ライフサイエンスコーパス: receptor-interacting protein

1 HLA-B-associated transcript 3) as a TGF-beta receptor-interacting protein.

2 The Cannabinoid Receptor Interacting Protein 1 (Cnrip1) was discovered a

3 report that alsin interacted with glutamate receptor interacting protein 1 (GRIP1) both in vitro and

4 A SNP within the glutamate receptor interacting protein 1 (GRIP1) gene presented a

5 subunit of the AMPAR and requires glutamate receptor interacting protein 1 (GRIP1) interaction with

6 Glutamate receptor interacting protein 1 (GRIP1) is a neuronal sca

7 P90/DLG/ZO-1 (PDZ) domain 2 of the glutamate receptor interacting protein 1 (GRIP1) through its intra

8 ch repeat and Ig-like domain-containing Nogo receptor interacting protein 1 (LINGO-1) is a negative r

9 ch repeat and Ig-like domain-containing Nogo receptor interacting protein 1 (LINGO-1) is a negative r

10 The recent discovery of the role of receptor interacting protein 1 (RIP1) kinase in tumor ne

11 The death domain kinase receptor interacting protein 1 (RIP1) plays a key role i

12 2 is necessary for the polyubiquitination of receptor interacting protein 1 (RIP1) that then serves a

13 In this study, we show that the receptor interacting protein 1 (RIP1), a central compone

14 d both K48- and K63-linked ubiquitination of receptor interacting protein 1 (RIP1).

15 We found that receptor interacting protein 1 and 3 (RIP1 and RIP3) kin

16 singly, TRADD is required for recruitment of receptor interacting protein 1 and TNFR-associated facto

17 ation (c.279delG, p.Trp93fs*) of the nuclear receptor interacting protein 1 gene (NRIP1) in all seven

18 oxicity was also abolished by necrostatin or receptor interacting protein 1 knock down.

19 uit fly, the PDZ7 domain of GRIP1 (glutamate receptor interacting protein 1) from rat and the PDZ2 do

20 naling complex and for the ubiquitination of receptor interacting protein 1.

21 Glutamate receptor-interacting protein 1 (GRIP1) and GRIP2 are clo

22 (LBD) and a peptide from the glucocorticoid receptor-interacting protein 1 (GRIP1) coactivator compl

23 r recruitment to the receptor/Glucocorticoid Receptor-Interacting Protein 1 (GRIP1) complex that bind

24 Overexpression of glutamate receptor-interacting protein 1 (GRIP1) rescues ephrinB3

25 lating the formation of a complex containing receptor-interacting protein 1 (RIP1) and receptor-inter

26 We reveal that receptor-interacting protein 1 (RIP1) and tumor necrosis

27 f Casp8 prevents proteolytic cleavage of the receptor-interacting protein 1 (RIP1) in hepatocytes and

28 ily promote K11-linked polyubiquitination of receptor-interacting protein 1 (RIP1) in vitro and in vi

29 f necroptosis, which could be blocked by the receptor-interacting protein 1 (RIP1) inhibitor, necrost

30 Although receptor-interacting protein 1 (RIP1) is well known as a

31 retroviral expression of TAK1, inhibition of receptor-interacting protein 1 (RIP1) kinase activity wi

32 tatin-1 (Nec-1), a specific inhibitor of the receptor-interacting protein 1 (RIP1) kinase domain, pre

33 Finally, the receptor-interacting protein 1 (RIP1) may be involved in

34 Ubiquitination and deubiquitination of receptor-interacting protein 1 (RIP1) play an important

35 NF) receptor-associated factor 2 (TRAF2) and receptor-interacting protein 1 (RIP1) play critical role

36 has been attributed to ubiquitin editing of receptor-interacting protein 1 (RIP1) to influence activ

37 Using macrophages in which receptor-interacting protein 1 (RIP1) was knocked down b

38 rongly reduced in murine fibroblasts lacking receptor-interacting protein 1 (RIP1), a known M45-inter

39 mbly of a death-signaling complex containing receptor-interacting protein 1 (RIP1), FADD, and caspase

40 Here we report that receptor-interacting protein 1 (RIP1)-regulated interleu

41 romises the formation of the downstream TRIF/receptor-interacting protein 1 (RIP1)/TBK1 complex.

42 e of necrostatin-1, a selective inhibitor of receptor-interacting protein 1 (RIP1, also known as RIPK

43 additional cell death pathways, necroptosis (receptor-interacting protein 1 [RIP1] and RIP3 mRNAs) an

44 sis, a form of programmed necrosis involving receptor-interacting protein 1 and the mixed lineage kin

45 king the inhibition of polyubiquitination of receptor-interacting protein 1 in TNF receptor 1 complex

46 mice and THP-1 macrophages in vitro with the receptor-interacting protein 1 kinase (RIP1) inhibitor n

47 In contrast, the reduction of receptor-interacting protein 1 suppressed the loss of De

48 id receptor coactivator-1 and glucocorticoid receptor-interacting protein 1 to inhibit hCAR activity.

49 Although TNFalpha-induced receptor-interacting protein 1 ubiquitination is indeed

50 he absence of TRAF2 and TRAF5 expression and receptor-interacting protein 1 ubiquitination.

51 ges leads to reduced ubiquitination of RIP1 (receptor-interacting protein 1), suggesting a role for L

52 formation of 2 cell death complexes, RIP 1 (receptor-interacting protein 1)-FADD (Fas-associated pro

53 ys, such as hypoxia-inducing factor-1-alpha, receptor-interacting protein 1, and apoptosis-inducing f

54 factor 6, TNF receptor-associated factor 2, receptor-interacting protein 1, Hemo-oxidized iron regul

55 PMN lysis was dependent on receptor-interacting protein 1, suggesting that PMN-SA u

56 receptor coactivator 1 or the glucocorticoid receptor-interacting protein 1.

57 nd hepatocyte death in vitro, independent of receptor-interacting protein 1.

58 teractions with TNFR-associated factor 6 and receptor-interacting protein 1.

59 riments show that WNV-E acts at the level of receptor-interacting protein 1.

60 ation is dependent upon its interaction with receptor-interacting protein 1.

61 itochondria, the role of necroptosis through receptor-interacting proteins 1 and 3 (RIPK1 and RIPK3)

62 Mechanistically, Traf2 critically regulates receptor-interacting proteins 1 and 3 and mixed lineage

63 nd resulted in the downregulation of nuclear receptor interacting protein-1 (NRIP1), which influences

64 We identified receptor interacting protein-1 (RIP1) as a novel API2-MA

65 how that the AMPAR-binding protein glutamate receptor-interacting protein-1 (GRIP1) is essential for

66 NF-alpha/Fas-induced cell death is a type of receptor-interacting protein-1 (RIP-1)-dependent program

67 -associated death domain protein (TRADD) and receptor-interacting protein-1 (RIP1) in TRAIL signaling

68 We identified TRIP12 (thyroid hormone receptor-interacting protein 12), an E3 ubiquitin-protei

69 In this study, we demonstrated that thyroid receptor interacting protein 13 (TRIP13) is a critical m

70 0-Mad2 subcomplex and identify it as Thyroid Receptor Interacting Protein 13 (TRIP13), an AAA-ATPase

71 The AAA-ATPase thyroid hormone receptor interacting protein 13 (TRIP13), jointly with t

72 In this study, we found receptor interacting protein 140 (RIP140) can regulate t

73 Receptor interacting protein 140 (RIP140) is a nuclear r

74 Receptor interacting protein 140 (RIP140), a ligand-depe

75 T3 repression of Crabp1 requires receptor interacting protein 140 (RIP140).

76 oregulator peptide representing a portion of receptor interacting protein 140 (RIP140).

77 ment of histone deacetylase-containing/GRIP1/receptor-interacting protein 140 (RIP140) complex in dif

78 onal carcinoma cell line P19, which involves receptor-interacting protein 140 (RIP140) for heterochro

79 urons induces rapid translocation of nuclear receptor-interacting protein 140 (RIP140) to the cytopla

80 coactivator 1alpha or 1beta and repressed by receptor-interacting protein 140.

81 Cannabinoid receptor interacting protein 1a (CRIP1a) is a CB1 recept

82 Cannabinoid receptor-interacting protein 1a (CRIP1a) binds to the CB

83 icated in the breakdown of 2-AG; cannabinoid receptor-interacting protein 1a (CRIP1a), a protein that

84 it a higher expression of IRAK1, IRAK-M, and receptor interacting protein 2 (Rip2).

85 nd their common downstream adaptor molecule, receptor interacting protein 2 (RIP2; also known as RIPK

86 now show that MDP induces ubiquitylation of receptor- interacting protein 2 (RIP2) in primary macrop

87 Receptor-interacting protein 2 (Rip2) is a transcription

88 1 (NOD1) interacts with its adaptor protein receptor-interacting protein 2 (RIP2) to propagate immun

89 pensable for recruiting a downstream kinase, receptor-interacting protein 2 (RIP2), that activates nu

90 ient in the gene coding for the NLR adaptor, receptor-interacting protein 2 (RIP2).

91 8 as well as NOD1 and its signaling molecule receptor-interacting protein 2 (RIP2).

92 on of MAPKs, and deficient ubiquitination of receptor-interacting protein 2 in response to MDP.

93 reduced in the absence of NOD proteins, but receptor-interacting protein 2 is not involved in CD8 si

94 protein 2) binds to the protein kinase RIP2 (receptor-interacting protein 2) to coordinate NF-kappaB

95 njury was seen with a deficiency of Nod2 and receptor-interacting protein 2, and the simultaneous def

96 receptor-associated kinase-like 2, glutamate receptor-interacting protein 2, and ubiquitin) that inte

97 of IkappaB kinase (IKKbeta) and its upstream receptor-interacting protein 2, whereas IKKbeta inhibito

98 , and in their downstream signaling molecule receptor-interacting protein 2.

99 ulation and NF-kappaB activation, indicating receptor-interacting protein 2/IKKbeta signaling plays c

100 Receptor interacting protein-2 (RIP2) is a caspase recru

101 Responses were mediated through receptor interacting protein-2 (RIP2)- and tumor necrosi

102 ifferentiation primary response gene 88, and receptor-interacting protein-2 but reduced the expressio

103 ne receptor-associated protein 220/vitamin D receptor-interacting protein 205/mediator 1, an anchor f

104 rated that a super-low dose of LPS activates receptor interacting protein 3 kinase (RIP3), a key mole

105 IF) and led to persistent phosphorylation of receptor-interacting protein 3 (Rip3) kinase, which resu

106 was correlated with increased expression of receptor-interacting protein 3 (RIP3), a master regulato

107 ia and that Gpx4 is essential for preventing receptor-interacting protein 3 (RIP3)-dependent necropto

108 ng receptor-interacting protein 1 (RIP1) and receptor-interacting protein 3 (RIP3).

109 lly, inhibition of STIM1 signaling prevented receptor-interacting protein 3-dependent (RIP3-dependent

110 lpha and CD95 ligand, but was independent of receptor-interacting protein 3.

111 +) exchange regulatory factor 2, and thyroid receptor interacting protein 6, which has been shown pre

112 of two proteins in the zyxin family, thyroid receptor-interacting protein 6 (TRIP6) and lipoma-prefer

113 In this study, a role for thyroid hormone receptor-interacting protein 6 (TRIP6) in sealing zone f

114 LIM domain-containing TRIP6 (Thyroid Hormone Receptor-interacting Protein 6) is a focal adhesion mole

115 Fisetin also inhibited TNF-induced TAK1 and receptor-interacting protein activation, events that lie

116 e by the "death" adaptor protein caspase and receptor interacting protein adaptor with death domain (

117 partners, we found that the aryl hydrocarbon receptor-interacting protein (AIP) directly associates w

118 homozygous deletion of the aryl hydrocarbon receptor-interacting protein (AIP), a survivin-associate

119 cluding Hsp90, p23, and the aryl hydrocarbon receptor-interacting protein (AIP), also known as ARA9 a

120 showed the expression of p50, p52, p100 and receptor-interacting protein; all linked with NF-kappaB

121 These G protein-coupled receptor-interacting proteins also facilitate fine-tunin

122 kermit 2 (also recently described as an IGF receptor interacting protein and named XGIPC).

123 re, we have identified AIP (aryl hydrocarbon receptor interacting protein) as a new binding partner o

124 ng protein adaptor with death domain (CRADD)/receptor interacting protein-associated ICH-1/CED-3 homo

125 -induced protein with a death domain (PIDD), receptor-interacting protein-associated ICH-1/CED-3 homo

126 r show that RIP1 (the Ser/Thr protein kinase receptor-interacting protein) associates with the GAP do

127 primary keratinocytes (KCs) as the vitamin D receptor-interacting protein complex.

128 of two structurally related CB1 cannabinoid receptor interacting proteins (CRIP1a and CRIP1b) that b

129 factor receptor (TNFR) superfamily, through receptor-interacting protein-dependent K63-linked ubiqui

130 Using VDR affinity beads, the vitamin D receptor interacting protein (DRIP)/mediator complex was

131 kout mice and chimeras revealed that Eph-Eph receptor-interacting proteins (ephrins) are expressed bo

132 We identified a novel NMDA receptor-interacting protein, GIPC (GAIP-interacting pro

133 with one of the PDZ domains of the Glutamate receptor interacting protein (Grip), another factor requ

134 -associated protein (GABARAP), and glutamate receptor interacting protein (GRIP).

135 on, two AMPAR-interacting proteins-glutamate receptor-interacting protein (GRIP) and protein interact

136 Glutamate receptor-interacting protein (GRIP) is a neuronal scaffo

137 brane through its interaction with glutamate receptor-interacting protein (GRIP), a PDZ domain protei

138 g domains for left-handed helix (Z-form) and receptor-interacting protein homotypic interaction motif

139 tead, PGRP-LC and PGRP-LE signaled through a receptor-interacting protein homotypic interaction motif

140 usion protein was used to isolate and purify receptor-interacting proteins in cell lysates prepared f

141 of RAMPs highlight the need to consider all receptor-interacting proteins in future drug development

142 PRIP (peroxisome proliferator-activator receptor interacting protein) is a nuclear receptor coac

143 ty of NEMO to bind to K63-linked polyUb RIP (receptor-interacting protein) is necessary for efficient

144 we report that the death domain kinase, RIP (receptor-interacting protein), is important for DNA dama

145 Our findings suggest that BAT3, a TGF-beta receptor-interacting protein, is capable of modulating T

146 Programmed necrosis mediated by receptor interacting protein kinase (RIP)3 (also called

147 d cIAP2 (cIAP1/2), leading to the release of receptor interacting protein kinase (RIPK1) from the act

148 Degradation of cIAPs triggers the release of receptor interacting protein kinase (RIPK1) from TNF rec

149 he necroptosis inhibitor Nec-1, which blocks receptor interacting protein kinase 1 (RIP kinase 1), al

150 The receptor interacting protein kinase 1 (RIP1) is essentia

151 D and caspase 8, and the necroptosis kinases receptor interacting protein kinase 1 (RIPK1) and RIPK3.

152 emonstrated that the scaffolding function of receptor interacting protein kinase 1 (RIPK1) and tumor

153 In contrast, inhibition and knockdown of receptor interacting protein kinase 1 (RIPK1) had no eff

154 Receptor interacting protein kinase 1 (RIPK1) has an ess

155 Receptor interacting protein kinase 1 (RIPK1) is a criti

156 iven by two serine threonine kinases, RIPK1 (Receptor Interacting Protein Kinase 1) and RIPK3, and a

157 ction model, such variant procaspase-1 binds receptor interacting protein kinase 2 (RIP2) via Caspase

158 Receptor interacting protein kinase 2 (RIPK2) is critica

159 itment of additional proteins (such as RIP2, receptor interacting protein kinase 2), which is further

160 Recent findings suggest that the receptor interacting protein kinase 3 (RIP3) acts as a s

161 orm of nonapoptotic cell death driven by the receptor interacting protein kinase 3 (RIPK3) and its su

162 sis is a cell death pathway regulated by the receptor interacting protein kinase 3 (RIPK3) and the mi

163 ptosis, functions as a negative regulator of receptor interacting protein kinase 3 (RIPK3), an essent

164 l survival, caspase-8-mediated apoptosis, or receptor interacting protein kinase 3 (RIPK3)-dependent

165 pases, while necroptosis is dependent on the receptor interacting protein kinase 3 (RIPK3).

166 ll death defined by activation of the kinase receptor interacting protein kinase 3 and its downstream

167 ed phosphorylation and decreased cleavage of receptor interacting protein kinase-1 (Rip1), leading to

168 8, revealing a new regulatory axis affecting receptor interacting protein kinase-1 (RIPK1)>CASPASE-8

169 Receptor Interacting Protein Kinase-1 (RIPK1), a key pla

170 osis is mediated by the kinase activities of receptor interacting protein kinase-1 and receptor inter

171 ch is transduced by the kinase activities of receptor interacting protein kinase-1 and receptor inter

172 Receptor interacting protein kinase-3 (RIP3) is an essen

173 ath pathway downstream of the protein kinase Receptor Interacting Protein Kinase-3 (RIPK3).

174 nt mice is completely rescued by ablation of receptor interacting protein kinase-3 (RIPK3).

175 of receptor interacting protein kinase-1 and receptor interacting protein kinase-3, eventually leadin

176 of receptor interacting protein kinase-1 and receptor interacting protein kinase-3, which eventually

177 Receptor-interacting protein kinase (RIP) 3 (also called

178 Because receptor-interacting protein kinase (RIP) 3-mediated nec

179 Inhibition of receptor-interacting protein kinase (RIP)1 by necrostati

180 NF-kappaB is shown to depend on the adaptor receptor-interacting protein kinase (RIP)1, acting via a

181 es neutrophil extracellular traps (NETs) via receptor-interacting protein kinase (RIPK) 1/3- and mixe

182 Receptor-interacting protein kinase (RIPK)-1 is involved

183 ochondrial permeability transition (MPT) and receptor-interacting protein kinase (RIPK)1-mediated nec

184 cells by inhibiting the interaction between receptor-interacting protein kinase 1 (RIP1) and RIP3, a

185 ck the Fas-associated death domain (FADD) or receptor-interacting protein kinase 1 (RIP1) cell death

186 pases, necrotic cell death, which depends on receptor-interacting protein kinase 1 (RIP1), and NF-kap

187 noic acid receptor gamma (RARgamma) controls receptor-interacting protein kinase 1 (RIP1)-initiated c

188 Receptor-interacting protein kinase 1 (RIPK1) and RIPK3

189 Receptor-interacting protein kinase 1 (RIPK1) induces ap

190 Receptor-interacting protein kinase 1 (RIPK1) is an impo

191 Inhibition of receptor-interacting protein kinase 1 (RIPK1) kinase act

192 Knock-in expression of kinase inactive receptor-interacting protein kinase 1 (RIPK1) prevented

193 Receptor-interacting protein kinase 1 (RIPK1) promotes c

194 ed apoptosis requires the kinase activity of receptor-interacting protein kinase 1 (RIPK1), a key reg

195 programmed cell death and innate immunity is receptor-interacting protein kinase 1 (RIPK1), which has

196 Rather, receptor-interacting protein kinase 1, a kinase also lin

197 Activation of macrophages required DR5 and receptor-interacting protein kinase 1.

198 n to directly binding its downstream targets receptor-interacting protein kinase 2 (RIP2) and autopha

199 reporting conflicting requirements for RIP2 (receptor-interacting protein kinase 2) in autophagy indu

200 Ly6/PLAUR domain-containing protein 6, in a receptor-interacting protein kinase 2-TGF-beta-activated

201 Receptor-interacting protein kinase 3 (RIP3 or RIPK3) ha

202 Receptor-interacting protein kinase 3 (RIP3) and its sub

203 We further demonstrate that receptor-interacting protein kinase 3 (RIP3), which is h

204 shed by caspase-8 compromise and mediated by receptor-interacting protein kinase 3 (RIP3).

205 The receptor-interacting protein kinase 3 (RIP3/RIPK3) has e

206 tein expression of the necroptosis mediators receptor-interacting protein kinase 3 (RIPK3) and mixed

207 otein with death domain (FADD) combined with receptor-interacting protein kinase 3 (RIPK3) deficiency

208 Receptor-interacting protein kinase 3 (RIPK3) is a serin

209 Receptor-interacting protein kinase 3 (RIPK3) mediates n

210 RNA interference for receptor-interacting protein kinase 3 (RIPK3) or mixed l

211 The receptor-interacting protein kinase 3 (RIPK3) plays cruc

212 Receptor-interacting protein kinase 3 (RIPK3)-mediated n

213 c approach, we demonstrate the presence of a receptor-interacting protein kinase 3 (RIPK3)-mixed line

214 rammed necrosis, or "necroptosis", driven by receptor-interacting protein kinase 3 (RIPK3).

215 m of cell death that critically requires the receptor-interacting protein kinase 3 (RIPK3).

216 Receptor-interacting protein kinase 4 (RIPK4) and interf

217 Receptor-interacting protein kinase 4 (RIPK4) is require

218 ted kinase (PKK), which is also known as the receptor-interacting protein kinase 4, as a suppressor o

219 D), Fas-associated death domain protein, and receptor-interacting protein kinase proteins form the si

220 nteract via a CARD-CARD interaction with the receptor-interacting protein kinase RIP2, an inducer of

221 at MLKL-mediated death is independent of the receptor-interacting protein kinase RIPK3.

222 Instead, robust cross-priming required receptor-interacting protein kinase-1 (RIPK1) signaling

223 because the molecules initiating it [such as receptor-interacting protein kinase-1 (RIPK1), RIPK3, or

224 Receptor-interacting protein kinase-3 (RIP3 or RIPK3) is

225 nonapoptotic form of cell death initiated by receptor-interacting protein kinase-3 (RIPK3) and mixed

226 Receptor-interacting protein kinase-3 (RIPK3) is an acti

227 Here, we show that modulating the receptor-interacting protein kinase-3 (Ripk3) pathway ma

228 ological cell suicide mechanism initiated by receptor-interacting protein kinase-3 (RIPK3) phosphoryl

229 ation of mixed lineage kinase-like (MLKL) by receptor-interacting protein kinase-3 (RIPK3) results in

230 l approach, we demonstrate the presence of a receptor-interacting protein kinase-3 (RIPK3)-a mixed li

231 tosis, in which dying cells are generated by receptor-interacting protein kinase-3 and caspase-8 dime

232 ting Fas-associated death domain protein and receptor-interacting protein kinase.

233 The receptor interacting protein kinases-1 (RIPK1) and RIPK3

234 Recent studies have demonstrated that receptor-interacting protein kinases (RIPKs) RIPK1 and R

235 of programmed necrosis that is regulated by receptor-interacting protein kinases (RIPs).

236 dence points to unregulated signaling by the receptor-interacting protein kinases-1 (RIPK1) and RIPK3

237 LCA) caused by mutations in Aryl hydrocarbon receptor interacting protein like-1 (Aipl1) is a severe

238 photoreceptor-specific gene aryl hydrocarbon receptor interacting protein-like 1 (Aipl1) are associat

239 the photoreceptor-expressed aryl hydrocarbon receptor interacting protein-like 1 (AIPL1) are associat

240 P351Delta12 human isoform, aryl hydrocarbon receptor interacting protein-like 1 (hAIPL1) mice demons

241 IPL1 and the mouse isoform, aryl hydrocarbon receptor interacting protein-like 1 (mAipl1).

242 ptor-specific gene encoding aryl hydrocarbon receptor-interacting protein-like 1 (AIPL1) are clinical

243 Aryl hydrocarbon receptor-interacting protein-like 1 (AIPL1) is a distinc

244 Aryl hydrocarbon receptor-interacting protein-like 1 (AIPL1) is a photore

245 the enzyme itself or AIPL1 (aryl hydrocarbon receptor-interacting protein-like 1), leads to retinal d

246 RICK (receptor-interacting protein-like interacting caspase-li

247 Defects in aryl hydrocarbon receptor interacting protein-like1 (AIPL1) are associate

248 identify LRR and Ig domain-containing, Nogo receptor-interacting protein (LINGO-1) as a potent axona

249 eviously, LRR and Ig domain-containing, Nogo receptor-interacting protein (LINGO-1) has been identifi

250 iated factor 2, TGF-beta-activated kinase 1, receptor-interacting protein, NF-kappaB-inducing kinase,

251 n induced by TNF, TNFR1, TRADD, TRAF2, TAK1, receptor-interacting protein, NIK, and IkappaBalpha kina

252 Nuclear receptor interacting protein (Nrip1), also known as RIP1

253 We have identified an N receptor-interacting protein, NRIP1, that directly inter

254 the recruitment of PPARalpha and the nuclear receptor-interacting protein, nuclear receptor corepress

255 rtate (NMDA) receptor and intracellular NMDA receptor-interacting proteins of the glutaminergic synap

256 ct of multiple ligands, association with the receptor-interacting protein receptor activity-modifying

257 f TLR2, TLR3, TLR4, TLR5, or TLR9 results in receptor interacting protein (RIP) 3 kinase-dependent pr

258 Wild-type C57BL/6 and receptor interacting protein (RIP) 3(-/-) mice were rand

259 y autophagy proteins such as ATG7, ATG8, and receptor interacting protein (RIP) blocks ROS accumulati

260 Here, we show that receptor interacting protein (RIP) kinase-mediated necro

261 It is known that receptor interacting protein (RIP) kinases, RIP1 and RIP

262 Receptor interacting protein (RIP)3 kinase (also called

263 e cytomegalovirus M45 protein interacts with receptor-interacting protein (RIP) 1 and RIP3 via a RIP

264 Receptor-interacting protein (RIP) and cellular Fas-asso

265 The receptor-interacting protein (RIP) family kinase RIP4 in

266 Receptor-interacting protein (RIP) has been implicated i

267 of IFN-regulatory factors (DAI) that contain receptor-interacting protein (RIP) homotypic interaction

268 Because this involves receptor-interacting protein (RIP) kinase 1/3, this stud

269 In this study, we show that receptor-interacting protein (RIP) kinase mediates necro

270 The complex interplay between caspase-8 and receptor-interacting protein (RIP) kinase RIP 3 (RIPK3)

271 Here we provide genetic evidence for a receptor-interacting protein (RIP) kinase-caspase-8-depe

272 , which plays a key role in the execution of receptor-interacting protein (RIP) kinase-dependent necr

273 type I interferon (IFN-I) response to induce receptor-interacting protein (RIP) kinase-mediated necro

274 ase-11, IL-1R-associated kinases (IRAK), and receptor-interacting protein (RIP) kinases contribute to

275 Receptor-interacting protein (RIP) plays a critical role

276 Here, we show that the receptor-interacting protein (RIP), a key mediator of tu

277 /Fas-mediated signaling pathway regulated by receptor-interacting protein (RIP), a kinase that shuttl

278 N-TNF receptor-associated factor (TRAF)2, DN-receptor-interacting protein (RIP), DN-transforming grow

279 -kinase do show an increase in the amount of receptor-interacting protein (RIP), while cells with red

280 ern recognition receptor that assembles with receptor-interacting protein (RIP)-2 kinase in response

281 e-2, or the caspase-2 adaptor protein RAIDD (receptor-interacting protein (RIP)-associated Ich-1/CED

282 ase-8, TNFR-associated factor 2 (TRAF2), and receptor-interacting protein (RIP).

283 n chains to signalling intermediates such as receptor-interacting protein (RIP).

284 t the principal components of the necrosome, receptor-interacting protein (RIP)1 and RIP3, are highly

285 were identified through its association with receptor-interacting protein (RIP)1 and TNFR-associated

286 and type II (gamma) IFNs induce precipitous receptor-interacting protein (RIP)1/RIP3 kinase-mediated

287 The pronecrotic kinase, receptor interacting protein (RIP1, also called RIPK1) m

288 Receptor-interacting protein (RIP1) and p38 mitogen-acti

289 Receptor-interacting protein (RIP1) is a serine/threonin

290 The receptor-interacting protein RIP140 interacts with the t

291 ced anti-inflammation engineered by lowering receptor interacting protein (RIP140) expression in macr

292 milar to programmed necrosis that depends on receptor interacting protein (Ripk1).

293 In particular, we focus on the roles of receptor-interacting proteins, scaffold proteins, synapt

294 n ID-related gene encoding the synaptic NMDA-receptor interacting protein synapse-associated protein

295 TO-ETHYLENE SENSITIVITY1 (RTE1), an ethylene receptor interacting protein that regulates the activity

296 In an attempt to discover novel receptor interacting proteins, the C-terminal tail of th

297 Two such proteins, mannose 6-phosphate receptor-interacting protein TIP47 and Arfaptin2, were f

298 tic Wnt-receiving cell to target dGRIP, a Wg-receptor-interacting protein, to postsynaptic sites.

299 y, we cloned the tomato ortholog of TGF-beta Receptor Interacting Protein (TRIP1), which was previous

300 uman PIMT (peroxisome proliferator-activated receptor-interacting protein with methyltransferase) pro