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1 impact on lactotrophs and a lesser impact on thyrotrophs.
2 he circannual timer may reside within the PT thyrotroph and is encoded by a binary switch timing mech
3 tes that resistance at the level of both the thyrotroph and the hypothalamic TRH neurons are required
4  coordination of T4-to-T3 conversion between thyrotrophs and tanycytes.
5 stages, including corticotrophs, rostral-tip thyrotrophs, and gonadotrophs, were generated, while hor
6 orticotrophs, rostral-tip POU1F1-independent thyrotrophs, and, interestingly, lactotrophs and gonadot
7 tropin beta (hTSHbeta) gene is restricted to thyrotrophs, at least in part, by silencing.
8 was not due to a reduced number of pituitary thyrotrophs because thyrotroph cell number, as assessed
9 he thyrotropin beta-subunit gene promoter in thyrotrophs by as much as 50 percent, an effect similar
10                   We show that individual PT thyrotrophs can be in one of two binary states reflectin
11 SH and TH synthesis but is not necessary for thyrotroph cell development.
12 y relevant cell system, the TalphaT1.1 mouse thyrotroph cell line.
13  development of somatotroph, lactotroph, and thyrotroph cell lineages and regulation of the temporal
14 uced number of pituitary thyrotrophs because thyrotroph cell number, as assessed by counting TSH immu
15 ut to this rhythm, wherein melatonin acts on thyrotroph cells of the pituitary pars tuberalis (PT), l
16 well as severe disruption of somatotroph and thyrotroph differentiation.
17                                       Murine thyrotroph embryonic factor (mTEF) was isolated and was
18 two other members of the PAR bZIP subfamily, thyrotroph embryonic factor (TEF) and albumin promoter D
19 rolled by the circadian transcription factor thyrotroph embryonic factor (Tef).
20 responding to two distinct isoforms of mouse thyrotroph embryonic factor (TEFalpha and TEFbeta) were
21 n erythroid cells, only 2 of these proteins, thyrotroph embryonic factor and hepatic leukemia factor,
22  by microarray and knockdown of two of them, thyrotroph embryonic factor and protein tyrosine phospha
23 ification of the somatotroph, lactotroph and thyrotroph lineages and in the activation of GH1, PRL an
24 markable specificity of D2 expression in the thyrotrophs of the anterior pituitary coupled with the r
25     The silencing activity was overcome in a thyrotroph-specific manner by an unknown enhancer locate
26 increased 2.2-fold, whereas gonadotrophs and thyrotrophs were unchanged.
27 s, such as somatotrophs and POU1F1-dependent thyrotrophs, were severely reduced.

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