ライフサイエンスコーパス: thalamic nuclei

1 ior thalamus (pulvinar) and the medio-dorsal thalamic nuclei.

2 signals reach the cortex via sense-specific thalamic nuclei.

3 cortices, pars opercularis and motor-related thalamic nuclei.

4 d RGC axons in adjacent non-retino-recipient thalamic nuclei.

5 nisms influence the formation of postmitotic thalamic nuclei.

6 ative dorsal (DP) and central posterior (CP) thalamic nuclei.

7 and a PCS pattern that involved the ventral thalamic nuclei.

8 n selectively and collectively from multiple thalamic nuclei.

9 tors preferentially give rise to caudodorsal thalamic nuclei.

10 ogeneity contributes to the specification of thalamic nuclei.

11 h interval and was localized to the anterior thalamic nuclei.

12 aracentral nucleus (OPC) of the intralaminar thalamic nuclei.

13 tum, hippocampus, dentate gyrus and specific thalamic nuclei.

14 orsal, ventral anterior, and anterior medial thalamic nuclei.

15 lamic regions, as well as particular midline thalamic nuclei.

16 signals reach the cortex via sense-specific thalamic nuclei.

17 atosensory cortex originate in various relay thalamic nuclei.

18 rain, including the amygdala and the midline thalamic nuclei.

19 These connectivity patterns differed between thalamic nuclei.

20 ates, expression is most abundant in various thalamic nuclei.

21 MGN nucleus is very similar to that in other thalamic nuclei.

22 se reciprocal projections with corresponding thalamic nuclei.

23 antigens for calbindin and Cat-301 to reveal thalamic nuclei.

24 odality inhibitory modulation between dorsal thalamic nuclei.

25 innervates cortex and possibly originates in thalamic nuclei.

26 , and specific sensory relay and association thalamic nuclei.

27 disperse activity across cortical areas and thalamic nuclei.

28 ct to the parafascicular and central lateral thalamic nuclei.

29 M), ventral lateral (VL), and posterior (Po) thalamic nuclei.

30 to regions that roughly reflected individual thalamic nuclei.

31 A1 region of the hippocampus, but not in two thalamic nuclei.

32 arietal areas are differently connected with thalamic nuclei.

33 e ventral posterior, and caudal ventromedial thalamic nuclei.

34 e size and our inability to measure discrete thalamic nuclei.

35 , caudal PC, oval paracentral, and reticular thalamic nuclei.

36 o most of the other intralaminar and midline thalamic nuclei.

37 al paracentral, parafascicular, and rhomboid thalamic nuclei.

38 s well as neurons in the medial habenula and thalamic nuclei.

39 nces between the auditory thalamus and other thalamic nuclei.

40 on of first (dLGN) and high-order (pulvinar) thalamic nuclei.

41 ITCc dendrites from nociceptive intralaminar thalamic nuclei.

42 amatergic outputs that differentially target thalamic nuclei.

43 ange feedback projections to primary sensory thalamic nuclei.

44 ular nuclei, sensory nuclei, and nonspecific thalamic nuclei.

45 he BG to directly excite neurons in specific thalamic nuclei.

46 ngling of the neurons that innervate the two thalamic nuclei.

47 rlap of cells projecting to the two anterior thalamic nuclei.

48 idus, subthalamic nucleus, and ventral motor thalamic nuclei.

49 reuniens (Re) is the largest of the midline thalamic nuclei.

50 levels of EphA7 in CT axons and ephrin-As in thalamic nuclei.

51 istributed among six cortical layers and two thalamic nuclei.

52 em form extensive projections to a number of thalamic nuclei.

53 raeeminentialis) and diencephalic (posterior thalamic) nuclei.

54 ecting to as few as 5, and to as many as 15, thalamic nuclei; (2) most nuclei received projections fr

55 campus make the reuniens and rhomboid (ReRh) thalamic nuclei a putatively major functional link for r

56 vide evidence for differential inhibition of thalamic nuclei across brain states, where the TRN separ

57 neuronal firing via monosynaptic afferents, thalamic nuclei act as a relay station routing prefronta

58 s received bilateral lesions of anterodorsal thalamic nuclei (ADN), postsubiculum (PoS), or sham lesi

59 a PCC/RSC pattern that involved the anterior thalamic nuclei, an MPC pattern that involved the latera

60 Coupled with a lack of input from principal thalamic nuclei and a minimal layer 4, these observation

61 lso seen in various basal forebrain regions, thalamic nuclei and amygdaloid nuclei.

62 ticular nucleus in conjunction with specific thalamic nuclei and are modulated by corticothalamic and

63 axons to the designated areas within target thalamic nuclei and by progressive increase of axonal pr

64 ctivity of the suprachiasmatic and reticular thalamic nuclei and choroid epithelial cells diminished,

65 ion with the pattern of connectivity between thalamic nuclei and cortical areas or deep nuclei), whic

66 th potential synaptic relays in the anterior thalamic nuclei and dorsolateral entorhinal cortex.

67 e paraventricular, supraoptic, and reticular thalamic nuclei and in the ventromedial hypothalamic nuc

68 io between higher PDE10A expression in motor thalamic nuclei and lower PDE10A expression in striatopa

69 n the low-frequency (1-4 Hz) oscillations in thalamic nuclei and neocortical areas are essentially th

70 ow that the projections that do form between thalamic nuclei and neocortical domains have a shifted t

71 eract with primary and higher-order specific thalamic nuclei and nonspecific thalamic nuclei to carry

72 tional links between the auditory-responsive thalamic nuclei and PAmp.

73 reciprocal connections between the anterior thalamic nuclei and retrosplenial cortex, another region

74 rdependent relationship between the anterior thalamic nuclei and retrosplenial cortex, given how dysf

75 reciprocal connections between the anterior thalamic nuclei and retrosplenial/pre- and parasubicular

76 (ii) It includes multiple thalamic nuclei and six-layered cortical microcircuitry

77 nd unconditioned stimuli in the multisensory thalamic nuclei and that these BF shifts are augmented a

78 ominent connections between the intralaminar thalamic nuclei and the basal ganglia has long been esta

79 icates the anterior and mediodorsal (limbic) thalamic nuclei and the reciprocally interconnected area

80 ith potential synaptic relays in the midline thalamic nuclei and the rostral caudomedial entorhinal c

81 s, permanent retinal projections to auditory thalamic nuclei and to visual thalamic nuclei that norma

82 cells that are densest in the "nonspecific" thalamic nuclei and usually target layer 1 (L1) of multi

83 bust IPSCs or IPSPs in other specific dorsal thalamic nuclei and vice versa.

84 nkey brain, with the greatest density in the thalamic nuclei and with moderate to low binding in the

85 nucleus, parafascicular and paraventricular thalamic nuclei, and a few brainstem nuclei (e.g., the i

86 ells, a subset of striatal neurons, selected thalamic nuclei, and a subset of interneurons in the ven

87 i of the torus innervate central and lateral thalamic nuclei, and all have a weak reciprocal connecti

88 yramidal cells in hippocampal regions CA1-3, thalamic nuclei, and olfactory bulb.

89 o fibers and neuropil in the analyzed dorsal thalamic nuclei, and presented no differences between ge

90 development, the cortical dependence of many thalamic nuclei, and the phenomenon of transsynaptic deg

91 ulation of cells in two or more other dorsal thalamic nuclei, and TRN-mediated inhibitory inputs can

92 Projections to the RAIC from medial thalamic nuclei are associated with motivational/affecti

93 rly and late in the period of gestation when thalamic nuclei are becoming histologically differentiat

94 aps in the cerebral cortex and corresponding thalamic nuclei are genetically prespecified to a large

95 Our recent studies found that these thalamic nuclei are in fact derived from molecularly het

96 current study examined whether the anterior thalamic nuclei are involved in attentional processes ak

97 Midline/intralaminar/posterior dorsal thalamic nuclei are involved in forebrain circuits relat

98 shown that responses in first-order sensory thalamic nuclei are modulated by cortical areas.

99 ons between different cortical areas via the thalamic nuclei are no longer functional, and there is a

100 Within this system, the anterior thalamic nuclei are prominent, both for their vital cont

101 Thalamic nuclei are thought to funnel sensory informatio

102 ns of CP and projections of all other dorsal thalamic nuclei are to the subpallium, however.

103 n, segregation, or putative absence in given thalamic nuclei are unknown.

104 ern of performance reveals that the anterior thalamic nuclei are vital for attending to those stimuli

105 ts, characteristic features of other sensory thalamic nuclei, are not encountered.

106 ipal trigeminal and ventral-posterior-medial thalamic nuclei, are substantially modulated by touch.

107 cond postnatal week, but it extends to other thalamic nuclei as well.

108 ness of CT axons to the ephrin-A gradient in thalamic nuclei, as well as by the matching levels of Ep

109 tal, intralaminar, parafasicular, posterior, thalamic nuclei, as well as the ventral medial, ventral

110 least in part, from a loss of inhibition to thalamic nuclei associated with both the sensory-discrim

111 inar (LP-pulvinar complex) are the principal thalamic nuclei associated with the elaborate developmen

112 a 3a receives the majority of its input from thalamic nuclei associated with the motor system, poster

113 SorCS3 expression marks thalamic nuclei at embryonic and early postnatal stages.

114 ent, convergent evidence places the anterior thalamic nuclei at the heart of diencephalic amnesia.

115 Selective inactivation of anterior thalamic nuclei (ATN) by microinfusion of muscimol or fl

116 bilateral cytotoxic lesions in the anterior thalamic nuclei (ATN) or transection of the fimbria-forn

117 ponent of the limbic circuitry, the anterior thalamic nuclei (ATN), on the generation of new neurons

118 to the hippocampal network via the anterior thalamic nuclei (ATN).

119 Infusion of beta-FNA near specific medial thalamic nuclei attenuated morphine-induced c-Fos expres

120 gene expression were seen between different thalamic nuclei but not between diagnostic groups.

121 Input to sensory thalamic nuclei can be classified as either driver or mo

122 Relay neurons in dorsal thalamic nuclei can fire high-frequency bursts of action

123 late and frontal cortices, amygdala, midline thalamic nuclei, cerebellum, and in several brainstem re

124 striatum and pallidum and increased in motor thalamic nuclei, compared to a group of matched healthy

125 iodorsal nucleus of the thalamus and midline thalamic nuclei, consistent with findings in the rhesus

126 Ventral motor thalamic nuclei contain exclusively excitatory projectio

127 gests that pTH-C is the only major source of thalamic nuclei containing neurons that project to the c

128 al and parietal cortical regions and related thalamic nuclei, correlated with skill.

129 eover, TRN-mediated switching between dorsal thalamic nuclei could provide a mechanism for the select

130 alterations are focally located in specific thalamic nuclei depending on the initial infarct locatio

131 remains one of the least explored among the thalamic nuclei despite occupying the most thalamic volu

132 cal waves, whereas neurons from higher-order thalamic nuclei display "hub dynamics" and thus may cont

133 wever, the neuronal precursors for different thalamic nuclei display temporally distinct Gbx2 express

134 Thus, our study shows that: (1) different thalamic nuclei do not establish projections independent

135 m (NCM), the core or shell regions of dorsal thalamic nuclei, dopaminergic cell groups in the mesence

136 euronal generation was also evident in other thalamic nuclei (e.g., the lateral geniculate nucleus).

137 theless, how a distinct array of postmitotic thalamic nuclei emerge from this single developmental un

138 nteraction with ligands in the somatosensory thalamic nuclei; EphA4 affects only cortical neuronal mi

139 idespread oscillations and render subsets of thalamic nuclei especially vulnerable to pathological sy

140 In mammals, different thalamic nuclei establish highly ordered projections wit

141 that stimulation of cells in specific dorsal thalamic nuclei evokes robust IPSCs or IPSPs in other sp

142 As in the hypothalamus, all thalamic nuclei examined displayed at least light immuno

143 also projected to all the other intralaminar thalamic nuclei, except for the central lateral thalamic

144 suggests that innervation from PV-containing thalamic nuclei extends across superficial and middle la

145 We conclude that sensory thalamic nuclei follow the propagating cortical waves, w

146 ic correlations between the cortex and these thalamic nuclei followed the known patterns of anatomica

147 Appreciating the importance of the anterior thalamic nuclei for memory and attention provides a more

148 bodies and their projections to the anterior thalamic nuclei for spatial memory.

149 to test whether the hippocampus and anterior thalamic nuclei form functional components of the same s

150 te receptor expression was determined in six thalamic nuclei from 12 subjects with DSM-III-R diagnose

151 ers of sensory-motor cortex, throughout most thalamic nuclei, globus pallidus, and spinal cord.

152 that delta frequency bursting in particular thalamic nuclei has a causal role in producing WM defici

153 on of the specific (VB) and nonspecific (CL) thalamic nuclei has been proposed as the basis for the t

154 Lesions involving the intralaminar thalamic nuclei have been associated with impairments in

155 Dorsal thalamic nuclei have been categorized as either "first-o

156 The lateral posterior and posterior thalamic nuclei have been implicated in aspects of visua

157 inputs bifurcates to innervate both anterior thalamic nuclei highlights the potential for parallel in

158 tor mRNA levels were found in several areas (thalamic nuclei, hippocampal CA3) with parallel increase

159 lands of Calleja, cerebral cortex, striatum, thalamic nuclei, hippocampus, amygdala, substantia nigra

160 Because cells in the multisensory thalamic nuclei, hypothalamic, and forebrain areas were

161 ostral or caudal regions of the intralaminar thalamic nuclei (i.e. the central lateral or parafascicu

162 The intralaminar thalamic nuclei (IL) probably serves as a central hub in

163 The rostral intralaminar thalamic nuclei (ILn) are organized to activate pathways

164 sults indicate a limited role for the medial thalamic nuclei in coding for pain intensity and the aff

165 anding of the involvement of ventral midline thalamic nuclei in cognitive processes: they point to a

166 anization of connections of NRT with sensory thalamic nuclei in other species in that the terminal fi

167 ial, ventral posterior and lateral posterior thalamic nuclei in patients assessed by the Glasgow Outc

168 , EAAT2, and EAAT3 was performed in discrete thalamic nuclei in persons with schizophrenia and compar

169 ntralaminar, midline, relay, and associative thalamic nuclei in rats.

170 r from a variety of specific and nonspecific thalamic nuclei in relation to the phase of global EEG s

171 d both the dorsomedial and ventral posterior thalamic nuclei in severely disabled and vegetative head

172 tive visual cortical areas and corresponding thalamic nuclei in the embryonic rhesus monkey (Macaca m

173 -brain functional connectivity of the visual thalamic nuclei in the various populations of subjects u

174 and mediodorsal, ventrolateral and pulvinar thalamic nuclei, in both the patients and the healthy mu

175 o area 1 were highly convergent from several thalamic nuclei including the ventral lateral nucleus (V

176 jections in area 3b were also found in other thalamic nuclei including: anterior pulvinar (Pa), ventr

177 as received connections from the same set of thalamic nuclei, including main inputs from the ventral

178 lesions of the contralateral medial auditory thalamic nuclei, including the medial division of the me

179 Thalamic nuclei, including the reticular nucleus, expres

180 al geniculate complex (MGC) and multisensory thalamic nuclei, including the suprageniculate (Sg), lim

181 the highest levels of binding were in select thalamic nuclei, including those implicated in hypoxic d

182 g stimuli that evoke strong responses in the thalamic nuclei innervating the cortex.

183 The thalamic nuclei investigated included LP, laterodorsal t

184 unction of corticothalamic pathways to relay thalamic nuclei is attention-dependent modulation of tha

185 onkey reticular nucleus to the motor-related thalamic nuclei is organized differently from what is kn

186 r thalamic stroke, and the role of different thalamic nuclei is unclear.

187 rough their innervation of a wide variety of thalamic nuclei, is effective in controlling absence sei

188 ultrastructural features of the intralaminar thalamic nuclei (ITN) projections to the globus pallidus

189 b) was injected into one of the intralaminar thalamic nuclei-lateral parafascicular, medial parafasci

190 the mediodorsal nucleus, suggests that these thalamic nuclei, like RE, represent important output sta

191 iculum and parasubiculum, amygdaloid nuclei, thalamic nuclei, locus coeruleus, and nucleus ambiguous

192 Thus, the lateral posterior thalamic nuclei (LP/pulvinar) appear important for vario

193 ution supports the emerging view that limbic thalamic nuclei may contribute critically to adaptive re

194 The medial auditory thalamic nuclei may modulate activity in a short-latency

195 Future analysis of individual thalamic nuclei may reveal important, specific relations

196 ventricle or laterally adjacent mediodorsal thalamic nuclei (MD).

197 dial nucleus-or into one of the intralaminar thalamic nuclei-medial parafascicular, lateral parafasci

198 creases neuron number in three associational thalamic nuclei: mediodorsal (MD), anterior, and pulvina

199 ned to determine how the auditory-responsive thalamic nuclei might activate the HPA axis.

200 Although the midline and intralaminar thalamic nuclei (MITN) were long believed to project "no

201 ence, an increased activity of ventral motor thalamic nuclei nicely explains the refractoriness of PR

202 imulus-response properties across and within thalamic nuclei, normalize responses to diverse sensory

203 in many areas of the hypothalamus and dorsal thalamic nuclei, nucleus intercollicularis and ventricul

204 lls in midline/intralaminar/posterior dorsal thalamic nuclei of male Fmr1 knockout mice.

205 ion, retinofugal projections to midbrain and thalamic nuclei of Monodelphis domestica were investigat

206 ateral entorhinal cortex and ventral midline thalamic nuclei of neonatal rats.

207 and motor pathways within ventrolateral (VL) thalamic nuclei of the motor thalamus of macaque monkeys

208 roposterior medial, and the posterior medial thalamic nuclei of the trigeminal somatosensory pathways

209 ndin (CB) expression in cortical regions and thalamic nuclei of these pathways.

210 As is typical of primary inputs to other thalamic nuclei, parabrachiothalamic terminals are over

211 central medial nucleus-or one of the midline thalamic nuclei-paraventricular (PVT), intermediodorsal

212 a (CTb) was injected into one of the midline thalamic nuclei-paraventricular, intermediodorsal, rhomb

213 trong mPFC projections to several additional thalamic nuclei, particularly to the mediodorsal nucleus

214 ne/intralaminar (M/IL) and ventromedial (VM) thalamic nuclei placed to spare the anterior nuclei.

215 tanding theoretical prediction that specific thalamic nuclei play a key role in controlling the spotl

216 , alongside dorsal portions of the posterior thalamic nuclei (Po), multisensory processing of informa

217 re- and postnatal development, with distinct thalamic nuclei projecting to specific cortical regions.

218 nal pathways to limbic structures and medial thalamic nuclei provide direct inputs to brain areas inv

219 BDA into these auditory-responsive posterior thalamic nuclei provided further evidence of projections

220 ior intralaminar nuclei (AILN) and posterior thalamic nuclei (PTN) to all cortical regions of the PMC

221 t both lemniscal and extralemniscal auditory thalamic nuclei receive significant corticofugal input.

222 ses a similar pathological increase in other thalamic nuclei regulated by the ZI, specifically the me

223 n pulvinar complex is a collection of dorsal thalamic nuclei related to several visual and integrativ

224 the development and organization of diverse thalamic nuclei remain largely unknown.

225 distribution of double-labeled cells in the thalamic nuclei resembled that of single-labeled cells f

226 In contrast, "higher-order" thalamic nuclei respond poorly to peripheral inputs, and

227 ng evidence suggest that the ventral midline thalamic nuclei (reuniens and rhomboid) might play a sub

228 hat the auditory thalamus (and perhaps other thalamic nuclei) serves mainly a relay function underest

229 cipal extrinsic cholinergic source for these thalamic nuclei, showed a marked degree of collateraliza

230 l magnetic resonance imaging to identify the thalamic nuclei, specifically implicated in the generati

231 ing or AAC-projecting cells varied among the thalamic nuclei studied, ranging from 2.9% up to 42.4%.

232 or no expression of beta(3) was observed in thalamic nuclei, substantia nigra, globus pallidus, infe

233 pothalamus, centromedian and paraventricular thalamic nuclei, substantia nigra, reticular formation,

234 y attributed to widespread connectivity from thalamic nuclei such as the pulvinar.

235 However, higher order thalamic nuclei, such as the pulvinar, interconnect with

236 s of interconnections of cortical fields and thalamic nuclei suggest that the somatosensory system ma

237 re, the presence of input from somatosensory thalamic nuclei suggests that it plays an important role

238 in sites including the striatum, associative thalamic nuclei, superior colliculus, zona incerta, pont

239 lamus, the spinal trigeminal nuclei, and the thalamic nuclei supports a role for NPFF in pain modulat

240 he laterorostral part of LP (LPLR) and other thalamic nuclei surrounding LP project to dorsolateral t

241 ough beta4 was more prominently localized in thalamic nuclei than beta2.

242 calcium current underlies burst responses in thalamic nuclei that are important to spindle propagatio

243 y segregated pathways arising from the other thalamic nuclei that are interconnected with the frontal

244 pressed, respectively, in cortical areas and thalamic nuclei that are interconnected.

245 ults from an increase in the excitability of thalamic nuclei that have lost normal ascending inputs a

246 nformation to deeper layers of the SC and to thalamic nuclei that modulate visually guided behaviors.

247 ns to auditory thalamic nuclei and to visual thalamic nuclei that normally receive little direct reti

248 layer 6 send a dense feedback projection to thalamic nuclei that provide input to sensory neocortex.

249 Moreover, the thalamic nuclei that provide major inputs to RWA are the

250 n those regions within the VPL and posterior thalamic nuclei that receive somatosensory information f

251 ntral medial nuclei), as well as the midline thalamic nuclei (the paraventricular, intermediodorsal,

252 as been provided via electrodes implanted in thalamic nuclei, the cerebellum and the hippocampus usin

253 laterodorsal, anteroventral, and parateanial thalamic nuclei, the fasciculus retroflexus of Meynert,

254 eus, lateral septum, midline and mediodorsal thalamic nuclei, the lateral parvocellular part of the b

255 , the hypothalamus, midline and intralaminar thalamic nuclei, the medial geniculate body, the periaqu

256 setup, an additional involvement of another thalamic nuclei, the parafascicular nucleus, when correc

257 ricular hypothalamic nucleus, several visual thalamic nuclei, the paranigral nucleus, several pretect

258 re relayed to the neocortex by "first-order" thalamic nuclei, the responses of which are determined b

259 We expressed ChR2 in two thalamic nuclei, the whisker motor cortex and local exci

260 l, and central medial nuclei; in the midline thalamic nuclei-the paraventricular, intermediodorsal, m

261 Three thalamic nuclei--the mediodorsal nucleus, pulvinar, and

262 l inhibitory connections with several dorsal thalamic nuclei, thereby controlling attention, sensory

263 auses dramatic reorganization of postmitotic thalamic nuclei through altering the positional identity

264 der specific thalamic nuclei and nonspecific thalamic nuclei to carry out attentive visual learning a

265 the anatomical connection from the anterior thalamic nuclei to retrosplenial cortex, and the involve

266 ntral anterior (VA) and ventral lateral (VL) thalamic nuclei to the striatum, suggesting that these n

267 o the paraventricular and medial mediodorsal thalamic nuclei; to the subthalamic and parasubthalamic

268 ions and specifications of connectivity with thalamic nuclei together with upcoming studies of cortic

269 Responses in ventrolateral and anterior thalamic nuclei tracked learning of the predictiveness o

270 Neurons in first-order thalamic nuclei transmit sensory information from the pe

271 waves that propagate among sensory-modality thalamic nuclei up to the cortex and that provide a mean

272 Upon determination that thalamic nuclei VM and VL were of primary interest in th

273 number of c-Fos(+) neurons in ventral motor thalamic nuclei was higher in PRS rats than in unstresse

274 the molecular basis of the specification of thalamic nuclei, we analyzed the expression patterns of

275 c feed-forward inhibition to the rest of the thalamic nuclei, we examined the effect of PCP on RtN ac

276 nization of "first-order" and "higher-order" thalamic nuclei, we followed bias-corrected sampling met

277 s have demonstrated microglial activation in thalamic nuclei well before the onset of retrograde neur

278 ents with pain, mainly lateral and posterior thalamic nuclei were affected, whereas a more anterior-m

279 ecting to the anteromedial and anteroventral thalamic nuclei were closely intermingled, with often on

280 projections to the intralaminar and midline thalamic nuclei were examined in rats.

281 he brainstem to the midline and intralaminar thalamic nuclei were examined in the rat.

282 projections to the midline and intralaminar thalamic nuclei were examined in the rat.

283 hial nucleus to the midline and intralaminar thalamic nuclei were examined in the rat.

284 ically in first-order and higher-order relay thalamic nuclei were juxtacellularly filled with an ante

285 itive for Ca(v) 3.3, while neurons in dorsal thalamic nuclei were nonimmunoreactive.

286 Neuron numbers and volumes in these limbic thalamic nuclei were normal in the schizophrenia and bip

287 s were missing, and most of the major dorsal thalamic nuclei were not identifiable.

288 gnificant volume reductions of the following thalamic nuclei were observed in migraineurs: central nu

289 Both thalamic nuclei were significantly smaller in the high-r

290 ceives input from diverse cortical areas and thalamic nuclei which are themselves interconnected.

291 tum and reticular and ventral posterolateral thalamic nuclei, which all showed synaptogyrin 1 labelin

292 The anterior thalamic nuclei, which are closely interconnected with t

293 and reduced activity in the medial group of thalamic nuclei, which may indicate loss of functional i

294 l, depolarized alpha in posterior-projecting thalamic nuclei while (2) they engage a new, hyperpolari

295 The thalamic nuclei with abnormal volumes are densely connec

296 generation suggest possible abnormalities in thalamic nuclei with connections to other brain regions

297 a differential effect of anesthetic drugs on thalamic nuclei with disparate spatial projections, i.e.

298 ia circuitry primarily associate the ventral thalamic nuclei with relaying basal ganglia output to th

299 striatal projections from LP and surrounding thalamic nuclei, with a focus on projections to DCS.

300 ity and function of the numerous and diverse thalamic nuclei within cortical-subcortical circuits con