination including skin prick testing.
22 hinitis but without positive results on skin prick testing.
23 were examined for eczema and underwent skin prick testing.
24 ic IgE was 10.1% (95% CI: 9.4-10.8) and skin prick test 2.7% (95% CI: 2.4-3.0), food challenge positi
26 stic testing, 47.3% was assessed with a skin prick test, 39.9% with a serum specific IgE test, and 20
27 allergens: OR = 1.81, 95% CI 0.80-4.24; skin prick test/4+ allergens: OR = 2.27, 95% CI 1.34-3.95).
30 , and allergic disease was evaluated by skin prick test and clinical examination at 12 months of age.
32 AID in history were tested first with a skin prick test and if negative challenged with the culprit N
39 d for selected cases where the history, skin prick test and/or specific IgE are not definitive for th
40 vention (structured allergy history and skin prick testing and appropriate advice on allergy avoidanc
41 tervention (structured allergy history, skin prick testing and appropriate allergy avoidance advice)
43 ined as one or more positive results on skin prick testing and clinically relevant symptoms of rhinit
44 kers measured, small wheal diameters on skin-prick testing and increases in egg-specific IgG4 antibod
45 correlates included end point titration skin prick testing and measurement of CM-specific IgE and IgG
49 aking a structured allergy history with skin prick testing and tailored advice on allergy avoidance r
51 lergic subjects (group 4) by performing skin prick tests and APTs with rBet v 1 and hypoallergenic rB
55 aking a structured allergy history with skin prick tests and giving tailored advice on allergy avoida
58 Associations with allergic diseases, skin prick tests and lung function assessed at 12 and 18 year
59 Clinic evaluation, which consisted of skin prick tests and OFC where eligible, was undertaken if st
62 d 5276 infants (74% participation) with skin prick tests and sensitized infants underwent food challe
65 ites was diagnosed longitudinally using skin prick tests and specific IgE measurements at (1/2), 1(1/
66 , because of the limited sensitivity of skin prick tests and specific IgE tests to meat extracts.
69 d consent; evidencing of an allergen by skin prick tests and/or serum-specific IgE dosages; being abl
70 atopy (grass, house dust mite, and cat skin prick test) and atopic vs. non-atopic asthma at the age
71 y outcomes were desensitization, peanut skin prick test, and specific IgE and specific IgG4 measureme
73 -demographic questionnaire, spirometry, skin prick test, and specific IgE to aeroallergens were done
75 Opishorchis felineus and specific IgE, skin prick testing, and atopic symptoms in Western Siberia, w
76 interviewer-administered questionnaire, skin prick testing, and measurement of lung function from the
77 ar in children positive and negative on skin prick testing, and were not appreciably altered by the e
79 voluntary hyperpnea challenge, allergy skin prick tests, and bronchoscopy with bronchial biopsies.
80 c immunoglobulin E-antibodies in serum, skin prick tests, and double-blind, placebo-controlled food c
82 e of testing (specific IgE measurement, skin prick tests, and oral food challenges), and the timing a
83 allergic sensitization were measured by skin prick tests, and physician-diagnosed inhalant and food a
87 itive ELISA results correlated with the skin prick test areas with the whole body and the setae extra
89 (5-17 years old) with asthma underwent skin prick tests at baseline and had clinical data collected
90 In two patients who showed positive skin prick test but negative for challenge test, titer of spe
91 itization (FAS) was identified by using skin prick tests conducted between 1 and 18 years of age to a
103 outcomes examined: at least 1 positive skin prick test from the panel of 10 allergens (21.7% vs. 11.
104 d at age 12 months: food sensitization (skin prick test >/= 2 mm) and allergy (oral food challenge) t
107 ve set of recommendations on the use of skin prick tests in allergic rhinitis-conjunctivitis and asth
109 etermine whether C+ assayed by means of skin prick tests influenced AR symptom severity in controlled
111 unction samples, we performed histamine skin prick tests, investigated the contribution of STAT3 to a
115 -bronchodilator spirometry (n = 1,389), skin prick testing, lung volumes, and diffusing capacity meas
118 Inclusion criteria included a positive skin prick test of 6 mm or more (wheal diameter, above the ne
119 med challenge test in 41 cases with positive prick test of Glupearl 19S(R), a major allergic HWP foun
120 which was determined with the use of a skin-prick test--one consisting of participants with no measu
121 appear to contain factors that enhance skin prick test or atopy patch test responses to house dust m
122 ding detection of milk-specific IgE (by skin prick test or serum assay), diagnostic elimination diet,
123 levant sensitizations are elucidated by skin prick testing or by the determination of specific IgE in
124 TG and DM allergen based on results of skin prick tests or nasal disk challenges (P < .01 and P < .0
126 using 6 predictors: sex, age, history, skin prick test, peanut specific immunoglobulin E (sIgE), and
127 ion, milk-specific IgE levels, and milk skin prick test performed at enrollment, 6 months, 12 months,
128 o hundred eighty-one children had valid skin prick tests performed, and 14% (39/281) were atopic.
129 ed to measure geographical variation in skin prick test positivity and assess whether it was explaine
130 raphical variation in the prevalence of skin prick test positivity in Europe is unlikely to be explai
131 tted for allergic sensitization (either skin prick test positivity or serum-specific IgE >/= 0.35 kU/
133 and severity of atopic dermatitis, and skin-prick-test positivity at 6 mo of age were not different
138 ociations of NVAS and atopy (defined as skin prick test reaction of >/=3 mm) were analysed using bino
141 ctions to peanut were reported in 1.5%, skin prick test reactivity in 2.0%, and IgE sensitization (>/
143 es demonstrated that exercise increases skin prick test reactivity to and bioavailability of the alle
144 tal IgE, grass pollen-specific IgE, and skin prick test reactivity to grass pollen were all reduced c
145 dren with eczema, wheeze, or a positive skin prick test response before ending exclusive breast-feedi
146 (n = 38) was defined as specific IgE or skin prick test response positivity to Aspergillus fumigatus,
148 mold at age 1 year and child's positive skin prick test response to aeroallergens and molds at age 7
151 POIT was associated with reduced peanut skin prick test responses and peanut-specific IgE levels and
154 ble by using routinely available peanut skin prick test responses or specific IgE levels, but this si
156 total IgE levels, specific IgE levels, skin prick test responses to common aeroallergens, and IgG4 a
157 ith allergic disease) but with negative skin prick test responses to common allergens at randomizatio
160 history of ragweed allergy and positive skin prick test responses to ragweed were randomized and rece
162 ure and sensitization (as determined by skin prick test responses) was analyzed in more than 1000 ref
163 eanut- and Ara h 2-specific IgE levels, skin prick test responses, basophil activation, and TH2 cytok
164 but have peanut-specific IgE, positive skin prick test responses, or both represents a significant d
166 By the end of therapy, titrated CM skin prick test results and CD63 and CD203c expression decrea
167 om sample of participants with negative skin prick test results attended a hospital-based food challe
168 their judgment on clinical history and skin prick test results before and after obtaining the ISAC r
170 tivenoms and cetuximab induced positive skin prick test results in patients with sIgE to alpha-gal.
171 llergy was positive in only 28% and positive prick test results were present in 55% of the 49 VKC-lik
172 Based on available clinical data and skin prick test results, 922 (73%) patients would have been i
174 er IgG4 values (P = .001) and lower egg skin prick test scores (P = .0002) over time and a lower medi
176 g M+ participants tracked the following skin prick test sensitization statuses: M+P+C- > M+P+C+ > M+P
177 of "atopic eczema," "any positive SPT [skin-prick test]," "sensitization to egg," and "sensitization
186 ze in last year, atopy assessed both by skin prick test (SPT) and by the measurement of allergen-spec
189 d the highest AUC (0.79), comparable to skin prick test (SPT) and sIgE to soy extract (0.76 and 0.77,
191 ht to determine the association between skin prick test (SPT) and specific IgE (sIgE) to egg proteins
192 nonallergic (n = 25) children underwent skin prick test (SPT) and specific IgE (sIgE) to peanut and i
193 e, were all negative.The results of the skin prick test (SPT) for Citrus unshiu and specific IgE test
194 iking of the ImmunoCAP, and size of the skin prick test (SPT) for hazelnut were determined, also in r
195 c sensitization was determined based on skin prick test (SPT) of five mites, three molds, and nine ot
197 ws' milk-specific IgE antibodies (IgE), skin prick test (SPT) reactivity and double-blind, placebo-co
198 bioactivity were predictive of allergen skin prick test (SPT) reactivity for infants at high risk of
199 ic IgE against aeroallergens (sIgE) and skin prick test (SPT) reactivity for the most common local al
203 n to egg, milk, or both with a positive skin prick test (SPT) response to the trigger food and/or (2)
204 , milk allergy, or both with a positive skin prick test (SPT) response to the trigger food and/or (2)
205 years of age and develop thresholds for skin prick test (SPT) results and specific IgE (sIgE) levels
206 peanut allergy, and the implications of skin prick test (SPT) screening before peanut introduction.
209 ile atopic dermatitis and preceding egg skin prick test (SPT) sensitization, we found a strong and si
211 276 one-year-old children who underwent skin prick test (SPT) to 4 food allergens and those with dete
212 A total of 433 patients with positive skin prick test (SPT) to birch pollen were analyzed regarding
214 a, egg allergy, or both but 0-mm peanut skin prick test (SPT) wheal responses (n = 542); group III, p
215 o poly-sensitized athletes according to skin prick test (SPT) with different allergic phenotypes (ast
216 Q-5D) health questionnaire, spirometry, skin prick test (SPT), exhaled nitric oxide (FeNO), smell tes
217 Patients with CMA and/or RA underwent skin prick test (SPT), intracutaneous test (ICT), and, when r
222 blot and IgE-ELISA were complemented by Skin Prick Testing (SPT) and mediator release assay to determ
226 eta lactam testing with 17% undertaking skin prick testing (SPT) only, 77% SPT followed by intra-derm
227 infants (HealthNuts), infants underwent skin prick testing (SPT) to egg white at 12 months of age.
228 y fever, eczema, food allergy, positive skin prick testing (SPT), or elevated allergen-specific serum
233 Conjunctival provocation tests (CPT), skin prick tests (SPT), BAT, and sIgE determination including
235 inst common allergens was determined by skin prick tests (SPT); specific immunoglobulin E (sIgE) tite
238 hey answered a questionnaire, underwent skin prick tests (SPTs) for common aeroallergens, and provide
239 invited to a parental questionnaire and skin prick tests (SPTs) to ten airborne allergens, and 2148 (
240 eactions was obtained, and standardized skin prick tests (SPTs) using finely ground tree-nut solution
241 10-fold dilutions of milk protein, and skin prick tests (SPTs) were performed to commercial milk ext
243 al work-up included a detailed history, skin prick tests (SPTs) with IVIP, and basophil activation te
244 lowing outcomes at age 2 years: eczema, skin prick tests (SPTs), increased allergen-specific IgE leve
245 h mollusc tolerance) were studied using skin prick tests (SPTs), specific IgEs (sIgEs) and SDS-PAGE i
247 o initially had negative results on the skin-prick test, the prevalence of peanut allergy at 60 month
248 o-controlled oral food challenge (OFC), skin prick test titration (SPTT), and basophil histamine rele
250 culture for varicella-zoster virus, and skin prick test to common food and animal allergens were nond
251 nts aged 18 to 65 years with a positive skin prick test to Dactylis glomerata pollen were exposed to
252 al allergic rhinitis (SAR) and positive skin prick test to grass and olive pollens and evaluate how k
253 action after peanut ingestion, positive skin prick test to peanuts, and positive by double-blind plac
254 e and a sputum cell differential count; skin prick testing to both common aeroallergens and an extend
255 of 5276 one-year-old infants underwent skin prick testing to peanut, egg, sesame, and cow's milk or
259 on between 10 loci and specific IgE and skin prick tests to individual allergens and poly-sensitizati
264 or scorings of symptoms and medication, skin prick testing, total IgE, specific IgE, and Der p 1-spec
281 ood-specific serum IgE measurements and skin prick tests were performed before initiating the diet.
282 E inhibition, ImmunoCAP inhibition, and skin prick tests were performed using samples from selected p
286 3 kUA /l (7.2-120.2), and median peanut skin prick test wheal 11.3 mm (6.5-18)]; four experienced no
287 ubjects ingesting baked egg, EW-induced skin prick test wheal diameter and EW-, ovalbumin-, and ovomu
294 s pollen allergic individuals underwent skin prick testing with allergen alone, allergen plus Bet-APE
295 cted to topical cowhage provocation and skin prick testing with histamine and assessed for difference
297 , we studied the possibility to perform skin prick tests with cetuximab, which carries the alpha-gal
300 es with standardized doses of rMal d 1, skin prick tests with recombinant allergens, and measurements
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